ライフサイエンスコーパス: T helper 1 cell

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1 ly expressed in hematopoietic tissues and in T helper 1 cells.

2 pha production from noninflammatory cells to T helper 1 cells.

3 ollagen-specific, interferon-gamma-producing T helper 1 cells.

4 ich then generate interferon gamma-producing T helper-1 cells.

5 lerant and have increased AT macrophages and T-helper 1 cells.

6 obligatory signal for the differentiation of T-helper-1 cells.

7 ocytes, provoking a shift from T helper 2 to T helper 1 cells and an increase in lymphocyte-derived t

8 ceptor that is expressed on a subset of CD4+ T helper 1 cells and natural killer T cells, is involved

9 ight inhibit apoptosis, promote expansion of T helper 1 cells and play a key role in acute liver reje

10 inal fluid (CSF) that enhance recruitment of T-helper 1 cells and/or myeloid cells to the central ner

11 Myelin-reactive T helper 1 cells are induced but nonpathogenic in the IL

12 Colitis-inducing CD4+ T-helper 1 cell clones can be obtained by enrichment thr

13 ors Runx3 and GATA3 in the network promoting T helper 1 cell development.

14 hages that serves as an essential inducer of T helper 1 cell development.

15 ter extent in T helper 2 (Th2) cells than in T helper 1 cells during polarization.

16 IL-27-conditioned T helper 1 cells exhibit reduced effector function and a

17 (2016) show that dendritic cells help CD4(+) T helper 1 cell immunity against malaria through PD-L2's

18 demonstration of a beneficial role for CD4+ T helper 1 cells in this disease.

19 lper 2 cells directly inhibited MBP-specific T helper 1 cells in vitro through the release of interle

20 ines known to target CD8+ T cells and CD4(+) T-helper 1 cells in the lungs of C3H/HeN mice infected w

21 sponses in vitro and in vivo and conferred a T-helper-1-cell-like phenotype.

22 unity and suggest that NK cells, like CD4(+) T helper 1 cells, may acquire immunoregulatory functions

23 n the pathogenesis of celiac disease (CD), a T helper 1 cell-mediated enteropathy induced by gluten.

24 e due only to the production of cytokines by T-helper 1 cells or T-helper 2 cells, we obtained bone m

25 depletion of naive cells, T cytotoxic 1 and T helper 1 cells, or by enrichment of regulatory T cells

26 i) B cells suppressed the differentiation of T helper 1 cells, partially via the provision of interle

27 d interferon-gamma (IFNgamma)-producing CD4+ T helper 1 cells participate in MG postnatal organogenes

28 Highly activated T-helper 1 cells, predominantly expressing interferon ga

29 lationship between IL-17-producing cells and T helper 1 cells remain unclear.

30 his effect of GLA-SE was shown to regulate a T-helper 1 cell response upon challenge with live influe

31 T helper 1 cell responses involved in cell-mediated immu

32 es from increased production of IFN-gamma by T helper 1 cells, resulting from enhanced secretion of I

33 In vitro differentiated T helper 1 cells show slight but significant hypersecret

34 The additional presence of T-helper 1 cells specific for streptococcal or staphyloc

35 ributed predominantly to innate and adaptive T-helper 1 cell (TH1) immune responses, whereas the role

36 ribe altered ligands for mature mouse CD4(+) T helper 1 cells that lead to T cell apoptosis by the se

37 ceptor (TCR)-induced cell death (TCR-ICD) in T helper 1 cells, the molecular mechanisms mediating TCR

38 nnate lymphoid cell (ILC) counterpart to the T helper 1 cell type exists.

39 ma (IFN-gamma) and interleukin 2 produced by T-helper 1 cells when comparing patients with AOT or POT

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