ライフサイエンスコーパス: TBEV

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1 TBEV/DEN4Delta30 and YF 17D demonstrated remarkably simi

2 Substitutions were made within a TBEV reverse genetic system and recovered mutants were c

3 izing antibodies against LGT TP21 as well as TBEV and were completely protected against subsequent LG

4 vations for development of a live attenuated TBEV vaccine are discussed.

5 for further development of a live attenuated TBEV vaccine.

6 vaccine candidate against disease caused by TBEV.

7 evaluation in mice and monkeys, the chimeric TBEV/DEN4Delta30 virus, carrying the prM and E protein g

8 mpared the neuropathogeneses of the chimeric TBEV/DEN4Delta30 virus; Langat virus (LGTV), a former li

9 sights into the NK cell response to clinical TBEV infection.

10 e encephalitis, and tick-borne encephalitis (TBEV) viruses are important neurotropic human pathogens,

11 at this modulatory role may be important for TBEV survival in nature, where the virus circulates by n

12 elated and more pathogenic viruses including TBEV, Louping ill virus, Omsk hemorrhagic fever virus (O

13 ted extensively in clinical trials as a live TBEV vaccine and was found to induce a protective, durab

14 30 virus; Langat virus (LGTV), a former live TBEV vaccine; and yellow fever 17D virus vaccine (YF 17D

15 Nevertheless, TBEV/DEN4Delta30 virus exhibited higher neurovirulence i

16 ty of an effective vaccine, the incidence of TBEV is increasing worldwide.

17 chemokine receptor Ccr5 in a mouse model of TBEV infection using the naturally attenuated tick-borne

18 rovirulence and abolish neuroinvasiveness of TBEV, namely substitution of structural protein genes of

19 ed with the highly virulent Sofjin strain of TBEV.

20 uctural protein genes of a highly pathogenic TBEV.

21 ntified two separate clines, suggesting that TBEV spread both east and west from a central point.

22 ot observed in any of these systems, and the TBEV and WNV systems did not yield any viable recombinan

23 r-128a, mir-218, or let-7c microRNA into the TBEV/DEN4 genome was sufficient to prevent the developme

24 (LGTV), a naturally attenuated member of the TBEV serogroup.

25 This TBEV-induced NK cell activation was restricted predomina

26 indings demonstrate that NK cells respond to TBEV infection with characteristics that are distinct fr

27 al analysis of the human NK cell response to TBEV infection in a cohort of infected individuals from

28 i.p. 50% lethal doses of the highly virulent TBEV.

29 imeric tick-borne encephalitis/dengue virus (TBEV/DEN4) that contained the structural protein genes o

30 embers of the tick-borne encephalitis virus (TBEV) complex, was firstly isolated from Ixodes granulat

31 member of the tick-borne encephalitis virus (TBEV) complex, was tested extensively in clinical trials

32 Tick-borne encephalitis virus (TBEV) is a flavivirus that is transferred to humans by i

33 Tick-borne encephalitis virus (TBEV) is a vector-transmitted flavivirus that causes pot

34 ern strain of tick-borne encephalitis virus (TBEV) on the backbone of a nonneuroinvasive dengue type

35 Tick-borne encephalitis virus (TBEV), a member of the Flaviviridae family, is a leading

36 sociated with tick-borne encephalitis virus (TBEV), the most virulent of the tick-borne flaviviruses.

37 derived from tick-borne encephalitis virus (TBEV), West Nile virus (WNV), and Japanese encephalitis

38 with those of tick-borne encephalitis virus (TBEV), yellow fever virus (YFV), and Japanese encephalit

39 most virulent tick-borne encephalitis virus (TBEV).

40 apsid gene of tick-borne encephalitis virus (TBEV, genus Flavivirus).

41 studies on tick-borne encephalitis viruses (TBEV), based on partial envelope gene sequences, predict

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