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1 es induced by naive and in vivo-primed human T helper 2 cells.
2 ad an impaired ability to differentiate into T helper 2 cells.
3 4, restricted to the appropriately polarized T helper 2 cells.
4 ng the differentiation of naive T cells into T helper 2 cells.
5         Expression of chemokine receptors on T helper 2 cells and eosinophils has been postulated to
6               Experimental studies implicate T helper 2 cells and the cytokines interleukin-4 and -13
7 endritic cells to promote differentiation of T-helper 2 cells and production of their cytokines (IL4,
8                                      Primary T helper 2 cells are heterogeneous, expressing subsets o
9 pment of tolerogenic NKT cells with a marked T helper 2 cell bias that, in turn, regulated the differ
10 e Chemoattractant Receptor Homologous to the T helper 2 cell (CRTH2), a G protein-coupled receptor, p
11 mast cell protease MCPT1, as well as splenic T-helper-2 cell-derived cytokine production.
12 ll as to antigen-experienced T cells, induce T helper 2 cell development, cytokine production or both
13                        Stat6 is critical for T-helper 2 cell differentiation, B-cell Ig class switch,
14                             Peptide-specific T helper 2 cells directly inhibited MBP-specific T helpe
15                                     Atopy--a T helper 2 cell driven hypersensitivity to innocuous ant
16 IL)-4- and IL-13-expressing cells, including T-helper-2 cells, eosinophils and basophils.
17 firm a direct relation between obesity and a T helper 2 cell immune response in women.
18  inflammation, characterized by expansion of T-helper 2 cells in the colon and lung, and infiltration
19 f naive CD4+ T lymphocytes into pro-allergic T-helper 2 cells induces interleukin (IL)4 expression an
20 or other fibrotic diseases associated with a T-helper 2 cell-mediated immune response.
21  helper 17 cells distinct from T helper 1 or T helper 2 cells, participates in the pathogenesis of in
22 erferons, proinflammatory cytokines, and the T-helper-2 cell pathway in predisposed people.
23 migratory dendritic cell subset required for T helper 2 cell polarization following cutaneous challen
24                                              T helper 2 cells regulate inflammatory responses to helm
25                                This modified T-helper-2 cell response should be regarded as a form of
26 ve eczema formation, C. bovis induced robust T helper 2 cell responses.
27 tions but generate stronger canonical CD4(+) T helper 2 cells responses (IL-4, IgE, mast cells, and b
28 munization was developed to produce specific T helper 2 cell (T(H)2)-type antibodies to provide an ef
29 rleukin (IL) 4 and 13 production from CD4(+) T helper 2 cells (T(H)2) and innate lymphoid type 2 cell
30                                 We show that T helper 2 cells (T(H)2), but not other T cell subsets,
31 le of autoimmune T cells toward a protective T helper 2 cell (Th2) type.
32  infections requires the rapid activation of T helper 2 cells (Th2 cells).
33 ted T cells (ATCs) in the lung, particularly T helper 2 cells (Th2), are strongly associated with ind
34 el of allergic asthma, OSPM caused increased T helper 2 cells (Th2), peribronchiolar inflammation, an
35 amily, functions as an important mediator of T helper 2 cell-type (type 2) responses.
36 oduction of cytokines by T-helper 1 cells or T-helper 2 cells, we obtained bone marrow-derived macrop
37                                Cloned murine T helper 2 cells were treated with Pca, N-acetylprocaina

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