ライフサイエンスコーパス: TG

1 TG and HDL-C concentrations were not associated with ris

2 TG biosensors work ideally within 2.5-2700s, in pH range

3 TG disposal and acylcarnitine production during lipid ov

4 TG level is below 150mg/dL in healthy persons.

5 TG mice demonstrated significant age-associated increase

6 plasma glucose (p = 0.008), TG (p = 0.003), TG: HDL-C ratio (p = 0.010), and vWF levels (p = 0.004).

7 (all p < 0.001), plasma glucose (p = 0.008), TG (p = 0.003), TG: HDL-C ratio (p = 0.010), and vWF lev

8 itracer PET imaging in transgenic APPPS1-21 (TG) mice.

9 In addition to canonical LTR-RTs with 5'-TG...CA-3' termini, LTR_retriever also identifies noncan

10 elf-assembly reaction is compatible with a 6-TG-modified DNA duplex and provides a straightforward me

11 helical chains, characterized as {Au(I)(mu-6-TG)} n , extending many mum in length that are structura

12 e (MNNG) and antimetabolite 6-thioguanine (6-TG).

13 tion polymer derived from 6-thioguanosine (6-TG-H), a sulfur-containing analog of a natural nucleosid

14 d TG <100 mg/dL (odds ratio 1.3 [1.0, 1.6]), TG >/=100 mg/dL and LDL-C <100 mg/dL (odds ratio 1.3 [1.

15 A TG mouse line secreting high levels of hNGF protein in i

16 ermined how the CXCL10/CXCR3 pathway affects TG- and cornea-resident CD8(+) T cell responses to recur

17 ndent thrombin generation (TG) and amplified TG initiated by low concentrations of tissue factor.

18 calorimetry (TG-DSC), evolved gas analysis (TG-DSC-FTIR) and High-resolution mass spectra (HRMS).

19 Dga1p, a diacylglycerol acyltransferase, and TG accumulation were both 30-35% lower in the absence of

20 6.0-11.0, temperature 25-39.5 degrees C and TG concentration range, 0.001-100mM, the detection limit

21 D and BOP, PD and TC, PD and TG, and CAL and TG in each group (P <0.01).

22 e displayed decreased plasma cholesterol and TG levels and reduced atherosclerotic lesions.

23 ocked all of these changes in the cornea and TG.

24 -C was accompanied by LDL-C >/=100 mg/dL and TG <100 mg/dL (odds ratio 1.3 [1.0, 1.6]), TG >/=100 mg/

25 tly after stimulation of cells with 5-HT and TG.

26 noted between PD and BOP, PD and TC, PD and TG, and CAL and TG in each group (P <0.01).

27 sory neurons of trigeminal ganglia (TG), and TG-resident CD8(+) T cells play a critical role in preve

28 Wild-type (WT) and TG mice received vehicle or BACE inhibitor (60 mg/kg) st

29 th FT-IR, SEM, EDX, TEM, UV-Visible, XRD and TG/DTA techniques.

30 ion-specific z-scores for WC, BP, HDL-C, and TGs.

31 tive associations between prenatal PFHxS and TGs z-score [for a doubling of exposure, beta=0.11; 95%

32 ge between the negative of HDL-C z-score and TGs z-score to give similar weight to lipids and the oth

33 -truncating) variant in APOC3 reported to be TG lowering and protective against CHD.

34 all detected pathologic alterations between TG and WT mice, only (18)F-AV45 could detect an effect o

35 ences between SB6 and commercial TG (Biobond TG-M).

36 ed to maintain circadian amplitudes of blood TG and FA in mice.

37 Furthermore, both TG and DSC results support the formation of inclusion co

38 Neuronal nucleus staining was lower in both TG groups in the thalamus and cortex.

39 d transcription factor CREBH is activated by TG accumulation and induces FGF21, which suppresses adip

40 l properties of the CPCMs were determined by TG and DSC.

41 h the amount of surface oxygen determined by TG-MS.

42 hased with respect to the histone octamer by TG motifs.

43 metry and differential scanning calorimetry (TG-DSC), evolved gas analysis (TG-DSC-FTIR) and High-res

44 s mainly associated with greater circulating TG disposal, lower systemic lipolysis, and better fatty

45 variant in APOC3 causes reduced circulating TG levels and, hence, protects from CHD.

46 ry obese mice lowers hepatic and circulating TGs and normalizes hyperglycemia.

47 ith the tip generation/substrate collection (TG/SC) mode of scanning electrochemical microscopy (SECM

48 cated differences between SB6 and commercial TG (Biobond TG-M).

49 s was seen for SB6 as compared to commercial TG.

50 B deficient (ETB def) or transgenic control (TG-con) rats were used in the presence or absence of ETA

51 nclusion, we uncovered here an active cornea-TG axis, driven by PEDF-R activation, that fosters axon

52 onding cell bodies of the DRG or the cranial TG.

53 ial remodeling and development of AF in CREM-TG mice.

54 s using the alpha-Gal analytes CTX and Bos d TG confirms the history of MA patients in 91% and 88% of

55 ng analytes CTX, bovine thyroglobulin (Bos d TG), and human serum albumin (HSA)-conjugated alpha-Gal.

56 s-reactivity profile (IgE against CTX, Bos d TG, and HSA-alpha-Gal) was identified, which is associat

57 d(TG(Br)GG(Br)GAC7) forms long linear quadruplex wires und

58 sign of two oligonucleotides d(TG4AC7) and d(TG(Br)GG(Br)GAC7) that self-assemble to form quadruplex

59 n in response to ATII, whereas PGC-1alpha EC TG mice were protected.

60 ng the VE-cadherin promoter to create ECIRS1 TG mice, which elevated pAkt activation and expressions

61 ersus WT mice, which were improved in ECIRS1 TG mice on normal chow or HF diet.

62 r out of season, were stimulated with either TG extract or a pool of previously identified immunodomi

63 ell-tolerated phenotype persisted in elderly TG with no indications of cardiac pathology or premature

64 Elevated TGs level in blood over 500mg/dL is a biomarker for card

65 er triglyceride (TG) as a result of enhanced TG hydrolysis and subsequent FA oxidation.

66 l adenine, consistent with trafficking of ER TG through the autophagic pathway before oxidation.

67 idly the virus reactivated following explant TG-induced reactivation.

68 cific ApoC-III-targeting ASO reduces fasting TG levels through a mechanism that is dependent on low-d

69 Besides transporting fat, TGs also act as stored fat in adipose tissue, which is u

70 DL-C was analyzed with higher thresholds for TG (>/=150 mg/dL) and LDL-C (>/=130 mg/dL), results were

71 r HDL-C (6.6% of variance), and 140 SNPs for TGs (5.9% of variance).

72 hing depended on the wheat protein fraction, TG amount and its origin.

73 Additionally, saliva collected from TG mice and containing unpurified hNGF was able to signi

74 upports the concept that salivary gland from TG animals is an efficient system for production of valu

75 s (TG), human primary hepatocytes (HPH) from TG, and mouse liver/HepG2 results from the Gene Expressi

76 l role in preventing HSV-1 reactivation from TG and subsequent virus shedding in tears that trigger r

77 Furthermore, TG secreted from human thyrocyte cultures hyperstimulate

78 By engineering loss- (Grem2(-/-)) and gain- (TG(Grem2)) of-Grem2-function mice, we discovered that Gr

79 activity in dental pulp, trigeminal ganglia (TG) neurons, and their nerve fibers.

80 ells maintain latency in trigeminal ganglia (TG) of mice latently infected with herpes simplex virus

81 t ganglia (DRG), and the trigeminal ganglia (TG) of streptozotocin-diabetic and healthy control rats.

82 n in the eye, latency in trigeminal ganglia (TG), and markers of T cell exhaustion in the TG were det

83 ithin sensory neurons of trigeminal ganglia (TG), and TG-resident CD8(+) T cells play a critical role

84 ection in the neurons of trigeminal ganglia (TG), cycling between productive infection and latency.

85 al root ganglia (DRG) or trigeminal ganglia (TG), respectively.

86 , were induced in bovine trigeminal ganglia (TG), which correlated with reduced beta-catenin protein

87 nal peptide (vip) in the trigeminal ganglia (TG).

88 nfected human and rabbit trigeminal ganglia (TG).

89 e sensory neurons of the trigeminal ganglia (TG).

90 Both sensory trigeminal ganglion (TG) and sympathetic superior cervical ganglion (SCG) neu

91 y expressed in the same trigeminal ganglion (TG) neuron during reactivation and cooperatively stimula

92 genetically manipulate trigeminal ganglion (TG) neurons would be useful in the study of the craniofa

93 PH) from Drug Matrix (DM) and open TG-GATEs (TG), human primary hepatocytes (HPH) from TG, and mouse

94 gered contact-dependent thrombin generation (TG) and amplified TG initiated by low concentrations of

95 Timothy grass (TG) pollen is a common seasonal airborne allergen associ

96 ering (SD-TIRS) with a transmission grating (TG).

97 d minerals by coupling a thermal gravimeter (TG) and a cavity ringdown laser spectrometer (CRDS).

98 nstrated that Acot1 knockdown led to greater TG turnover and FA oxidation, suggesting that ACOT1 is i

99 ntinuation group (IDG) or the tapered group (TG).

100 density lipoprotein (HDL) triglycerides (HDL-TG) predicted LVEF (beta=1.90 [95% confidence interval (

101 ide transfer protein (MTP) activity, hepatic TG content increased dramatically; however, CREBH proces

102 k the insulin receptor in the brain, hepatic TG secretion was reduced compared with wild-type litterm

103 pla3(148M/M) and Pnpla3(+/+) mice in hepatic TG synthesis, utilization, or secretion.

104 ic diet-fed mice displayed increased hepatic TG content, which was highly correlated (r(2) = 0.95) wi

105 he third ventricle acutely increased hepatic TG secretion.

106 These results reveal that hepatic TG content, per se, does not regulate CREBH processing.

107 LD1 deficiency led to an increase in hepatic TGs and liver weight.

108 Higher TG concentrations at baseline were associated with an in

109 induced in another set of control and hREG3A-TG mice by administration of trinitrobenzene sulfonic ac

110 Pnpla3(148M/M) mice being caused by impaired TG mobilization from LDs.

111 activator of PKCeta, significantly improved TG activity in epidermis of AD17(DeltaKC) mice.

112 CD103(high)CD8(+) tissue-resident T cells in TG of latently infected HLA-A*0201-transgenic mice and r

113 apparent feeble numbers of CD8(+) T cells in TG remains a challenge for immunotherapeutic strategies.

114 of this study was to characterize changes in TG-specific T cell responses as a function of seasonalit

115 e, delivery of exogenous CXCL10 chemokine in TG of CXCL10(-/-) mice, using the neurotropic adeno-asso

116 lutamate respiration rates were decreased in TG mice.

117 ncreased collagen and glycogen deposition in TG mice.

118 ific CXCR3(+) CD8(+) T cells was detected in TG and corneas of protected C57BL/6 (B6) mice, but not i

119 h reduced beta-catenin protein expression in TG neurons 6 h after dexamethasone treatment.

120 s were able to drive transgene expression in TG neurons.

121 iodination in vitro, de novo T3 formation in TG was decreased in mice lacking TSHRs.

122 but no significant differences were found in TG neurons.

123 tected a significant age-related increase in TG mice (P < 0.0001) but did not detect the treatment-in

124 al microbiota occurred after a few months in TG mice heterozygous for REG3A that harbored a wild-type

125 s of protected C57BL/6 (B6) mice, but not in TG and corneas of nonprotected CXCL10(-/-) or CXCR3(-/-)

126 n of viral progeny in SCG neurons but not in TG neurons.

127 lized with these receptors in SCG but not in TG neurons.

128 irmed reduction of amyloid-beta pathology in TG-BACE mice.

129 a(+) DCs in blocking explant reactivation in TG of mice latently infected with avirulent or virulent

130 tribute to HSV-1 latency and reactivation in TG of ocularly infected mice.

131 ome P450 isozymes are rapidly upregulated in TG neurons after orofacial inflammation and increase the

132 no effect on VLDL secretion, heparin-induced TG reduction, or uptake of lipids into heart and skeleta

133 ctivation was induced from latently infected TG by UV-B light.

134 -specific CD8(+)T cells in latently infected TG, thus allowing for increased viral reactivation.

135 ocation induced by catecholamine injections, TG animals were resistant to triggered ventricular arrhy

136 Instead, TG mobilization into the endoplasmic reticulum for nasce

137 omicrons, these lipids are reesterified into TG, primarily through the monoacylglycerol pathway.

138 8, and 9 trafficked from the whiskerpad into TG neurons and expressed transgenes within cell bodies a

139 Intracellular TG storage was induced in the cells by sodium lactate.

140 cordingly, Ca(2+)-spark analysis in isolated TG cardiomyocytes revealed remarkably reduced Ca(2+) lea

141 ase risk for type 2 diabetes (T2D); and (iv) TG-lowering alleles involved in hepatic production of TG

142 IV gene expression, and secretion of larger, TG-enriched VLDL, despite a lower rate of TG secretion a

143 SV-1 or with LAT-rescued mutant (i.e., LAT(+)TG) is significantly higher than TG latently infected wi

144 ells from LAT(-)TG, CD8(+)T cells from LAT(+)TG (i) recognized a broader selection of nonoverlapping

145 y infected with LAT-null mutant (i.e., LAT(-)TG).

146 Compared to CD8(+)T cells from LAT(-)TG, CD8(+)T cells from LAT(+)TG (i) recognized a broader

147 However, the ApoC-III ASO did not lower TG levels in mice lacking both LDLR and LRP1.

148 These biosensors measured TG level in fruit juices, beverages, sera and urine samp

149 isoform CREM-IbDeltaC-X in transgenic mice (TG) leads to spontaneous-onset AF preceded by atrial dil

150 ave been transferred to the ER by microsomal TG transfer protein (MTP), inducing ER stress.

151 slocation in WT mice was exacerbated in MIOX-TG mice but absent in MIOX(-/-) mice.

152 Similarly, MIOX-TG mice had the highest and MIOX(-/-) mice had the lowes

153 itional MIOX-overexpressing transgenic (MIOX-TG) mice and MIOX-knockout (MIOX(-/-)) mice with tubule-

154 wild-type (WT) mice, cisplatin-treated MIOX-TG mice had even greater increases in urea, creatinine,

155 tokines in kidneys of cisplatin-treated MIOX-TG mice.

156 Moreover, TG demonstrated improved cardiac function after myocardi

157 Most TG nerve endings are subepithelial, so this colonization

158 ithin the carboxyl-terminal portion of mouse TG, T3 is formed de novo independently of deiodination f

159 jor regulators of HSV-1 latency in the mouse TG.

160 tiple ASYMP epitopes (prime) and neurotropic TG delivery of the T cell-attracting chemokine CXCL10 (p

161 al microbiota from REG3A-TG mice protect non-TG mice from induction of colitis.

162 urthermore, fasting acyl carnitines in obese TG mice were decreased, indicating that increased GLUT4-

163 obtained to produce 6.074+/-0.019UmL(-1) of TG from a novel source; Streptomyces sp.

164 Additions of TG to wheat protein and flour based doughs revealed that

165 To define what aspects of TG homeostasis regulate hepatic CREBH processing and apo

166 al inflammation and increase the capacity of TG neurons to generate OLAMs.

167 further improvement and commercialization of TG biosensors are discussed.

168 operating principles, merits and demerits of TG biosensors, specifically nanomaterials based biosenso

169 ype of pre-treatment on the detectability of TG were found.

170 antation is indicative of the development of TG in patients with dnDSA (P = 0.001).

171 Disruption of TG neuronal ecto-nucleotidase expression and axonal term

172 ovo T3 that can be formed upon iodination of TG secreted from PCCL3 (rat thyrocyte) cells was augment

173 ophagy and subsequent lysosomal lipolysis of TG, followed by mitochondrial oxidation of released FA,

174 of PNPLA3-148M and impaired mobilization of TG from LDs.

175 s developed using tryptic marker peptides of TG (five markers), and bovine and porcine fibrinogen (si

176 hibition in vivo increases the percentage of TG neurons expressing deltaR on the surface and allows e

177 The reactivation phenotype of TG that are latently infected with wild-type HSV-1 or wi

178 ng alleles involved in hepatic production of TG-rich lipoproteins (TM6SF2 and PNPLA3) tracked with hi

179 r, TG-enriched VLDL, despite a lower rate of TG secretion and a similar or reduced rate of apoB100 se

180 To identify novel regulators of TG levels, we carried out a high throughput screen (HTS)

181 e, but not control sera, led to secretion of TG with an increased intrinsic ability to form T3 upon i

182 adipose tissue constitutes a major source of TG in the liver of patients with nonalcoholic fatty live

183 Pre-treatment of TG-con rats with the ETA blocker ABT-627 for 1 week prio

184 indicate that ApoC-III inhibits turnover of TG-rich lipoproteins primarily through a hepatic clearan

185 I: 0.026, 0.154; P = 0.007) and the ratio of TGs to high-density lipoprotein (HDL) cholesterol (beta

186 us methods are accessible for recognition of TGs, among them, most are cumbersome, time-consuming, re

187 fore AF onset) and old (TGo, after AF onset) TG mice were investigated by mRNA microarray profiling i

188 tocytes (RPH) from Drug Matrix (DM) and open TG-GATEs (TG), human primary hepatocytes (HPH) from TG,

189 C <100 mg/dL (odds ratio 1.3 [1.1, 1.5]), or TG and LDL-C >/=100 mg/dL (odds ratio 1.6, [1.2, 2.2]),

190 in single phenotype rare variant analyses (P TG = 6.5 x 10(-8) and P FI = 0.27).

191 provides stronger support for association (P TG+FI = 1.8 x 10(-9)) than observed in single phenotype

192 vely reduced body weight, hepatic and plasma TG, and total cholesterol (TC) levels.

193 ynthesis pathway and resulted in high plasma TG content.

194 CIII (ApoC-III) is a key regulator of plasma TG levels through regulation of lipolysis and lipid synt

195 ApoC-III ASO treatment lowered plasma TGs in mice lacking lipoprotein lipase (LPL), hepatic he

196 for ApoC-III ASO-induced reduction of plasma TGs in mice fed a high-fat diet, in postprandial clearan

197 Increased LDL-C, HDL-C, and possibly TG levels are associated with a lower risk of diabetes.

198 NINDS Intramural Research Program; TG Therapeutics Inc.

199 The developed quantitative TG-MS analysis was used for the determination of the sur

200 It was proved that the quantitative TG-MS analysis could be a useful tool for the characteri

201 D8(+)T cells within the latency/reactivation TG site.

202 REG3A TG mice developed only mild colonic inflammation after e

203 used and germ-free mice fed feces from REG3A-TG mice and given DSS developed less-severe forms of col

204 Fecal samples from REG3A-TG mice had lower levels of ROS than feces from control

205 Fecal microbiota from REG3A-TG mice protect non-TG mice from induction of colitis.

206 compared with mice not fed feces from REG3A-TG mice.

207 2-O-tetradecanoylphorbol-13-acetate restored TG activity in the epidermis of keratinocyte-specific Ad

208 that an 80-mer minicircle DNA using the same TG-motifs faithfully reproduces the CPD pattern in the n

209 ino acid signature was associated with serum TG and with the risk of hypertriglyceridemia after 2 yea

210 whereas Sike-overexpressing transgenic (Sike-TG) mice are protected from hypertrophic stimuli.

211 rial were carried out by Raman spectroscopy, TG-MS, UV/vis, and fluorescence spectroscopy.

212 present data suggesting that TSH-stimulated TG phosphorylation contributes to enhanced de novo T3 fo

213 Serum levels of total cholesterol (TC), TG, low-density lipoprotein, and high-density lipoprotei

214 ef and pork were performed using a technical TG mixture (Activa, Ajinomoto), and bovine and porcine p

215 xperiments were performed with two technical TG mixtures with and without caseinate.

216 i.e., LAT(+)TG) is significantly higher than TG latently infected with LAT-null mutant (i.e., LAT(-)T

217 through a similar mechanism as thapsigargin (TG), involving increased cytosolic calcium.

218 form T3 Our data support the hypothesis that TG processing in the secretory pathway of TSHR-hyperstim

219 The TG effectively isolated the scattering signals in first-

220 ne to the analysis of the DrugMatrix and the TG-GATEs, two of the largest toxicogenomics resources av

221 The results obtained from the TG-MS analysis imply the good agreement with the results

222 -1 replication in the eye and latency in the TG by modulating immune components, specifically, by alt

223 protocol showed benefits, as patients in the TG experienced less relapses and lower accumulation of M

224 Levels of vector genomes in the TG increased with input dose, and multiple transgenes co

225 CD8, IFN-gamma, and PD-1 transcripts in the TG of latently infected mice.

226 latency and, thus, T-cell exhaustion in the TG of ocularly infected mice.IMPORTANCE Our findings dem

227 TG), and markers of T cell exhaustion in the TG were determined.

228 s titers in the eye, had less latency in the TG, and took a longer time to reactivate than virulent s

229 , CD8, IFN-gamma,and PD-1 transcripts in the TG.

230 T cells seem to play a bystander role in the TG.

231 ely in the infected ophthalmic branch of the TG.

232 sensory neurons in all three branches of the TG.

233 etion of Bacteroidetes (Prevotellaceae); the TG mice developed less-severe colitis following administ

234 f relapses in the IDG (n=28) compared to the TG (n=8) over 12 months from the last infusion (p=0.007)

235 virus (AAV) vectors for gene delivery to the TG after intradermal whiskerpad delivery in mice.

236 ing the last natalizumab infusion, while the TG received two more natalizumab infusions, at 6 and 8 w

237 plexation, as revealed by thermogravimetric (TG) analysis (13.3% for beta-CD, 2.5-6.5% for complexes)

238 l stability as analyzed by thermogravimetry (TG) and differential thermal analysis (DTA).

239 hermogravimetry/derivative thermogravimetry (TG/DTG), differential scanning calorimetry coupled with

240 to the ion source by using thermogravimetry (TG) hyphenated to REMPI time-of-flight mass spectrometry

241 ccessfully purified from the saliva of these TG mice and its identity was verified.

242 results in photooxidation of 1-thioglycerol (TG) mediated by Os-PVP complex on the surface of graphit

243 h photocatalyze oxidation of 1-thioglycerol (TG), generating photocurrent as the readout signal.

244 PF) in stomach carcinoma, and thyroglobulin (TG) in thyroid carcinoma.

245 de for the detection of human Thyroglobulin (TG), a protein marker of differentiated thyroid cancer.

246 on results from iodination of thyroglobulin (TG) at residues Tyr(5) and Tyr(130), whereas thyroidal T

247 multiple transgenes could be co-delivered to TG neurons by separate AAV vectors.

248 V1 vectors are suitable for gene delivery to TG sensory neurons following intradermal whiskerpad inje

249 significantly decreased in fat-1 transgenic (TG) mice (P < 0.001), which exhibited decreased cyclooxy

250 In this study, we generated 18 transgenic (TG) founder mice each carrying a salivary gland specific

251 art-specific phosphodiesterase 2-transgenic (TG) mice showed a marked reduction in resting and in max

252 By using a transgenic (TG) mouse with cardiac-specific overexpression (3.5-fold

253 enhancement of cardiac function, transgenic (TG) mice expressing non-phosphorylatable TnI protein kin

254 We investigated whether transgenic (TG) expression of the human regenerating family member 3

255 the human GLUT4 promoter (i.e., transgenic [TG] mice) are resistant to obesity-induced insulin resis

256 the detection of microbial transglutaminase (TG) from Streptomyces mobaraensis in different types of

257 ous detection of microbial transglutaminase (TG) from Streptomyces mobaraensis, and bovine and porcin

258 The popularity of transglutaminase (TG) by the food industry and the variation in functional

259 1a, which possesses TP and transglycosylase (TG) activities.

260 ide hydrolases (GHs) into transglycosylases (TGs), i.e., from enzymes that hydrolyze carbohydrates to

261 Regardless of treatment, TG mice demonstrated significantly lower (18)F-FDG uptak

262 ized by droplets containing triacylglycerol (TG).

263 eptors expressed on terminals of trigeminal (TG) nociceptive afferent neurons.

264 combinant human histones H3 and H4 triggered TG in recalcified human plasma in a platelet-dependent m

265 Elevated triglyceride (TG) levels are well-correlated with the risk for cardiov

266 ose overload (2h-OGTT), HbA1c, triglyceride (TG) levels and HOMA-IR and positively with free fatty ac

267 ion is correlated with hepatic triglyceride (TG) content in mouse models of chronic hepatosteatosis,

268 n of the key genes involved in triglyceride (TG) and fatty acid (FA) metabolism and is required to ma

269 exercise induces intramuscular triglyceride (TG) accumulation and promotes mitochondrial maintenance

270 (sHDL), accompanied by larger, triglyceride (TG)-rich VLDL, and a higher lipoprotein insulin resistan

271 Acot1 knockdown reduced liver triglyceride (TG) as a result of enhanced TG hydrolysis and subsequent

272 the D25V-carriers exhibit low triglyceride (TG) and apolipoprotein C-III levels, and low very-low-de

273 esponsible for accumulation of triglyceride (TG) and PNPLA3 in hepatic lipid droplets (LDs).

274 C-III, a critical inhibitor of triglyceride (TG) lipolysis and remnant TRL clearance.

275 ciation, if any, between serum triglyceride (TG) levels and gemfibrozil consumption with periodontal

276 of total cholesterol (TC) and triglycerides (TG)).

277 tein cholesterol (LDL-C), and triglycerides (TG).

278 erized by the accumulation of triglycerides (TG) as lipid droplets in the liver.

279 s is to increase secretion of triglycerides (TG) packaged as VLDLs.

280 use of MARV with analysis of triglycerides (TG), fasting insulin (FI) and waist-to-hip ratio (WHR) i

281 n cholesterol (LDL-C), and/or triglycerides (TG).

282 P = 3.84 x 10(-31)) and serum triglycerides (TG) (beta = 0.067, P = 4.5 x 10(-21)).

283 ic accumulation of unsecreted triglycerides (TG).

284 Triglycerides (TGs) are the major transporters of dietary fats througho

285 fic z-scores for cholesterol, triglycerides (TGs), high-density (HDL-C), and low-density lipoprotein

286 of lean organs to circulating triglycerides (TGs) and nonesterified fatty acids (NEFAs), ultimately l

287 associated with a decrease in triglycerides (TGs) (beta = 0.090; 95% CI: 0.026, 0.154; P = 0.007) and

288 ovascular disease, and plasma triglycerides (TGs) correlate strongly with plasma apolipoprotein C-III

289 ates with steatosis and serum triglycerides (TGs) in humans.

290 Fasting lipid fractions (triglycerides [TGs], high-density lipoprotein cholesterol [HDL-C], low-

291 l exploitation was compared to commonly used TG, from Streptomyces mobaraensis.

292 orm of nitric oxide synthase (iNOS) from VCP TG mouse and by pharmacological inhibition of iNOS in is

293 KO animals due to a 3-fold decrease in VLDL-TG secretion rate without any associated reduction in he

294 Plasma VLDL-TG levels were reduced in the KO animals due to a 3-fold

295 d with 20% to 40% lower CVD risk except when TG and LDL-C were elevated.

296 isk for T2D, and lower risk for CAD, whereas TG-lowering alleles involved in peripheral lipolysis (LP

297 x 10-8), and one novel locus associated with TG near WDR11-FGFR2 (P = 2.7 x 10-10).

298 These results are consistent with TG accumulation in the Pnpla3(148M/M) mice being caused

299 memory CD8(+) T cells (TCM), locally within TG, and improved protection against recurrent herpesviru

300 d us to isolate and evaluate a high-yielding TG strain.