ライフサイエンスコーパス: TENS

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1 TENS inhibits IL-1beta-induced synthesis of IL-1beta, IL

2 TENS reduces hyperalgesia through activation of receptor

3 TENS-L is a potent inhibitor of interleukin (IL)-1 beta-

4 ontrol groups, who were not influenced about TENS.

5 extracellular fluid before, during and after TENS and analyzed GABA in dialysates with high performan

6 tic machines, and more recently with TMS and TENS.

7 ory and proinflammatory signals generated by TENS of various magnitudes.

8 rs prevents the antihyperalgesia produced by TENS in rats with joint inflammation.

9 In contrast, TENS-H is a proinflammatory signal that induces I-kappaB

10 We show that high frequency TENS increases extracellular GABA concentrations in the

11 Thus, high frequency TENS increases release of GABA in the deep dorsal horn o

12 glycine in response to low or high frequency TENS.

13 hypothesis that either high or low frequency TENS applied to the inflamed knee joint increases GABA i

14 hyperalgesia by both high and low frequency TENS is prevented by spinal blockade of GABA(A) receptor

15 spinal cord, and both high and low frequency TENS reduce primary hyperalgesia by activation of GABA(A

16 BA do not occur in response to low frequency TENS, and there are no increases in glycine in response

17 y, whereas tensile strain of high magnitude (TENS-H) is proinflammatory and catabolic.

18 equibiaxial tensile strain of low magnitude (TENS) provokes potent anti-inflammatory signals in PDL c

19 enerated by tensile strain of low magnitude (TENS-L) are antiinflammatory, whereas tensile strain of

20 l group was conditioned about the effects of TENS with a surreptitious amplification of the visual fe

21 our therapeutic modalities (sham-PENS, PENS, TENS, and exercise therapies) were each administered for

22 scores after each treatment than sham-PENS, TENS, and exercise therapies (after-treatment mean +/- S

23 and 2.6+/-1.2 pills per day with sham-PENS, TENS, and exercise, respectively.

24 posttreatment function more than sham-PENS, TENS, and exercise.

25 dditionally, as an anti-inflammatory signal, TENS induces IL-10 synthesis in the presence and absence

26 The most common reported etiologies for SJS/TENS were antibiotics (n = 25), ibuprofen (n = 15), and

27 5 males and 51 females with a history of SJS/TENS; median age was 36 years.

28 transcutaneous electrical nerve stimulation (TENS) applied on the first dorsal interosseus would indu

29 Transcutaneous electrical nerve stimulation (TENS) is a commonly utilized non-pharmacological, non-in

30 ntrolled study, PENS was more effective than TENS or exercise therapy in providing short-term pain re

31 eached higher levels of force, believed that TENS had been effective and expected to perform better c

32 servations are the first to demonstrate that TENS antagonizes IL-1beta actions on PDL cells by (i) in

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