ライフサイエンスコーパス: T-cell line

1 infects bovine mammary epithelial cells (MAC-T cell line).

2 cleic acid (DNA) from an HIV-infected Jurkat T cell line.

3 onotype found in the poly(F,Y,A,K)n-specific T cell line.

4 and proliferation by an epitope-specific CD4 T cell line.

5 essed in primary cells and an IL-2-dependent T cell line.

6 of the Notch target gene Hes1 in a leukemia T cell line.

7 ncrease in transfer of HIV-1(BaL) to a human T cell line.

8 from a peptide-independent alloreactive CD8+ T cell line.

9 active Cre recombinase into a loxP-reporter T cell line.

10 on factors indicated that this is a CD4-1(+) T cell line.

11 establishment of an IL-15-dependent CD4-1(+) T cell line.

12 ncorporation in HeLa cells and in the Jurkat T-cell line.

13 mma interferon (IFN-gamma) production by the T-cell line.

14 ng to the GATA3 promoter in the human Jurkat T-cell line.

15 n that against the Cdt-hypersensitive Jurkat T-cell line.

16 e occupancy of three ETS proteins in a human T-cell line.

17 (mac239)) replication in a transformed human T-cell line.

18 apoptotic cell death of the Tax2-established T cell lines.

19 ion in latently infected primary T cells and T cell lines.

20 ppaB kinase and JNK activation in both B and T cell lines.

21 that human GIMAP6 is expressed primarily in T cell lines.

22 primary human T cells but not in transformed T cell lines.

23 through luciferase reporter assays in Jurkat T cell lines.

24 CYLD silencing increases HIV replication in T cell lines.

25 CD4(+) T lymphocytes, but not in some CD4(+) T cell lines.

26 peptide specificity of the in vitro induced T cell lines.

27 ress membrane TRAIL, and induce apoptosis of T-cell lines.

28 n purified CD4+ T cells and Jurkat and HSB-2 T-cell lines.

29 ral blood mononuclear cells and EBV-specific T-cell lines.

30 of both integrins in murine lymphocytes and T-cell lines.

31 orylated in a panel of HTLV-1-infected human T-cell lines.

32 s infectious, yet it spreads poorly in other T-cell lines.

33 compared to the other five SIV-specific CD8+ T-cell lines.

34 o a small number of allergic subject-derived T-cell lines.

35 eases and their functional targets in B- and T-cell lines.

36 in a manner comparable to established mouse TS cell lines.

37 ells, and failure to yield trophoblast stem (TS) cell lines.

38 174 cells and the immortalized rhesus monkey T-cell line 221.

39 more limited neutralizing capacity against a T-cell line-adapted SIV compared to those of the milk Ig

40 positive and -negative clones of the WEHI7.2 T cell line after T cell receptor (TCR) activation by an

41 tosis in primary mouse T cells and in the H9 T cell line after TCR cross-linking.

42 tiation, specificity, or clonal diversity of T cell lines after ex vivo production.

43 ns decreased the survival of HTLV-1-infected T-cell lines, although no cell death was observed after

44 ing RNA in a VEGF receptor-expressing Jurkat T cell line and by SU5416, a pharmacological KDR inhibit

45 ll chemoattractant (CxCL13), both in a model T cell line and in primary human CD4(+) T cells.

46 lock proliferation of an IL-1alpha-dependent T cell line and inhibit production of TNF-alpha by activ

47 Seventeen of these T cell lines and clones reacted to a broad range of stud

48 with IgE to both allergens, Amb a 1-induced T cell lines and clones responded weakly to Art v 6.

49 proliferation of intestinal gliadin-specific T cell lines and clones were measured as evidence of T c

50 ent for ARF-6 was observed in Nef-expressing T cell lines and in HIV-infected primary T cells.

51 gical inhibition reactivated latent HIV-1 in T cell lines and in primary CD4(+) T cells.

52 drugs that primed latent HIV-1 infection in T cell lines and in primary T cells for reactivation and

53 t SAMHD1 expression varies among four CD4(+) T cell lines and is transcriptionally regulated.

54 assays, both in terms of number of specific T cell lines and number of responding donors.

55 0-induced apoptosis was further confirmed in T cell lines and peripheral blood mononuclear cells usin

56 is indeed required for HIV-1 reactivation in T cell lines and primary CD4 T cells.

57 from infecting multiple human macrophage and T cell lines and primary cells by hT5Cyp, as were HIV-2R

58 letion or overexpression of hnRNPLL in B and T cell lines and primary T cells resulted in reciprocal

59 lowed the quantitative comparison of the two T cell lines and provided predictions for the conversion

60 USP9X silencing in both a human T-cell line and mouse primary T cells reduced T-cell rec

61 of Bim partially protected an IL-7-dependent T-cell line and peripheral T cells, especially cells wit

62 udy, we expressed HDAC7 mutant proteins in a T-cell line and use DNA microarrays to identify transcri

63 ects on the functional phenotype of specific T-cell lines and clones by means of flow cytometry, real

64 We generated intestinal T-cell lines and clones from 7 patients with HLA-DQ2.2 (

65 The existing paradigm is that T-cell lines and clones from children differ from those

66 with myeloma and generated peptide-specific T-cell lines and clones from HLA-A*0201-positive (HLA-A*

67 s their recognition by EphA2-specific CD8(+) T-cell lines and clones in vitro via a mechanism that is

68 A panel of human GAD (hGAD65)-specific CD4 T-cell lines and hybridomas was generated to serve as de

69 miRNA expression in HTLV-1-transformed human T-cell lines and primary peripheral blood mononuclear ce

70 oduced robust cytotoxicity against malignant T-cell lines and primary tumors and were protective in a

71 uced the rapid degradation of MCPIP1 in both T-cell lines and quiescent human CD4+ T cells.

72 -cell responses against gluten by generating T-cell lines and T-cell clones from intestinal biopsies

73 l-2 family, is restricted to HTLV-1-infected T-cell lines and to T-cells expressing both Tax and HBZ

74 oth resting T cells and the nonactivated EL4 T cell line, and was up-regulated in both types of T cel

75 ding HIV-1 infection was also normal in most T cell lines, and flap mutant viruses replicated equival

76 cytokines produced by IFX-specific T cells, T cell lines, and T cell clones were evaluated at the mR

77 n increased Rac1 activity in both B-cell and T-cell lines, and its suppression was able to abrogate p

78 Feline fibroblasts, T-cell lines, and primary peripheral blood mononuclear c

79 9 induced the greatest proliferation in CD4+ T-cell lines, and VirB9-specific CD4+ T-cell clones resp

80 n activation, however, some of the malignant T-cell lines are able to coexpress IL-17A and IL-17F, le

81 assays were performed using the Jurkat E6.1 T cell line as a model of T lymphocytes and Caco2-BBE mo

82 s associated with Tam resistance in the MCF7-T cell line as opposed to the Tam-sensitive MCF7 cell li

83 rosis factor alpha and interferon-gamma in a T cell line as well as in thymocytes.

84 rong promoter activity in mature NK cell and T cell lines as well as in immature NK cells.

85 ncing increases HIV infection in transformed T cell lines as well as primary CD4(+) T cells.

86 We observed that myelin-reactive CD4 T cell lines, as well as short-term PHA-activated CD4 T

87 ltivars Ayacuchana and Pasankalla stimulated T cell lines at levels similar to those for gliadin and

88 cassette with flanking loxP sites in a human T-cell line at the precise location of vector integratio

89 rived from rabbits immunized with the Jurkat T-cell line (ATG-Fresenius) or thymus cells (Thymoglobul

90 ify the APOBEC3 repertoire in multiple human T-cell lines, bulk leukocytes and leukocyte subsets, and

91 ecent phosphoproteomic studies of the Jurkat T cell line but difficult to reconcile with former bioch

92 were measured in an HIV-1-inducible reporter T cell line by flow cytometry.

93 SAMHD1 expression was induced in CD4(+) T cell lines by blocking DNA methyltransferase activity,

94 the CD4 T cell response to CCNB1, we derived T cell lines by multiple weekly rounds of stimulation wi

95 by hamster kidney)-21 and an HTLV-1-infected T cell line, C8166, physiologically relevant to HTLV-1-i

96 m, we show that autologous BK virus-specific T cell lines can be reliably generated from viremic KTR.

97 Cell encapsulation studies using an H9 T-cell line (CD4+) were conducted to evaluate feasibilit

98 We show that in an IL-7-dependent T cell line, cells protected from apoptosis nevertheless

99 mRNAs indicated that, compared to the human T-cell lines CEM and H9, prostate cell lines LNCaP and D

100 parental Jurkat cells or an unrelated human T cell line (CEM391) inhibited SOCE and led to sensitiza

101 ral blood mononuclear cells (PBMC) and a CD4 T-cell line compared to monocyte-derived macrophages, or

102 NF expression is elevated in HTLV-1-infected T-cell lines compared to uninfected T cells.

103 The pathogenic C3Bir CD4(+) T-cell line contained more cells producing IL-17 than th

104 ased in a single round of replication from a T cell line containing A3G complexes (CEM cells) after i

105 ipid C-stimulated mouse iNKT cells and human T-cell lines containing NKTs primed with CD1d+C1R transf

106 re autoantigen-nonspecific, in that the same T cell line could suppress autoimmunity induced by three

107 28-35)SL8- and Gag(181-189)CM9-specific CD8+ T-cell lines could suppress the replication of an escape

108 Using mutants of the Jurkat T-cell line deficient for key components of the T-cell r

109 of mutant CARD11 expression constructs into T cell lines demonstrated both loss-of-function and domi

110 n experiments using latently infected Jurkat T-cell lines demonstrated that the HKMT enhancer of Zest

111 T-cell lines derived from lung biopsy specimens of asthm

112 Human T-cell lines derived from nut-allergic patients produced

113 ix mutant replicated efficiently in the MT-4 T-cell line despite maintaining an MVB-targeting phenoty

114 d therefore these findings and the new model T cell line developed will contribute to a greater under

115 viral efficacies of 26 epitope-specific CD8+ T-cell lines directed against seven SIV epitopes in Tat,

116 processed A. fumigatus and the multispecific T-cell lines, directed against all 3 proteins, especiall

117 Using a CD8(+) T-cell line displaying potent noncytotoxic HIV-1 suppres

118 Using a CD1c-reactive T cell line (DN6) to complete an organism-wide survey of

119 ly, silencing of MOV10 expression in a human T cell line enhanced HIV-1 replication.

120 CD4(+) T-cell lines enriched for VZV specificity were generated

121 Here, we show that a set of malignant T-cell lines established from patients with cutaneous T-

122 Tax-positive HTLV-1-transformed T-cell lines express elevated levels of p21(CIP1/WAF1),

123 Interestingly, stable T cell lines expressing dominant-negative RhoA mimicked

124 We also show that T cell lines expressing modified U1 snRNAs exhibit reduc

125 NS5 protein inhibits HIV replication, CD4(+) T cell lines expressing this protein were generated.

126 We generated CD4(+) T cell lines expressing varying levels of CCR5 on the ce

127 , in contrast to its parental virus, infects T-cell lines expressing low levels of cell surface CCR5.

128 was highlighted by data showing that CEM-SS T-cell lines expressing near-physiologic levels of APOBE

129 fers the possibility to generate BK-specific T cell lines for adoptive immunotherapy.

130 senting cells to an M1 epitope-specific CD8+ T-cell line for specific lysis.

131 Furthermore, when generating T-cell lines for adoptive T-cell therapy, it avoids the

132 itopes, establishing antigen-specific memory T-cell lines for identifying CD8(+) and CD4(+) T-cell ep

133 ated a large number of MAGE-A3-specific CD4+ T cell lines from all individuals tested, enabling full

134 oted that all bronchoalveolar lavage-derived T cell lines from HLA-DP2-expressing CBD patients contai

135 T cell lines from KTR and healthy control showed similar

136 tion and CTL activity of PBMCs and of CD8(+) T cell lines from patients with MS.

137 ved through addition of IL-2 to HPV-specific T cell lines from RRP patients.

138 nical-scale protocol to generate BK-specific T cell lines from viremic KTR.

139 esent study, we established cytotoxic CD4(+) T cell lines from VV-immune donors, which recognize epit

140 us (HTLV; now known as HTLV-1) produced by a T-cell line from a lymphoma patient.

141 T-cell lines from all donors, vaccinated from 1 month to

142 ng was observed in mice injected with CD4(+) T-cell lines from diabetic donors.

143 MBC4 was able to activate T-cell lines from donors with birch pollen allergy and f

144 significantly less frequent in EBV-specific T-cell lines from patients with EBV-associated nasophary

145 We developed a method to generate Ts cell lines from freshly isolated lamina propria lymph

146 In contrast to previous reports in T-cell lines, FTY720-induced toxicity in the Raji cell l

147 In addition, CD8+ T cell lines generated by gD53-61, gD70-78, and gD278-28

148 peripheral blood mononuclear cell (PBMC) and T cell lines generated from healthy donors.

149 is of 50 of a total of 236 CD4(+) and CD8(+) T cell lines grown from individual handpicked islets or

150 The majority of the T cell lines had high Foxp3 expression and secreted both

151 The resulting CD4(+) regulatory T cell lines had marked inhibition on autologous myelin

152 Studies with CD8+ T cell lines have suggested that Mtb Ags gain access to

153 These findings demonstrate that in human T cell lines, HIV-1 and simian immunodeficiency virus ca

154 ll as a TCR derived from a CD1b-autoreactive T-cell line (HJ1Tg).

155 oth kill thymocytes, peripheral T cells, and T cell lines; however, we have found that galectin-9 and

156 In primary T cells and T-cell lines, HRF triggered a high but nonsustained peak

157 stimulation in PBMCs, CD4(+) T cells, or the T cell line HuT78 activates the Notch pathway by nuclear

158 We report that in the human T cell line HuT78 and in primary murine T lymphocytes, s

159 Using a previously established T-cell line immortalized with an HTLV-1 molecular clone

160 ectrometry was performed on the human Jurkat T cell line in the presence of U0126, an inhibitor of ER

161 imed in vitro a large number of specific CD4 T cell lines in all the donors.

162 , we analyzed the dynamics of the two CD4(+) T cell lines in mice during infection with L. monocytoge

163 enuate the proliferation of disease-specific T cell lines in response to gluten antigens and, therefo

164 ng DSBs, in benign Barrett's epithelial (BAR-T) cell lines in vitro and in patients with Barrett's es

165 t and in response to signaling in a cultured T-cell line in a manner which temporally correlates with

166 HDR donors in hESCs to generate an isogenic TS cell line in a scarless manner and to model the 16p11

167 issue-derived stem cells, and a human Jurkat T cell line) in vitro.

168 cific overexpression of either SOD in Jurkat T cell lines increases intracellular production of H2O2

169 eceptors from the regulatory IL-10-secreting T cell line induced by the random amino acid copolymers

170 We enumerated the Lipo5-specific T cell lines induced in vitro by weekly rounds of specif

171 OCS1 and SOCS3, but not SOCS2, in the Jurkat T cell line inhibited IFN-alpha-induced phosphorylated S

172 -tRNA(Val) observed in the homoplasmic 1624C>T-cell lines is caused by a rapid degradation of the dea

173 driving IL-17 secretion in short-term CD4(+) T cell lines isolated from human peripheral blood, altho

174 ramer(+) cells and cloned them from uncloned T cell lines isolated from spleen and lymph nodes of dia

175 nes were derived from HIV-reactive CD28+CD8+ T-cell lines isolated from 7 HIV+ HAART-treated patients

176 In Jurkat T-cell lines, it has been shown that TAC, in addition to

177 replication in a latently HIV-infected human T-cell line (J1.1).

178 on of endogenous c-Myc by Runx1 in the human T cell line Jurkat and murine primary hematopoietic cell

179 s increased after restimulation in the human T cell line Jurkat, in human memory and Th2 cells, and i

180 The T-cell line Jurkat ceased proliferation and died shortly

181 Our earlier work showed that a somatic T-cell line Jurkat mutant H123 bearing a defect in Ca(2+

182 GBV-C NS5A proteins were expressed in a CD4+ T cell line (Jurkat), and both induced a potent, dose-de

183 ent with SS, an acute lymphoblastic leukemia T-cell line (Jurkat), and JFL (a FAS-low variant of Jurk

184 (LBRM-33), similar to its effects on a human T-cell line (Jurkat), but did not inhibit IL-2 productio

185 en analyzed in physiologically more relevant T-cell lines (Jurkat and CEM), NC mutant viruses remaine

186 nfection of peripheral blood lymphocytes and T-cell lines (Jurkat and CEMX174).

187 f the herpesvirus saimiri-transformed CD8(+) T-cell line, K#1 50K, has potent HIV-1-inhibitory activi

188 interleukin-2 (IL-2) production by a murine T-cell line (LBRM-33), similar to its effects on a human

189 , two different murine CD4(+) TCR transgenic T cell lines, LLO118 and LLO56, both specific for the sa

190 The CD4(+) T cell lines lysed VV-infected, Ag- and peptide-pulsed t

191 Based on studies with activated T cell lines maintained in vitro, IL-2 is known to activ

192 d Ag-specific cytotoxic activity of a CD8(+) T cell line manifested in a 4-h assay (10-20% increase),

193 Here, by using a T cell line model, we demonstrate that Id1 expression st

194 ant process, p101 was overexpressed in human T-cell lines Molt-4 and Jurkat.

195 B-HCV") has been shown to infect established T cell lines (Molt-4 and Jurkat) and primary human naive

196 sociation with DRMs in 293T cells and in the T-cell line, MOLT 4.

197 at we cloned from the MDV-transformed CD4(+) T-cell line MSB-1.

198 ell lymphotrophic virus (HTLV)-1-transformed T cell lines, MT-2, MT-4, SLB-1, and ATL-2.

199 and irradiated cells of an HTLV-1-producing T-cell line, MT2 was used to determine the permissivity

200 HIV-specific T cell lines obtained from vaccine recipients confirmed

201 e also analyzed A3 editing of HTLV-1 in five T-cell lines obtained from HTLV-1-infected patients.

202 44 internalization varied among cultured CD8 T cell lines of different specificities, and correlated

203 using HLA-A2 matched effector cells (CD8(+) T cell line or clone) and target cells supporting full H

204 al strains in pairwise competition assays in T-cell lines, primary cells, and the ecotropic human imm

205 Furthermore, T-cell lines primed in vitro from the blood of melanoma

206 Other malignant T-cell lines produce IL-17A but not IL-17F.

207 AK kinase inhibitors have depressed leukemic T cell line proliferation.

208 Most of the T cell lines reacted with the peptides 9-23 and 14-28, l

209 we demonstrate that M. leprae-specific mouse T-cell lines recognize several of these antigens, with t

210 exogenous NY-ESO-1 protein, only one CD4(+) T cell line recognized NY-ESO-1(+) HLA class II-expressi

211 However, we found that virus-specific T cell lines recognized up to 10% of a panel of 44 HLA d

212 This CD4(+) T cell line recognizes target cells infected with influe

213 e, we showed that silencing IFN-lambda1 in T/T cell line reduced basal ISG expression and improved an

214 te more potently activated a CD1b-restricted T cell line relative to Mo-DCs pulsed with free lipid Ag

215 These rat TS cell lines represent valuable new in vitro models for

216 Moreover, Art v 6-induced T cell lines responded stronger to Amb a 1.

217 xt to an "optimal" Kozak sequence in a human T cell line resulted in enhanced translation of a single

218 of complete STLV genome sequences from these T cell lines revealed them to be closely related but dis

219 dhesive properties of the human mature B and T cell lines RPMI 8866, Daudi and Jurkats.

220 These CD4+CD25+T cell lines secrete high levels of IL-10 and IL-13 but

221 Analysis of expanded Vgamma2Vdelta2 T-cell lines showed the abundant presence of TLR2 mRNA,

222 In this study, we isolated a CD4 T cell line specific for CF that produces inflammatory c

223 e of A/Japan/305/1957 (H2N2), we generated a T cell line specific to this epitope.

224 nalyzed two naturally occurring human CD4(+) T cell lines specific for different peptides from cytoso

225 A-DR-typed healthy donors, we derived CD4(+) T cell lines specific for eight MDK peptides.

226 In contrast, most of the T cell lines specific for Lipo5 reacted with G2, reveali

227 IL-10-secreting regulatory T cell lines specific to glatiramer acetate [poly(Y,E,A,

228 were characterized in vitro by using a human T-cell line specific for the immunodominant epitope of B

229 T-cell receptor (TCR) cDNAs from murine CD8 T-cell lines specific for either pIRS-21097-1105 or pCDC

230 CD8 T-cell lines specific for NS3-1073 and NS5-2594 were exp

231 with collagen-induced arthritis (CIA), this T cell line specifically enhanced the severity of autoim

232 ed a CpG island that is methylated in CD4(+) T cell lines (such as Jurkat and Sup-T1), resulting in t

233 timulation of the Fas receptor in the Jurkat T-cell line, suggesting that Hip is a substrate unique t

234 ty of interactions are common to both B- and T-cell lines, suggesting interactions may be highly cell

235 xposed IKpDC induced apoptosis of the CD4(+) T cell line SupT1 via the TRAIL pathway.

236 xamined the fine specificity of VSG-specific T-cell lines, T-cell hybridomas, and Th cells activated

237 Dau c 1-specific T-cell lines (TCL) and clones (TCC) established from PBM

238 re detected in 83% of Art v 125-36 -reactive T-cell lines (TCL) from mugwort-allergic individuals, bu

239 Can f 4-specific CD4(+)CD45RO(+) T-cell lines (TCLs) from allergic and healthy subjects w

240 In primary tumors and TCL1-transfected T-cell lines, TCR engagement leads to rapid recruitment

241 on of TrkB is also higher in HTLV-1-infected T-cell lines than in uninfected T cells.

242 rt hairpin RNA in both primary T cells and a T cell line that recapitulates the stability phase of re

243 ical to those recently found in a regulatory T cell line that secretes both IL-4 and IL-10 induced in

244 This approach identified several independent T cell lines that proliferated robustly in response to I

245 s among the drug, HLA, and TCR, we generated T cell lines that react to ALP or its metabolite oxypuri

246 sion with either HSV or VZV enriches for CD4 T cell lines that recognize the other agent at the whole

247 ild-type virus replication, a Tet-off Jurkat T-cell line that expressed approximately 15-fold-higher

248 t express human or macaque CD16 and a CD4(+) T-cell line that expresses luciferase from a Tat-inducib

249 cell carcinoma (RCC), we identified a CD4(+) T-cell line that showed TCR-mediated recognition and lys

250 H5N1 peptide-specific T-cell lines that did not cross-react with H1 or H3 infl

251 lus fumigatus proteins, Aspergillus-specific T-cell lines that have a broad specificity and favorable

252 at it is possible to establish clonal CD8(+) T-cell lines that represent the most abundant specificit

253 spleen and lymph node cells in vitro yielded T-cell lines that specifically produced interferon-gamma

254 1) vif compromise virus replication in human T-cell lines that stably express APOBEC3F (A3F) or APOBE

255 by ELF5 will be instrumental to derive human TS cell lines that truly reflect early placental trophob

256 and enables derivation of trophoblast stem (TS) cell lines that, when injected into blastocysts, chi

257 epared M. tuberculosis-specific gamma9delta2 T cell lines to study their direct protective effects an

258 l antigen-specific C3H/HeJBir (C3Bir) CD4(+) T-cell line to C3H/HeSnJ SCID mice.

259 have used an IA(b)/OMP-19(107-122)-specific T-cell line to monitor antigen display ex vivo during ac

260 abilities of different epitope-specific CD8+ T-cell lines to control simian immunodeficiency virus (S

261 tor Vbeta genes in expanded microbe-reactive T-cell lines to determine their clonal diversity.

262 firmed the responsiveness of these cytotoxic T-cell lines to seven peptides described previously and

263 re examined in parallel with mouse and human T cell lines transfected with CD40.

264 ate the effect: an anti-myelin basic protein T-cell line treated with cefuroxime or penicillin was mo

265 s the major coreceptor for cellular entry of T-cell line-tropic (X4) HIV-1 strains.

266 olyclonal vaccinia virus-polyspecific CD8(+) T cell line, two conserved vaccinia-derived TAP-independ

267 D147-dependent phosphoproteome in the Jurkat T cell line upon treatment with T cell stimulating agent

268 cytosis in the adherent cell 293T and Jurkat T cell lines using a fusion protein of extracellular CD4

269 ects, allowing the isolation of HPV-specific T cell lines using tetramers.

270 f a beryllium-responsive, HLA-DP2-restricted T cell line was seen after the induction of 4-1BB ligand

271 One T cell line was stimulated by HLA-compatible MDK-transfe

272 strated that forced expression of GRAIL in a T cell line was sufficient for conversion of these cells

273 n of food allergen-reactive Bet v 1-specific T-cell lines was assessed.

274 +), OT-II CD4+ T cells, and the human Jurkat T cell line, we show that physiological levels of H(2)S

275 human T cells and a reporter assay in Jurkat T cell lines, we dissected the regulation of TLR10, a TL

276 ted CD4(+) T cells from spleen and lung, and T cell lines, we found that the majority of these T cell

277 Using an early T-cell line, we describe two branches of this network.

278 a/beta pairs cloned from single cells of the T cell line were inserted into a retrovirus vector linke

279 The T cell lines were antigen-specific and showed no nonspec

280 CD4-expressing T cell lines were constructed to constitutively express

281 Continuously growing T cell lines were established from two baboons, animals

282 Polyclonal T cell lines were generated from either normal mice or m

283 In this study, CD4(+) regulatory T cell lines were generated from patients with multiple

284 By contrast, the peptide 14-28-specific T cell lines were not stimulated in any of these conditi

285 CD4(+) T cell lines were rendered virtually uninfectable, with

286 T cell lines were specific for 15 immunodominant peptide

287 The peptide 9-23-specific T cell lines were specifically stimulated by autologous

288 Allergen-specific T-cell lines were analyzed for epitope recognition.

289 28-35)SL8- and Gag(181-189)CM9-specific CD8+ T-cell lines were consistently superior at suppressing v

290 For epitope mapping, Mal d 1-specific T-cell lines were stimulated with overlapping synthetic

291 chemical genetic inhibitor system in Jurkat T cell lines, where the inhibitor blocked ZAP-70-depende

292 This 9-mer serves to direct cytolysis by T cell lines, whereas a related 10-mer (E7(11-20)), prev

293 Unexpectedly, IL-27 did not inhibit HIV-1 in T cell lines, whereas IL-2 inhibited HIV-1 replication i

294 ired for proliferation and survival of these T cell lines whether or not JAKs or STATs were mutated.

295 gIIFPLA(2) inhibited the proliferation of a T cell line, which was not seen with group IIA PLA(2).

296 interrogated vaccinia-reactive CD4 in vitro T cell lines with vaccinia protein fragments expressed f

297 age is observed upon infection of the Jurkat T-cell line with vesicular stomatitis virus G glycoprote

298 We screened 25 human cytotoxic T-cell lines with adenovirus specificity to extensively

299 In addition, treatment of malignant T-cell lines with peg-Arg I significantly impaired their

300 CR-alpha LCR-linked transgenes into existing T cell lines yields incomplete LCR activity.