ライフサイエンスコーパス: TG neuron

1 but no significant differences were found in TG neurons.

2 n of viral progeny in SCG neurons but not in TG neurons.

3 more highly attenuated in MRC5 cells than in TG neurons.

4 capsaicin responses in both male and female TG neurons.

5 ng that tonic NO levels inhibit M-current in TG neurons.

6 he mutation resulted in hyperexcitability of TG neurons.

7 +) channels (VGCCs) in 23% of small-diameter TG neurons.

8 lized with these receptors in SCG but not in TG neurons.

9 PRLRs), short and long, were also present in TG neurons.

10 n gene-related peptide release from cultured TG neurons.

11 , the truncated promoters did not express in TG neurons.

12 e PCR assay for the viral genome on purified TG neurons.

13 alpha 4 and beta 2) were localized in intact TG neurons.

14 s were able to drive transgene expression in TG neurons.

15 HEK293T cells and mouse trigeminal ganglion (TG) neurons.

16 se (PI3K) pathways in female rat trigeminal (TG) neurons.

17 spine density in Mecp2(WT_EGFP) transgenic (TG) neurons.

18 er of BMP4 signaling in trigeminal ganglion (TG) neurons.

19 establishes latency in trigeminal ganglion (TG) neurons.

20 Compared with non-Tg neurons, 3xTg-AD neurons had more mitochondria/neuron

21 h reduced beta-catenin protein expression in TG neurons 6 h after dexamethasone treatment.

22 ome P450 isozymes are rapidly upregulated in TG neurons after orofacial inflammation and increase the

23 By 24 h, staining was also seen in a few TG neurons and by 96 h their number had greatly increase

24 8, and 9 trafficked from the whiskerpad into TG neurons and expressed transgenes within cell bodies a

25 expressed mutant TRESK subunits in cultured TG neurons and observed a significant decrease in the la

26 PRL expression was restricted to TG neurons and was highly overlapped with transient pote

27 discharges from injured trigeminal ganglion (TG) neurons and thalamocortical reorganization are possi

28 activity in dental pulp, trigeminal ganglia (TG) neurons, and their nerve fibers.

29 g patterns in the IAN and V2 branches of the TG neurons; and (3) the receptive field expanded, the mo

30 sed green fluorescent protein efficiently in TG neurons but did not induce HSV-1 reactivation.

31 multiple transgenes could be co-delivered to TG neurons by separate AAV vectors.

32 Taken together, these data show that adult TG neurons can mount an effective antiviral response onl

33 anscript (LAT)-positive trigeminal ganglion (TG) neurons coexpressed SSEA3, 71% coexpressed Trk(A) (t

34 These data reveal that rat TG neurons contain the entire spectrum of mammalian NnAC

35 stochemical studies revealed that almost all TG neurons contained alpha 7-LI and alpha 4-LI, and that

36 tribution of LAT-positive, latently infected TG neurons contrasted sharply with (i) the overall distr

37 ught to further investigate this response in TG neurons cultured from adult mice deficient in a varie

38 mouse, may be such an intrinsic modulator of TG neuron development.

39 Cultured PTPRO(-/-) TG neurons display enhanced axonal outgrowth and branchi

40 y expressed in the same trigeminal ganglion (TG) neuron during reactivation and cooperatively stimula

41 Direct mechanical stimulation of duck TG neurons evokes high-amplitude depolarizing current wi

42 Subsets of TG neurons express different receptors for growth factor

43 hibition in vivo increases the percentage of TG neurons expressing deltaR on the surface and allows e

44 root ganglion (DRG) and trigeminal ganglion (TG) neurons expressing the cold-sensitive TRPM8 channel

45 mbrane currents evoked in piperine-sensitive TG neurons far exceeded the algebraic sum of the respons

46 ling in a heterologous expression system and TG neurons from PRL receptor (PRLR)-null mutant mice by

47 In PTPRO mutant mice, subsets of TG neurons grow longer and more elaborate axonal branche

48 n part to a dramatic increase in the loss of TG neurons in animals infected with the LAT mutants.

49 he rabbit efficiently transduced >70% of the TG neurons in the optic tract.

50 frequencies from latently infected explanted TG neurons in the presence or absence of CD45(+) cells.

51 RPM8 antagonists, and were absent in DRG and TG neurons isolated from Trpm8(-/-) mice.

52 the state of persisting HSV genomes in some TG neurons may be more dynamic and more easily activated

53 Furthermore, the majority of BK-responsive TG neurons may have a potential to become responsive to

54 The predominant PRLR form in TG neurons/nerves from rats and humans is PRLR-S.

55 C/EBP-alpha protein expression is induced in TG neurons of infected calves and after dexamethasone-in

56 About 1.8% of the TG neurons of mice infected with 17/tBTK(-) harbored the

57 spontaneous activities, first in the injured TG neurons of the IAN (2-30 d), followed by uninjured V2

58 ifferences in the growth rates of TMN and DM-TG neurons on L1.

59 d WIN 55,212-2 (WIN) to cultured trigeminal (TG) neurons or isolated skin biopsies rapidly and signif

60 nfection with HSV and antiviral signaling in TG neurons produce an unorthodox autophagic response.

61 These studies indicate that SP production in TG neurons projecting to the nasal epithelium is transie

62 Despite these findings, TG neurons responded to IFN-beta pretreatment with STAT1

63 We report that ~50% of trigeminal ganglion (TG) neurons retrogradely labeled from the DP express TRP

64 TG neurons showed decreased basal cytosolic calcium leve

65 re pervasive latent HSV-1 infection of human TG neurons than originally thought.

66 e immunosurveillance of trigeminal ganglion (TG) neurons that harbor latent HSV-1.

67 nd L1, were markedly different on TMN and DM-TG neurons, these differences were not sufficient to cau

68 al inflammation and increase the capacity of TG neurons to generate OLAMs.

69 Capsazepine blocked the response of TG neurons to piperine at both physiological and acidic

70 The relative permissiveness of TG neurons to viral gene expression near the joint regio

71 In this study, HSV-1 infection of murine TG neurons triggered unusual clusters of autophagosomes,

72 levels and redox state in nontransgenic (non-Tg) neurons until middle age, followed by a decline in o

73 (MRC5) and adult murine trigeminal ganglion (TG) neurons using the Illumina platform for cDNA sequenc

74 d on cultured adult rat trigeminal ganglion (TG) neurons voltage-clamped near their resting membrane

75 nt sites established in trigeminal ganglion (TG) neurons was determined using a single-cell quantitat

76 issociated adult murine trigeminal ganglion (TG) neurons were assessed for relative permissiveness fo

77 e of spread from the olfactory epithelium to TG neurons, while SSMs blocked systemic spread.

78 Treatment of TG neurons with epileptogenic compound-PTZ led to a mark

79 genetically manipulate trigeminal ganglion (TG) neurons would be useful in the study of the craniofa