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1 , and ectopic expression of which results in T cell leukaemia.
2 lated from a chromosomal breakpoint in human T-cell leukaemia.
3 ate that sustained Zeb2 expression initiates T-cell leukaemia.
4 a range of human diseases including AIDS and T-cell leukaemias.
7 discovered via chromosomal translocations in T-cell leukaemia and shown normally to be essential for
8 nduces a high incidence of myeloid or B- and T-cell leukaemia and the proviral integration sites in t
9 rotein LMO2 is expressed aberrantly in acute T-cell leukaemias as a result of the chromosomal translo
10 us Type 1 (HTLV-1) infection to lethal Adult T-cell Leukaemia (ATL); a progression that is more likel
11 Notably, reactivation of PTEN mainly reduced T-cell leukaemia dissemination but had little effect on
13 , activated by chromosomal translocations in T-cell leukaemias, is normally expressed in haematopoies
14 sly, PTEN expression in the PTEN-null Jurkat T cell leukaemia line was shown to cause reduced prolife
15 or, in Japanese patients with relapsed adult T-cell leukaemia-lymphoma and other peripheral T-cell ly
17 -2, documented diagnosis of aggressive adult T-cell leukaemia-lymphoma or other peripheral T-cell lym
18 Of 13 assessable patients (nine with adult T-cell leukaemia-lymphoma, four with other peripheral T-
20 linically linked to the development of adult T-cell leukaemia/lymphoma (ATL), an aggressive blood can
22 ations were seen among the 114 patients with T-cell leukaemia (p=0.61, 0.35, and 0.53, respectively).
24 rare, sporadic leukaemia similar to a mature T-cell leukaemia seen in some patients with Ataxia Telan
25 and downstream of the splice donor in human T cell leukaemia virus type 1 (HTLV-1) was inserted into
26 ruses human immunodeficiency virus and human T-cell leukaemia virus are resistant to human complement
27 t enforced expression of Lmo1 and Lmo2 cause T cell leukaemias with long latency and that Lmo2 expres
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