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1 se mice are determined by factors other than TCR gene rearrangement.
2 tment is determined before or independent of TCR gene rearrangement.
3 YDbSmcy share an invariant Vbeta8.2-Jbeta2.3 TCR gene rearrangement.
4 by measuring the excisional DNA products of TCR-gene rearrangement.
5 well as analysis of several sequential human TCR gene rearrangements.
6 circles created by alphabeta and gammadelta TCR gene rearrangements.
7 vival of TN cells and its role in regulating TCR gene rearrangements.
8 urface antigens, and clonal T-cell receptor (TCR) gene rearrangements.
9 81/CD42, and all had clonal T-cell receptor (TCR) gene rearrangements.
11 erogeneous in developmental potential before TCR gene rearrangement and suggest that in some precurso
12 previously shown to impair T cell receptor (TCR) gene rearrangement and to cause a partial block in
13 based on immunoglobulin/T-cell receptor (Ig/TCR) gene rearrangements and with quantification of IKZF
15 by polymerase chain reaction analysis of Ig/TCR gene rearrangements, and patients were assigned to a
17 of immunoglobulin (Ig) and T-cell receptor (TCR) gene rearrangement are reflected in the accessibili
18 that differ from the mouse are the status of TCR gene rearrangements at the nonexpressed loci, the ti
19 cal Ig heavy chain (IgH) or T-cell receptor (TCR) gene rearrangement at initial diagnosis and subsequ
20 Progenitor cells undergo T cell receptor (TCR) gene rearrangements during their intrathymic differ
21 Quantitative RT-PCR assessment of different TCR gene rearrangement events revealed lower levels in M
22 itored indirectly by measuring the levels of TCR gene rearrangement excision circles in peripheral T
23 p study of the junctional diversity of these TCR gene rearrangements focuses on characterization of t
24 studies that have assessed T-cell receptor (TCR) gene rearrangements (GRs) present at different anat
25 e negative, DN) thymocytes is independent of TCR gene rearrangement; however, induction of CD5 surfac
26 cular, we now have a better understanding of TCR gene rearrangement in endomysial T cells, regulation
27 flow cytometry and by the presence of clonal TCR gene rearrangements in four patients' posttreatment
29 flow cytometry, and clonal T-cell receptor (TCR) gene rearrangements in two of two pretreatment bloo
30 tailed, comprehensive computer simulation of TCR gene rearrangement, incorporating the interaction of
31 , these findings demonstrate that productive TCR gene rearrangement is associated with events that ca
33 el of lineage commitment in which sequential TCR gene rearrangements may influence alphabeta/gammadel
34 sity, as detected by Ig and T-cell receptor (TCR) gene rearrangements, may represent a very useful pr
37 Immunoglobulin (Ig) and T-cell receptor (TCR) gene rearrangements provide clonal markers useful f
38 pment of T cell precursors in the absence of TCR gene rearrangement, recombinase-activating gene-defi
39 tromal signals that induce functions such as TCR gene rearrangement reside mainly in the outer half o
40 ised DNA products of baboon T-cell receptor (TCR) gene rearrangement (signal-joining TCR excision cir
41 excisional DNA products of T-cell-receptor (TCR) gene rearrangement to measure thymic output directl
42 in all cases and an identical clonal IgH or TCR gene rearrangement was found on PCR analysis of DNA
43 ment expressing a repertoire biased to early TCR gene rearrangements, we developed a mouse model in w
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