ライフサイエンスコーパス: TEG

1 TEG functionalization of the hydrazone switch allows for

2 usion strategy or standard of care (SOC; 1:1 TEG:SOC).

3 hours from arrival (21.8% CCA group vs 7.1% TEG group) (P = 0.032).

4 ), and more platelets units [CCA: 0.0 (0-1), TEG: 0.0 (0-0)] (P = 0.041) in the first 2 hours of resu

5 units as the TEG patients [CCA: 5.0 (2-11), TEG: 4.5 (2-8)] (P = 0.317), but more plasma units [CCA:

6 317), but more plasma units [CCA: 2.0 (0-4), TEG: 0.0 (0-3)] (P = 0.022), and more platelets units [C

7 2.5 mg TAM implant + 1000 ppm genistein (2.5 TEG); E2 implant + 5 mg TAM implant (5 TE), and E2 impla

8 ant +5 mg TAM implant +1000 ppm genistein (5 TEG).

9 full factorial optimization technique and a TEG configuration for simultaneous optimization of power

10 tests except platelet count (PLT) and in all TEG parameters, on the first day of infection compared w

11 nfection resolved, while the r, k, and alpha TEG parameters significantly worsened in the 8 patients

12 ere included in an intent-to-treat analysis (TEG = 56, CCA = 55).

13 without ischemic events by aggregometry and TEG (p < 0.001 for both measurements).

14 t reactivity as measured by aggregometry and TEG were the only variables significantly related to isc

15 t reactivity at baseline by aggregometry and TEG, respectively.

16 evaluated with routine hemostasis tests and TEG on admission and/or the first day with signs of infe

17 show the least sequence conservation between TEG sulfotransferases.

18 yielded correction to normal ranges in both TEG and CBT end points at 5 to 15 mg/kg and 15 to 20 mg/

19 encing cocktail in an immobilization buffer (TEG/ethanol), the DNA fragments demonstrated a high affi

20 /- 2% after PCI (p = NS), and aggregation by TEG was 5 +/- 9% before PCI and 6 +/- 14% after PCI (p =

21 fter aspirin (p < 0.001), and aggregation by TEG was 86 +/- 14% before and 5 +/- 7% at 24 h after asp

22 0% aggregation by LTA or >50% aggregation by TEG.

23 be managed either by an MTP goal directed by TEG or by CCA (ie, international normalized ratio, fibri

24 es in viscoelasticity were then monitored by TEG.

25 SF from the excimer state of Me, C6, TEG, and EH takes place in tauSF = 22, 336, 195, and 120

26 hexaethylene glycol hydrophilic units (DEG, TEG, and HEG, respectively).

27 Utilization of a goal-directed, TEG-guided MTP to resuscitate severely injured patients

28 DOX-TEG-TAM retains 60% of the affinity of 4-hydroxytamoxife

29 DOX-TEG-TAM uptake was inhibited in a dose-dependent manner

30 DOX-TEG-TAM was also taken up by the AEBS-negative, ER-posit

31 DOX-TEG-TAM was taken up by four AEBS-positive cell lines to

32 xorubicin-formaldehyde conjugate, called DOX-TEG-TAM, was found to possess superior cell growth inhib

33 The lead compound, DOX-TEG-TAM, bearing a triethylene glycol tether, binds the

34 ine as a function of time showed initial DOX-TEG-TAM localization in cytosol, in contrast to initial

35 illator-embedded triboelectric generator (FO-TEG) is applicable for both impulse excitation and sinus

36 nstrate the energy harvesting behavior of FO-TEG, lighting of an array of LEDs is demonstrated using

37 from the inherently packaged structure of FO-TEG.

38 obic "B" blocks and hydrophilic PFS-b-(PEO-g-TEG) "A" segments were prepared and their hierarchical s

39 PFS-b-(PEO-g-TEG) cylindrical micelles of controlled length with low

40 ne glycol (TEG), abbreviated as PFS-b-(PEO-g-TEG).

41 that consists of a triboelectric generator (TEG) and a thin-film electroluminescent (TFEL) lamp.

42 ed that segmented thermoelectric generators (TEGs) can operate over large thermal gradient and thus p

43 study, segmented thermoelectric generators (TEGs) have been simulated with various state-of-the-art

44 h biotin (bioactive) and triethylene glycol (TEG) (antifouling) functionality.

45 core and two hydrophilic triethylene glycol (TEG) chains on the other.

46 S-b-PAGE) decorated with triethylene glycol (TEG), abbreviated as PFS-b-(PEO-g-TEG).

47 thyl (Me), n-hexyl (C6), triethylene glycol (TEG), and 2-ethylhexyl (EH) substituents at the 2,5-posi

48 duced using 400 MW PEG (polyethylene glycol),TEG (triethylene glycol), alpha-MG (methyl-alpha-glucosi

49 n independent predictor of a hypercoagulable TEG profile and platelet count, endotoxin, Protein C and

50 hrombin III deficiencies and hypercoagulable TEG parameters were prevalent among patients with VTE.

51 i-Factor Xa deficiencies and hypercoagulable TEG parameters, including elevated coagulation index (>3

52 e independent predictors of a hypocoagulable TEG profile.

53 INR > 3 was associated with: hypocoagulable TEG profile with heparin-like effect; falls in thrombin

54 In TEG assays, tPA significantly decreased clot strength (m

55 Clotting rapidity (angle alpha in TEG) was decreased from a baseline value of 73.3 +/- 1.1

56 Clot strength (maximum amplitude in TEG) was decreased from a baseline value of 72.2 +/- 1.4

57 Objective: To evaluate the effectiveness of TEG-adjusted prophylactic enoxaparin dosing among trauma

58 gle x-ray scattering done in the presence of TEG suggests that for further deformation-beyond a 9% de

59 It is demonstrated that multiple types of TEGs are applicable to the self-powered system, indicati

60 e statistical tools, we provide near-optimum TEG configuration with only 25 experiments as compared t

61 oelastic coagulation tests (such as ROTEM or TEG) have emerged as practical, rapid and sensitive diag

62 rospective way to construct high performance TEGs with greatly enhanced efficiency and output power d

63 Preoperative TEG may reliably identify group of recipients at greater

64 The maximum amplitude (MA) on preoperative TEG was significantly higher in patients diagnosed with

65 ant coagulopathy before invasive procedures, TEG-guided transfusion strategy leads to a significantly

66 r-TEG estimated percent lysis was categorized as PF when >

67 r-TEG was performed by adding tissue factor to uncitrated

68 In addition, r-TEG identified patients with an increased risk of early

69 All had admission r-TEG and CCTs.

70 We have previously shown that admission r-TEG results are available faster than CCTs and predict p

71 Coagulopathy was defined as r-TEG clot strength = G < 5.3 dynes/cm.

72 We correlated r-TEG values [activated clotting time (ACT), r, k, alpha,

73 The charge for r-TEG ($317) was similar to the 5 CCTs ($286).

74 Overall, r-TEG correlated with CCTs.

75 The r-TEG data was clinically superior to results from 5 CCTs.

76 Recently, rapid thrombelastography (r-TEG) has become recognized as a comprehensive assessment

77 Rapid thrombelastography (r-TEG) offers point of care comprehensive assessment of th

78 Admission CCTs can be replaced with r-TEG.

79 We hypothesized that r-TEGs more reliably predict blood component transfusion t

80 As this resolved, TEG demonstrated that both developed a marked hypercoagu

81 However, segmented TEGs are still in early stages of development due to the

82 tal factors on the optimization of segmented TEGs is also studied.

83 for optimizing the performance of segmented TEGs.

84 ize output power and efficiency of segmented TEGs.

85 procedure bleeding was rare, indicating that TEG thresholds should be reevaluated.

86 The TEG gene cluster, a glycopeptide biosynthetic gene clust

87 The TEG group would receive FFP if the reaction time (r) was

88 The TEG provides high-voltage alternating electric output, w

89 imilar number of red blood cell units as the TEG patients [CCA: 5.0 (2-11), TEG: 4.5 (2-8)] (P = 0.31

90 Induced charges pumped onto the lamp by the TEG generate an electric field that is sufficient to exc

91 d blood product transfusions versus 5 in the TEG group (100% vs. 16.7%; P < 0.0001).

92 he CCA group (36.4%) compared with 11 in the TEG group (19.6%) (P = 0.049).

93 Survival in the TEG group was significantly higher than the CCA group (l

94 In the TEG group, none received FFP alone (P < 0.0001 vs. SOC),

95 f compatibility factor s from one end of the TEG leg to the other, even if s values of two ends diffe

96 gh considered potentially detrimental to the TEG performance, these effects, if well-regulated, do no

97 en heparinase and standard thrombelastogram (TEG) is associated with a decreased risk of VTE.

98 ivated clotting time, and thrombelastograph (TEG).

99 Thrombelastography (TEG) is a method that is used to conduct global assays t

100 et aggregation (LTA) and thrombelastography (TEG) platelet mapping were performed on the blood of hea

101 ittance aggregometry and thrombelastography (TEG) will be at increased risk for poststenting ischemic

102 ine hemostasis tests and thrombelastography (TEG), a global test of hemostatic function.

103 y the viscoelastic assay thrombelastography (TEG) improves survival compared with an MTP guided by co

104 od samples were drawn for thrombelastograpy (TEG) and active metabolite assay.

105 through pretransplant thromboelastographic (TEG) data among other known risk factors, to identify ri

106 Thromboelastography (TEG) provides a more comprehensive global coagulation as

107 At 12 h thromboelastography (TEG) demonstrated increased clot formation rate, associa

108 ostasis as exhibited in thromboelastography (TEG) and cuticle bleeding time (CBT) tests.

109 scribe the first use of thromboelastography (TEG) in the management of 2 cases of Ebola virus disease

110 Sixty patients were randomly allocated to TEG-guided transfusion strategy or standard of care (SOC

111 ciency up to 11%) as compared to traditional TEGs, comprising of single thermoelectric (TE) material.

112 Two TEG parameters between the case and control groups were

113 red standard dosing (30 mg twice daily) with TEG-adjusted enoxaparin dosing (35 mg twice daily) for 1