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1 insight into how deficiencies in E2A lead to T lymphoma.
2 ranscription when these cells are fused to a T lymphoma.
3 s essential for the efficient development of T lymphoma.
4 ible to N-methyl-N-nitrosourea (MNU)-induced T lymphomas.
5 ation and the ability of the virus to induce T lymphomas.
6 on EBV-transformed B cells, but not on B or T lymphomas.
7 c-myb and outside the transcription unit in T-lymphomas (Ahi-1, fit-1, and Mis-2) and monocytic and
8 CD44 to cell rolling, a CD44-negative mouse T lymphoma AKR1 was transfected with wild type (WT) or m
9 m during PDT-induced apoptosis, Jurkat human T lymphoma and Chinese hamster ovary cells were labeled
10 highly oncogenic herpesvirus that can cause T lymphomas and peripheral nerve demyelination in chicke
11 are present on the surface of a murine EL-4 T-lymphoma and a human carcinoma cell (HeLa), 2) arrive
12 c leukemia (T-ALL), T-lymphoid blast crisis, T-lymphoma, and B-cell chronic lymphocytic leukemia (B-C
13 for the lineage marker CD5 selectively kill T-lymphoma but not normal T cells, although both express
14 nterfering RNA treatment of the Jurkat human T lymphoma cell line or human peripheral blood T cells d
15 emosensitivity assays, using the L5178 mouse T lymphoma cell line transfected with the human MDR1 gen
17 e expression differences between two related T lymphoma cell lines, HuT78 and H9, were identified.
20 in domain of MDC-L supported adhesion of the T-lymphoma cell line, Jurkat, in a concentration- and di
22 RNA) stability decreases rapidly when murine T lymphoma cells are treated with the synthetic glucocor
23 we show that Fas-dependent killing of Jurkat T lymphoma cells by SW620 colon cancer cells requires ca
26 n human epidermoid carcinoma A431 and Jurkat T lymphoma cells that are, respectively, rich in or lack
27 rs (IL-2R) in plasma membranes of Kit 225 K6 T lymphoma cells were investigated by fluorescence reson
29 se required for galectin-1 susceptibility of T lymphoma cells, indicating that similar O-glycan ligan
33 ot induce significant cytotoxicity to feline T-lymphoma cells (3201B) or human T-lymphoma cells (CEM)
36 current studies, we used wild-type (WT) S49 T-lymphoma cells and the kin(-) variant (which lacks pro
37 blocks T-cell-receptor-mediated apoptosis in T-lymphoma cells does not block anti-mu-induced apoptosi
38 c domain of TNFR2 (p75) and is detectable in T-lymphoma cells stably transfected with the TRAF2A cDNA
43 when cultured in vitro with the human Jurkat T lymphoma, CHL-1 melanoma, and SBC-3 small cell lung ca
44 lymphocyte-specific genes in plasmacytoma x T lymphoma hybrids can be prevented by preserving Oct-2
45 on factor on the phenotype of plasmacytoma x T lymphoma hybrids established a critical role for Oct-2
46 the most oncogenic herpesviruses and induces T lymphomas in chickens within weeks after infection.
48 Rac1 is mediated through the suppression of T lymphoma invasion and metastasis 1 (Tiam1) and its ups
49 potential miR-10b targets identified Tiam1 (T lymphoma invasion and metastasis 1), a guanidine excha
50 c guanine nucleotide exchange factor, Tiam1 (T lymphoma invasion and metastasis 1), in transducing a
51 observed for the amino-terminal PH domain of T lymphoma invasion and metastasis protein (Tiam-1), the
52 -specific guanine nucleotide exchange factor T-lymphoma invasion and metastasis 1 (Tiam1) to speciali
54 al structure of the DH and PH domains of the T-lymphoma invasion and metastasis factor 1 (Tiam1) prot
55 d increase in protein and mRNA expression of T-lymphoma invasion and metastasis gene 1 (Tiam1), a gua
58 r of c-myb was provided by one of the murine T-lymphoma lines bearing an insertion at Ahi-1 (p/m16i)
59 pment of hemopathies and is downregulated in T-lymphomas, such as anaplastic large-cell lymphoma (ALC
61 mice also develop at lower frequencies B and T lymphomas with antigen receptor locus translocations.
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