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1 TNF beta expression correlated significantly with the oc
2 TNF-beta polymorphisms within the donor liver did not co
3 TNF-beta that is encoded by lymphotoxin-alpha gene (LTA)
5 nes tumor necrosis factor alpha (TNF-alpha), TNF-beta, and interleukin-1 alpha and chemokines macroph
7 thway that is independent of Fas, TNF alpha, TNF beta, and ATP, but is dependent upon extracellular c
9 re analyzed for the expression of TNF alpha, TNF beta, and their receptors (p55 and p75 TNFR), utiliz
10 we suggest that lymphotoxin's (LT, LT-alpha, TNF-beta) crucial role in these processes is pivotal and
11 IL-1beta, tumor necrosis factor (TNF) alpha, TNF-beta, and IL-6 and in the IL-1 receptor antagonist (
12 ective in tumor necrosis factor (TNF)-alpha, TNF-beta, lymphotoxin-beta, or TNFR1, TNFR2, Fas, or dea
13 dd, fas, fas ligand (fas L), ice, TNF-alpha, TNF-beta, TNFR1, TNFR2, TRAF1, TRAF2, TRAF3, cIAP2, and
14 decreased activity of this Fas-, TNF alpha-, TNF beta-, ATP-independent pathway associated with very
17 mphokines: interferon-gamma, IL-2, IL-4, and TNF-beta) in tissues obtained at autopsy from subjects w
18 rease of proinflammatory cytokines, IL-6 and TNF-beta, indicating that exosomes containing TAR RNA co
19 n was also achieved when IL-1beta, IL-6, and TNF-beta were added to macrophages with cross-linked cos
20 yet it elicited production of TNF-alpha and TNF-beta, and induced TCR Vbeta-specific proliferation s
21 , which contains the genes for TNF-alpha and TNF-beta, is located on chromosome 6 within the major hi
23 Tumor necrosis factor alpha (TNF-alpha) and TNF-beta mRNAs were upregulated in advanced stages of di
24 tumor necrosis factor alpha (TNF-alpha) and TNF-beta, which is consistent with functional stimulatio
26 ses the release of IFN-gamma, TNF-alpha, and TNF-beta through direct binding to multiple conserved re
27 te-macrophage-CSF, IFN-gamma, TNF-alpha, and TNF-beta) release by both TALL-104 and LAK cells, ligati
28 eta (IL-1 beta), tumor necrosis factor-beta (TNF-beta), or interleukin-6 (IL-6), messenger RNA was el
29 1-like cells, but the cytokine modulation by TNF-beta and the clinical significance of this cytokine
32 ker (MSM) upstream of the TNFB gene encoding TNF-beta and at a restriction fragment length polymorphi
36 We have examined the ability of lymphotoxin (TNF-beta), to stimulate rolling, adhesion, and transmigr
48 the crystal structures of TNF alpha and the TNF beta/TNF-receptor(I) complex and a model of an anti-
51 pha gene construct, which contains the whole TNF-beta gene with 1.2 kb of 5' flanking sequence, 1.1 k
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