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1 (prostate), MDA-MB-231 (breast), and Jurkat (T-cell leukemia).
2 a spectrum of human malignancies, including T cell leukemia.
3 d retroviruses with the former causing adult T cell leukemia.
4 pathy/tropical spastic paraparesis and adult T cell leukemia.
5 ons in both the induction and maintenance of T cell leukemia.
6 perturbation of its activity often leads to T cell leukemia.
7 and MAPK kinase activities in CD3-activated T cell leukemia.
8 ll lines and dampens global transcription in T cell leukemia.
9 ackfan anemia (DBA), congenital asplenia and T cell leukemia.
10 of thymocytes, and subsequent generation of T-cell leukemia.
11 ors was associated with improved outcomes in T-cell leukemia.
12 e 1 (HTLV-1) is the causative agent of adult T-cell leukemia.
13 humans and is etiologically linked to adult T-cell leukemia.
14 cell transformation and development of adult T-cell leukemia.
15 event in the process of ZNF198-FGFR1-induced T-cell leukemia.
16 irus type-1 is the causative agent for adult T-cell leukemia.
17 nt of the aggressive and fatal disease adult T-cell leukemia.
18 1 (HTLV-1) is the etiological agent of adult T-cell leukemia.
19 infects CD4+ T lymphocytes and causes adult T-cell leukemia.
20 rus may play a role in the etiology of adult T-cell leukemia.
21 I (HTLV-1) is the etiological agent of adult T-cell leukemia.
22 onses in the majority of patients with adult T-cell leukemia.
23 1 (HTLV-1) is the etiologic agent for adult T-cell leukemia.
24 investigate the role of this interaction in T-cell leukemia.
25 ed ribosomal mutation in acute lymphoblastic T-cell leukemia.
26 y reduced in CD19(+) spleen B cells from Emu-T cell leukemia 1 (TCL1) transgenic mice relative to the
27 tudies revealed that ROR1 could complex with T-cell leukemia 1 (TCL1) in CLL, we crossed these animal
33 ogenesis related to the progression of adult T cell leukemia and HTLV-1-associated myelopathy/tropica
35 ic virus type 1 (HTLV-1) mainly causes adult T cell leukemia and predominantly immortalizes/transform
36 genetic aberrations that promote acute B and T cell leukemias and the mechanisms of cell transformati
39 e 1 (HTLV-1) is the causative agent of adult T-cell leukemia and HTLV-1-associated myelopathy/tropica
40 virus type 1 (HTLV-1) infection causes adult T-cell leukemia and is associated with a variety of lymp
41 mature lymphocytes, develop multiple B- and T-cell leukemia and lymphoma subtypes, supporting an onc
42 -1), an etiological factor that causes adult T-cell leukemia and lymphoma, has a crucial role in init
46 virus type 1 (HTLV-1) infection causes adult T-cell leukemia and several lymphocyte-mediated inflamma
47 kemia virus type 1 (HTLV-1)-associated adult T-cell leukemia and T-cell lymphoma (ATL) are aggressive
48 with several serious diseases, such as adult T-cell leukemia and tropical spastic paraparesis/myelopa
49 ry tumor virus (MMTV) variant, often induces T-cell leukemias and lymphomas by c-myc activation follo
50 est that FBW7 is a novel tumor suppressor in T cell leukemia, and implicate the loss of FBW7 function
52 and constitutive Notch signaling potentiates T cell leukemia as well as Drosophila neuroblast tumors.
55 of pathologic abnormalities, including adult T cell leukemia (ATL) and HTLV-1-associated myelopathy/t
59 1 (HTLV-1) is the etiological agent of adult T-cell leukemia (ATL) and HTLV-1-associated myelopathy/t
60 e 1 (HTLV-1) is the causative agent of adult T-cell leukemia (ATL) and HTLV-1-associated myelopathy/t
62 s-1 (HTLV-I) is the etiologic agent of adult T-cell leukemia (ATL) and the neurological disorder HTLV
63 oup that first identified a cluster of adult T-cell leukemia (ATL) cases in Japan, provided conclusiv
64 -1)-transformed lymphocyte cell lines, adult T-cell leukemia (ATL) cells, and in other hematologic ma
72 s are implicated in the development of adult T-cell leukemia (ATL), a T-cell malignancy caused by HTL
73 rus type 1 (HTLV-I) is associated with adult T-cell leukemia (ATL), an aggressive lymphoproliferative
74 e-I (HTLV-I) is the etiologic agent of adult T-cell leukemia (ATL), an aggressive lymphoproliferative
75 irus type 1 (HTLV-1) that recapitulate adult T-cell leukemia (ATL)-like leukemic symptoms and display
84 distinct signaling proteins in a panel of 13 T-cell leukemia cell lines treated with a gamma-secretas
87 GBP with selective cytotoxicity toward human T-cell leukemia cells and indicate its potential use in
88 zomib (an agent whose cytotoxicity in Jurkat T-cell leukemia cells is dependent on Noxa) was examined
89 ol for the incubation of Jurkat (human acute T-cell leukemia) cells with Ac5ManNTGc and the quantitat
90 ked to a spectrum of diverse diseases: adult T cell leukemia, encephalomyelopathy, and predisposition
91 tudies, performed in translational models of T cell leukemia, establish a mechanism-based rationale f
92 HTLV) type 1, the etiological agent of adult T-cell leukemia, expresses the viral oncoprotein Tax1.
95 oncogene (encoding the transcription factor T cell leukemia homeobox protein-1) has a major role in
96 n proposed to have tumor suppressor roles in T-cell leukemia homeobox 1/3-transformed human T-ALL cel
97 NA binding site for the poorly characterized T-cell leukemia homeobox 3 (TLX3) TF was confirmed with
101 ell leukemia virus 1 (HTLV-1)-mediated adult T cell leukemia is associated with the ability of viral
102 ood, Nagai et al provide evidence that adult T-cell leukemia is hierarchically organized and sustaine
103 leukemia virus type 1 (HTLV-1)-induced adult T-cell leukemia is linked to the expression of the viral
104 lective and potent cytotoxicity toward human T-cell leukemia Jurkat cells compared with a panel of ca
107 , pTa, Gata3, and Runx1, in both Ikaros null T cell leukemia lines and Ikaros null primary thymocytes
112 kemia virus type 1 (HTLV-1)-associated adult T cell leukemia/lymphoma (ATL) cells as a model system.
117 ype 1 (HTLV-1) is a causative agent of adult T cell leukemia/lymphoma and a variety of inflammatory d
120 virus type 1 (HTLV-1) infection causes adult T-cell leukemia/lymphoma (ATL) and is associated with a
121 d with the lymphoproliferative disease adult T-cell leukemia/lymphoma (ATL) and the neurodegenerative
125 -cell leukemia virus type 1-associated adult T-cell leukemia/lymphoma (ATL) typically has survivals m
126 sactivator initiates transformation in adult T-cell leukemia/lymphoma (ATL), a highly aggressive chem
127 e 1 (HTLV-1) is the causative agent of adult T-cell leukemia/lymphoma (ATL), a malignancy of CD4(+) T
129 leukemia (3), hairy cell leukemia (1), adult T-cell leukemia/lymphoma (ATLL) (1), marginal zone leuke
130 ), anaplastic large-cell lymphoma, and adult T-cell leukemia/lymphoma (ATLL) and a lower incidence of
131 r A (RHOA) hotspot mutations among the adult T-cell leukemia/lymphoma (ATLL) category have opposite b
134 ic virus type-1 (HTLV-1) that initiate adult T-cell leukemia/lymphoma (ATLL) remain unclear, in part
135 We recently reported the first case of adult T-cell leukemia/lymphoma (ATLL) that responded rapidly t
136 CD8(+) T cells, yet this virus causes adult T-cell leukemia/lymphoma (ATLL) that typically has a CD4
140 ouse studies indicate that activation of the T-cell leukemia/lymphoma 1 (TCL1) oncogene is a contribu
141 mmon human leukemia and dysregulation of the T-cell leukemia/lymphoma 1 (TCL1) oncogene is a contribu
145 ive anaplastic large cell lymphoma, 14 adult T-cell leukemia/lymphoma and 44 extranodal NK/T-cell lym
146 e 1 (HTLV-1) is the causative agent of adult T-cell leukemia/lymphoma and HTLV-1-associated myelopath
147 causes two distinct pathologies termed adult T-cell leukemia/lymphoma and tropical spastic paraparesi
148 virus type 1 (HTLV-1) is the cause of adult T-cell leukemia/lymphoma as well as tropical spastic par
149 To study the impact of oncogenic K-Ras on T-cell leukemia/lymphoma development and progression, we
158 1 (HTLV-1) is the etiological agent of adult T-cell leukemia/lymphoma, and it encodes a number of non
159 l lymphoma, cutaneous T-cell lymphoma, adult T-cell leukemia/lymphoma, and other peripheral T-cell ly
160 ided highly diverse results on the issues of T-cell leukemia/lymphoma-initiating cells (T-LICs) and p
167 ferent human cancers including lymphoblastic T-cell leukemia, pancreatic cancer, melanoma and rhabdom
170 Adult patients with acute lymphoblastic T cell leukemia (T-ALL) have a very poor prognosis and f
171 nsmembrane protein highly expressed in acute T-cell leukemia (T-ALL) and in a subset of peripheral T-
172 ly in primary and secondary tissues, we used T-cell leukemia (TCL)1 cells reactive with the autoantig
185 ced apoptosis, we used TRAIL-resistant human T cell leukemia virus type 1 (HTLV-1)-associated adult T
186 cytes) to the total viral burden in 22 human T cell leukemia virus type 1 (HTLV-1)-infected individua
189 Furthermore, the Tax oncoprotein of human T cell leukemia virus type I targeted this complex for i
190 l virus, human immunodeficiency virus, human T cell leukemia virus, human papilloma virus, hepatitis
192 recognizes the foreign Ag Tax from the human T cell leukemia virus-1 when presented by the class I MH
193 sible link with the autoimmune disease human T cell leukemia virus-1-associated myelopathy/tropical s
197 ly discovered the antisense protein of human T-cell leukemia virus (HTLV) type 2 (APH-2), whose messe
198 ntification of the genes necessary for human T-cell leukemia virus (HTLV-1) persistence in humans may
200 cation and partial characterization of human T-cell leukemia virus (HTLV; now known as HTLV-1) produc
207 ence techniques in real time with both human T-cell leukemia virus type 1 (HTLV-1) and human immunode
211 et al report that vaccination against human T-cell leukemia virus type 1 (HTLV-1) basic leucine zipp
213 ally integrated form of the retrovirus human T-cell leukemia virus type 1 (HTLV-1) contains identical
214 rs proposed a model for this region of human T-cell leukemia virus type 1 (HTLV-1) Env in which expul
216 iculoendotheliosis virus A (REV-A) and human T-cell leukemia virus type 1 (HTLV-1) exhibit PCE activi
217 nt data showing that neither HIV-1 nor human T-cell leukemia virus type 1 (HTLV-1) expresses signific
219 PPY motif in the matrix (MA) domain of human T-cell leukemia virus type 1 (HTLV-1) Gag associates wit
228 Surprisingly, the NC protein from human T-cell leukemia virus type 1 (HTLV-1) is an extremely po
231 ablished that cell-free infection with human T-cell leukemia virus type 1 (HTLV-1) is less efficient
242 d, Fujikawa et al demonstrate that the human T-cell leukemia virus type 1 (HTLV-1) oncoprotein Tax in
243 In the present study, we show that the Human T-cell Leukemia Virus Type 1 (HTLV-1) oncoprotein Tax is
244 uggested to be largely dispensable for human T-cell leukemia virus type 1 (HTLV-1) particle biogenesi
245 ecently shown to bind and activate the human T-cell leukemia virus type 1 (HTLV-1) promoter at bases
249 -risk human papillomavirus (HPV) E6 or human T-cell leukemia virus type 1 (HTLV-1) Tax oncoproteins.
250 lishment of humanized mice infected by human T-cell leukemia virus type 1 (HTLV-1) that recapitulate
253 The retroviral oncoprotein Tax from human T-cell leukemia virus type 1 (HTLV-1), an etiological fa
254 udy, we found that HBZ, encoded by the Human T-cell Leukemia Virus type 1 (HTLV-1), binds to multiple
255 of the deltaretroviruses, for example, human T-cell leukemia virus type 1 (HTLV-1), have been evaluat
256 , which can result from infection with human T-cell leukemia virus type 1 (HTLV-1), is associated wit
257 infection by HIV-1, HIV-1Deltavif, and human T-cell leukemia virus type 1 (HTLV-1), while significant
266 s level of BIC by up to 70% in EBV- or human T-cell leukemia virus type 1 (HTLV1)-transformed cell li
268 The Tax oncoprotein encoded by the human T-cell leukemia virus type 1 plays a pivotal role in vir
270 Unlike the resistance of HIV-1 and human T-cell leukemia virus type 1 to hA3G, the resistance of
271 ion of additional human tumor viruses--human T-cell leukemia virus type 1, hepatitis C virus, and Kap
275 viruses in the transplant population: human T-cell leukemia virus type 1; hepatitis E virus; bocavir
284 date tumor suppressor, is repressed in human T-cell leukemia virus type-1 (HTLV-1)-transformed lympho
287 nduces a severe telomere shortening in human T-cell leukemia virus type-1-infected cells which signal
294 omain are highly conserved among the primate T cell leukemia viruses, but this extension is absent in
296 sease onset in murine models of Notch-driven T-cell leukemia, whether Dicer1-processed miRNAs are ess
297 ted mAb by immunizing mice with Jurkat acute T cell leukemia, which binds ILT3.Fc to its membrane.
298 vating IKK, yet only HTLV-1 infection causes T cell leukemia, which correlates with persistent activa
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