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1 level of cIAP1 and, in some cases, cIAP2 and TNF receptor-associated factor 2.
2 rs or dominant-negative forms of p38 MAPK or TNF receptor-associated factor 2.
3 by TNF receptor-associated death domain and TNF receptor-associated factor-2.
4 am of receptor-interacting protein (RIP) and TNF receptor- associated factor 2.
5 , TNF receptor 2 (TNF-R2) interacts with the TNF receptor associated factors 2/1 (TRAF2/TRAF1) hetero
6 at beta-arrestin-1 is associated with TRAF2 (TNF receptor-associated factor 2), an adaptor protein of
7 B-inducing kinase-IKK was interrupted at the TNF receptor associated factor 2 and NF-kappa B-inducing
8 cells, along with the selective reduction of TNF receptor-associated factor 2 and impairment in the a
9 e/IkappaBalpha kinase was interrupted at the TNF receptor-associated factor 2 and NF-kappaB-inducing
11 cytoplasmic domain of CD40, while binding of TNF receptor-associated factors 2 and 3 is dispensable,
12 ter gene expression activated by TNF, TNFR1, TNF receptor-associated factor 2, and NF-kappaB-inducing
13 paB regulator, TRAF2 (tumor necrosis factor (TNF) receptor-associated factor 2), as an oncogene that
15 hibitor of apoptosis-1), cIAP-2, and TRAF-2 (TNF receptor-associated factor-2)) in an NF-kappaB-depen
16 d IL-6, and the cytoplasmic adaptor molecule TNF receptor-associated factor 2 is involved in both R-8
17 eptor 1-TNF receptor associated death domain-TNF receptor associated factor 2 NF-kappa B-inducing kin
18 eptor, TNF receptor-associated death domain, TNF receptor-associated factor 2, NF-kappa B-inducing ki
19 e activated by receptor-interacting protein, TNF receptor-associated factor 2, NF-kappaB-inducing kin
20 tor 1, TNF receptor-associated death domain, TNF receptor-associated factor-2, NF-kappaB-inducing kin
21 tor 1, TNF receptor-associated death domain, TNF receptor-associated factor-2, NF-kappaB-inducing kin
22 These data suggest that TNFR1, through a TNF receptor-associated factor 2-NF-kappaB signaling pat
23 eptor 1/TNF receptor-associated death domain/TNF receptor-associated factor 2/NF-kappaB-inducing kina
24 osis protein 1, Bcl-2, Bcl-xL, survivin, and TNF receptor-associated factor 2), proliferation (cyclin
25 Tumor necrosis factor alpha (TNF-alpha) and TNF receptor-associated factor 2 protein were moderately
26 both knockdowns reduce expression of TRAF2 (TNF receptor-associated factor-2) protein, and small int
27 IKK-beta, TNF receptor-associated factor 6, TNF receptor-associated factor 2, receptor-interacting p
28 ulin were related to increased expression of TNF receptor-associated factor 2, the product of an NF-k
30 RADD (TNF receptor-associated death domain), TNF receptor-associated factor-2 (TRAF-2), the Ser/Thr k
31 ptor proteins, including the docking protein TNF receptor-associated factor-2 (TRAF-2), which is beli
33 ding of CD154 (gp39, CD40L) to CD40 recruits TNF receptor- associated factor 2 (TRAF2) and TRAF3 to t
34 induces IRAK1 sumoylation in the presence of TNF receptor-associated factor 2 (TRAF2) and intracellul
35 B and JNK activity associated with increased TNF receptor-associated factor 2 (TRAF2) and NF-kappaB e
38 of the Fn14-TRAF domain site or depletion of TNF receptor-associated factor 2 (TRAF2) expression by s
40 In addition, we also demonstrated that the TNF receptor-associated factor 2 (TRAF2) plays little ro
44 we discovered that GAPDH interacts with the TNF receptor-associated factor 2 (TRAF2), a protein requ
45 and CD160 engagement induced recruitment of TNF receptor-associated factor 2 (TRAF2), but not TRAF3,
46 ptosis signal-regulating kinase 1 (ASK1) and TNF receptor-associated factor 2 (TRAF2), regulation of
47 aling by TRANCE-R appears to be dependent on TNF receptor-associated factor 2 (TRAF2), since JNK indu
48 aprolin (TTP) is Lys-63-polyubiquitinated by TNF receptor-associated factor 2 (TRAF2), suggesting a r
49 wn to be important for CD40 association with TNF receptor-associated factor 2 (TRAF2), TRAF3, and TRA
65 (E2) Ubc13 for binding to the RING domain of TNF-receptor associated factor 2 (TRAF2), thereby inhibi
69 IP (receptor-interacting protein), or TRAF2 (TNF receptor-associated factor-2) were caused by apoptog
70 F-alpha induced the ubiquitination of TRAF2 (TNF receptor-associated factor 2), which interacts with
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