ライフサイエンスコーパス: T7 phage

1 cessful display was achieved using the lytic T7 phage.

2 played library generated from C4-2B cells in T7 phage.

3 used to construct a phage display library in T7 phage.

4 s C approach levels observed using wild-type T7 phage.

5 4 protein that cannot support the growth of T7 phage.

6 strategy for quantifying miRNAs by employing T7 phage-a bacteria-specific virus nanoparticle-as a sur

7 T7 phage also express a 56-kDa truncated gp4 lacking the

8 genes for their ability to inhibit growth of T7 phage and >90% of the host genes for their ability to

9 of the portal complexes of Phi29, SPP1, T3, T7 phages and herpes simplex virus.

10 nuclease subunits, EcoKI is inhibited by the T7 phage antirestriction protein ocr, a DNA mimic.

11 titer IgG autoantibodies for biopanning of a T7 phage breast cancer cDNA display library.

12 Together, these findings suggest that T7 phage display can be used to rapidly and selectively

13 ing a high-throughput method, we developed a T7 phage display cDNA library derived from mRNA isolated

14 For this, we generated T7 phage display libraries of N-terminally and C-termina

15 n be identified through immunocreenings of a T7 phage display library with high accuracy, which may h

16 A T7 phage display screen against full-length human ERalph

17 Through the use of a T7 phage display, we discovered a novel interaction betw

18 e in vitro genetic selection method based on T7 phage display.

19 to achieve successful in vitro selection of T7 phage-displayed peptides that recognize markers expre

20 consistent with cryo-electron micrographs of T7 phage DNA.

21 The T7 phage-encoded small protein Gp2 is a non-DNA-binding

22 roughput method involved the assembly of 938 T7 phages encoding potential breast cancer autoantigens.

23 T7 phage gp4 protein also drives DNA branch migration, s

24 Although T7 phage grow better when both forms are present, the ro

25 amino acid substitution(s) in this region on T7 phage growth and on the interaction of the polymerase

26 We show here that inhibition of T7 phage growth by udk overexpression can be overcome by

27 oduct inhibits host RNA polymerase, restores T7 phage growth on hosts overexpressing udk.

28 e, which explains why A257T does not support T7 phage growth.

29 T7 phage is a lytic phage with a broad host range specif

30 d are inefficient in complementing growth of T7 phage lacking gene 3.

31 able the 56-kDa gp4 to support the growth of T7 phage lacking gene 4 (T7Delta4).

32 containing these mutations cannot complement T7 phage lacking gene 4 for T7 growth.

33 the altered proteins to complement growth of T7 phage lacking gene 4.

34 lyzed for their ability to support growth of T7 phage lacking gene 4.

35 yielded a protein that could not complement T7 phage lacking gene 5 (T7Delta5) to grow on E. coli ha

36 eduction in its ability to support growth of T7 phage lacking gene 5.

37 Infection of Escherichia coli by the T7 phage leads to rapid and selective inhibition of the

38 al genome is transcribed by a single-subunit T7 phage-like RNA polymerase (mtRNAP), structurally unre

39 We isolated 95 T7 phage mutants that were resistant to ddT.

40 t RNA polymerase, restores the burst size of T7 phage on udk-overexpressing hosts to normal.

41 ability of short interfering RNAs, including T7 phage polymerase-synthesized RNA (PRNA), which like s

42 istidine fusion protein under control of the T7 phage promoter was expressed in HT-1080 cells and UMR

43 We describe here an RNA, derived from the T7 phage R1.1 RNase III substrate, that is resistant to

44 When gene 1.7 was expressed from a plasmid, T7 phage resistant to ddT still arose; analysis of 36 of

45 placed from the template strand by an active T7 phage RNA polymerase.

46 roof-of-concept for the use of bioengineered T7 phage strains to increase the sensitivity of phage am

47 Overexpression using the vaccinia virus/T7 phage system resulted in secretion of approximately 3

48 Selection of T7 phage that can recognize these altered receptors has

49 e mRNA as bait, we could selectively amplify T7 phage that display either the spliceosomal protein U1

50 However, T7 phages that grow at a similar rate provided with eith

51 ng protein, over the detection of replicated T7 phage viron itself, and a greater then 100-fold incre

52 A synthesis in cells infected with wild-type T7 phage was inhibited by ddT, suggesting that it result

53 age of B. malayi expressed on the surface of T7 phage was sequentially screened with sera samples fro

54 T7 phage with gene 2.5 deleted can grow only on Escheric