ライフサイエンスコーパス: TEWL

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1 TEWL and FES demonstrated a significant difference betwe

2 TEWL could be used for stratifying infants in the first

3 TEWL measurements obtained at the time of the second imm

4 TEWL was measured on unaffected forearm skin.

5 The inverse of TEWL (1/TEWL) and removed SC thickness yielded a highly linear c

6 children with FA at 2 years of age had day 2 TEWL in the upper quartile.

7 Lowest quartile day 2 TEWL was protective against AD at 12 months.

8 itis (AD), infants with upper-quartile day 2 TEWL were 3.5 times more likely to have FA at 2 years th

9 Although TEWL was normal in the basal state, following disruption

10 Attenuated TEWL correlated with decreased expression of the pro-inf

11 e barrier formation is well characterized by TEWL assay.

12 The results show that capacitance, TEWL, and absorption-desorption rates had larger values

13 ct was independent of FLG mutation carriage, TEWL, and AD phenotype (flexural vs. non-flexural).

14 In contrast, TEWL changed little as the outer SC layers were stripped

15 sence of MyD88 alleviated disease (decreased TEWL, skin thickness, proinflammatory cytokines), wherea

16 an experimental model of atopic dermatitis, TEWL, allergic sensitization, and epidermal thickness we

17 ce for association of this SNP with elevated TEWL also.

18 ared to NC skin (both P < 0.001), and higher TEWL and pH compared to CSU lesions.

19 expression in the skin, along with increased TEWL, epidermal thickness, and skin inflammation, all of

20 using UPLC-MS/MS, transepidermal water loss (TEWL) and epidermal pH.

21 skin resistance, transepidermal water loss (TEWL) and Fourier transform infrared (FTIR) spectroscopi

22 quantified using transepidermal water loss (TEWL) and were correlated with the immune responses.

23 oppler imaging, a transepidermal water loss (TEWL) device and a skin thermometer in a 28 h session.

24 birth cohort had transepidermal water loss (TEWL) measured in the early newborn period and at 2 and

25 Transepidermal water loss (TEWL) measures were collected at 12 months from a subset

26 ther increases in transepidermal water loss (TEWL) predate the development of clinical AD.

27 ts of the rate of transepidermal water loss (TEWL) were recorded sequentially in vivo in human subjec

28 RCM, transepidermal water loss (TEWL), and fluorescence excitation spectroscopy (FES) we

29 ux across the SC, transepidermal water loss (TEWL), in six women, in vivo.

30 ring capacitance, transepidermal water loss (TEWL), rates of absorption-desorption as well as Raman s

31 nb3a-null mice on transepidermal water loss (TEWL), sensitization, and inflammation.

32 rmatitis (AD) and transepidermal water loss (TEWL).

33 ssed by measuring transepidermal water loss (TEWL).

34 4, P=0.0004) with transepidermal water loss (TEWL).

35 ity barrier using transepidermal water loss (TEWL).

36 An upper quartile 2 month TEWL was also strongly predictive of AD at 12 months (ar

37 The inverse of TEWL (1/TEWL) and removed SC thickness yielded a highly

38 Measurements of TEWL were made at birth (day 2) and at 2 and 6 months.

39 The variation of TEWL as a function of SC removal behaved in a manner ent

40 istic regression model, day 2 upper quartile TEWL measurement was significantly predictive of AD at 1

41 Day 2 upper-quartile TEWL (>9 g water/m(2)/h) was a significant predictor of

42 RCM, TEWL, and FES are valuable non-invasive tools to quantit

43 impedance, apparently proceeded faster than TEWL decreased to the prestripping control.

44 cance of these observations was tested using TEWL to evaluate the permeability barrier function of th

45 temperature and skin blood flow but not with TEWL.

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