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1 levels of PAI-1 antigen, PAI-1 activity, and TPA antigen.
2 ivity are associated with increases in total TPA antigen.
3 plate assay), tissue plasminogen activator (tPA) antigen (Ag) and activity (Act), plasminogen activa
4 lues were 9.0 versus 7.4 ng/mL (P<0.001) for tPA antigen and 27.6 versus 23.4 ng/mL (P=0.001) for D-d
5 es in blood flow and local release of plasma tPA antigen and activity (P<0.001 for all) but had no ef
6 de (P<0.001), although coronary sinus plasma tPA antigen and activity concentrations increased only d
8 , effectively slowing the clearance of total TPA antigen and explaining in part why high levels of PA
9 n stratified by HT, the relationship between tPA antigen and incident coronary heart disease was simi
12 tigen and tissue-type plasminogen activator (tPA) antigen and activity in 31 patients taking an ACE i
13 ps of elevated tissue plasminogen activator (tPA) antigen and D-dimer with subsequent first coronary
15 ith glucose intolerance, levels of PAI-1 and tPA antigen in men and women (P<.01 for trend) and vWF a
16 n forearm blood flow (P=0.03) and release of tPA antigen (P=0.04) and activity (P<0.001) caused by su
17 activity: tissue-type plasminogen activator (TPA) antigen, plasminogen activator inhibitor-1 (PAI-1)
20 iod followed by measurement of TPA activity, TPA antigen, TPA/PAI-1, TPA/C1 inhibitor, PAI-1 activity
21 tio in the highest versus lowest quartile of tPA antigen was 3.5 (95% CI, 2.1 to 5.8; P trend <0.001)
23 rance of total tissue plasminogen activator (TPA) antigen, we studied the clearance of active TPA and
24 ltivariate-adjusted geometric mean values of TPA antigen were 6.3 and 7.3 ng/mL among current users a
26 -1 antigen and tissue plasminogen activator (TPA) antigen were measured before randomization and on d
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