ライフサイエンスコーパス: T3

1 T3 binding to TRbeta increased DC viability and augmente

2 T3 binds to thyroid hormone receptor, which heterodimeri

3 T3 concentrations were increased in tadpoles exposed to

4 T3 is believed to affect development by regulating targe

5 T3 promoted O4(+) cell differentiation in mouse, but not

6 T3 reduces occupancy of SMAD-binding elements in respons

7 T3+Dex enhanced elementary Ca release measured by Ca spa

8 T3-mediated gene regulation was analyzed in human HCC ce

9 T3/T4 therapy was associated with procurement of signifi

10 3217 patients, T2 in 1128 patients (35.1%), T3 in 789 patients (24.5%), and T4 in 185 patients (5.8%

11 final product (T0), and after 1 (T1) and 3 (T3) months of ageing, respectively).

12 et sigma1 of serotype 1 (T1) and serotype 3 (T3) reoviruses.

13 onuclear Type 2 (T2) and a binuclear Type 3 (T3) site, arranged in a trinuclear copper cluster (TNC),

14 subcutaneous (SC) VRC01 (treatment group 3 [T3], n = 20); placebo (placebo group 3 [P3], n = 4) dose

15 1%), 5 of 17 T2b lesions (29.4%), and 3 of 6 T3 lesions (50.0%).

16 r tumors were associated with higher Ca19.9, T3-T4 and N1, higher grade, perineural invasion, R1 rese

17 expression, and Dot1L in turn functions as a T3 receptor (TR) coactivator to promote vertebrate devel

18 ced metamorphosis and that Dot1L is itself a T3 target gene.

19 ut not neuroblasts, express high levels of a T3-inactivating deiodinase, Dio3.

20 For patients with locally advanced (T3, T4) disease, organ-preservation surgery, combined ch

21 pares the cardiovascular system from adverse T3 action.

22 After T3-SCI, the MCA had more collagen I (42%), collagen III

23 pressures, inward remodelling occurred after T3-SCI with a 40% reduction in distensibility (both P <

24 ring multilamellar vesicles entrapping alpha-T3 resulted in a dramatically improved (by at least 52-f

25 As expected, alpha-tocotrienol (alpha-T3) and alpha-tocopherol (alpha-T) were the predominant

26 genome-wide binding of TR in the control and T3-treated tadpole intestine.

27 mpounds to changes in thyrocyte function and T3/T4 production.

28 SNS activity and plasma leptin, ghrelin, and T3 and their changes with CR were not related to AT.

29 Coordinated glucagon and T3 actions synergize to correct hyperlipidemia, steatohe

30 ynchronized signaling driven by glucagon and T3 reciprocally minimizes the inherent harmful effects o

31 We conclude that hypoxia and T3 enhance the functionality of hESC-VCMs and their engi

32 and two nonenveloped bacteriophages (MS2 and T3) in raw wastewater samples.

33 5.5% (+/-24.5%) for the nonenveloped MS2 and T3, respectively.

34 nduced at T3 target genes during natural and T3-induced metamorphosis and that Dot1L is itself a T3 t

35 Lymphatic spread and T3 stage were predictive of recurrence (univariate, P =

36 rs removed had predominantly T-stages T2 and T3 (32% and 45%, respectively).

37 ntrol (irrigated at 100% ETc) and T1, T2 and T3 (50% ETc at phases I, II and III of fruit growth, res

38 D and generalized anxiety disorder at T2 and T3 and of major depressive episode at T2.

39 e based on specific substrates of the T2 and T3 enzymes, elements in or near the enzyme recognition s

40 Subsequent field testing of T2 and T3 generations of transgenic NtMYC2a overexpression line

41 ine trait was stably inherited to the T2 and T3 generations, indicating the important role that NtMYC

42 o-opted to uncouple the morphology of T2 and T3 wings and to act as a general modifier of hindwings,

43 At T2 and T3, physical QOL was slightly lower than the preoperativ

44 different pathways being affected by T2 and T3.

45 o be examined for patients with T1b, T2, and T3 disease, respectively.

46 iated with maternal concentrations of T4 and T3 during pregnancy.

47 hene oxide (Ag@fGO-T3) as a carrier and anti-T3 antibody-tris(2,2'-bipyridyl) ruthenium(II) (Ru(bpy)3

48 late tetramer (LiOPin)4.HMPA3 abbreviated as T3.

49 as not observed when patients were staged as T3, N0 by 8th edition criteria.

50 Median survival in patients staged as T3, N0 by the 7th edition definitions was different betw

51 ASO-T3 enhances white fat browning, decreases genes for fatt

52 Both NNMT-ASO-T3 (NAT3) and ApoB-ASO-T3 (AAT3) enhance thyroid hormone receptor activity.

53 Both NNMT-ASO-T3 (NAT3) and ApoB-ASO-T3 (AAT3) enhance thyroid hormone

54 We hypothesize that ASO-T3 conjugates may knock down target genes and enrich T3

55 eness increased from 38.6% at T0 to 66.3% at T3.

56 significantly increased over time to 31% at T3.

57 ined 0.28 servings/d higher than baseline at T3 among those who received the intervention.

58 (total) remained significantly different at T3.

59 ted/adjacent teeth displayed GRD increase at T3 (P <0.001).

60 that H3K79 methylation levels are induced at T3 target genes during natural and T3-induced metamorpho

61 In grafted sites, KT at T3 remained stable compared with T1 value (4.1 mm, P <0.

62 rvention group had better quality of life at T3.

63 , respectively) and, for FCRI total only, at T3 ( P = .018), and from T0 to T1 on three FCRI subscale

64 In Drosophila, mimicking phosphorylation at T3 decreased HTTex1 aggregation both in larvae and adult

65 results demonstrate that phosphorylation at T3 stabilizes the alpha-helical conformation of the N-te

66 This difference was sustained at T3 ( P < .001).

67 ible with 100% aqueous polar phase (Atlantis T3), and the ESI enhancement by using UHPLC-MS purity gr

68 igen-specific CD8(+) T cells in mice bearing T3 methylcholanthrene-induced sarcomas that are suscepti

69 rnatively, when this fully reduced binuclear T3 site is oxidized via the T1 Cu, a different thermodyn

70 form of a high-potential MCO, the binuclear T3 Cu(II) site can be reduced via the 700 mV T1 Cu.

71 cific antibody inhibited hemagglutination by T3 virions but not ISVPs, indicating that the antibody-

72 Hyperthyroidism induced by T3 treatment caused up-regulation of TRbeta1 and of its

73 e climax of metamorphosis and are induced by T3.

74 e degraded by DNA exonucleases or ligated by T3 and T4 DNA ligases.

75 iptional regulation of the alphav monomer by T3 and of beta3 monomer by both hormones and documented

76 a cell proliferation in vitro was reduced by T3 in a dose-dependent manner and increased by insulin a

77 d evidence that these genes are regulated by T3 and likely involved in the T3-induced formation of ad

78 96 and 349 genes were uniquely regulated by T3, whereas 22, 40 and 929 were exclusively regulated by

79 to either positive or negative regulation by T3.

80 ibution and PD-L1 expression was revealed by T3 analysis of the whole tumors.

81 circumstances, only one-fifth of circulating T3 is directly released by the thyroid, but in states of

82 Cluster T3 is similar to cluster T2 in terms of chronic airflow

83 Seven weeks after complete T3 spinal cord transection (T3-SCI, n = 15) or sham inju

84 s or C57BL/6 mice were fed a diet containing T3 for 2-7 days.

85 Converting T3 to the unphosphorylatable residue alanine (H3T3A) or

86 ds to the TH receptor, whereas DIO3 degrades T3 and T4.

87 ne with T1b disease and another with diffuse T3 disease, underwent secondary enucleation.

88 Liver-directed T3 action offsets the diabetogenic liability of glucagon

89 Patients with locally advanced disease (T3/T4) presented with more symptoms (p < 0.01) and more

90 hat survive through metamorphosis) or during T3-induced metamorphosis.

91 e been studying intestinal remodeling during T3-dependent Xenopus metamorphosis as a model for organ

92 -CM treated with T3+Dex, but not with either T3 or Dex alone, developed an extensive T-tubule network

93 The system employed T3-conjugated, silver nanoparticle-decorated carboxylic

94 gates may knock down target genes and enrich T3 action in fat and liver.

95 utralizing alphavbeta3 antibodies, excluding T3-induced beta3 messenger RNA, suggesting subspecializa

96 owed by a rapid increase in Mct8 expression (T3/T4 transport), peaking early-September before gradual

97 For selected patients with extensive T3 or large T4a lesions and/or poor pretreatment larynge

98 Plasma free (F) T3 and T4 were measured at baseline, and at 9 days and 1

99 The Ag@fGO-T3 and Ru(bpy)3(2+) complex could be mobilized rapidly t

100 -decorated carboxylic graphene oxide (Ag@fGO-T3) as a carrier and anti-T3 antibody-tris(2,2'-bipyridy

101 ents, and we provide evidence that bona-fide T3 phosphorylation alters Huntingtin exon 1 protein conf

102 % CI, 73%-80%) and 68% (95% CI, 60%-74%) for T3 tumors, and 61% (95% CI, 49%-71%) (5-year only) for T

103 strate that endogenous Dot1L is critical for T3-induced activation of endogenous TR target genes whil

104 howed an increased intrinsic ability to form T3 Our data support the hypothesis that TG processing in

105 with an increased intrinsic ability to form T3 upon in vitro iodination.

106 ally and functionally closely related GalNAc-T3 homolog did not show compensatory functionality for e

107 To test whether D2-generated T3 plays a role in exercise-induced PGC-1a expression, m

108 This indicates H3 T3-Phos/H2A T120-Phos is a universal epigenetic signatur

109 The bank vole HBA-T3 gene is distinguished from each HBA-T1 and HBA-T2 by

110 e-oligonucleotides (ASO) and thyroid hormone T3 conjugates for obesity treatment.

111 t combines glucagon with the thyroid hormone T3 lowers lipid levels, improves glucose tolerance, and

112 However, the thyroid hormone T3 up-regulated mitoIK, ATP, conferring diazoxide protec

113 Thyroid hormone (T3) affects development and metabolism in vertebrates.

114 Glucagon and thyroid hormone (T3) exhibit therapeutic potential for metabolic disease

115 es taking place when plasma thyroid hormone (T3) levels are high.

116 We have been studying thyroid hormone (T3)-dependent amphibian metamorphosis in two highly rela

117 Here we report that a thyroid hormone (T3)-free window, with or without a demyelinating insult,

118 not only transform our understanding of how T3 orchestrates adult brain lineage decisions, but also

119 An Acquity HSS T3 C18 column resulted in a baseline separation, a calib

120 ma was staged as stage I (T1, T2), stage II (T3, M1, M2, C1), and stage III (C2).

121 sed higher levels of inhibitory molecules IL-T3 and PD-L1.

122 cal to identify novel mechanisms that impact T3/T4 production.

123 ;2, BoiPIP2;3) were significantly changed in T3 of RNAi plants.

124 External maturation was delayed in T3 when severe stress occurred during the first part of

125 pling between L-type Ca channels and RyR2 in T3+Dex-treated cells.

126 man ventricular cardiomyocytes, T-tubules in T3+Dex-treated hiPSC-CM were less organized and had more

127 ri-iodothyronine (T3) production or increase T3 degradation preserves cones.

128 AEG-1 inhibited T3-mediated gene regulation in human HCC cells and mouse

129 injecting suspensions of tri-iodothyronine (T3) in coconut oil into the midbrain ventricle or into t

130 IOs) to suppress cellular tri-iodothyronine (T3) production or increase T3 degradation preserves cone

131 io of free thyroxine:free tri-iodothyronine [T3], low concentration of total reverse T3, high concent

132 te that only constant delivery of L-T4 and L-T3 fully normalizes T3-dependent metabolic markers and g

133 postinstallation (T2) until 6 months later (T3).

134 ater (T2), and approximately 9 months later (T3).

135 after intervention (T2) and 6 months later (T3).

136 Local T3 also dramatically increased dendritic arbor growth in

137 Low T3 with normal T4 was observed in the sera of HCC patien

138 While many T3 response genes have been identified in different anim

139 T0) and after 4 (T1), 6 (T2), and 10 months (T3) of follow-up.

140 n=113), 6 months (T2; n=106), and 18 months (T3; n=84) after transplantation.

141 post-therapy (T1), and 3 (T2) and 6 months (T3) later.

142 Moreover, T3 stimulated the ability of DCs to cross-present antige

143 Moreover, T3-induced in vivo inhibition of the NRF2 pathway accomp

144 [AG]n /[TC]n , [A2 G]n /[T2 C]n , [A3 G]n /[T3 C]n , [A4 G]n /[T4 C]n , [A9 G]n /[T9 C]n , [GATC]n /

145 t delivery of L-T4 and L-T3 fully normalizes T3-dependent metabolic markers and gene expression profi

146 ition, novel data indicated that T4, but not T3, controls integrin's outside-in signaling by phosphor

147 found that upon iodination in vitro, de novo T3 formation in TG was decreased in mice lacking TSHRs.

148 ion substrate in a way that enhances de novo T3 formation, contributing to the relative T3 toxicosis

149 sphorylation contributes to enhanced de novo T3 formation.

150 Conversely, de novo T3 that can be formed upon iodination of TG secreted fro

151 At 5 years, 25% of T3 patients presented with metastasis; overall and speci

152 As an application of T3, 3D mapping and analysis revealed a heterogeneous dis

153 of Dio3 provides double-pronged blockage of T3 action during glial lineage commitment.

154 sensitive to physiological concentrations of T3 , insulin and leptin.

155 K9 or K15, reversed the inhibitory effect of T3 phosphorylation.

156 our results establish an adjuvant effect of T3-TRbeta signaling in DCs, suggesting an immediately tr

157 At the end of T3, 83% of the 64 treated sites showed recession reducti

158 with SMAD3 and SMAD4 that is independent of T3-mediated transcriptional activity but requires residu

159 r and inner ring deiodination (O and IRD) of T3 and 3,3'-T2 formation from T4, respectively, with an

160 ll as the rest of the brain, the majority of T3 present is generated locally by T4 deiodination via t

161 Quantitative measurement of T3 could be achieved in the range from 0.1 pg/mL to 0.8

162 n premetamorphic tadpoles in the presence of T3.

163 ces gene activation by TR in the presence of T3.

164 characteristics, with a lower proportion of T3 and T4 lesions (27.9% v 39.8%), fewer patients with p

165 T1 sigma1, the carbohydrate-binding site of T3 sigma1 is located in the tail domain, distal to the a

166 ssenger RNA, suggesting subspecialization of T3 and T4 binding to the integrin receptor pocket.

167 P, conferring diazoxide protective effect on T3-treated hESC-VCMs.

168 hesis in liver, and shows limited effects on T3 target genes in heart and muscle.

169 aggregation by regulating phosphorylation on T3.

170 Most patients presented with T2 (37 %) or T3 (29 %) cancer.

171 Interventions: Patients with TanyN+M0 or T3-4N0M0 LA-SCCHN were randomized 1:1 to receive standar

172 Patients with TanyN+M0 or T3-4N0M0 LA-SCCHN were randomized 1:1 to receive standar

173 tate but with a positive surgical margin) or T3 (with histologic extension beyond the prostatic capsu

174 ts on vegetable or fruit purchasing at T2 or T3.

175 cantly different between the groups at T2 or T3.

176 ilapia treated with equimolar doses of T2 or T3.

177 with intermediate-thickness melanomas (T2 or T3; Breslow thickness of > 1.0 to 4.0 mm).

178 rrays of TTAGGG repeats flanked by the T7 or T3 promoters.

179 rent thermodynamically favored half oxidized T3 form, i.e., the AR site, is generated.

180 RB1 (P = .037) predicted risk of pathologic T3 disease in an ethnicity-dependent manner.

181 rs), and at the end of the follow-up period (T3) over 25 years.

182 Pharmacological T3 treatment increases energy expenditure and causes wei

183 eriod characterized by high levels of plasma T3.

184 rong repression of more than 43% of positive T3 (3,3',5-triiodothyronine) targets in hypothyroid mice

185 effects sustained at 6 mo postintervention (T3).

186 lec or T4lec during ppGalNAc-T2 and ppGalNAc-T3 catalytic reaction had a clear inhibitory effect on G

187 In cells and mice, it blocked ppGalNAc-T3-mediated glycan-masking of FGF23 thereby increasing i

188 a cell-based fluorescence sensor of ppGalNAc-T3 but not on a sensor of ppGalNAc-T2.

189 irectly binds and inhibits purified ppGalNAc-T3 with no detectable activity against either ppGalNAc-T

190 o identify a compound targeting the ppGalNAc-T3 isozyme, we screened libraries to find compounds that

191 invasiveness driven by upregulated ppGalNAc-T3 suggesting the inhibitor might be anti-metastatic.

192 04 to 2014 was queried for patients with pT2/T3 gallbladder adenocarcinoma who underwent resection.

193 rming a high energy, metastable half reduced T3 state, is followed by the rapid delivery of a second

194 phosphorylatable residue in the N17 region (T3, S13 and S16) towards huntingtin exon 1 (HTTex1) olig

195 Rs), patients develop a syndrome of relative T3 toxicosis.

196 o T3 formation, contributing to the relative T3 toxicosis of Graves' disease.

197 Physiologically relevant T3 (1 nM) and T4 (100 nM) concentrations in OVCAR-3 (hig

198 nal modification (phosphorylation at residue T3) of a protein associated with polyglutamine repeat ex

199 ine [T3], low concentration of total reverse T3, high concentration of creatine kinase, mild anaemia)

200 ely associated with overall RT (central, S3, T3, I3, and N3 sectors, P = .004-.024) and the thickness

201 Serum T3 and T4 levels were checked in Alb/AEG-1 mice and huma

202 wed undetectable serum T4 and very low serum T3 levels when fed a diet supplying the minimum I(-) req

203 e monotherapy to universally normalize serum T3 levels.

204 advanced invasive breast cancer (tumor size T3/T4), inflammatory breast cancer, or ductal carcinoma

205 s 2 and (2) monosomy 3 and large tumor size (T3-4 by American Joint Committee on Cancer classificatio

206 s. 57%; P < 0.001), had smaller tumor sizes (T3-4 56% vs. 83%; P = 0.001), and were younger.

207 Eligible patients had clinical-stage T3 rectal adenocarcinoma within 12 cm of the anal verge

208 s the proportions with combined tumour stage T3 and T4 remained constant.

209 For adults initially with stage T3 HCC who received LRT, there were three studies report

210 Strategy T3 advanced internal ripening when moderate water stress

211 ricular cardiac micro-tissue (hvCMT) system, T3 substantially enhanced the developed tension by 3-fol

212 s and 18 days of exposure, whereas total (T) T3 and TT4, thyroid histology and hepatic T4-ORD were de

213 institutions (Gleason scores: 6-9, Stage: T1-T3).

214 New Zealand randomized 475 patients with T1-T3 rectal adenocarcinoma less than 15 cm from the anal v

215 Among patients with T1-T3 rectal tumors, noninferiority of laparoscopic surgery

216 The severe asthma clusters (T2, T3, and T4) had higher sputum eosinophilia than cluster

217 mend extended/radical cholecystectomy for T2/T3 gallbladder cancer; however, many tumors are discover

218 rapy prolongs survival after resection of T2/T3 tumors.

219 ne networks; these presumably enabled the T2/T3 wing's increased size and functionality.

220 -expression differences in ectopic T1 vs. T2/T3 wings suggest that the transition from flaps to wings

221 ging, only 5% of tumors were high stage (T2b/T3), but they accounted for 60% (95% CI, 50% to 69%) of

222 nd measuring changes in thyroid hormone (T4, T3, rT3, and 3,3'-T2) concentrations.

223 in D2 ubiquitination account for the high T4/T3 serum ratio in adult thyroidectomized (Tx) rats chron

224 yronine (T3) and high serum thyroxine/T3 (T4/T3) ratio.

225 eement with this transcriptional cross-talk, T3 is able to antagonize fibrotic processes in vivo.

226 n the carboxyl-terminal portion of mouse TG, T3 is formed de novo independently of deiodination from

227 We find that T3 phosphorylation is greatly reduced in samples from Hu

228 d two-dimensional NMR techniques reveal that T3 is the major species in hydrocarbon solution when mor

229 These suggest that T3 induces Dot1L expression, and Dot1L in turn functions

230 The T3-specific antibody inhibited hemagglutination by T3 vi

231 eanwhile, examination of the leaves from the T3 of RNAi transformants indicated reduction of cell exp

232 e regulated by T3 and likely involved in the T3-induced formation of adult intestinal stem cells duri

233 xpressed the HIV-1 Gag-p24 on the tip of the T3 pilus of Streptococcus pyogenes as a fusion to the Cp

234 (TRbetaPV) to establish the relevance of the T3-TRbeta system in vivo.

235 cally favorable electron to fully reduce the T3 site.

236 Under the salt stress, the T3 of RNAi plants showed significant higher resistance.

237 apable of initiating transcription using the T3 promoter in <10 h.

238 Using the T3-dependent metamorphosis in Xenopus tropicalis as a mo

239 s to examine the influence of high-thoracic (T3 spinal segment) SCI on cerebrovascular structure and

240 y of patients diagnosed as having T1 through T3 M0 pancreatic adenocarcinoma between January 1, 2004,

241 regulating target gene transcription through T3 receptors (TRs).

242 Thus, T3 may be of therapeutic relevance to stimulate beta-cat

243 idues Tyr(5) and Tyr(130), whereas thyroidal T3 production may originate in several different ways.

244 iponectin (-49%), 3,5,3'-tri-iodo-thyronine (T3) (-39%), and testosterone (-11%).

245 L-thyroxine, T4; 3,5,3'-triiodo-L-thyronine, T3) and is overexpressed in ovarian cancer.

246 oid hormones triiodothyronine and thyroxine (T3/T4) can impact metabolism, body composition, and deve

247 iiodothyronine (T3) and high serum thyroxine/T3 (T4/T3) ratio.

248 sed whole-body D2-dependent T4 conversion to T3, D2 activity in the hypothalamus was only minimally a

249 e same cells are differentially sensitive to T3 at different time points.

250 Follow-up effects (T0 to T3) were assessed in assessment completers.

251 T1 (P < .001), T1 to T2 (P = .03), and T2 to T3 (P < .001) but no significant gain thereafter through

252 adjacent untreated teeth improved from T2 to T3 (P <0.001).

253 The accelerated conversion of T4 to T3 within myocytes mediates part of the PGC-1a induction

254 I 5'-deiodinase (DIO1), which converts T4 to T3, contributes to NTIS.

255 ponsible for conversion of thyroxine (T4) to T3.

256 tion of peripheral deiodinase-mediated T4-to-T3 conversion provided a physiologic means to justify l-

257 e we describe transparent tissue tomography (T3) as a tool for rapid, three-dimensional, multiplexed

258 (PFDeA) was associated with lower cord total T3.

259 s after complete T3 spinal cord transection (T3-SCI, n = 15) or sham injury (Sham, n = 10), rats were

260 active marginal zone precursor, transitional T3, and PDL-2(+)CD80(+) memory B cells, with significant

261 2), which generates 3,5,3'-triiodothyronine (T3 ), the active thyroid hormone.

262 elatively low serum 3,5,3'-triiodothyronine (T3) and high serum thyroxine/T3 (T4/T3) ratio.

263 Although 3,5,3'-triiodothyronine (T3) is considered to be the primary bioactive thyroid ho

264 erized by low serum 3,5,3'-triiodothyronine (T3) with normal l-thyroxine (T4) levels, is associated w

265 pression and plasma 3,3',5-triiodothyronine (T3) concentrations in tadpoles treated at higher tempera

266 nstructed to detect 3,3',5-triiodothyronine (T3).

267 yroxine (T4) (100 nM), and triiodothyronine (T3) (100 pM) alter intrafollicular mitochondrial energy

268 ormones thyroxine (T4) and triiodothyronine (T3) averaged 46.9 and 64%, respectively, of the levels e

269 plasma thyroxine (T4), and triiodothyronine (T3), and hepatic outer ring deiodination (T4-ORD).

270 in both thyroxine (T4) and triiodothyronine (T3), were the first pharmacologic treatments available a

271 le for the thyroid hormone triiodothyronine (T3) in controlling the maturation and antitumor function

272 w that the thyroid hormone triiodothyronine (T3), through binding to its nuclear receptors (TRs), is

273 role of one such hormone, triiodothyronine (T3), in triggering the differentiation of bone marrow-de

274 thyroid hormone, 3,5,3'-L-triiodothyronine (T3).

275 The effects of triiodothyronine (T3 ), insulin and leptin on beta cell proliferation rate

276 utcome had lower levels of triiodothyronine (T3) and higher thyroxine (T4).

277 dothyronine (T4), and some triiodothyronine (T3).

278 converts thyroxine (T4) to triiodothyronine (T3), which binds to the TH receptor, whereas DIO3 degrad

279 ated with lower cord total triiodothyronine (T3) and total T4 levels, and maternal perfluorodecanoic

280 ngly associated with total triiodothyronine (T3), free T4, or thyroid-stimulating hormone (TSH).

281 y response to vaccination, triiodothyronine (T3), hepatic biotransformation (7-ethoxyresorufin-O-deet

282 n include thyroid hormone (triiodothyronine [T3] or levothyroxine [T4]), corticosteroids, antidiureti

283 on of levels of active TH (triiodothyronine, T3), through spatiotemporal expression of thyroid hormon

284 g regulation of the muscle-specific Troponin T3 (TNNT3) mutually exclusive exons 16 and 17 in OPMD sa

285 PDX) engraftment in locally advanced tumors (T3-T4 or N+) predict poor prognosis in patients with bla

286 AJCC and UICC high-stage tumors (T3/T4) were rare at 0.3% and 3% of the cohort, respectiv

287 1-T2), and a 6-mo no-intervention follow-up (T3).

288 es were present in 19% of cases and 23% were T3 lesions.

289 ignificantly enriched at binding sites where T3 induced an increase or decrease in TRbeta1 binding, r

290 gnalling and transcriptional pathways, while T3 regulated pathways related to cell signalling, the im

291 nt and tumor characteristics: 263 (80%) with T3 or T4 disease, 215 (66%) N1, and 62 (19%) with celiac

292 ight loss were significantly associated with T3/T4 disease.

293 and ApoB-ASO are chemically conjugated with T3 using a non-cleavable sulfo-SMCC linker.

294 e, consecutive cohort study of patients with T3-4 choroidal melanomas according to the 7th edition of

295 Patients with T3-4, N0/Nx, M0 prostate cancer or T1-2 disease with eit

296 cal differentiation method supplemented with T3 (triiodothyronine) and/or Dex (dexamethasone) during

297 hiPSC-CM treated with T3+Dex, but not with either T3 or Dex alone, developed a

298 VA mouse model of melanoma, vaccination with T3-stimulated DCs inhibited tumor growth and prolonged h

299 metamorphic tadpoles treated with or without T3 and for chromatin immunoprecipitation assays with the

300 end of the follow-up period (18 to 35 years, T3).