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1 ions of adhesion proteins from the bacterium Streptococcus pyogenes.
2 urface of invasive M3-type strain MGAS315 of Streptococcus pyogenes.
3 treptolysin O (SLO), a virulence factor from Streptococcus pyogenes.
4 e 8 charged residues in YtgP, a homolog from Streptococcus pyogenes.
5 lycoside-hydrolase secreted by the bacterium Streptococcus pyogenes.
6  plays an important role in the virulence of Streptococcus pyogenes.
7 occus pneumoniae, Staphylococcus aureus, and Streptococcus pyogenes.
8 using two rapid antigen detection assays for Streptococcus pyogenes.
9 ins are critical for the in vivo survival of Streptococcus pyogenes.
10 , including the clinically relevant pathogen Streptococcus pyogenes.
11 d to effectively identify DNA signatures for Streptococcus pyogenes.
12 phil peptide (HNP-1) with the human pathogen Streptococcus pyogenes.
13 AgI/II-family proteins are also expressed by Streptococcus pyogenes.
14 gous to the myosin cross-reactive antigen of Streptococcus pyogenes.
15  share homology with the mitogenic toxins of Streptococcus pyogenes.
16  with PJI, namely, Staphylococcus aureus and Streptococcus pyogenes.
17 hat mimics that of many virulence factors of Streptococcus pyogenes.
18 tia marcescens and, to a lesser extent, with Streptococcus pyogenes.
19 lence factor secretion in the human pathogen Streptococcus pyogenes.
20  a heme ABC transporter in the Gram positive Streptococcus pyogenes.
21 ogens including Streptococcus pneumoniae and Streptococcus pyogenes.
22  to make up the machinery for heme uptake in Streptococcus pyogenes.
23 t of the heme acquisition machinery found in Streptococcus pyogenes.
24  functional upstream domain of protein F1 of Streptococcus pyogenes.
25  Cas9 enzymes from Staphylococcus aureus and Streptococcus pyogenes.
26 ting infection with the riboflavin auxotroph Streptococcus pyogenes.
27 can enter human cells and kill intracellular Streptococcus pyogenes.
28 toxins secreted by Staphylococcus aureus and Streptococcus pyogenes.
29 yte activation in response to infection with Streptococcus pyogenes.
30 s II-bound peptide from the mucosal pathogen Streptococcus pyogenes.
31 s aureus (10 [43.5%] vs 4 [12.9%]; P = .02), Streptococcus pyogenes (2 [8.7%] vs 19 [61.3%]; P < .001
32  mutagenesis approach, we have identified in Streptococcus pyogenes a gene that exhibits a receptor-l
33 r, pharyngitis resulting from infection with Streptococcus pyogenes (a group A Streptococcus [GAS]) c
34     Having no known environmental reservoir, Streptococcus pyogenes, a bacterium responsible for a wi
35                                           In Streptococcus pyogenes, a common and potentially deadly
36                                              Streptococcus pyogenes, a human-restricted pathogen, acc
37                                              Streptococcus pyogenes, a multiple-auxotrophic human pat
38                                              Streptococcus pyogenes activates IFN-I production in inn
39                  The Gram-positive bacterium Streptococcus pyogenes (also called group A Streptococcu
40                                              Streptococcus pyogenes , also known as group A streptoco
41 ing a positive result for samples containing Streptococcus pyogenes and a negative result for those w
42 p protein (Cpa) of the T3 pilus derived from Streptococcus pyogenes and expressed this fusion protein
43     We examined the cluster 9 protein Lsp of Streptococcus pyogenes and found that specific deletion
44      Cysteine protease SpeB is secreted from Streptococcus pyogenes and has been studied as a potenti
45 tudy, we created an RNase III null mutant of Streptococcus pyogenes and its RNA sequencing (RNA-Seq)
46           The type II CRISPR/Cas system from Streptococcus pyogenes and its simplified derivative, th
47 ce against lethal soft tissue infection with Streptococcus pyogenes and prevented bacterial dissemina
48 al effects against Staphylococcus aureus and Streptococcus pyogenes and protected against staphylococ
49 ive clinical diagnose include Staphylococci, Streptococcus pyogenes and Pseudomonas aeruginosa in ble
50  has never, or rarely, been reported for the Streptococcus pyogenes and S. bovis groups of species, e
51                                         Both Streptococcus pyogenes and Streptococcus pneumoniae are
52 which includes common human pathogens, e.g., Streptococcus pyogenes and Streptococcus pneumoniae.
53            Only two CRISPR-Cas systems (from Streptococcus pyogenes and Streptococcus thermophilus),
54      Here, we combine the CRISPR system from Streptococcus pyogenes and synthetic antisense RNAs (asR
55 ular elements of patients with GP respond to Streptococcus pyogenes and whether this initial immune r
56  Neisseria gonorrhoeae, Salmonella enterica, Streptococcus pyogenes and Xenorhabdus nematophila.
57 utagenesis of EndoS (an endoglycosidase from Streptococcus pyogenes ) and were found to be capable of
58 ccus epidermidis, Staphylococcus aureus, and Streptococcus pyogenes) and Gram-negative (Klebsiella pn
59 oth gram-positive (Staphylococcus aureus and Streptococcus pyogenes) and gram-negative bacteria (Pseu
60 m Staphylococcus aureus, streptolysin O from Streptococcus pyogenes, and anthrolysin O from Bacillus
61 aphylococcus aureus, Pseudomonas aeruginosa, Streptococcus pyogenes, and Candida albicans.
62 us influenzae (NTHi), Moraxella catarrhalis, Streptococcus pyogenes, and culture-negative OM.
63  three datasets from Francisella tularensis, Streptococcus pyogenes, and Escherichia coli.
64 ease onset in children and associations with Streptococcus Pyogenes, and influenza A H1N1-infection a
65 tein 9 (Cas9) from Staphylococcus aureus and Streptococcus pyogenes, and recombinant Cas9 and develop
66 in 8 patients, and Streptococcus agalactiae, Streptococcus pyogenes, and Streptococcus salivarius in
67                                              Streptococcus pyogenes, and to a lesser extent, Staphylo
68 eus, methicillin-resistant S. aureus (MRSA), Streptococcus pyogenes, and vancomycin-resistant Enteroc
69                                              Streptococcus pyogenes AP1, a strain of the highly virul
70 li of the nasty gram-positive human pathogen Streptococcus pyogenes are assembled as single, micromet
71    The heme-binding proteins Shp and HtsA of Streptococcus pyogenes are part of the heme acquisition
72 coccus aureus, Streptococcus pneumoniae, and Streptococcus pyogenes, are most commonly isolated.
73 organisms, such as Staphylococcus aureus and Streptococcus pyogenes, are the dominant organisms isola
74  HIV-1 Gag-p24 on the tip of the T3 pilus of Streptococcus pyogenes as a fusion to the Cpa protein (L
75  inhibits growth of the pathogenic bacterium Streptococcus pyogenes as effectively as melittin create
76 e success of the group A streptococcus (GAS, Streptococcus pyogenes) as an important human pathogen.
77 i, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subtilis, Staphylococcu
78                 The BC-GP detected a case of Streptococcus pyogenes bacteremia but failed to detect 2
79  (FUD), a polypeptide based on F1 adhesin of Streptococcus pyogenes, binds by anti-parallel beta-stra
80                             A gene unique to Streptococcus pyogenes, called vfr, that negatively regu
81 ustom DNA-binding modules, the nuclease-dead Streptococcus pyogenes Cas9 (dCas9) protein, which recog
82 e-wide off-target effects of the widely used Streptococcus pyogenes Cas9 (SpCas9) are imperfect, poss
83          Here we show that the commonly used Streptococcus pyogenes Cas9 (SpCas9) can be modified to
84                               The success of Streptococcus pyogenes Cas9 (SpCas9) has led to the disc
85                                          The Streptococcus pyogenes Cas9 (SpCas9) nuclease can be eff
86                                              Streptococcus pyogenes Cas9 (SpCas9), a CRISPR-associate
87 SaCas9), which is significantly smaller than Streptococcus pyogenes Cas9 (SpCas9), to facilitate effi
88 ss and efficiency as previously reported for Streptococcus pyogenes Cas9 (SpCas9).
89 lls comparable with those of the widely used Streptococcus pyogenes Cas9 (SpCas9).
90 in engineering to improve the specificity of Streptococcus pyogenes Cas9 (SpCas9).
91 o 70 degrees C, compared to 45 degrees C for Streptococcus pyogenes Cas9 (SpyCas9), which expands the
92 icity, selectivity, and reaction kinetics of Streptococcus pyogenes Cas9 activity, we challenged libr
93                                Structures of Streptococcus pyogenes Cas9 alone or bound to single-gui
94                                              Streptococcus pyogenes Cas9 cleavage of the viral genome
95                        Here we show that the Streptococcus pyogenes Cas9 DNA endonuclease and single
96 DNA vectors expressing maize codon-optimized Streptococcus pyogenes Cas9 endonuclease and single guid
97     Here, we report the crystal structure of Streptococcus pyogenes Cas9 in complex with sgRNA and it
98 g guide RNA (stgRNA) that repeatedly directs Streptococcus pyogenes Cas9 nuclease activity toward the
99 also compared the cleavage efficiency of the Streptococcus pyogenes Cas9 protein based on expression
100 cular structures of the catalytically active Streptococcus pyogenes Cas9 R-loop that show the displac
101 hese inhibitors also blocked the widely used Streptococcus pyogenes Cas9 when assayed in Escherichia
102 protein containing a catalytically defective Streptococcus pyogenes Cas9, a cytidine deaminase, and a
103 ing method using only the well-characterized Streptococcus pyogenes Cas9, by incorporating MS2 or PP7
104    However, in M. tuberculosis, the existing Streptococcus pyogenes Cas9-based CRISPRi system is of l
105                                              Streptococcus pyogenes Cas9-guide RNA (gRNA) was success
106 n of the protospacer-adjacent motif (PAM) of Streptococcus pyogenes Cas9.
107 ence required for target site recognition by Streptococcus pyogenes Cas9.
108 ons, with most studies to date utilizing the Streptococcus pyogenes Cas9.
109               For the Gram-positive pathogen Streptococcus pyogenes, catabolism of the amino acid arg
110 d expression and activity of SLO, DNase, and Streptococcus pyogenes cell envelope protease in vitro.
111                                          The Streptococcus pyogenes cell-surface protein Scl2 contain
112                                       In the Streptococcus pyogenes collagen-like protein Scl2, a V d
113 etween sequence, stability, and folding, the Streptococcus pyogenes collagenous domain CL (Gly-Xaa-Ya
114                    Staphylococcus aureus and Streptococcus pyogenes colonize mucosal surfaces of the
115       The CRISPRi system is derived from the Streptococcus pyogenes CRISPR (clustered regularly inter
116                    The Cas9 protein from the Streptococcus pyogenes CRISPR-Cas acquired immune system
117      Type-II CRISPR-Cas systems, such as the Streptococcus pyogenes CRISPR-Cas9 system, can be adapte
118                         The Mga regulator of Streptococcus pyogenes directly activates the transcript
119        Macrolide- and tetracycline-resistant Streptococcus pyogenes emm12 isolates represent the majo
120                          FCT region genes of Streptococcus pyogenes encode surface proteins that incl
121 reveal that a bacterial endoglycosidase from Streptococcus pyogenes , EndoS, is complementary to othe
122 ant bacterial pathogens in humans, including Streptococcus pyogenes, express surface proteins that bi
123 -residue functional upstream domain (FUD) of Streptococcus pyogenes F1 adhesin interacts with fibrone
124                                              Streptococcus pyogenes fibronectin-binding protein FbaB
125 to the surface of the human pathogen group A Streptococcus pyogenes (GAS) and subsequent hPg activati
126        Dimeric M-proteins (M-Prt) in group A Streptococcus pyogenes (GAS) are surface-expressed virul
127 ow that PS increased the severity of group A Streptococcus pyogenes (GAS) cutaneous skin infection in
128            Staphylococcus aureus and group A Streptococcus pyogenes (GAS) express superantigen (SAg)
129                  A emm53 subclass of Group A Streptococcus pyogenes (GAS) interacts tightly with huma
130                         Infection by Group A Streptococcus pyogenes (GAS) is a leading cause of sever
131                                      Group A Streptococcus pyogenes (GAS) is a leading human pathogen
132                                      Group A Streptococcus pyogenes (GAS) strain AP53 is a primary is
133               A skin-tropic invasive group A Streptococcus pyogenes (GAS) strain, AP53, contains a na
134 -mediated opsonophagocytosis enables group A Streptococcus pyogenes (GAS) to establish infection.
135 rt a population genomic study of the group A Streptococcus pyogenes (GAS), a human pathogen with high
136 The normalized fluorescence intensity of the Streptococcus pyogenes gave a signal that is up to 16.4
137                       Genomic analysis of 12 Streptococcus pyogenes genomes representing six differen
138                         Infections caused by Streptococcus pyogenes (group A Streptococcus [GAS]) are
139 Rgg2 and Rgg3 (Rgg2/3) regulatory circuit of Streptococcus pyogenes (group A streptococcus [GAS]) is
140                                              Streptococcus pyogenes (group A Streptococcus [GAS]) is
141                                              Streptococcus pyogenes (group A streptococcus [GAS]) is
142                 The important human pathogen Streptococcus pyogenes (group A Streptococcus [GAS]) pro
143                                              Streptococcus pyogenes (group A streptococcus [GAS]) res
144 e role of zinc efflux in the pathogenesis of Streptococcus pyogenes (group A Streptococcus [GAS]), a
145 15,783-bp genome of a serotype M49 strain of Streptococcus pyogenes (group A streptococcus [GAS]), st
146 d an RNase Y ortholog has been identified in Streptococcus pyogenes (group A streptococcus [GAS]).
147 a, including one (stk) in the human pathogen Streptococcus pyogenes (group A streptococcus [GAS]).
148  major surface feature of the human pathogen Streptococcus pyogenes (group A streptococcus [GAS]).
149 t to which glucose alters gene expression in Streptococcus pyogenes (group A streptococcus) and the c
150                     All sequenced genomes of Streptococcus pyogenes (Group A Streptococcus, GAS) enco
151  the Gram-positive human-restricted pathogen Streptococcus pyogenes (Group A Streptococcus, GAS) has
152                 The important human pathogen Streptococcus pyogenes (group A streptococcus, GAS) init
153                The human-restricted pathogen Streptococcus pyogenes (Group A Streptococcus, GAS) is r
154  genes that are crucial for the virulence of Streptococcus pyogenes (group A Streptococcus, GAS).
155                         The CovR/S system in Streptococcus pyogenes (Group A Streptococcus, or GAS),
156                                              Streptococcus pyogenes (Group A Streptococcus; GAS) is a
157                                              Streptococcus pyogenes [group A Streptococcus; (GAS)] is
158 rammed genome editing using CRISPR/Cas9 from Streptococcus pyogenes has enabled rapid and accessible
159 , formed autocatalytically, in the pili from Streptococcus pyogenes has highlighted the role that suc
160 mbinant immunoglobulin G-degrading enzyme of Streptococcus pyogenes (IdeS) followed by chemical reduc
161        The IgG-degrading enzyme derived from Streptococcus pyogenes (IdeS), an endopeptidase, cleaves
162  A bacterial enzyme, IgG-degrading enzyme of Streptococcus pyogenes (IdeS), was shown to specifically
163  use of immunoglobulin G-degrading enzyme of Streptococcus pyogenes (IdeS), which is capable of diges
164 A reactivity against IgG-degrading enzyme of Streptococcus pyogenes (IdeS)- or pepsin-generated F(ab'
165 ors of the bacterial IgG degrading enzyme of Streptococcus pyogenes , IdeS.
166 ld activate NF-kappaB in response to bRNA or Streptococcus pyogenes in an RNA-specific manner.
167                                              Streptococcus pyogenes, in addition to causing fulminant
168 pha2 exhibited considerable activity against Streptococcus pyogenes, indicating a role of PSMs in the
169 rate that type I interferons produced during Streptococcus pyogenes infection are required to prevent
170 ated as an adjunctive treatment for clinical Streptococcus pyogenes infection however, the protein ta
171 ontrast to infection of superficial tissues, Streptococcus pyogenes infection of deeper tissue can be
172 ses that are associated with severe invasive Streptococcus pyogenes infections.
173                   The gram-positive pathogen Streptococcus pyogenes injects a beta-NAD(+) glycohydrol
174  (CMT) pathway of the Gram-positive pathogen Streptococcus pyogenes injects effector proteins into th
175              To obtain new information about Streptococcus pyogenes intrahost genetic variation durin
176 owed that the adhesive is capable of killing Streptococcus pyogenes introduced subcutaneously at the
177       While many virulence factors promoting Streptococcus pyogenes invasive disease have been descri
178                                              Streptococcus pyogenes is a gram-positive human pathogen
179                                              Streptococcus pyogenes is a major bacterial pathogen and
180                                              Streptococcus pyogenes is a major cause of pharyngitis i
181                                              Streptococcus pyogenes is a significant bacterial pathog
182  The ExPortal protein secretion organelle in Streptococcus pyogenes is an anionic phospholipid-contai
183                                              Streptococcus pyogenes is an exclusively human pathogen.
184                                              Streptococcus pyogenes is an important human pathogen th
185                                              Streptococcus pyogenes is an important human pathogen wi
186                                              Streptococcus pyogenes is an important pathogen that cau
187 epeats (CRISPR)-associated protein Cas9 from Streptococcus pyogenes is an RNA-guided DNA endonuclease
188 ession of many virulence-associated genes in Streptococcus pyogenes is controlled in a growth phase-d
189         The transcriptional regulator Rgg of Streptococcus pyogenes is essential for expression of th
190 e factors by the group A streptococcus (GAS) Streptococcus pyogenes is important in this pathogen's a
191 of maternal death in the United Kingdom, and Streptococcus pyogenes is the leading pathogen.
192                  A key attribute of invasive Streptococcus pyogenes is their ability to resist phagoc
193 cription of several key virulence factors of Streptococcus pyogenes is under the control of Mga and N
194              The group A Streptococcus (GAS, Streptococcus pyogenes) is a Gram-positive bacterial pat
195              The Group A Streptococcus (GAS, Streptococcus pyogenes) is a Gram-positive human pathoge
196                  Group A Streptococcus (GAS, Streptococcus pyogenes) is a human-restricted pathogen w
197 egulator of the group A streptococcus (GAS) (Streptococcus pyogenes), is a transcriptional regulator
198          The group A streptococcus (GAS), or Streptococcus pyogenes, is a strict human pathogen of me
199             The group A streptococcus (GAS), Streptococcus pyogenes, is an important human pathogen t
200 m Staphylococcus aureus and SrtA(strep) from Streptococcus pyogenes, is exploited for site-specific l
201             While screening the clonality of Streptococcus pyogenes isolates from an outbreak of eryt
202 C-reactive protein (CRP), and M protein from Streptococcus pyogenes, it has been hypothesized that th
203 LDH2 < E. faecalis LDH1 < L. lactis LDH1 </= Streptococcus pyogenes LDH.
204 h year, millions of people are infected with Streptococcus pyogenes, leading to an estimated 500,000
205 ombined with focused experimental testing in Streptococcus pyogenes, led to a better understanding of
206 f a catalytically inactive Cas9 (dCas9) from Streptococcus pyogenes loaded with single guide RNAs (sg
207 r Staphylococcus aureus (</=0.12 microg/mL), Streptococcus pyogenes (&lt;/=0.12 microg/mL), Streptococcu
208                                     However, Streptococcus pyogenes LTA essentially devoid of D-alani
209 h six of the seven loci corresponding to the Streptococcus pyogenes MLST scheme.
210                                           In Streptococcus pyogenes, mutation of the peroxide sensor
211                                          The Streptococcus pyogenes NAD(+) glycohydrolase (SPN) is se
212 ected mutagenesis of an endoglycosidase from Streptococcus pyogenes of serotype M49 (Endo-S2) and the
213                                              Streptococcus pyogenes, one of the most common human pat
214                        Group A streptococci (Streptococcus pyogenes or GAS) freshly isolated from ind
215 rtant human pathogen, group A Streptococcus (Streptococcus pyogenes or GAS).
216                                              Streptococcus pyogenes (or group A streptococcus [GAS])
217                                              Streptococcus pyogenes, or group A Streptococcus (GAS),
218                                              Streptococcus pyogenes, or group A Streptococcus (GAS),
219 re, and clears infection in a mouse model of Streptococcus pyogenes peritonitis.
220         The Gram-positive bacterial pathogen Streptococcus pyogenes produces a C3 family ADP-ribosylt
221                           The human pathogen Streptococcus pyogenes produces pili that are essential
222             The pathogenic bacterium Group A Streptococcus pyogenes produces several extracellular DN
223                                  SpyCEP is a Streptococcus pyogenes protease that cleaves CXCL8/IL-8
224 nstrate that the group A Streptococcus (GAS; Streptococcus pyogenes) protease SpyCEP (S. pyogenes cel
225 and several bacterial ligands: M6 protein of Streptococcus pyogenes, PspC of Streptococcus pneumoniea
226                                              Streptococcus pyogenes ranks among the main causes of mo
227                   The surface protein Shp of Streptococcus pyogenes rapidly transfers its hemin to Ht
228 tudy of the two lactose metabolic operons of Streptococcus pyogenes, reported in this issue of Molecu
229 ar analysis conducted on streptolysin O from Streptococcus pyogenes revealed that this CDC also has g
230                                              Streptococcus pyogenes Rgg is a regulatory protein that
231                                              Streptococcus pyogenes Rgg is a transcriptional regulato
232 en was used to identify mutations in rgg2 of Streptococcus pyogenes (rgg2Sp ) that conferred pheromon
233                                        Using Streptococcus pyogenes RopB as a model, we demonstrated
234 indigenous to the human mouth, as well as in Streptococcus pyogenes, S. agalactiae and S. suis.
235 The AF, but not the meconium SALSA, bound to Streptococcus pyogenes, S. agalactiae, S. gordonii, and
236 resent in pathogenic streptococci, including Streptococcus pyogenes, S. agalactiae, S. pneumoniae, an
237                           The human pathogen Streptococcus pyogenes secretes a highly cytolytic toxin
238                                              Streptococcus pyogenes secretes many toxins that facilit
239              The globally prominent pathogen Streptococcus pyogenes secretes potent immunomodulatory
240 human pathogens Streptococcus pneumoniae and Streptococcus pyogenes, SEER identifies relevant previou
241 cluding that in group A Streptococcus (GAS) (Streptococcus pyogenes Ser/Thr kinase (SP-STK)), regulat
242  the eukaryote-type serine/threonine kinase (Streptococcus pyogenes serine/threonine kinase; SP-STK)
243       The first genome sequence of a group A Streptococcus pyogenes serotype M23 (emm23) strain (M23N
244                      Here, we used Scl2 from Streptococcus pyogenes serotype M28 strain MGAS6274 as a
245 The CRISPR-associated endonuclease Cas9 from Streptococcus pyogenes (spCas9) along with a single guid
246 (CRISPR)-associated endonuclease (Cas)9 from Streptococcus pyogenes (SpCas9) can be used to edit sing
247     The RNA-guided CRISPR-Cas9 nuclease from Streptococcus pyogenes (SpCas9) has been widely repurpos
248   In vitro assays demonstrate that Cas9 from Streptococcus pyogenes (SpCas9) is more active in creati
249 (CRISPR)-associated endonuclease (Cas)9 from Streptococcus pyogenes (SpCas9) is used to deplete VEGFR
250 ver, the size of the commonly used Cas9 from Streptococcus pyogenes (SpCas9) limits its utility for b
251 yC, a bacteriophage-encoded endolysin, lyses Streptococcus pyogenes (Spy) on contact.
252 for entry of other virulent pathogens (e.g., Streptococcus pyogenes, Staphylococcus aureus, and poten
253 arisons of the 1.84-Mb genome of serotype M5 Streptococcus pyogenes strain Manfredo with previously s
254    We identified a unique genetic element in Streptococcus pyogenes strain SF370 that controls MMR vi
255                           The FCT regions of Streptococcus pyogenes strains encode a variety of cell
256  identifying variation, we pooled DNA of 100 Streptococcus pyogenes strains of different emm types in
257 eruginosa, S. aureus, Enterococcus faecalis, Streptococcus pyogenes, Streptococcus agalactiae, and vi
258 ria monocytogenes, Streptococcus pneumoniae, Streptococcus pyogenes, Streptococcus agalactiae, Escher
259  pathogens, including Staphylococcus aureus, Streptococcus pyogenes, Streptococcus pneumoniae, Helico
260  against local and/or systemic infections by Streptococcus pyogenes, Streptococcus pneumoniae, Lister
261 rsA homologues encoded by Bacillus subtilis, Streptococcus pyogenes, Streptococcus pneumoniae, Strept
262  reproduce the canonical PAM preferences for Streptococcus pyogenes, Streptococcus thermophilus CRISP
263 ally increase mortality when associated with Streptococcus pyogenes superinfection.
264 of superantigens and superantigen-containing Streptococcus pyogenes supernatants, although not by iso
265                The important human pathogen, Streptococcus pyogenes, synthesizes a key antigenic surf
266 -ribulose 5-phosphate 3-epimerase (RPE) from Streptococcus pyogenes that catalyzes the equilibration
267 pacity factor (SOF), a substance produced by Streptococcus pyogenes that turns mammalian serum cloudy
268 ity factor (SOF), a virulence determinant of Streptococcus pyogenes that turns serum opaque.
269 /SpyCatcher), based on a protein domain from Streptococcus pyogenes, that locks itself together via s
270  toxin production and increased virulence in Streptococcus pyogenes The nature of the polymorphism is
271 type M1 and M3 strains of the human pathogen Streptococcus pyogenes (the group A Streptococcus [GAS])
272            As an exclusively human pathogen, Streptococcus pyogenes (the group A streptococcus [GAS])
273           The impressive disease spectrum of Streptococcus pyogenes (the group A streptococcus [GAS])
274 ortant in regulation of virulence factors of Streptococcus pyogenes (the group A streptococcus, GAS),
275 l streptococci, including the human pathogen Streptococcus pyogenes(the group A Streptococcus[GAS]),
276 r model organisms, Staphylococcus aureus and Streptococcus pyogenes, the activity is dependent on pri
277 toxins produced by Staphylococcus aureus and Streptococcus pyogenes, the superantigens (SAgs) are the
278            In the human-pathogenic bacterium Streptococcus pyogenes, the tagatose bisphosphate aldola
279  protein against the Gram-positive bacterium Streptococcus pyogenes This protein is composed of two d
280  lysin PlyC to permeabilize the cell wall of Streptococcus pyogenes to antibodies, thereby allowing t
281                               The ability of Streptococcus pyogenes to infect different niches within
282   We previously used crystal coordinates for Streptococcus pyogenes UGDH to generate a model of the h
283                                              Streptococcus pyogenes utilizes numerous mechanisms for
284 n-mediated translocation (CMT), performed by Streptococcus pyogenes, utilizes the cholesterol-depende
285 nature-tagged mutagenesis (STM), to identify Streptococcus pyogenes virulence genes important for pat
286                        The Cas9 protein from Streptococcus pyogenes was pre-complexed with a single g
287 ermore, utilization of supplied 5-CHO-THF by Streptococcus pyogenes was shown to require expression o
288 ion of streptococcal pyrogenic exotoxin A by Streptococcus pyogenes was unaffected by alpha and beta
289  synthetase proteins from the human pathogen Streptococcus pyogenes, we demonstrate the in vitro reco
290    Studying the pilus tip adhesin Spy0125 of Streptococcus pyogenes, we developed a single molecule a
291 elical peptide epitope from the M protein of Streptococcus pyogenes, were designed by exchanging one
292 h a fluorescent antibody complex specific to Streptococcus pyogenes, were volumetrically normalized a
293 cterial enzyme IdeS (IgG-degrading enzyme of Streptococcus pyogenes), which selectively cleaves IgG a
294  betabetaalpha/metal-dependent nuclease from Streptococcus pyogenes, which is encoded by the SF370.1
295 e also analyzed promiscuity of epitopes from Streptococcus pyogenes, which is known to exhibit epitop
296 tting a pilin subunit from a human pathogen, Streptococcus pyogenes, which usually undergoes intramol
297 lence of many bacterial pathogens, including Streptococcus pyogenes, which utilizes the cholesterol-d
298 e interactions of the group A streptococcus, Streptococcus pyogenes, with the extracellular matrix pr
299 ine development and mucosal immunity against Streptococcus pyogenes would benefit from the availabili
300 e immune response against the human pathogen Streptococcus pyogenes, yet the innate immune response a

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