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1 imicrobials and vaccine formulations against Streptococcus pneumoniae.
2 ia, including the major respiratory pathogen Streptococcus pneumoniae.
3 ongitudinal sampling of individuals carrying Streptococcus pneumoniae.
4 o efficacy against Staphylococcus aureus and Streptococcus pneumoniae.
5 ying protein interaction network in the host Streptococcus pneumoniae.
6 y intranasal infection with A66.1 serotype 3 Streptococcus pneumoniae.
7 eks later with PR8 influenza virus and A66.1 Streptococcus pneumoniae.
8 peron to copper homeostasis and virulence in Streptococcus pneumoniae.
9 esponse for the recognition and clearance of Streptococcus pneumoniae.
10 pathogens, namely, Staphylococcus aureus and Streptococcus pneumoniae.
11 neutrophils process IL-1beta in response to Streptococcus pneumoniae.
12 critical role in preventing pneumonia due to Streptococcus pneumoniae.
13 dent cytolysin (CDC) and virulence factor of Streptococcus pneumoniae.
14 cell wall biosynthesis and cell division of Streptococcus pneumoniae.
15 cluster in the Gram-positive human pathogen, Streptococcus pneumoniae.
16 pathogens, e.g., Streptococcus pyogenes and Streptococcus pneumoniae.
17 ent of pneumonia to the common lung pathogen Streptococcus pneumoniae.
18 suppresses the host immune responses against Streptococcus pneumoniae.
19 eumolysin (Ply) is a key virulence factor of Streptococcus pneumoniae.
20 of the most frequent bacterial colonizers is Streptococcus pneumoniae.
21 vasive bacterial infections mainly caused by Streptococcus pneumoniae.
22 of infection with either influenza virus or Streptococcus pneumoniae.
23 sing approach for efficient vaccines against Streptococcus pneumoniae.
24 ndary bacterial pneumonia, particularly from Streptococcus pneumoniae.
25 PD and challenged with opsonized serotype 14 Streptococcus pneumoniae.
26 so abrogating the virulence of the pathogen Streptococcus pneumoniae.
27 Th) 17 cells are important in the control of Streptococcus pneumoniae.
28 concept of this approach for 2 serotypes of Streptococcus pneumoniae.
29 owth of the major human respiratory pathogen Streptococcus pneumoniae.
30 y erm(B) confer most macrolide resistance in Streptococcus pneumoniae.
31 es was correlated with relative abundance of Streptococcus pneumoniae.
32 e the agglutination of specific serotypes of Streptococcus pneumoniae.
33 re used to test 10 Staphylococcus aureus, 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and
34 operative bile and wound infection cultures (Streptococcus pneumoniae, 114 cultures [47.9%] in instit
35 s: Neisseria meningitidis (1350 cases, 22%), Streptococcus pneumoniae (1143, 18%), Staphylococcus aur
37 respiratory syncytial virus (60 [5.6%]), and Streptococcus pneumoniae (57 [5.3%]) were most commonly
39 ryngeal infection by S. pyogenes, but not by Streptococcus pneumoniae, a bacterium that does not prod
44 he pneumonococcal capsular polysaccharide of Streptococcus pneumoniae and against the AD-2S1 peptide
45 y infection with influenza A virus (IAV) and Streptococcus pneumoniae and are the basis of effective
46 ureus in keratitis; Streptococcus viridians, Streptococcus pneumoniae and Coagulase negative Staphylo
48 States for instance, Staphylococcus aureus, Streptococcus pneumoniae and Haemophilus influenzae are
49 emonstrate the efficacy of this technique in Streptococcus pneumoniae and highlight the potential for
50 we use Ab-binding assays to demonstrate that Streptococcus pneumoniae and house dust mite (HDM) bear
54 al), with the major pathogens involved being Streptococcus pneumoniae and non-typeable Haemophilus in
56 us aureus, Coagulase negative Staphylococci, Streptococcus pneumoniae and Pseudomonas aeruginosa are
57 ng affects both susceptibility to subsequent Streptococcus pneumoniae and Staphylococcus aureus infec
58 ions of genomes of the major human pathogens Streptococcus pneumoniae and Streptococcus pyogenes, SEE
59 ed several hundred-fold both by a bacterial (Streptococcus pneumoniae) and a viral infection (influen
60 istant Staphylococcus aureus, penicillin for Streptococcus pneumoniae, and an update on cephalosporin
61 pneumonia caused by Pseudomonas aeruginosa, Streptococcus pneumoniae, and Aspergillus fumigatus when
62 stillation of MS-WF, mice were infected with Streptococcus pneumoniae, and bronchoalveolar lavage flu
64 coli derived lipopolysaccharide, heat-killed Streptococcus pneumoniae, and Mycobacterium tuberculosis
65 fections caused by antibiotic-nonsusceptible Streptococcus pneumoniae (ANSP) continue to present an i
68 e molecular identification and serotyping of Streptococcus pneumoniae are useful for culture-negative
70 pecific IgG responses to intact, heat-killed Streptococcus pneumoniae, as well as a soluble OVA-polys
71 ecalis ATCC 29212 (0.03 to 0.12 mug/ml), and Streptococcus pneumoniae ATCC 49619 (0.004 to 0.015 mug/
72 terococcus faecalis ATCC 29212 (broth only), Streptococcus pneumoniae ATCC 49619 (disk and broth), an
75 The pathogenesis of the disease caused by Streptococcus pneumoniae begins with colonization of the
76 lla catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, but not other bacterial pathog
77 CXCL14 contributed to enhanced clearance of Streptococcus pneumoniae, but not Pseudomonas aeruginosa
78 Most children are transiently colonized with Streptococcus pneumoniae, but very few develop invasive
79 24%) were tested for respiratory viruses and Streptococcus pneumoniae by real-time polymerase chain r
84 ng toxins, such as Staphylococcus aureus and Streptococcus pneumoniae, cause a substantial burden of
89 is, respectively, in the mid-cell regions of Streptococcus pneumoniae cells at different stages of di
90 screen for mutations affecting the growth of Streptococcus pneumoniae cells when the aPBP synthase PB
94 Beta-lactam resistant clinical isolates of Streptococcus pneumoniae contain altered penicillin-bind
96 he predominant P2X7R-expressing cells during Streptococcus pneumoniae corneal infection, and P2X7R wa
97 this study, we use glycoconjugates of type 3 Streptococcus pneumoniae CPS (Pn3P) to assess whether th
101 we have shown that the respiratory pathogen Streptococcus pneumoniae disables neutrophils by exploit
104 lococcus aureus, Staphylococcus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Ent
108 neumococcal disease, defined as isolation of Streptococcus pneumoniae from blood, cerebrospinal fluid
110 est and MALDI-TOF for the differentiation of Streptococcus pneumoniae from other mitis group streptoc
111 n their entirety or reliably differentiating Streptococcus pneumoniae from viridans streptococci.
113 ding proteins that Staphylococcus aureus and Streptococcus pneumoniae, Gram-positive bacterial pathog
114 strate that the opportunistic human pathogen Streptococcus pneumoniae grows in medium supplemented wi
115 owed a predominance of bacteria, principally Streptococcus pneumoniae, >75% being of serotypes covere
116 bocavirus) and 3 pathogenic airway bacteria (Streptococcus pneumoniae, Haemophilus influenzae, and Mo
117 Viral diagnostics and quantitative PCR for Streptococcus pneumoniae, Haemophilus influenzae, and Mo
118 us, coagulase-negative staphylococci (CoNS), Streptococcus pneumoniae, Haemophilus influenzae, and Ps
119 and Acanthamoeba), six bacterial pathogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisse
121 ein response, and innate immune responses to Streptococcus pneumoniae has not been fully elucidated.
122 vaccines against Haemophilus influenzae and Streptococcus pneumoniae has virtually eliminated the co
123 targeting Haemophilus influenzae type b and Streptococcus pneumoniae have dramatically altered the e
124 (e.g., MRSA, VRE, PRSP (penicillin-resistant Streptococcus pneumoniae)); however, there are currently
125 domonas aeruginosa, Streptococcus mitis, and Streptococcus pneumoniae in a dose-dependent manner in p
128 g the efficacy of geOMVs as vaccines against Streptococcus pneumoniae in mice, and against Campylobac
130 ing of the spleen, we identify a tropism for Streptococcus pneumoniae in this organ mediated by tissu
132 Haemophilus influenzae, Aspergillus species, Streptococcus pneumoniae) in bronchoalveolar lavage flui
134 reased risk of pulmonary infection caused by Streptococcus pneumoniae, in particular during severe as
138 ncoding the only PP2C Ser/Thr phosphatase in Streptococcus pneumoniae, indicating that GpsB plays a k
141 of the alveolar barrier in a mouse model of Streptococcus pneumoniae-induced pneumonia, and ex vivo
145 MP), and generated better protection against Streptococcus pneumoniae infection than 2'3'-cGAMP adjuv
146 o regulate the pathogen-host response during Streptococcus pneumoniae infection, but the role Chil1 p
147 es in cerebrospinal fluid from children with Streptococcus pneumoniae infection, compared with childr
152 d PCV13) induce immunological memory against Streptococcus pneumoniae infections caused by vaccine se
155 ia meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae, inflicts a substantial burden
156 stridium difficile, anaerobes, Candida spp., Streptococcus pneumoniae, influenza, Mycobacterium tuber
158 s in the pre-PCV period, 34% were mixed with Streptococcus pneumoniae IRRs (95% confidence interval)
183 resolution microscopy, report that PBP2x of Streptococcus pneumoniae is directed to a discrete locat
184 el et al. (2014) report that colonization of Streptococcus pneumoniae is facilitated by coinfection w
185 at the activation of macrophage NF-kappaB by Streptococcus pneumoniae is highly diverse, with a prepo
199 cial cell wall constituent of the pathobiont Streptococcus pneumoniae, is bound to peptidoglycan (wal
200 We analyzed whole genome sequences of 1,680 Streptococcus pneumoniae isolates from four independent
201 ection of human airway epithelial cells with Streptococcus pneumoniae leads to induction of endoplasm
202 followed by the noninvasive EF3030 strain of Streptococcus pneumoniae, leads to a significant decreas
203 phis infected with the common lung pathogens Streptococcus pneumoniae, Legionella pneumophila, or Myc
205 systematically sampled genomes, we show that Streptococcus pneumoniae lineages are typically characte
206 r macrophages for host immunity during early Streptococcus pneumoniae lung infection is well establis
207 the role of IL-22-IL-22R is understudied in Streptococcus pneumoniae lung infection, a prevalent pat
208 fection on bacterial carriage and density of Streptococcus pneumoniae, Moraxella catarrhalis, Haemoph
209 se bacterial species: Staphylococcus aureus, Streptococcus pneumoniae, Mycobacterium tuberculosis, Sa
210 s were Streptococcus viridans (n = 47; 71%), Streptococcus pneumoniae (n = 13; 21%), and beta-hemolyt
211 [n = 5], Haemophilus influenzae [n = 5], and Streptococcus pneumoniae [n = 5]), and transport medium
213 tination for bacterial antigen) and qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Ha
214 so-called conventional bacterial infections (Streptococcus pneumoniae, Neisseria meningitidis, Haemop
217 philus influenzae, Moraxella catarrhalis, or Streptococcus pneumoniae (odds ratio [OR], 3.27; 95% con
218 ls were stimulated in vitro with heat-killed Streptococcus pneumoniae or CD3/CD28 antibodies and stai
220 al [CI] = 3.27-5.37; n = 2432 participants), Streptococcus pneumoniae otitis media (OR = 2.51; 95% CI
221 ue hypoxia-ischemia are strongly linked with Streptococcus pneumoniae pathogenesis in patients with s
222 f Enterococcus faecium (fnm), a homologue of Streptococcus pneumoniae pavA, in the genomes of E. faec
223 acet of the interaction between the pathogen Streptococcus pneumoniae (pneumococcus) and its human ho
231 he least understood aspects of the bacterium Streptococcus pneumoniae (pneumococcus) is its transmiss
235 ped ovococcus bacteria, such as the pathogen Streptococcus pneumoniae (pneumococcus), side-wall (peri
236 e-wall like) peptidoglycan (PG) synthesis in Streptococcus pneumoniae (pneumococcus); yet, mechanisms
237 These studies tested the role of Nrf2 during Streptococcus pneumoniae pneumonia and identified Nrf2-d
241 ediated hemolysis of ES PspCN, a CFH-binding Streptococcus pneumoniae protein domain, binds CFH tight
242 spectrometry, that the N-terminal domain of Streptococcus pneumoniae protein PspC (PspCN) not only b
244 tion of energy, the important human pathogen Streptococcus pneumoniae relies on host-derived sugars,
246 on of infant rats with increasing inocula of Streptococcus pneumoniae resulted in a dose-dependent in
247 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae (S. pneumoniae), and Haemophilu
253 eak was due to multiple pathogens, including Streptococcus pneumoniae serotype 5 and influenza viruse
256 The divergent epidemiological behavior of Streptococcus pneumoniae serotypes suggests that serotyp
257 ble PCR primers were designed to distinguish Streptococcus pneumoniae serotypes within serogroup 18 f
258 ting invasive pneumococcal disease caused by Streptococcus pneumoniae Some components of the S. pneum
261 ly cover only 13 of the over 90 serotypes of Streptococcus pneumoniae (Sp), so nonvaccine serotypes a
263 with the family 98 glycoside hydrolase from Streptococcus pneumoniae SP3-BS71 (Sp3GH98), which cleav
264 topoisomerase IV (ParE) from a G(+) strain (Streptococcus pneumoniae (sParE)) and a G(-) strain (Pse
266 virus URIs on the frequency of AOM caused by Streptococcus pneumoniae (Spn) and nontypeable Haemophil
267 y we characterize the PaaI thioesterase from Streptococcus pneumoniae (SpPaaI), including structural
268 ane disruption, against the airway pathogens Streptococcus pneumoniae, Staphylococcus aureus, Nontype
270 teria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus agalactiae, cyto
271 y Bacillus subtilis, Streptococcus pyogenes, Streptococcus pneumoniae, Streptococcus mutans, Staphylo
273 ng Escherichia coli, Bacillus anthracis, and Streptococcus pneumoniae, studies in mycobacteria have n
279 s is a prerequisite for the human pathobiont Streptococcus pneumoniae (the pneumococcus) to cause sev
282 their ability to combat microbes, including Streptococcus pneumoniae, the most common cause of CAP i
284 In a number of bacterial species, including Streptococcus pneumoniae, the prevalence of resistance h
285 bset of pathogenic microorganisms, including Streptococcus pneumoniae, the recognition and degradatio
287 c domain comprising a fragment of GH101 from Streptococcus pneumoniae TIGR4, SpGH101, in the absence
288 ative data for the pattern of disease due to Streptococcus pneumoniae, trends in the serotype of inva
289 related to the capsular polysaccharide from Streptococcus pneumoniae type 37, which consists of a be
290 lysin pneumolysin, a key virulence factor of Streptococcus pneumoniae, using single cell studies.
291 MRSA and 22 (1.0%) with MSSA; 115 (5.1%) had Streptococcus pneumoniae Vancomycin or linezolid was adm
292 he commensal genus Neisseria and the species Streptococcus pneumoniae was associated with lower EAC r
296 ildren (107/6769); Staphylococcus aureus and Streptococcus pneumoniae were most commonly identified.
298 ity-acquired pneumonia is commonly caused by Streptococcus pneumoniae, which is associated with exces
299 beta-lactam and co-trimoxazole resistance in Streptococcus pneumoniae with accuracies ranging from 88
300 PspC) are key players for the interaction of Streptococcus pneumoniae with matricellular hTSP-1.
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