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1 TMJ arthritis was present in the majority of patients wi
2 TMJ clicking was the most common clinical symptom.
3 TMJ disk displacement was less prevalent and was of a di
4 TMJ intra-articular status was determined by 3 blinded,
5 TMJ reconstruction with standard alloplastic prosthesis
6 TMJ tissues were removed, and expression of TNF-alpha an
10 that mice deficient in Gli2 display aberrant TMJ development such that the condyle loses its growth-p
14 To assess if estrogens act rapidly to affect TMJ-responsive neurons, we applied 17beta-estradiol (E2)
16 the prognosis of using standard alloplastic TMJ prostheses for the treatment of TMJ ankylosis in Chi
18 ble working to balancing muscle activity and TMJ reaction ratios during biting to those observed in v
20 sociation between MRI-depicted effusions and TMJ arthralgia, and (2) a PPT of 1 pound does not discri
22 HS sulfation is critical for mandibular and TMJ development and allows HS-PGs to exert their roles v
27 ermal fibroblasts, a mixture of the two, and TMJ disc cells in a scaffoldless tissue-engineering appr
29 ge-dependent OA-like changes in the knee and TMJs of Col9a1-/- mice starting at the age of 3 months.
31 ciceptive response of rats with an arthritic TMJ and reduced the amount of the proinflammatory cytoki
35 o consider the potential association between TMJ remodeling and mandibular repositioning under orthop
46 ular joint (TMJ) intra-articular disorders ("TMJ intra-articular status"), representing a transition
47 he activating FgfR3(P244R) mutation disturbs TMJ developmental processes, likely by reducing hedgehog
48 importance of regulated RTK signaling during TMJ development and suggest multiple skeletal origins fo
49 clinical symptoms were used to classify each TMJ data sample as healthy control ( n = 124) or TMJOA (
51 stages of the disease, using an established TMJ OA genetic mouse model deficient in 2 extracellular
54 nd tissue defects in young bgn(-/0)fmod(-/-) TMJ subchondral bone are likely attributed to increased
57 ted from 3-week-old WT and bgn(-/0)fmod(-/-) TMJs with an intact cartilage/subchondral bone interface
58 ifferentially expressed in bgn(-/0)fmod(-/-) TMJs, including 5 genes involved in osteoclast activity/
60 t biglycan and fibromodulin are critical for TMJ subchondral bone integrity and reveal a potential ro
63 Our data indicate that Prg4 is needed for TMJ disc integrity and function and that its absence lea
64 ne integrity and reveal a potential role for TMJ subchondral bone turnover during the initial early s
67 od can serve as a primary screening tool for TMJ pathology, before proceeding to complicated, time co
69 s in tissue stiffness and ultrastructure for TMJ components is critical to understanding joint functi
71 dence of TMJ inflammation received CT-guided TMJ injections of corticosteroid (triamcinolone acetonid
73 with a chamber in the exact shape of a human TMJ was designed for controllable perfusion throughout t
75 ile and compressive) properties of the human TMJ disc, and also discs from the cow, goat, pig, and ra
81 electrical and solute transport behaviors in TMJ discs under mechanical loading and aids in the under
84 hat a reduction in FcgammaRIII expression in TMJ tissues would reduce the nociceptive and inflammator
87 ll death and severe functional impairment in TMJ chondrocytes, and warrant in vivo testing to explore
88 Fos expression suggests that an increase in TMJ C-fiber input after inflammation and robust central
89 -AR signal-mediated subchondral bone loss in TMJ osteoarthritisis associated with increased RANKL sec
91 upport the idea that Wnt5a/Ror2 signaling in TMJ subchondral BMSCs enhanced by UAC promoted BMSCs to
99 poro-mandibular or temporomandi-bular joint (TMJ), a highly specialized synovial joint that permits m
101 thritis (OA) of the temporomandibular joint (TMJ) and to explore the role of stromal cell-derived fac
105 Inflammation of the temporomandibular joint (TMJ) can alter behavioral responses such as food intake
106 We selected the temporomandibular joint (TMJ) condylar bone as our tissue model, because of its c
110 -engineering of the temporomandibular joint (TMJ) disc aims to provide patients with TMJ disorders an
111 gitudinal course of temporomandibular joint (TMJ) disc displacement (DD) and degenerative joint disea
112 nt of patients with temporomandibular joint (TMJ) disc displacement without reduction (DDwoR), but th
113 or individuals with temporomandibular joint (TMJ) disc displacement without reduction with limited mo
118 ransport in porcine temporomandibular joint (TMJ) discs using the electrical conductivity method.
120 uency and impact of temporomandibular joint (TMJ) disorders necessitate research in characterizing th
122 the hypothesis that temporomandibular joint (TMJ) eminence shapes develop ideally to minimize joint l
125 advanced stages of temporomandibular joint (TMJ) intra-articular disorders ("TMJ intra-articular sta
128 ee that the primate temporomandibular joint (TMJ) is loaded compressively during function and that co
129 s) derived from the temporomandibular joint (TMJ) mandibular condyle that generates cartilage anlagen
130 usly shown in a rat temporomandibular joint (TMJ) model that injection of 15d-PGJ2 into the rat TMJ c
131 erally into the rat temporomandibular joint (TMJ) or perioral (PO) skin to produce inflammation in de
132 gy and treatment of temporomandibular joint (TMJ) osteoarthritis (TMJOA) remain complex and unclear.
134 seek treatment for temporomandibular joint (TMJ) symptoms, typically occurring with anterior disc di
135 ive response of the temporomandibular joint (TMJ) to mandibular advancement, while others have report
136 affecting the human temporomandibular joint (TMJ), but the underlying molecular mechanisms remain obs
143 erived from bovine temporomandibular joints (TMJ), were examined for matrix loss and the expression o
144 Knee joints and temporomandibular joints (TMJs) from Col9a1-/- mice and their wild-type (Col9a1+/+
145 ke lesions in mice temporomandibular joints (TMJs), displaying as subchondral trabecular bone loss.
146 the right and left temporomandibular joints (TMJs), or in the cisterna magna, respectively, to induce
153 emonstrate that DDR2 is necessary for normal TMJ condyle development and homeostasis and that these D
155 equation model estimated the association of TMJ intra-articular status with the latent measure TMD i
157 a) characterized individual-specific data of TMJ stress-field mechanics to determine ED (ED = W/ Q mJ
159 conductivity, as well as ion diffusivity, of TMJ discs was determined under confined compression with
160 ages 4-16 years with JIA and MRI evidence of TMJ inflammation received CT-guided TMJ injections of co
164 l brainstem site for synaptic integration of TMJ sensory signals, while recording single neuron activ
166 ng pathways is critical for the integrity of TMJ development and for the maintenance of cellular orga
167 ay led to a dramatic and progressive loss of TMJ articular integrity and osteoarthritis-like changes.
168 e technique, previously validated markers of TMJ pain or nociception (specifically, meal duration and
181 ly, the osteoblast activity in the tissue of TMJ subchondral trabecular bone of these UAC-treated rat
182 oplastic TMJ prostheses for the treatment of TMJ ankylosis in Chinese patients with severe mandibular
188 rogated the catabolic effects of IL-1beta on TMJ chondrocytes by inhibiting iNOS, COX-2, and MMP-1 mR
190 The effects of continuous passive motion on TMJ were simulated by exposing primary cultures of rabbi
192 Temporomandibular joint osteoarthritis (TMJ OA) is a degenerative disease that affects both cart
194 increased osteoclast activity and an overall TMJ subchondral trabecular bone loss in the UAC-treated
197 tation creep tests were performed on porcine TMJ condylar cartilage at 5 different regions-anterior,
198 ntact area were performed on 8 adult porcine TMJs at 5 different regions (anterior, posterior, centra
199 ential direct and indirect roles to preserve TMJ structural and cellular integrity over post-natal li
201 A-adrenergic signal in chondrocytes promotes TMJ degenerative remodelling by chondrocyte-mediated pro
203 lated by exposing primary cultures of rabbit TMJ fibrochondrocyte monolayers to cyclic tensile strain
204 The impact of PLGA MP concentration on rat TMJ function was quantified via computerized meal patter
205 odel that injection of 15d-PGJ2 into the rat TMJ can provide antinociceptive relief against a subsequ
206 15d-PGJ2 cream for 15min directly on the rat TMJ skin did not induce any significant antinociceptive
207 able for intra-articular delivery to the rat TMJ, a finding that has significant implications for the
208 y (within 10 minutes) and reversibly reduced TMJ-evoked neural activity at the Vc/C(1-2) region.
210 ession of hard tissue diagnoses in the right TMJ occurred in 15.2% of subjects (95% CI, 10.5% to 20.8
212 H rats also demonstrated larger implant-side TMJ discs and mandibular fossae in comparison with the o
213 hate-buffered physiological saline solution, TMJ = temporomandibular joint, mu(T) =tractional coeffic
216 The majority of children with symptomatic TMJ arthritis improved after intraarticular corticostero
217 advancement, while others have reported that TMJ adaptive responses are non-existent and negligible.
220 their mandibular ramus was elongated by the TMJ prosthesis and 2 patients were combined with Le Fort
222 ent an efficient approach to engineering the TMJ disc graft with anisotropic scaffold microstructure,
225 to dynamic behavior, perhaps implicating the TMJ disc as a structure primarily exposed to predominant
226 entified in this screen were examined in the TMJ and compared with those of other synovial joints, in
227 ated by type I and III collagen found in the TMJ and other fibrocartilages, has been associated with
230 ify changes in gene expression levels in the TMJ during early stages of the disease, using an establi
232 n of an osteoarthritis-like condition in the TMJ in the Col1-IL-1betaXAT mouse model resulted in up-r
235 RNA reduced the amount of FcgammaRIII in the TMJ tissues, and the transcript was cleaved in a manner
238 in conjunction with arthritic changes in the TMJ, assessed by histopathologic and immunohistochemical
240 th complete Freund's adjuvant (CFA) into the TMJ or served as uninjected controls and were killed two
241 CFA injection, the injection of CFA into the TMJ produced a significantly stronger inflammation assoc
251 ional forces following static loading of the TMJ disc: (1) increase with compressive strain at the st
253 for cellular and molecular evaluation of the TMJ during its adaptive response to biomechanical forces
257 The etiology of degenerative disease of the TMJ may involve fatigue produced by surface tractional f
259 ions in experimental JRA-like disease of the TMJ that are similar to those observed in humans with th
261 the synovial and subsynovial tissues of the TMJ, and plasma volume (PV) and permeability surface are
262 resulting from inflammatory arthritis of the TMJ, and siRNA has the potential to be an effective trea
263 tered craniofacial development in JRA of the TMJ, we characterized the gross morphologic adaptations
269 we performed micro-tribometry testing on the TMJ disc and condylar cartilage to obtain their region-
270 ut the effect of orthopedic treatment on the TMJ, it is necessary that we understand the biologic bas
273 bly, our work provides the evidence that the TMJ condyle and disc develop independently of the mandib
275 ologically and biochemically superior to the TMJ disc and dermal fibroblast constructs, and their com
276 structs would produce matrix relevant to the TMJ disc, but the mixture constructs were expected to ap
277 highly expressed in muscles attached to the TMJ, including the lateral pterygoid and temporalis musc
279 valbumin was used to induce arthritis in the TMJs of 10 previously sensitized adult white rabbits.
280 ficantly greater degenerative changes in the TMJs of 3- and 10-mo-old Ddr2(slie/slie) mice as compare
281 ighted magnetic resonance (MR) images of the TMJs in 58 patients with pain and dysfunction were analy
286 that these DDR2 functions are restricted to TMJ fibrocartilage and not seen in the hyaline cartilage
287 a transition from normal joint structure to TMJ disc displacement with and without reduction (DDwR a
291 number of posterior functional tooth units, TMJ disorder, and dentition status) on overall diet qual
292 stimated potential for cartilage fatigue via TMJ energy densities (ED) and jaw muscle duty factors (D
294 er fibrocartilages, has been associated with TMJ degeneration, but its role in normal joint developme
296 that primary treatment for individuals with TMJ closed lock should consist of medical management or
297 n a single-blind trial, 106 individuals with TMJ closed lock were randomized among medical management
299 int (TMJ) disc aims to provide patients with TMJ disorders an option to replace diseased tissue with
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