ライフサイエンスコーパス: TBHQ

1 TBHQ dose- and time-dependently decreased the growth of

2 tly stronger antioxidant activity than 0.02% TBHQ.

3 -tocopherol reduction were TBHQ-SOv (4.90%), TBHQ-SOt (53.62%), HOSOv (96.87%), HOSOt (99.76%).

4 TCNE photoelectrochemical sensor presented a TBHQ photocurrent about 13-fold higher and a charge tran

5 .0), GA (43.2; 249.9), MG (38.0; 237.5), and TBHQ (17.7; 214.4), respectively.

6 x detection and measurement of BHA, BHT, and TBHQ levels in complex food samples using a linear sweep

7 effect of 3-tert-butyl-4-hydroxyanisole and TBHQ in these tissues.

8 s activated by electrophile inducers such as TBHQ and is ER independent, and a second that is antiest

9 7 synthetic antioxidants including BHT, BHA, TBHQ and PG with regard to their ability to protect omeg

10 oxidants, and inhibition of JNK/SAP blocked TBHQ-increased PAI-1-luciferase expression.

11 xytoluene (BHT), and tert-butylhydroquinone (TBHQ) are synthetic antioxidants used in the food indust

12 mediated grafting of tert-butylhydroquinone (TBHQ) onto chitosan and further crosslinking to agave in

13 Tert-butylhydroquinone (TBHQ) was tested for potential cytotoxicity and genotoxi

14 he same extent as by tert-butylhydroquinone (TBHQ), a known activator of EpREs; however, only the sti

15 s than 0.02% (1.1mM) tert-butylhydroquinone (TBHQ).

16 :50, and 25:75), and tert-butylhydroquinone (TBHQ).

17 ic antioxidant (tertiary-butylhydroquinone) (TBHQ-SO).

18 (38.0; 2.0346) were considerably improved by TBHQ (201.1; 0.0267), followed by GA (163.8; 0.0837), MG

19 Occupation of this site by TBHQ stabilizes the neutral pH structure through intersu

20 d a limit of detection of 0.21mumolL(-1) for TBHQ, under optimized experimental conditions.

21 t dose tested (400 micromol/kg,i.v.) 5-(Gsyl)TBHQ and 6-(GSyl)TBHQ caused 2-fold increases in the uri

22 he half-wave oxidation potentials of 5-(Gsyl)TBHQ and 6-(GSyl)TBHQ were similar to that of TBHQ, wher

23 tyl-5-(glutathion-S-yl)hydroquinone [5-(GSyl)TBHQ], 2-tert-butyl-6-(glutathion-S-yl)hydroquinone [6-(

24 0 micromol/kg,i.v.) 5-(Gsyl)TBHQ and 6-(GSyl)TBHQ caused 2-fold increases in the urinary excretion of

25 ation potentials of 5-(Gsyl)TBHQ and 6-(GSyl)TBHQ were similar to that of TBHQ, whereas the half-wave

26 tyl-6-(glutathion-S-yl)hydroquinone [6-(GSyl)TBHQ], and 2-tert-butyl-3,6-bis-(glutathion-S-yl)hydroqu

27 3,6-bis-(GSyl)TBHQ (200 micromol/kg) was the most potent of the GSH co

28 In addition to nephrotoxicity, 3,6-bis-(GSyl)TBHQ increased the bladder wet weight 2-fold and caused

29 lf-wave oxidation potential of 3,6-bis-(Gsyl)TBHQ was approximately 100 mV higher than that of TBHQ.

30 glutathion-S-yl)hydroquinone [3,6-bis-(GSyl)-TBHQ] have been identified recently as metabolites of TB

31 oxidants decreased in the order ETX>TR>PG>AA>TBHQ>BHA.

32 st potent of these, tert-butyl hydroquinone (TBHQ), inhibits both the conformational change in HA fro

33 virus infectivity, tert-butyl hydroquinone (TBHQ), shows that the inhibitor binds in a hydrophobic p

34 xytoluene (BHT) and tert-butyl hydroquinone (TBHQ), were determined in different edible vegetable oil

35 -hydroxyanisole and tert-butyl-hydroquinone (TBHQ) are antioxidants known to promote renal and bladde

36 or determination of tert-butyl-hydroquinone (TBHQ) in edible vegetable oils, based on CdSe/ZnS core-s

37 entage decreased in all treatments except in TBHQ-SOv.

38 th a mixture of the Ca(2+)-ATPase inhibitor, TBHQ, and the calcium ionophore, A23187.

39 Like TBHQ, most of these molecules inhibit the conformational

40 nly the stimulation by antiestrogen, and not TBHQ, required ER and was repressed by estradiol, althou

41 gation does not impair the redox activity of TBHQ.

42 ggest that the toxicity of GSH conjugates of TBHQ to kidney and bladder may contribute to the promoti

43 e been identified recently as metabolites of TBHQ in the male rat, we investigated the effects of the

44 ests routes for the chemical modification of TBHQ that could lead to the development of more potent i

45 BHQ and 6-(GSyl)TBHQ were similar to that of TBHQ, whereas the half-wave oxidation potential of 3,6-b

46 was approximately 100 mV higher than that of TBHQ.

47 h blank (non-encapsulated) OLE and synthetic TBHQ antioxidant.

48 m) showed the same antioxidant activity than TBHQ, representing a potential alternative as natural an

49 The TBHQ-GSH conjugates also catalyzed the formation of 8- h

50 electrochemical sensor showed selectivity to TBHQ, with a high photocurrent for this antioxidant comp

51 A rings within the nucleus of cell's treated TBHQ.

52 Values of FFA, p-AV, TPC using TBHQ-SO with traditional frying were (0.201, 207.0, 25.0

53 rcentages of alpha-tocopherol reduction were TBHQ-SOv (4.90%), TBHQ-SOt (53.62%), HOSOv (96.87%), HOS

54 The EO was 2.44 and 7.95 with TBHQ-SO traditional and vacuum frying respectively, and

55 Sunflower oil with TBHQ (SOTBHQ) (200mg/kg) and without additives (SOcontro