ライフサイエンスコーパス: TMV

1 TMV additionally emerged as a prototypic model to invest

2 TMV also exemplifies how a model system furthers novel,

3 TMV CP affects the display of several specific virus and

4 TMV first accumulated in abaxial or external phloem-asso

5 TMV infection of transgenic BY-CP(T42W) protoplasts resu

6 TMV movement protein (MP) and replicase colocalized with

7 TMV MP and PAPK1 are colocalized within cross-walls in a

8 TMV particles collected from the filter could be used fo

9 TMV RNAs initially appeared as single-stranded threads o

10 TMV that carried epitope 31D from the RV nucleoprotein d

11 TMV VLPs are high surface area macromolecules with nanor

12 TMV was detected in asymptomatic tissue surrounding lesi

13 TMV was simulated with 1.5 diopters (D) of anisometropia

14 TMV-CP(T42W):GFP failed to produce visible infection sit

15 TMV-induced fluorescent lesions on Rca- or AtpC-silenced

16 ys-118 (TMIII), Cys-251 (TMVI), and Cys-202 (TMV)).

17 4.52) (TMIV), F150(4.56) (TMIV), F184(5.40) (TMV), and Y188(5.44) (TMV) was observed away from the li

18 6) (TMIV), F184(5.40) (TMV), and Y188(5.44) (TMV) was observed away from the ligand-binding pocket, b

19 A TMV helicase polypeptide also was capable of unwinding d

20 rotoplasts and in planta when supported by a TMV mutant that expressed the 183-kDa protein but not th

21 thogenicity, the replication and spread of a TMV mutant with a reduced ability to interact with speci

22 is interaction in the display of symptoms, a TMV mutant defective in the PAP1 interaction was identif

23 s of the LRR, is more prevalent 4-8 hr after TMV infection.

24 amatically suppressed in sun1-1 plants after TMV treatment compared to wild-type plants.

25 coded proteins in a defense response against TMV.

26 he induction of resistance responses against TMV.

27 partners for Tyr(187) in TMIV (Phe(171)) and TMV (Trp(194)).

28 virus (MHV) and giving rise to TMV-5B19 and TMV-5B19L, respectively.

29 Closer examination of the CMV and TMV populations revealed biases for particular types of

30 type CP, the resulting viruses TMV-RB19E and TMV-4D:31D infected N. tabacum Xanthi-nn plants and BY2

31 is the unproven assumption that the TVCV and TMV life cycles are identical.

32 As TMV poorly infects Arabidopsis thaliana, Turnip vein cle

33 virus (TVCV), a virus from the same genus as TMV, did not require intact microfilaments for normal sp

34 Because TMV has a wide host range, expressing TMV-TMOF in plants

35 s to the TMV and bridging of the BSA between TMV produces the attractions.

36 The interaction between TMV MP and calreticulin was confirmed in vivo and in vit

37 tion to isolate a tobacco protein that binds TMV MP and identified it as calreticulin.

38 t 200 fold 4 d after infection (dai) by both TMV and TRV.

39 s report that SIPK and WIPK are activated by TMV in a gene-for-gene-dependent manner, we conclude tha

40 WIPK activation by TMV depends on the disease-resistance gene N because inf

41 Like the HR elicited by TMV infection, transgenic expression of p50 induces a te

42 Interestingly, induction of both genes by TMV infection was still observed in resistant tobacco pl

43 ic plants confers resistance to infection by TMV and related tobamoviruses.

44 in plants confers resistance to infection by TMV and related tobamoviruses.

45 On plants expressing CP(T42W), infection by TMV-CP:GFP or TMV-MP:GFP-CP produced infection sites of

46 4 genes could be rapidly induced not only by TMV infection but also by SA or its biologically active

47 inability of dRNAs that can be replicated by TMV to move in plants.

48 unctional relationship between calreticulin, TMV MP, and viral cell-to-cell movement.

49 sidue plus the 5B19 epitope fused to the CP (TMV-RB19), resulted in the induction of necrotic local l

50 o mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cells it interacted with the TMV

51 Finally, we developed TMV and SNP formulations loaded with the chemotherapeuti

52 CMV had the highest levels of diversity, TMV had an intermediate level of diversity, and CCMV had

53 as very efficient with a 35S promoter-driven TMV replicon that lacked the TMV coat protein gene seque

54 versity of these guilds being unique to each TMV.

55 ecause TMV has a wide host range, expressing TMV-TMOF in plants can be used as a general method to pr

56 For TMV/PEO mixtures, attractions are entropically driven vi

57 l replicase- and VRC-enriched fractions from TMV-infected Nicotiana tabacum plants.

58 tein and the homologous 125-kDa protein from TMV and TVCV, respectively.

59 Furthermore, TMV encoding the MP mutant mimicking phosphorylation was

60 used to the green fluorescent protein (GFP) (TMV-CP:GFP, TMV-CP(T42W):GFP) and clones harboring TMV m

61 green fluorescent protein (GFP) (TMV-CP:GFP, TMV-CP(T42W):GFP) and clones harboring TMV movement prot

62 :GFP, TMV-CP(T42W):GFP) and clones harboring TMV movement protein (MP):GFP were followed in nontransg

63 However, TMV vectors are, in general, not efficiently delivered t

64 Hybrid TMV-5B19, administered by subcutaneous injections, elici

65 tibody, showed specific decoration of hybrid TMV particles, confirming the expression and display of

66 Plants that were infected with the hybrid TMV RNA accumulated TMOF to levels of 1.3% of total solu

67 In TMV-infected tobacco, these studies revealed that critic

68 Furthermore, in TMV-infected plant tissues overexpressing calreticulin,

69 monstrated that expression of these genes in TMV-inoculated leaves is mediated via an SA-independent

70 However, in TMV-infected tissues, PAP1 failed to accumulate to signi

71 sion size upon TMV infection, as observed in TMV-inoculated N gene-containing NahG tobacco plants.

72 Since EH-1 was expressed only in TMV-resistant tobacco after infection, and the encoded e

73 d-type tobacco, suggesting a role for PME in TMV systemic infection.

74 vestigate the role of these host proteins in TMV accumulation and plant defense, we used a Tobacco ra

75 nspiration rate was significantly reduced in TMV infected N. tabacum.

76 recognized by N-containing tobacco to induce TMV resistance.

77 cing of NbBRI1 and NbBSK1 blocked BR-induced TMV resistance, and silencing of NbBES1/BZR1 blocked Bik

78 ortance of specific domains of N in inducing TMV resistance, by examining various N deletion and poin

79 IAA-interacting protein was found to inhibit TMV accumulation and phloem loading selectively in flowe

80 subviral replication complexes that initiate TMV replication immediately after entry to adjacent cell

81 When the biosensor was infiltrated into TMV-inoculated leaves displaying HR lesions at 90 and 16

82 tapeptide sequence TLIAHPQ was inserted into TMV coat protein near the C end.

83 howing that non-viral expression of a 50 kDa TMV helicase fragment (p50) is sufficient to induce the

84 In AtpC- and Rca-silenced leaves TMV accumulation and pathogenicity were greatly enhanced

85 SIPK/WIPK pathway attenuated N gene-mediated TMV resistance.

86 proteins play a critical role in N-mediated TMV resistance.

87 Genetically modified TMV VLPs express both surface attachment-promoting cyste

88 plasm and have documented associations of MP(TMV) with endoplasmic reticulum (ER) membrane, microtubu

89 obacco mosaic virus (TMV) 30-kDa protein (MP(TMV)).

90 d program averages intensities from multiple TMV molecules for accurate standard determination, makes

91 Native TMV forms a hollow, high aspect-ratio nanotube measuring

92 culated leaves because of the lack of native TMV coat protein.

93 and the poly(A) tail but not with the native TMV 3'-UTR which contains an independent translational e

94 bicin, and we demonstrate the application of TMV rods and spheres for chemotherapy delivery targeting

95 TMV by temporal and quantitative control of TMV Cg CP (CgCP) gene expression using a simple, methoxy

96 The dependence of TMV, PVX, and TBSV on intact microfilaments for intercel

97 f vein classes in source leaves for entry of TMV, and the lack of equivalence of vein classes in sink

98 e of vein classes in sink leaves for exit of TMV.

99 the deposited model for the helical form of TMV at the lower-radius region (residues 88 to 109).

100 The history of TMV illustrates how pragmatic strategies to control an e

101 verexpressing calreticulin, the inability of TMV MP to reach plasmodesmata substantially impaired cel

102 th wild-type TMV infections: an inability of TMV to support dRNAs that can move in plants and the ina

103 E-binding region resulted in inactivation of TMV cell-to-cell movement.

104 provide evidence that a wild-type isolate of TMV is able to enter C71 cells grown in liquid medium, r

105 eral SIS genes in the uninoculated leaves of TMV-infected NahG plants was delayed and/or reduced, rai

106 the density map to build an atomic model of TMV.

107 Mutants of TMV lacking functional MP accumulated vRNA, but the dist

108 The use of amino acid deletion mutants of TMV MP showed that its domain was necessary and sufficie

109 ich could be correlated with the presence of TMV particles.

110 tein, directed by the subgenomic promoter of TMV coat protein in Av, supported systemic infection wit

111 To improve these properties of TMV vectors, the gene encoding the 30-kDa movement prote

112 The host range of TMV was extended to include spinach as a permissive host

113 Here we report a helical reconstruction of TMV in its calcium-free, metastable assembling state at

114 ction is delayed, and cell-to-cell spread of TMV and CaLCuV movement proteins is inhibited.

115 Previously, the structure of TMV was solved at atomic detail by X-ray fiber diffracti

116 lind Franklin, who had taken up the study of TMV.

117 ere successfully displayed on the surface of TMV, and viruses accumulated to high levels in infected

118 o display foreign peptides on the surface of TMV.

119 ositively charged epitopes on the surface of TMV.

120 ly interfered with plasmodesmal targeting of TMV MP, which, instead, was redirected to the microtubul

121 ne, and inhibits cell-to-cell trafficking of TMV and CaLCuV movement proteins, when tested in an Agro

122 ork, infection induced by RNA transcripts of TMV clones that contain wt CP or mutant CP(T42W) fused t

123 Systemic transport of TMV in these plants, however, was substantially delayed

124 h agrees with available experimental data on TMV reconstruction.

125 but not three other myosins, inhibited only TMV movement.

126 ressing CP(T42W), infection by TMV-CP:GFP or TMV-MP:GFP-CP produced infection sites of smaller size t

127 0.5 D, where it improved in MMV by 19% over TMV.

128 icacy of Tobacco mosaic virus-like particle (TMV VLP) sensing probes using an impedimetric microsenso

129 eover, Arabidopsis PAPK1 also phosphorylates TMV MP in vitro at its C terminus.

130 n nontransgenic and wt CP transgenic plants, TMV-CP:GFP produced expanding, highly fluorescent disk-s

131 -cell tobacco mosaic virus movement protein (TMV MP) mediates viral spread between the host cells thr

132 es of tobacco mosaic virus movement protein (TMV MP); this posttranslational modification has been sh

133 Mice that received TMV-5B19L intranasally developed serum IgG and IgA speci

134 Recombinant TMV functioned as an epitope tag recognizing streptavidi

135 Recombinant TMV was left on the filter.

136 Recombinant TMV-streptavidin complex was dissociated with 0.2M aceti

137 With a similar approach, a recombinant TMV vector carrying a gene for the ectopic expression of

138 The streptavidin-specific recombinant TMV system was applied successfully for purification of

139 streptavidin were incubated with recombinant TMV virions.

140 ncing of NbBES1/BZR1 blocked Bikinin-reduced TMV resistance.

141 We were able to obtain discrete rod-shaped TMV@MOF core-shell hybrids with good uniformity, and the

142 mers to the capsid exterior, organic-soluble TMV rods have been prepared.

143 low sulfate TMVs, while the highest sulfate TMV harboured 16S rRNA phylotypes associated with sulfur

144 Detailed studies demonstrate that TMV multiplies exclusively in the cytoplasm and have doc

145 icating vascular tissues, we determined that TMV could enter minor, major or transport veins directly

146 Combined, these findings indicate that TMV directs the reprogramming of auxin-regulated gene ex

147 These studies show that TMV can be an effective vaccine delivery vehicle for par

148 Combined, these findings suggest that TMV replicase-Aux/IAA interactions selectively enhance v

149 n in the PME antisense plants suggested that TMV systemic movement may be a polar process in which th

150 The TMV coat protein (CP) was fused to TMOF at the C terminu

151 The TMV hybrids were propagated in tobacco plants, and the v

152 The TMV-based vector also enabled C. acutatum to transiently

153 Although promoters containing the TMV Omega sequence exhibited slightly increased basal ex

154 t a regulatory mechanism for controlling the TMV MP-plasmodesmata interactions in a host-dependent fa

155 gA specific for the 5B19 epitope and for the TMV coat protein.

156 For the TMV/BSA mixtures, the BSA adsorbs to the TMV and bridgin

157 promoter-driven TMV replicon that lacked the TMV coat protein gene sequence.

158 mistries have been established to modify the TMV rod, such methods have not yet been described for th

159 m Tobacco mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cells it interacted with

160 Thus, both the structure and assembly of the TMV CP function as determinants in the induction of dise

161 des covering the interacting portions of the TMV helicase domain were expressed and purified.

162 These results suggest that features of the TMV helicase domain, independent of its enzymatic activi

163 hese host genes altered the phenotype of the TMV infection foci and VRCs, yielding foci with concentr

164 Introduction of the TMV Omega sequence into the 5'UTR resulted in a further

165 nted with alterations in at least one of the TMV parameters.

166 Transfer of the TMV Pumilio recognition sequences into the genome of pot

167 evious study indicated that a portion of the TMV replicase containing a putative helicase domain is i

168 The ability of the TMV replicase to interact with Aux/IAA proteins from div

169 pathogens, the helicase domain (p50) of the TMV replicase, the avirulence gene of N, was linked to s

170 ethyltransferase and helicase domains of the TMV replicase.

171 lay of the MHV epitope on the surface of the TMV.

172 Based on the TMV classification, bone turnover was normal in 48%, hig

173 en together, these data demonstrate that the TMV helicase domain interacts with itself to produce hex

174 Da proteins, these findings suggest that the TMV replicase may form a similar oligomer.

175 Combined, these data indicate that the TMV replicase protein interferes with the plant's auxin

176 The authors believe that the TMV-based affinity system can also be used for the purif

177 d/or synthesis inhibitors indicated that the TMV-induced expression of several SIS genes is independe

178 the TMV/BSA mixtures, the BSA adsorbs to the TMV and bridging of the BSA between TMV produces the att

179 ucleus, suggesting that interaction with the TMV replicase protein disrupts PAP1 localization.

180 )], in infected cells it interacted with the TMV RNA to form filamentous ribonucleoprotein (RNP) part

181 odern usage of the word "virus." Since then, TMV has been acknowledged as a preferred didactic model

182 Using this TMV expression vector, some foreign proteins were expres

183 pression studies show that in mature tissues TMV 126/183-kDa-interacting Aux/IAAs predominantly expre

184 In addition to TMV MP, PME is recognized by MPs of turnip vein clearing

185 Heat-transformation can be applied to TMV yielding spherical nanoparticles (SNPs) measuring ~5

186 ignificantly reduced with MMV as compared to TMV only at intermediate object distances, however was u

187 ines exhibit hypersensitive response (HR) to TMV and restrict virus spread to the inoculated site.

188 pleted in InsP6 and were hypersusceptible to TMV, turnip mosaic virus, cucumber mosaic virus and caul

189 assays, treatment with Intrepid-2F prior to TMV infection resulted in high levels of viral resistanc

190 Rar1- like gene for N-mediated resistance to TMV and some powdery mildew resistance genes in barley p

191 Here, we examined resistance to TMV by temporal and quantitative control of TMV Cg CP (C

192 the tobacco N gene can confer resistance to TMV in heterologous N. benthamiana.

193 results in very high levels of resistance to TMV infection in plants containing CP(T42W).

194 NIM1- like genes in N-mediated resistance to TMV using a TRV based VIGS approach.

195 d in attenuation of N-mediated resistance to TMV, indicating that these miRNAs have functional roles

196 K1/NQK1, attenuated N-mediated resistance to TMV.

197 me also compromises N-mediated resistance to TMV.

198 e necessary to confer complete resistance to TMV.

199 transcripts, exhibit complete resistance to TMV.

200 tein, fail to exhibit complete resistance to TMV.

201 ired for N to provide complete resistance to TMV.

202 g transcriptional alterations in response to TMV contain multiple auxin response promoter elements.

203 is transcriptionally induced in response to TMV infection, and its overexpression significantly redu

204 ts undergoing the hypersensitive response to TMV infection.

205 In response to TMV infections, ATAF2 and PR1 display increased transcri

206 ole in the N-mediated resistance response to TMV.

207 ine hepatitis virus (MHV) and giving rise to TMV-5B19 and TMV-5B19L, respectively.

208 InsP6 levels and enhanced susceptibility to TMV and to virulent and avirulent strains of the bacteri

209 -1 plants exhibit enhanced susceptibility to TMV, as well as to virulent pathogens.

210 roperties of the tobacco mosaic tobamovirus (TMV) coat protein (CP) make it possible to display forei

211 Today, TMV is used as a tool to study host-pathogen interaction

212 ' native aberrations and induce traditional (TMV) and modified (MMV) monovision corrections.

213 ain JHM, whereas mice administered wild-type TMV died 10 d post challenge.

214 ccurring dRNAs in association with wild-type TMV infections: an inability of TMV to support dRNAs tha

215 Unlike TMV, the PVX RNA was concentrated in distinctive 'whorls

216 ene N induces a hypersensitive response upon TMV infection and protects tobacco against systemic spre

217 ypersensitive response (HR) lesion size upon TMV infection, as observed in TMV-inoculated N gene-cont

218 helicase domain of the Tobacco mosaic virus (TMV) 126- and 183-kDa replicase proteins was previously

219 helicase domain of the Tobacco mosaic virus (TMV) 126- and/or 183-kDa replicase protein(s) was found

220 helicase domain of the Tobacco mosaic virus (TMV) 126-/183-kDa replicase protein(s) and the Arabidops

221 hese activities is the Tobacco mosaic virus (TMV) 30-kDa protein (MP(TMV)).

222 confers resistance to tobacco mosaic virus (TMV) and encodes a Toll-interleukin-1 receptor/nucleotid

223 confers resistance to Tobacco mosaic virus (TMV) and encodes a toll-interleukin-1 receptor/nucleotid

224 replication protein of tobacco mosaic virus (TMV) and phloem-specific auxin/indole acetic acid (Aux/I

225 to this approach using Tobacco Mosaic Virus (TMV) as a test specimen and obtained a map from 210,000

226 url virus (CaLCuV) and Tobacco mosaic virus (TMV) cell-to-cell movement proteins.

227 ical properties of the Tobacco mosaic virus (TMV) coat protein (CP) are addressed in relation to its

228 Expression of tobacco mosaic virus (TMV) coat protein (CP) in plants confers resistance to i

229 ion of a gene encoding tobacco mosaic virus (TMV) coat protein (CP) in transgenic plants confers resi

230 e replicase protein of Tobacco mosaic virus (TMV) disrupts the localization and stability of interact

231 The genome of tobacco mosaic virus (TMV) encodes replicase protein(s), movement protein (MP)

232 Tobacco mosaic virus (TMV) entry into minor, major and transport veins from no

233 vement protein (MP) of tobacco mosaic virus (TMV) facilitates the cell-to-cell spread of infection by

234 Tobacco mosaic virus (TMV) forms dense cytoplasmic bodies containing replicati

235 ackled the assembly of Tobacco mosaic virus (TMV) from its constituent RNA and protein subunits.

236 ence (called Omega) of tobacco mosaic virus (TMV) functions as a translational enhancer in plants.

237 n vectors based on the tobacco mosaic virus (TMV) genome are powerful tools for foreign gene expressi

238 Tobacco mosaic virus (TMV) has had an illustrious history for more than 100 ye

239 plant defense against Tobacco Mosaic Virus (TMV) in Nicotiana benthamiana.

240 ein was expressed from Tobacco mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cel

241 ory activities against tobacco mosaic virus (TMV) in vivo and in vitro.

242 Tobacco mosaic virus (TMV) induces the hypersensitive response (HR) in tobacco

243 ore and for 3 hr after tobacco mosaic virus (TMV) infection.

244 The tobacco mosaic virus (TMV) movement protein (MP) facilitates transport of viru

245 e protein shell of the tobacco mosaic virus (TMV) provides a robust and practical tubelike scaffold f

246 the helper virus (HV) Tobacco mosaic virus (TMV) replicase.

247 The dominant tobacco mosaic virus (TMV) resistance gene N induces a hypersensitive response

248 lectivity studies with tobacco mosaic virus (TMV) showed an excellent specificity for the targeted TN

249 age of a deconstructed tobacco mosaic virus (TMV) system, where the capsid protein (CP) gene is repla

250 Using tobacco mosaic virus (TMV) tagged with green fluorescent protein to follow inf

251 licase (p50) domain of Tobacco mosaic virus (TMV) through its TIR domain.

252 Cell-to-cell spread of tobacco mosaic virus (TMV) through plant intercellular connections, the plasmo

253 cell-to-cell spread of tobacco mosaic virus (TMV) through plant intercellular connections, the plasmo

254 ze the distribution of tobacco mosaic virus (TMV) viral RNA (vRNA) in infected tobacco protoplasts.

255 of the surface of the tobacco mosaic virus (TMV) virion with a mosquito decapeptide hormone, trypsin

256 irst discovered virus, tobacco mosaic virus (TMV) was at the center of virus research.

257 ast, susceptibility to tobacco mosaic virus (TMV) was not altered in CSN-silenced plants.

258 Recombinant tobacco mosaic virus (TMV) was used as an affinity matrix for isolation and pu

259 Hybrids of tobacco mosaic virus (TMV) were constructed with the use of fusion to the coat

260 ective RNAs (dRNAs) of Tobacco mosaic virus (TMV) were examined in planta with helper viruses that ex

261 e RNA from the helical tobacco mosaic virus (TMV) were extracted using phenol/chloroform.

262 s of the rodlike virus tobacco mosaic virus (TMV) with globular macromolecules such as polyethylene o

263 confers resistance to tobacco mosaic virus (TMV), and leads to induction of standard defense and res

264 bust protein template, tobacco mosaic virus (TMV), can be used to regulate the size and shape of as-f

265 is-like plant viruses, Tobacco mosaic virus (TMV), Cucumber mosaic virus (CMV), and Cowpea chlorotic

266 ll-to-cell movement of tobacco mosaic virus (TMV), is also required for the systemic spread of viral

267 ses is highlighted for tobacco mosaic virus (TMV), M13 bacteriophage, cowpea chlorotic mottle virus (

268 of known MPL, such as Tobacco Mosaic Virus (TMV), MPL of the fibrils in question can be determined.

269 from different genera [tobacco mosaic virus (TMV), potato virus X (PVX), tomato bushy stunt virus (TB

270 e (PAL) reduces SAR to tobacco mosaic virus (TMV), whereas overexpression of PAL enhances SAR.

271 Infection with Tobacco mosaic virus (TMV), which is known to activate the SIPK cascade and in

272 Tobacco mosaic virus (TMV)-based transient expression vectors can express very

273 protein of AlMV into a tobacco mosaic virus (TMV)-based vector Av.

274 A tobacco mosaic virus (TMV)-based vector carrying such inverted-repeats, homolo

275 ing SAR development in tobacco mosaic virus (TMV)-infected tobacco and Pseudomonas syringae-infected

276 leaves inoculated with tobacco mosaic virus (TMV).

277 confers resistance to tobacco mosaic virus (TMV).

278 nts after infection by tobacco mosaic virus (TMV).

279 resisting infection by tobacco mosaic virus (TMV).

280 confers resistance to tobacco mosaic virus (TMV).

281 llowing infection with tobacco mosaic virus (TMV).

282 ple viruses, including tobacco mosaic virus (TMV).

283 ed on the plant virus, tobacco mosaic virus (TMV).

284 is work focuses on the tobacco mosaic virus (TMV).

285 confers resistance to tobacco mosaic virus (TMV).

286 in the genomic RNA of tobacco mosaic virus (TMV).

287 the virulent pathogen tobacco mosaic virus (TMV).

288 that of wild-type CP, the resulting viruses TMV-RB19E and TMV-4D:31D infected N. tabacum Xanthi-nn p

289 The molecular pathway by which TMV MP interacts with the host cell is largely unknown.

290 llular surface for TMI, II, IV and VI, while TMV seems to be minimally involved in the ligand selecti

291 a benthamiana plants prior to challenge with TMV expressing green fluorescent protein.

292 ns was 3.5 +/- 0.5 D (mean) as compared with TMV (2.7 +/- 0.3 D).

293 1.0 D) than with negative SA, compared with TMV.

294 act with TMV MP, none of them coresides with TMV MP within plasmodesmata.

295 ions were isolated from plants infected with TMV(ORF3) or with GRV itself.

296 he effect in NN tobacco plants infected with TMV.

297 ost factors have been shown to interact with TMV MP, none of them coresides with TMV MP within plasmo

298 otiana glutinosa and Chenopodium quinoa with TMV).

299 eptible host systems (Nicotiana tabacum with TMV (Tobacco mosaic virus), and Arabidopsis thaliana wit

300 ble from that of the wild-type N plants, yet TMV was able to move systemically, causing a systemic hy