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1 1 expression was associated with an impaired T cell proliferative response.
2 63, that generated a low-level but clear-cut T cell proliferative response.
3 -gamma, TNF-alpha, and IL-6 in the augmented T cell-proliferative response.
4 mus nor everolimus could inhibit the CD4CD28 T-cell proliferative response.
5 ing a significant HER-2/neu protein-specific T-cell proliferative response.
6 atory mediators, and failed to induce robust T cell proliferative responses.
7 ed by significant adaptive CD4(+) and CD8(+) T cell proliferative responses.
8 remia was not associated with an increase in T cell proliferative responses.
9 ent spleen APC in vitro leads to normal CD4+ T cell proliferative responses.
10 ther, mIEC did not inhibit splenic alphabeta T cell proliferative responses.
11 cant reduction in both MBP- and PLP-specific T cell proliferative responses.
12 the potency of APCs and boost mitogen-driven T-cell proliferative responses.
13 re involved in the ability of FDC to inhibit T-cell proliferative responses.
14 for antibody binding and induction of human T-cell proliferative responses.
15 accompanied by diminished antigen-specific, T-cell proliferative responses.
16 eral blood lymphocytes and better priming of T-cell proliferative responses.
17 llergic subjects but did not change in vitro T-cell proliferative responses.
18 hanges paralleled reduced IL-2 secretion and T cell proliferative responses after TCR-CD28 stimulatio
19 construct produced antibodies and exhibited T-cell proliferative responses against core or envelope.
21 significantly increased autoantigen-induced T cell proliferative responses along with greater number
22 tion of innate immune responses and adaptive T cell proliferative responses, along with only transien
23 ptides containing these sequences stimulated T cell proliferative responses, although less intensely
24 decreased its effectiveness in costimulating T cell proliferative response and early IL-2 production
26 s characterized by antigen-specific impaired T cell proliferative responses and a distinct pattern of
27 ice treated with recombinant Map had reduced T cell proliferative responses and a significantly reduc
29 ing that anti-TNF treatment in vivo enhances T cell proliferative responses and cytokine production p
30 as significant levels of OVA-specific CD4(+) T cell proliferative responses and OVA-induced IFN-gamma
31 ion, significant levels of OVA-specific CD4+ T cell proliferative responses and OVA-induced IL-4 and
34 fold increase in the median p24-induced CD4+ T-cell proliferative response and a 57% increase in the
35 on of E4 and E1 had little impact on the CD4 T-cell proliferative response and cytolytic activity of
36 compared with Ab positivity, we assessed the T-cell proliferative responses and Ab responses (islet c
37 ograft histopathology as well as anti-HLA-A2 T-cell proliferative responses and anti-HLA-A2 antibody
40 neration of T-regulatory cells that suppress T-cell proliferative responses and induce tolerance.
41 pulsed DC can induce both E7-specific CD4(+) T-cell proliferative responses and strong CD8(+) CTL res
43 A and/or SRL both in vitro (by inhibiting of T-cell proliferative response) and in vivo (by inhibitin
44 -specific intrahepatic and peripheral CD4(+) T cell proliferative responses, and cytokines (enzyme-li
45 ion of maturation markers, IL-12 production, T cell proliferative responses, and IFN-gamma production
46 noglobulin A (IgA)- and IgG-secreting cells, T-cell proliferative responses, and gamma interferon sec
48 nt enhancement in anti-CD3-stimulated CD4(+) T-cell proliferative responses, and this proliferation w
50 n present on the surface of donor DCs, donor T cell proliferative responses are generated only in res
52 allergen-driven IL-4(+) CD4(+) T cells, and T-cell proliferative responses are detectable in the per
55 ion significantly increased the CEA-specific T-cell proliferative responses as well as the cytolytic
56 key positions induced enhanced specific CD4+ T cell proliferative responses at lower peptide concentr
58 ogeneic MLRs between DCs and T cells reduced T cell proliferative responses but did so less efficient
59 type, fail to mount a detectable FV-specific T-cell proliferative response but nevertheless produce F
60 or necrosis factor alpha failed to block the T cell proliferative responses, but anti-IL-2 was strong
61 stimulatory activity for primary and memory T-cell proliferative responses, but this was substantial
62 acterized by a lack of virus-specific CD4(+) T-cell-proliferative responses, but strong responses hav
65 pse formation prior to the induction of full T cell proliferative responses by concurrent indirect Ag
66 atory T cells that more efficiently suppress T cell proliferative responses by mixed leukocyte reacti
67 -shock-protein peptides elicited significant T-cell proliferative responses by the gamma delta subset
68 lls showed enhanced suppressive activity for T cell proliferative responses compared with freshly iso
69 cells, B cells, or natural killer cells, or T-cell proliferative response compared with interferon b
72 demonstrated CMV-specific CD4(+) and CD8(+) T cell proliferative responses from PBMC, with CD4(+)IFN
73 5-284, 295-314, and 305-324) elicited strong T-cell proliferative responses from all strains of mice
74 more, both the magnitude of p24-induced CD4+ T-cell proliferative responses from CD8-depleted PBMC an
76 Our results suggest that the inhibition of T cell proliferative response in microgravity culture is
77 -beta fusion cells failed to induce a strong T cell proliferative response in vitro, mainly due to th
79 d the CTLp frequency, anti-NP Ab titers, and T cell proliferative responses in mice that were injecte
80 alf-life becomes apparent during analyses of T cell proliferative responses in mice, particularly whe
82 tion of cell cycle progression and sustained T cell proliferative responses in naive T cell populatio
83 t epitope, peptide 5 (P5), stimulates strong T cell proliferative responses in subjects with delayed
84 and TILN immunization induced specific CD4+ T cell proliferative responses in the iliac lymph nodes,
86 ryptophan (1-mT) results in increased CD4(+) T-cell proliferative response in PBMCs from HIV-infected
90 nerally marked, but temporary suppression of T-cell proliferative responses in vitro to phytohemagglu
91 a low level of immunogenicity; (4) decreased T-cell proliferative responses in vitro, and (5) did not
96 inally, both internalization of CD26 and the T cell proliferative response induced by CD26-mediated c
99 tablished in male mice: ZP3 peptide-specific T cell proliferative response is reduced and AOD is abse
101 st dose levels of vaccine, peptide-specific, T-cell proliferative responses (n = 3) and/or DTH respon
103 cell deficiency did not significantly alter T cell proliferative response or cause a shift in the Th
104 and inguinal lymph nodes, without affecting T cell proliferative responses or levels of anticollagen
105 gammaRIIB only modestly affected initial CD4 T cell proliferative responses, suggesting that FcgammaR
106 s anergy to recall antigens and lower (<70%) T-cell proliferative responses than controls after activ
107 te resulted in abrogation of hapten-specific T cell proliferative responses that correlated with dimi
108 ood of infected individuals, inhibited human T cell proliferative response through interaction with t
109 d dendritic cell responses (flow cytometry), T-cell proliferative responses (thymidine incorporation)
110 ll peptide tolerance suppressed the in vitro T cell proliferative response to AChR and its dominant a
112 As little as 5 microM PAHA led to a 10-fold T cell proliferative response to chromatin in short term
114 iciency only partially reduced the naive CD8 T cell proliferative response to IL-15/IL-15Ralpha compl
115 was associated with a significantly reduced T cell proliferative response to mycobacterial Hsp65, wh
116 eduction in EAE correlated with a diminished T cell proliferative response to myelin basic protein in
119 h lowered serum autoantibody levels, reduced T cell proliferative responses to AChR, and an expansion
120 e immunocompromised, with reduced polyclonal T cell proliferative responses to alloantigen, defined p
121 cell functions including IL-2 secretion and T cell proliferative responses to CD28 plus T cell recep
124 We compare changes in lymphocyte subsets, T cell proliferative responses to disease-associated tar
126 oviding sufficient help to allow optimal CD8 T cell proliferative responses to exosomal protein.
127 First, neither APC type was able to initiate T cell proliferative responses to full-length native Top
131 helper lymphocytes, manifested by increased T cell proliferative responses to HIV-1 Gag and recall a
132 owever, it has been difficult to demonstrate T cell proliferative responses to human insulin in IDDM
133 n of 25 tested persons made antigen-specific T cell proliferative responses to L2E7, and peripheral b
134 IL-12 significantly increased PBMC and CD4+ T cell proliferative responses to p24 Ag in HIV-infected
136 bioavailability facilitated the induction of T cell proliferative responses to suboptimal stimuli.
138 Eleven of the 21 patients also developed T cell proliferative responses to the homologous self-Ag
140 Abs, inhibition of SIV replication, and CD4+ T cell proliferative responses to three of the extracell
142 row also had a reduced capacity to stimulate T cell proliferative responses to tubercle bacillus Ag 8
143 validated linear discriminant analysis using T cell proliferative responses to two regions of Tri r 2
144 immunization, respectively), in increases in T cell-proliferative response to HPV-16 L1 VLPs (P<.001)
145 lerosis, but had no inhibitory effect on the T cell-proliferative response to myelin basic protein (M
147 ient than untreated DCs in driving syngeneic T cell-proliferative responses to staphylococcal enterot
149 of T-cell tolerance was assessed by splenic T-cell proliferative response to antigen at 5 weeks.
150 protective effect is associated with reduced T-cell proliferative response to B-(9-23) in B-(9-23)-tr
152 l but two patients made primary antibody and T-cell proliferative responses to a foreign antigen admi
153 L-17 receptor (R):Fc fusion protein inhibits T-cell proliferative responses to alloantigens and prolo
154 rom four different colonies side-by-side for T-cell proliferative responses to an expanded panel of a
155 nfiltration into grafts but not with altered T-cell proliferative responses to donor stimulators.
156 R-/- --> F1 chimeras were also able to mount T-cell proliferative responses to foreign antigens equal
159 ulin, saliva IgA binding to insulin, or CD4+ T-cell proliferative responses to insulin were observed
161 After the transplantation of thymus tissue, T-cell proliferative responses to mitogens developed in
166 as detected by inhibition of the Ag-specific T cell proliferative response upon Ag presentation by IF
168 dendritic cells (DCs) to induce Ag-specific T cell proliferative responses was significantly reduced
169 r ability to transduce CLL cells, a vigorous T-cell proliferative response was obtained using cells t
170 well as anti-HLA-A2 antibody development and T cell proliferative responses were determined at days +
171 en delayed-type hypersensitivity and splenic T cell proliferative responses were examined, Peyer's pa
172 persistent, and vigorous HIV-1-specific CD4+ T cell proliferative responses were present, resulting i
174 Furthermore, specific CD4(+) and CD8(+) T cell proliferative responses were significantly increa
176 mature DCs were used to stimulate allogeneic T cells, proliferative responses were dampened (approxim
177 fied subsets of lymphocytes and quantitative T-cell proliferative response were assessed in an explor
178 her neutralizing antibodies nor SIV-specific T-cell proliferative responses were detectable in any of
179 troy islet tissue in vivo though spontaneous T-cell proliferative responses were observed in prediabe
180 he sole class II molecule generated a robust T cell-proliferative response when primed with peptide 2
181 l newborn blood specimens tested also showed T cell proliferative responses, which included a marked
182 ith specific impairment of ex vivo antidonor T cell proliferative responses, which was not reversed b
183 These data suggest that Ply induces CD4 T cell proliferative responses with production of IFN- g
184 the absence of strong HIV-1-specific, CD4(+) T-cell-proliferative responses, yet the mechanism underl
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