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1                                              TBARS (thiobarbituric acid reactive substances) test was
2                                              TBARS assay indicated that LA was more effective in prot
3                                              TBARS did not differ significantly between trials.
4 53g/100g, a TMAN value of 3.25mg/100g, and a TBARS value 0.983MDAmg/100g.
5              The models were used to analyze TBARS formation and oxidation of entrapped Fe(CN)(6)(4)(
6 thane (8.88 versus 1.71 pmol/L; P<.0001) and TBARS (24.0 versus 20.7 micromol/mL; P=.008) than nonsmo
7 nt association was observed between aMED and TBARS.
8 hromatography-mass spectrometry analysis and TBARS values.
9 d further to 7.0 lowered both Hb-binding and TBARS development.
10 her pH values studied (pH 5-7), lower CD and TBARS concentrations were detected in samples with 25-50
11 were collected for measurement of ethane and TBARS, respectively.
12 idant enzymes, decrease in ROS formation and TBARS generation, increase in the mitochondria membrane
13                   Based on hydroperoxide and TBARS analysis, sinapic acid and rutin (200 ppm) showed
14 FP (from 1.22 to 1.29 mmol peroxides/kg) and TBARS (from 0.37 to 0.40 mg MDA equivalents/kg mince).
15        The amounts of both reduced metal and TBARS reactivity are greatest when generated by A beta 1
16 ow, medium and high concentration of OFR and TBARS.
17 ate proved to affect both protein, ORAC, and TBARS in unexposed daphnids.
18                          However, the pH and TBARS values were higher in control samples irrespective
19 FA formation and induced formation of PV and TBARS.
20 isolated oleosome, as measured by the PV and TBARS.
21 ant decrease of lipid peroxides in serum and TBARS levels in plasma.
22 ng power, beta carotene bleaching system and TBARS assay) showed that the variety Chatos exhibited th
23 nced by the increases in the TVBN, TMAN, and TBARS contents; however, these values were very low.
24 ake, total cholesterol, triacylglycerol, and TBARS concentrations did not change significantly.
25        High correlation coefficients between TBARS values and linear aldehydes (C3-C7) measured by bo
26 significant differences were eliminated, but TBARS remained higher after fish-oil supplementation tha
27 ductions in oxidative stress, as assessed by TBARS.
28 rol/very low density lipoprotein cholesterol-TBARS (r = -0.16) and glutathione (r = -0.16), while FEV
29 holesterol (LDL cholesterol/VLDL cholesterol-TBARS) as indicators of lipid peroxidation and 2) compou
30 sed the total phenolic content and decreased TBARS and EC50 of breakfast cereal (p<0.05).
31 amples with 1mg/mL MFP which showed an early TBARS formation.
32                                          GCF TBARS level was elevated in CsA GO+ compared with other
33            Both diets reduced blood glucose, TBARS and hepatic NO.
34 re infused with DHA, which increased hepatic TBARSs and reduced VLDL-ApoB100 secretion.
35  20 minutes showed a significant increase in TBARS (1.8-fold) and gamma-glutamyl cysteine synthetase
36          The cooked beef showed an increased TBARS value compared to the raw meat, and the highest va
37 ot associated with increases in isoprostane, TBARS, 9-HODE, or 13-HODE levels.
38 th juice and extract had significantly lower TBARS values towards the end of the storage period compa
39         At the sampling end point, the lower TBARS values were obtained in samples with TEA and GRA e
40 /ml increased GSH levels up to 138%, lowered TBARS levels up to 25% and decreased ROS levels up to 41
41 st that bicyclic endoperoxides are the major TBARS active compounds present in cholesteryl arachidona
42                             With metoprolol, TBARS values decreased from 4.7+/-0.9 nmol/mL at baselin
43 eaction with triphenylphosphine but negative TBARS and FOX assays after this reaction.
44 a-3 fatty acids, but neither accumulation of TBARS nor formation of oxidized cholesterol forms was fo
45                                The amount of TBARS gradually increased during refrigerated storage wi
46 te analysis showed an inverse association of TBARS with forced expiratory volume in 1 second and forc
47                          The associations of TBARS and glutathione peroxidase with FVC% in men remain
48 gnificant effect on plasma concentrations of TBARS or oxidized protein.
49                                The effect of TBARS on events and procedures was also seen in a multiv
50 his analysis showed an independent effect of TBARS on major vascular events (p = 0.0149), nonfatal va
51 50=226mug/ml) at inhibiting the formation of TBARS and lipid hydroperoxides.
52             SNP also stimulated formation of TBARS in retinal homogenates, occurring to a greater ext
53 ne derivatives as antioxidant (inhibition of TBARS in brain membranes and thiol peroxidase-like activ
54 t OS, with p-anisidine, PV and inhibition of TBARS similar to that of MRPs.
55                              Serum levels of TBARS were strongly predictive of cardiovascular events
56 0 degradation via intracellular induction of TBARSs.
57 tent of free fatty acids (1.4-3.8 mg/g oil), TBARS values (8.8-10.2 nmol MDA/g), and carbonyl groups
58                       Significant effects on TBARS were noted on day (D) 1, 3, 4 and 7; increased wit
59 EV1% showed significantly higher levels of p-TBARS (p = 0.02) and lower levels of bilirubin (p = 0.04
60 ituric acid-reactive substances in plasma (p-TBARS) and in low and very low density lipoprotein chole
61  statistical significantly associated with p-TBARS (r = -0.19).
62 y associated with higher lipid peroxidation (TBARS) [exp(beta) = 1.09-1.78, p < 0.01-0.04)] and SOD a
63 (FRAP, ABTS), as well as lipid peroxidation (TBARS) were determined at the end of the experiment.
64 The fatty acid content, the physicochemical (TBARS and volatile compounds) and sensory parameters wer
65 a F(2)-isoprostanes and MDA, although plasma TBARS was higher than with sunflower-oil and safflower-o
66 e in oxidative stress on the basis of plasma TBARS concentrations after the consumption of EPA and DH
67 thout having any beneficial effect on plasma TBARS.
68 e were added to the diet, neither the plasma TBARS concentration nor the protein oxidation changed.
69  supplementation (P: = 0.04), whereas plasma TBARS were higher after fish-oil supplementation than af
70 chrotrophic count) and chemical (TVB-N, POV, TBARS, FFA) characteristics.
71 ation in synaptosomes caused by OH radicals (TBARS), and significant prevention of protein oxidation
72                            In diabetic rats, TBARS were elevated by 74% in retina and 87% in plasma.
73                       In galactose-fed rats, TBARS were significantly elevated in retina (P < 0.05),
74 s 2.8 microg/mg, P < or = 0.05), and retinal TBARS (6.2 nM/mg protein versus 2.2 nM/mg, P < or = 0.05
75                      The increase in retinal TBARS in group 2 indicates that moderate zinc deficiency
76  2 months prevented the elevation of retinal TBARS and the decrease of Na(+)-K(+)-ATPase and calcium
77 ein:lipid ratio was associated with a slower TBARS production and more rapid protein oxidation, sugge
78 by the thiobarbituric acid reactive species (TBARS) formation
79  of colour, texture and oxidative stability (TBARS) after processing and also after frozen storage.
80 onal thiobarbituric acid-reactive substance (TBARS) and ferrous oxidation in xylenol orange (FOX) ass
81  positive thiobarbituric-reactive substance (TBARS) assay, compatible with the generation of the hydr
82  and thiobarbituric acid-reactive substance (TBARS) contents.
83 y by thiobarbituric acid reactive substance (TBARS) formation in a membrane lipid peroxidation assay,
84  and thiobarbituric acid-reactive substance (TBARS) value of oil, in oleosome suspensions stored at 6
85 lyze thiobarbituric acid reactive substance (TBARS); ferric-reducing antioxidant power (FRAP); total
86 and thiobarbituric acid reacting substances (TBARS), in the plasma of postmenopausal women taking die
87 and thiobarbituric acid reactive substances (TBARS) (2.56mug/g) within 28days, and provided the highe
88  of thiobarbituric acid-reactive substances (TBARS) (P: = 0.0001) but not that of oxidatively modifie
89  of thiobarbituric acid-reactive substances (TBARS) and activation of the transcription factor NF-kB,
90  as thiobarbituric acid-reactive substances (TBARS) and alpha-tocopherol was measured by HPLC.
91 A), thiobarbituric acid reactive substances (TBARS) and fluorescent interaction compounds (OFR).
92 of, thiobarbituric acid-reactive substances (TBARS) and hexanal were formed in washed mince containin
93 V), thiobarbituric acid reactive substances (TBARS) and non-haem iron content throughout hydrolysis p
94  in thiobarbituric acid-reactive substances (TBARS) and p-anisidine value (AV) of lipids were noticea
95 N), thiobarbituric acid reactive substances (TBARS) and peroxide value (PV)], textural (i.e., hardnes
96  of thiobarbituric acid-reactive substances (TBARS) and protein carbonyls in the liver by at least 28
97 rom thiobarbituric acid reactive substances (TBARS) and sensory analysis indicate that oxidation can
98 and thiobarbituric acid-reactive substances (TBARS) as an indirect marker of free radical activity.
99 The thiobarbituric acid reactive substances (TBARS) assay is widely used to measure lipid oxidation a
100 the thiobarbituric acid reactive substances (TBARS) assay.
101 the thiobarbituric acid-reactive substances (TBARS) assay.
102  by thiobarbituric acid reactive substances (TBARS) assay.
103 and thiobarbituric acid reactive substances (TBARS) in 252 women from western New York State (2005-20
104  as thiobarbituric acid reactive substances (TBARS) in 634 patients with documented CAD using reverse
105 er, thiobarbituric acid reactive substances (TBARS) increased markedly (p < 0.05).
106  by thiobarbituric acid reactive substances (TBARS) on day (D) 1-8 of storage at 4 degrees C; and FA
107 the thiobarbituric acid reactive substances (TBARS) test.
108 ecreased thiobarbituric reactive substances (TBARS) values and hexanal.
109 and thiobarbituric acid reactive substances (TBARS) were analysed periodically during the hydrolysis
110 and thiobarbituric acid reactive substances (TBARS) were measured </= 8 times per cycle at visits sch
111  as thiobarbituric acid reactive substances (TBARS) were measured.
112     Thiobarbituric acid reactive substances (TBARS) were reduced by the addition of curing salts but
113 and thiobarbituric acid-reactive substances (TBARS), a marker of oxidative stress, were measured in b
114  of thiobarbituric acid-reactive substances (TBARS), and catalase and superoxide dismutase (SOD) in l
115 tic thiobarbituric acid reactive substances (TBARS), and hepatic TNF-alpha and IL-1beta contents in H
116 V), thiobarbituric acid reactive substances (TBARS), fluorescence compounds (OFR) and free fatty acid
117 ing thiobarbituric acid-reactive substances (TBARS), glutathione (GSH), glutathione peroxidase (GPX),
118 sma thiobarbituric acid-reactive substances (TBARS), glutathione, glutathione peroxidase, and 6-hydro
119 de, thiobarbituric acid reactive substances (TBARS), malondialdehyde and phytosterol oxidation produc
120  made of thiobarbituric reactive substances (TBARS), nitric oxide (NO), total antioxidant status (TAS
121  in thiobarbituric acid reactive substances (TBARS), p-anisidine value (AnV) and free fatty acid (FFA
122  retinal thiobarbituric reactive substances (TBARS).
123 and thiobarbituric acid reactive substances (TBARS).
124 and thiobarbituric acid reactive substances (TBARS, 0.30-0.38 mg malondialdehyde (MDA) equivalents/kg
125 and thiobarbituric acid-reactive substances (TBARS, an in vitro assay), were examined in 123 adults (
126 sed thiobarbituric-acid-reactive-substances (TBARS) assay to determine lipid oxidation in seafood may
127 sed with thiobarbituric-reactive-substances (TBARS), except in samples processed at 25 kGy.
128 and thiobarbituric acid reactive substances (TBARSs) production.
129 ed thio-barbituric acid reactive substances (TBARSs, an index of oxidized proteins) and an antioxidan
130  in thiobarbituric acid-reactive substances (TBARSs; a measure of lipid peroxidation products) and re
131  as thiobarbituric acid reactive substances [TBARS]) in retina and plasma.
132  sensitivity for antioxidant evaluation than TBARS.
133                                          The TBARS value of the turkey meat decreased for all the coo
134 onic acid) radical cation (ABTS(+)), and the TBARS system based on a linoleic acid emulsion.
135                 As an antioxidant assay, the TBARS test may lack acceptable reproducibility, and long
136 esents an overview of the current use of the TBARS test in food and physiological systems, before loo
137 cludes with proposals for development of the TBARS test so that it can be used as a rapid and robust
138 ntial limitations, there are features of the TBARS test that make it useful as a complement to popula
139 all the cooking methods in comparison to the TBARS value of the fresh meat.
140 d beta-carotene were primary contributors to TBARS activity.
141                                        Total TBARS concentrations increased 1 h after cream intake an
142 effect on fish quality by reducing pH value, TBARS and TVB-N contents, and retarding the softening of
143 ntaining filled hydrogel particles, in which TBARS levels were up to 62% lower than other systems con
144 the WQS index was positively associated with TBARS levels, with the three PCBs acting as the main con
145                   At baseline, patients with TBARS levels in the highest quartile had a relative risk

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