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1  allow cleavage by the metalloprotease TACE (TNF-alpha-converting enzyme).
2 ncreases the activity of the metalloprotease TNF-alpha-converting enzyme.
3 ed shedding, but shedding was independent of TNF-alpha-converting enzyme (a disintegrin and metallopr
4 tivation of the metalloprotease TACE/ADAM17 (TNF-alpha-converting enzyme), a previously unknown p53 t
5                                This enhanced TNF-alpha-converting enzyme activity significantly incre
6               The latter enzymes, as well as TNF alpha-converting enzyme and FasL-converting enzyme,
7 n CMV due to an enhanced activity of ADAM17 (TNF-alpha converting enzyme) and matrix metalloprotease
8 phil elastase, tumor necrosis factor [TNF]), TNF-alpha-converting enzyme, and matrix metalloproteinas
9 pparent deficiency in the level of the major TNF-alpha converting enzyme in adipose tissue to account
10                      This enzyme (called the TNF-alpha-converting enzyme, or TACE) is a new member of
11 ages in a manner requiring activation of the TNF-alpha-converting enzyme TACE.
12                                              TNF alpha converting enzyme (TACE) processes precursor T
13                   The tumor necrosis factor (TNF)-alpha converting enzyme (TACE/ADAM-17) is a transme
14                         Here, we report that TNF-alpha converting enzyme (TACE) (a disintegrin and me
15                                    Using the TNF-alpha converting enzyme (TACE) inhibitor, TAPI-1, TP
16 pyl dicarboxylate were examined as potential TNF-alpha converting enzyme (TACE) inhibitors.
17                                              TNF-alpha converting enzyme (TACE) is a membrane-bound m
18                                              TNF-alpha converting enzyme (TACE) is a multidomain, mem
19                                              TNF-alpha converting enzyme (TACE) is the protease respo
20                                              TNF-alpha converting enzyme (TACE) or disintegrin and me
21                  Endothelial deletion of the TNF-alpha converting enzyme (TACE) prevented the TNF-alp
22                         sTNF is generated by TNF-alpha converting enzyme (TACE) proteolytic release o
23 TNF-alpha have centered on the inhibition of TNF-alpha converting enzyme (TACE) through the use of hy
24 ane protein that is enzymatically cleaved by TNF-alpha converting enzyme (TACE) to generate a 17-kDa
25 ct with epithelial cells, and by 1 h induced TNF-alpha converting enzyme (TACE) transcription.
26                                          The TNF-alpha converting enzyme (TACE), a disintegrin and me
27    In the present study, we demonstrate that TNF-alpha converting enzyme (TACE), a disintegrin and me
28 esothelin sheddase activity is mediated by a TNF-alpha converting enzyme (TACE), a member of the matr
29 ly in fibroblasts and monocytes deficient in TNF-alpha converting enzyme (TACE), a sheddase involved
30 F-related apoptosis-inducing ligand (TRAIL), TNF-alpha converting enzyme (TACE), TRAIL receptor (TRAI
31 (solTNF) molecule after being cleaved by the TNF-alpha converting enzyme (TACE).
32 yzed for expression of AR and its activator, TNF-alpha converting enzyme (TACE).
33                                          The TNF-alpha converting enzyme (TACE; a disintegrin and met
34                                              TNF-alpha converting enzyme (TACE; ADAM17), a member of
35                 Tumor necrosis factor alpha (TNF-alpha) converting enzyme (TACE) is the first discove
36 ase (MAPK) and tumour necrosis factor-alpha (TNF-alpha) converting enzyme (TACE) were also assessed.
37 llagenase-1) or tumor necrosis factor-alpha (TNF-alpha) converting enzyme (TACE).
38 hown to involve tumor necrosis factor alpha (TNF-alpha) converting enzyme (TACE).
39                 Tumor necrosis factor alpha (TNF-alpha)-converting enzyme (TACE) is at the center of
40 uction, which corresponded to an increase in TNF-alpha-converting enzyme (TACE) activity.
41                                              TNF-alpha-converting enzyme (TACE) cleaves pro-TGF-alpha
42                         Shedding mediated by TNF-alpha-converting enzyme (TACE) results in an unattac
43               Metalloprotease inhibitors and TNF-alpha-converting enzyme (TACE) small interfering RNA
44 rophages, by suppressing the activity of the TNF-alpha-converting enzyme (TACE), arguing that MUC1 is
45 lecular mass 26K which can be processed by a TNF-alpha-converting enzyme (TACE), to generate secreted
46 thelial (NCI-H292) cells via metalloprotease TNF-alpha-converting enzyme (TACE)-dependent EGFR activa
47  mucus, and its expression is modulated by a TNF-alpha-converting enzyme (TACE)-EGF receptor pathway
48 ionally by inhibiting cellular processing by TNF-alpha-converting enzyme (TACE).
49                                              TNF-alpha-converting enzyme (TACE, herein denoted as Ada
50                     Targeting this protease, TNF-alpha-converting enzyme (TACE; also referred to as a
51                                     ADAM 17 (TNF-alpha converting enzyme, TACE) is a potential target
52 vation of the tmTNF cleavage metalloprotease TNF-alpha-converting enzyme via p38 MAP kinase activatio

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