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1 n ratio (p < 0.02) with a consequent fall in SvO2.
2 ared spectroscopy positively correlates with SvO2.
3 Hg, cardiac index 3.3 [2.7-4.0] L/min.m, and SvO2 68.3 [62.8-74.7]%), microvascular variables were ma
5 gen saturation with Perfusion, intravascular SvO2, and T2* may be a useful tool to investigate the pa
8 , p = 0.025), along with an increase in mean SvO2 (baseline = 75 +/- 3, 48 hour = 85 +/- 2, p = 0.018
9 reathing developed a progressive decrease in SvO2 caused by the combination of a relative decrease in
11 we recorded mixed venous oxygen saturation (SvO2) continuously in eight ventilator-supported patient
15 ass solely for low venous oxygen saturation (SvO2), ie, <55%, without regard to hematocrit (Hct), pos
16 Multivariate analysis identified continuous SvO2 monitoring as a factor favoring S1P survival (P=0.0
17 ding continuous superior vena cava oximetry (SvO2), phenoxybenzamine (POB), strategies to minimize th
20 na cava, right atrium, and pulmonary artery (SVO2) was measured by cooximetry in consecutive blood sa
22 O, PCCO measurements from each catheter, and SVO2 were compared with PATD CO at each mean arterial pr
23 d right atrial oxyhemoglobin saturations and SvO2 were compared, the ranges and 95% confidence limits
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