ライフサイエンスコーパス: TTF

1 TTF and OS were comparable in both arms.

2 TTF was independently associated with age, beta-2M, perc

3 TTF was longer in the GD arm.

4 TTF-1 dose-dependently activated alpha(7) transcription

5 TTF-1 is amplified in lung cancers, suggesting that it i

6 TTF-1 knockdown and overexpression studies showed that T

7 ification of thyroid transcription factor 1 (TTF-1 or NKX2-1) biochemical activity as a transcription

8 hat PPFP and thyroid transcription factor 1 (TTF-1) physically interact, and that these transcription

9 d binding of thyroid transcription factor 1 (TTF-1) to an upstream response element (TTF-1-binding el

10 dependent on thyroid transcription factor 1 (TTF-1).

11 Thyroid transcription factor 1 (TTF-1/Nkx-2.1) plays a critical role in lung morphogenes

12 f endogenous thyroid transcription factor 1 (TTF-1/Nkx2.1) to the miR-29ab1 promoter in HFL type II c

13 Thyroid transcription factor-1 (TTF-1/Nkx-2.1) is required for formation of the lung and

14 A binding of thyroid transcription factor-1 (TTF-1/Nkx2.1), a master regulator of lung development.

15 ,5-dioxynaphthalene (DNP) unit, as well as a TTF unit, encircled by a CBPQT(4+) ring.

16 , that contain a BIPY(2+) unit with either a TTF or DNP unit, is investigated.

17 The first example of a TTF-based self-assembled cage has been produced from thi

18 ed significantly less often in grafts with a TTF with low flow (259 of 363 [71.3%]) than in those wit

19 New electron-donor (D)-electron-acceptor (A) TTF architectures are presented in which two electron-do

20 icenter [TTF](*+)...[TTF](*+) bonds when all TTF groups host a 1+ au of charge, as in clip2(4+).

21 Although TTF-1 is amplified in primary human lung cancers, it inh

22 However, in multivariate analysis, TTF was not an independent risk factor for VA <0.3 (odds

23 nding between surface-adsorbed CBPQT(4+) and TTF.

24 miR-29ab1 promoter in HFL type II cells, and TTF-1 increased miR-29ab1 promoter-driven luciferase act

25 n of the CBPQT(4+) rings between the DNP and TTF recognition sites in the GSCC.

26 Protein-protein interactions between Erm and TTF-1 were demonstrated by mammalian two-hybrid assays a

27 NFATc3 and TTF-1 activated the Sftpd promoter, synergized transcrip

28 OS and TTF were not affected by the occurrence of irAEs or the

29 as independent prognostic factors for OS and TTF.

30 roid-specific transcription factors Pax8 and TTF-1, leading to expression of the thyroid-specific tar

31 P-ribose) polymerases (PARP-2 and PARP-1) as TTF-1 interacting proteins that influence its transcript

32 factor NK2 homeobox 1 (NKX2-1; also known as TTF-1).

33 e findings identify miR-29 family members as TTF-1-driven mediators of SP-A expression and type II ce

34 triangular magnetic lattice of spin-1/2 BEDT-TTF dimers that provides a prime example of a spin liqui

35 etween the n-orbitals of the S atoms in BEDT-TTF of the BEDT-TTF/C60 layer and the pi* orbitals of C

36 The layered molecular system kappa-(BEDT-TTF)(2)Cu(2)(CN)(3) is a Mott insulator with an almost i

37 organic molecular metals in the kappa-(BEDT-TTF)(2)X family, we reveal how the Nernst effect, a sens

38 The Mott-insulating state in the kappa-(BEDT-TTF)2X organic molecular metals can be tuned, without do

39 ng in a centimeter-sized free-standing (BEDT-TTF)C60 charge-transfer single crystal is demonstrated.

40 bis(ethylenedithio)tetrathiafulvalene (BEDT-TTF)/C60 and poly(3-dodecylthiophene-2,5-diyl) (P3DDT)/C

41 itals of the S atoms in BEDT-TTF of the BEDT-TTF/C60 layer and the pi* orbitals of C atoms in C60 of

42 EGJ cancer demonstrated significantly better TTF than did ECX.

43 ns, of dimers of bis-tetrathiafulvalene (bis-TTF)-functionalized diphenylglycoluril molecular clips (

44 a 1:1 stoichiometry between the anion-bound TTF-C4Ps and the complexed fullerenes.

45 ports the first miRNAs directly regulated by TTF-1 and clarifies how TTF-1 controls HMGA2 expression.

46 s revealed a high affinity of the calixarene-TTF receptors for planar electron-deficient guests, lead

47 In lung cancers, TTF-1 displays seemingly paradoxical activities.

48 g mixed-valence (TTF)2*+ and radical-cation (TTF*+)2 states inside the 'molecular flasks.' The experi

49 in the two catenanes, the radical cationic (TTF(*+))2 dimer in the [2]catenane occurs only fleetingl

50 ring system to either its radical cationic (TTF*+) or dicationic (TTF2+) counterparts, whereupon the

51 tacts, both involving the positively charged TTF group of one monomer and the negatively charged cent

52 On charging, TTF is oxidized to TTF(+) at the cathode surface; TTF(+)

53 e of the resulting supramolecular complexes (TTF-C4P + fullerene + halide anion + tetraalkylammonium

54 d SUMOylated BEND3 stabilizes NoRC component TTF-1-interacting protein 5 via association with ubiquit

55 e does topology play in catenanes containing TTF units?

56 Conversely, TTF-1 counters epithelial to mesenchymal transition in c

57 patient characteristics associated with CR, TTF, and OS establishes a baseline to compare and incorp

58 that the ability of pioglitazone to decrease TTF-1 expression contributes to its pro-adipogenic actio

59 Defining TTF as either starting a new treatment or death, estimat

60 If these newly designed TTF- and PMDI-based crystals with high polarizations are

61 ssociation of TTF+* to form the diamagnetic [TTF+,TTF+] dication and to also undergo the equally rapi

62 states of the two constitutionally different TTF units in the [2]catenane still experience long-range

63 formation between constitutionally different TTF units.

64 encapsulates the better pi-electron-donating TTF station.

65 ilicene with either a strong electron donor (TTF) or a strong electron acceptor (TCNQ) and demonstrat

66 Dithiophene-tetrathiafulvalene (DT-TTF) derivatives can be readily prepared through trialky

67 f TTF-1(+) lung cancer cells (designated EDM-TTF-1(+)) displayed an anti-angiogenic activity in the e

68 stimulating factor (GM-CSF) level in the EDM-TTF-1(+) conferred the antiangiogenic activities.

69 ecular slabs of (EDT-TTF-CONH(2))(2)(+) (EDT-TTF = ethylenedithiotetrathiafulvalene) and anionic [BAB

70 yl-ethylenedithio-tetrathiafulvalene (DM-EDT-TTF) 1 donors are synthesized by cross coupling followed

71 th highly conducting molecular slabs of (EDT-TTF-CONH(2))(2)(+) (EDT-TTF = ethylenedithiotetrathiaful

72 ithin the highly conducting crystals of (EDT-TTF-CONH(2))(2)(+)[BABCO(-)] are essentially "braked" at

73 r 1 (TTF-1) to an upstream response element (TTF-1-binding element [TBE]).

74 rahigh local concentration for the encircled TTF units to form stable dimers associated with their di

75 II cells decreased DNA binding of endogenous TTF-1, blocked cAMP stimulation of surfactant proteins,

76 activity but that Erm significantly enhanced TTF-1-mediated SP-C transcription.

77 hermore, in cells that lack TTF-1, exogenous TTF-1 expression dampened the inhibitory effect of TGF-b

78 the essential pulmonary transcription factor TTF-1 and suppressed by Egr-1 during pulmonary developme

79 lymerase I transcription termination factor (TTF-I), it has been speculated that the p19Arf-Mdm2-p53

80 ng epithelial (thyroid transcription factor (TTF)-1 and pro-surfactant protein-B (pro-SP-B), and mese

81 of lung developmental transcription factors (TTF-1, NKX2-8, and PAX9) that we recently discovered as

82 ly, prediction of time to treatment failure (TTF) and overall survival (OS) in mantle cell lymphoma (

83 AGC with a better time-to treatment failure (TTF) compared to ECX, ECX arm (ECX followed by FOLFIRI)

84 ent in the median time to treatment failure (TTF) compared with that reported for six cycles of R-CHO

85 ary criterion was time-to-treatment failure (TTF) of the first-line therapy.

86 se incidence, and time to treatment failure (TTF) were examined by race.

87 se duration (RD), time to treatment failure (TTF), clinical benefit rate (CBR), and safety.

88 primary end point-time to treatment failure (TTF), which included patients without a response after f

89 of the study was time to treatment failure (TTF).

90 urvival (OS), and time to treatment failure (TTF).

91 survival (OS) and time to treatment failure (TTF).

92 survival (OS) and time to treatment failure (TTF).

93 Time to treatment failure (TTF; defined as discontinuation, progressive disease, de

94 erexpression in murine tail tip fibroblasts (TTFs) and cardiac fibroblasts (CFs) from multiple lines

95 iPSCs from adult mouse tail-tip fibroblasts (TTFs) using retroviral vectors or virus-free piggyBac tr

96 Finally, TTF-1 knockdown conferred human lung cancer cells resist

97 Transit time flow (TTF) probes may be useful for predicting long-term graft

98 index (hazard ratio [HR], 2.0; P = .0054 for TTF, and HR, 2.1; P = .068 for OS).

99 08) for TTP, 0.99 (95% CI, 0.92 to 1.06) for TTF, and 0.98 (95% CI, 0.87 to 1.11) for OS.

100 MIPI was similarly prognostic for TTF.

101 ated in order to study their ability to form TTF radical dimers.

102 one van der Waals interaction when the four TTF groups host a 1+ charge, and (4) the net stabilizing

103 = 7.6E-08); a block of 23 SNPs in XPA/FOXE1 (TTF-2) associated with serum TSH (p = 5.5E-08 to 1.0E-09

104 er studies (MTNR1B, ZNF259/APOA5, XPA/FOXE1 (TTF-2), DARC, CCR3, ABO); 2) localized novel genes in pl

105 The thyroid transcription factor 1 gene (TTF-1 or NKX2-1) is essential to lung development; howev

106 c absorption studies suggest that they have [TTF](l+)...[TTF](m+) interactions that are preserved at

107 irectly regulated by TTF-1 and clarifies how TTF-1 controls HMGA2 expression.

108 readily obtainable dibromo-functionalized IF-TTF building block using palladium-catalyzed cross-coupl

109 enofluorene-extended tetrathiafulvalenes (IF-TTFs) with thioacetate end groups was prepared from a re

110 Furthermore, alpha(7) was not detected in TTF-1-null mice and markedly increased in TTF-1-overexpr

111 No significant differences in TTF or overall response to therapy were seen.

112 treatment, but experience no differences in TTF or overall response to therapy.

113 in TTF-1-null mice and markedly increased in TTF-1-overexpressing mice.

114 that the induction of GMT overexpression in TTFs and CFs is inefficient at inducing molecular and el

115 Including TTF in the assessment of risk for glaucoma blindness did

116 miR-200 antagonists inhibited TTF-1 and surfactant proteins and up-regulated TGF-beta2

117 in all cases, has three short interfragment [TTF](l+)...[TTF](m+) interactions.

118 Intraoperative TTF probe data may be helpful in predicting long-term pa

119 studies suggest that they have [TTF](l+)...[TTF](m+) interactions that are preserved at room tempera

120 , has three short interfragment [TTF](l+)...[TTF](m+) interactions.

121 Furthermore, in cells that lack TTF-1, exogenous TTF-1 expression dampened the inhibitor

122 t treatment end point correlated with longer TTF and OS.

123 We measured TTF and/or PI in 2738 grafts, and 1-year patency was det

124 Median TTF was similar at 9.24 months (ATA + TOR) versus 10.44

125 g a new treatment or death, estimated median TTF was 5.7 months.

126 fter a median follow-up of 31 months, median TTF was significantly longer with FOLFIRI than with ECX

127 a median follow-up of 72 months, the median TTF was 34.2 months.

128 ation of a maximum of two long, multicenter [TTF](*+)...[TTF](*+) bonds when all TTF groups host a 1+

129 the VEGF promoter element contains multiple TTF-1-responsive sequences.

130 ive electron-rich recognition units, namely, TTF and DNP, was investigated by electrochemistry and sp

131 est S...S interaction defined by neighboring TTF cores, which inversely correlates with the ionic rad

132 g that conjugation is disrupted by a neutral TTF unit.

133 f the TTF*+ or TTF2+ back to being a neutral TTF.

134 g that involves the oxidation of the neutral TTF ring system to either its radical cationic (TTF*+) o

135 the oxyphenylene station back to the neutral TTF station was slowed considerably by the longer linker

136 ield-switched low-symmetry structures of new TTF- and PMDI-based crystals.

137 ses epigenetic changes that permit access of TTF-1 and NF-kappaB to the SP-A promoter.

138 w miRNAs influence the oncogenic activity of TTF-1 and the role of TTF-1 in cholesterol metabolism.

139 The activity of TTF-1 is influenced by its interactions with other trans

140 stablish the spontaneous self-association of TTF+* to form the diamagnetic [TTF+,TTF+] dication and t

141 occludin, which represents another avenue of TTF-1-mediated metastasis suppression.

142 to the metastasis-critical signaling axis of TTF-1 to HMGA2, we used both reverse and forward strateg

143 The electrocatalytic behavior of TTF-TCNQ-ionic liquid gels toward oxidation of thiocholi

144 dent and is mediated by increased binding of TTF-1/Nkx2.1 and NF-kappaB to a critical response elemen

145 the cohort of patients with coactivation of TTF-1 and NKX2-8 pathways appears to be resistant to sta

146 or prognosis associated with coactivation of TTF-1 and NKX2-8 was validated in 2 other independent cl

147 RP-2 interacted via the C-terminal domain of TTF-1.

148 at TAZ binds to the NH(2)-terminal domain of TTF-1.

149 Surprisingly, the EDM of TTF-1(+) lung cancer cells (designated EDM-TTF-1(+)) dis

150 In human lung cancer, the expression of TTF-1 and GM-CSF exhibits a statistically significant an

151 -like morphology and decreased expression of TTF-1, aquaporin-5 (AQP5), zonula occludens-1 (ZO-1), an

152 ocked [2]catenanes controls the formation of TTF radical dimers within their structural frameworks, i

153 The frequency of TTF increased with stage of glaucomatous loss: 28.3% in

154 GA2) mediates the antimetastatic function of TTF-1.

155 d the combined N terminus and homeodomain of TTF-1 were critical for this interaction.

156 The relative influence of TTF on the risk of blindness in the 2 matched groups was

157 ion occurred through a direct interaction of TTF-1 with the OCLN and CLDN1 promoters.

158 gh TP53 expression was a strong predictor of TTF and inferior OS compared with low TP53 expression in

159 was associated with increased recruitment of TTF-1, NF-kappaB, PCAF, and CBP, as well as enhanced ace

160 cAMP and IL-1 stimulated the recruitment of TTF-1, p65, CBP, and steroid receptor coactivator 1 to t

161 Reversible oxidation and reduction of TTF moieties changes the linker flexibility, which in tu

162 Li2O2, which results in the regeneration of TTF.

163 -33a) is under direct positive regulation of TTF-1.

164 be under a direct and positive regulation of TTF-1.

165 oncogenic activity of TTF-1 and the role of TTF-1 in cholesterol metabolism.

166 The absorption spectra of TTF-OXD hybrids 10d and 11 are blue-shifted by 80 nm com

167 bypass grafting (CABG); however, studies of TTF probe use are limited.

168 Use of TTF probes to assess graft flow and pulsatility index (P

169 Through both gain- and loss-of-TTF-1 expression strategies, we show that TTF-1 positive

170 traoperative revision were compared based on TTF measurements (<20 [low flow] vs >/=20 mL/min [normal

171 ed for detection of carbamate drugs based on TTF-TCNQ-ionic liquid gel thiocholine sensor.

172 bital energy levels of the station it is on (TTF or DNP).

173 n-radical is formed after the initial HQ- or TTF-mediated electron transfer to the metallic carbene c

174 nd that such treatment does not affect OS or TTF.

175 CBPQT(4+) ring away from the singly oxidized TTF(+) unit by overcoming one of the thiomethyl (SMe) sp

176 has obvious correspondence with the oxidized TTF(+) distributions in the electric fields of the charg

177 ining 1.1 kb of the mouse alpha(7) promoter, TTF-1, and Egr-1.

178 te) to a tetrathiafulvalene calix[4]pyrrole (TTF-C4P) donor enforces a host conformation that favors

179 C70) by tetrathiafulvalene-calix[4]pyrrole (TTF-C4P) receptors and the nature of the resulting supra

180 The unifying theory regarding TTF-1 is that it exhibits both pro-oncogenic and anti-me

181 Surprisingly, pioglitazone repressed TTF-1 expression in PPFP-expressing thyrocytes.

182 zed molecular structure and an electron-rich TTF moiety.

183 n potential of the small, structurally rigid TTF-AB macrocycle is found to depend on the conformation

184 onverted to the unusual cation-radical salt, TTF+* CB- (where CB- is the non-coordinating closo-dodec

185 The separate TTF units in the two {1+1} macrocycles (each containing

186 (>50% positive lymphoma cells) had a shorter TTF and poor OS independent of both MIPI score and Ki-67

187 g adenocarcinomas, we observed that silenced TTF-1 expression down-regulated occludin, which we suppo

188 ) transcription in vitro by binding specific TTF-1 regulatory elements in the mouse alpha(7) promoter

189 Centimeter-sized segregated stacking TTF-C60 single crystals are crystallized by a mass-trans

190 s of the P-C60 dyad unit and the two-station TTF-DNP-based [2]rotaxane separately, using conventional

191 y combining the states of separate stations (TTF and DNP) with or without the (CBPQT)(PF(6))(4) shutt

192 s oxidized to TTF(+) at the cathode surface; TTF(+) in turn oxidizes the solid Li2O2, which results i

193 Tetrathiafulvalene (TTF) as the prototypical electron donor for solid-state

194 )) mobile ring between a tetrathiafulvalene (TTF) station and an oxyphenylene station, were synthesiz

195 as the ring), between a tetrathiafulvalene (TTF) unit and a 1,5-dioxynaphthalene (DNP) ring system l

196 y that consists of (i) a tetrathiafulvalene (TTF) unit as an efficient pi-electron-donor station, (ii

197 angement of redox-active tetrathiafulvalene (TTF) arms, which serve as the guest binding centers.

198 ontaining a redox-active tetrathiafulvalene (TTF) ring system within its rod section has been synthes

199 eneethynylene) (OPE) and tetrathiafulvalene (TTF) scaffolds, end-capped with acetyl-protected thiolat

200 oxynaphthalene (DNP) and tetrathiafulvalene (TTF) units along with a 4,4'-bipyridinium (BIPY(*+)) rad

201 thylferrocene (DMFc) and tetrathiafulvalene (TTF).

202 electron donors such as tetrathiafulvalene (TTF).

203 Macrocyclization between tetrathiafulvalene (TTF) dithiolates and bis-bromomethylazobenzenes/bis-brom

204 g a dumbbell, containing tetrathiafulvalene (TTF) and 1,5-dioxynaphthalene (DNP) recognition units wh

205 active stalks containing tetrathiafulvalene (TTF) units, (b) [2]pseudorotaxanes formed between these

206 ilized, redox-controlled tetrathiafulvalene (TTF) dimers, enabling their spectrophotometric and struc

207 (n)-OXD (n = 1) [Donor = tetrathiafulvalene (TTF), bithiophene, 9-(4,5-dimethyl-1,3-dithiol-2-ylidene

208 ransfer complexes, i.e., tetrathiafulvalene (TTF)- and pyromellitic diimide (PMDI)-based crystals, th

209 ntrolled to be on either tetrathiafulvalene (TTF) or naphthalene (NP) stations, either chemically ((1

210 ce of the redox mediator tetrathiafulvalene (TTF) in 1.0 m LiClO4 dissolved dimethyl sulfoxide electr

211 ion of a redox mediator, tetrathiafulvalene (TTF), enables recharging at rates that are impossible fo

212 ) and the guest molecule tetrathiafulvalene (TTF).

213 electronic properties of tetrathiafulvalene (TTF) can be tuned by attaching electron-donating or elec

214 present the evolution of tetrathiafulvalene (TTF) fused D-A systems and their potential applications

215 pect to the oxidation of tetrathiafulvalene (TTF), where the TTF(0/*+) process is used as a reversibl

216 as hydroquinone (HQ) or tetrathiafulvalene (TTF).

217 tems based on the parent tetrathiafulvalene (TTF).

218 nical bonding stabilizes tetrathiafulvalene (TTF) radical dimers, the question arises: what role does

219 via a vinylene-bridge to tetrathiafulvalene (TTF) units, is presented.

220 ient ring and either two tetrathiafulvalene (TTF) and 1,5-dioxynaphthalene (DNP) containing macrocycl

221 sopropylacrylamide) with tetrathiafulvalene (TTF) end groups complexed with CBPQT(4+) induced positiv

222 lectronic properties to tetrathiafulvalenes (TTFs).

223 Here, we demonstrate that TTF-1 transactivated the expression of the epithelial ti

224 atin immunoprecipitation, we determined that TTF-1 binds to the promoter of SREBF2, the host gene of

225 This study provides direct evidence that TTF truly plays a role in promoting the decomposition of

226 n summary, this study provides evidence that TTF-1 may reprogram lung cancer secreted proteome into a

227 Our data indicate that TTF-1 interacts with PPFP to inhibit the pro-adipogenic

228 of-TTF-1 expression strategies, we show that TTF-1 positively regulates vascular endothelial growth f

229 kdown and overexpression studies showed that TTF-1 inhibits PPFP target gene expression and impairs a

230 Collectively, these data suggest that TTF-1 transcriptionally regulates occludin, which repres

231 of HMGA2 without the 3'-UTR, suggesting that TTF-1 keeps the prometastasis gene HMGA2 in check via up

232 ulating cholesterol metabolism suggests that TTF-1 may be a modulator of cholesterol homeostasis in t

233 The TTF-1-dependent upregulation of VEGF was moderately sens

234 The TTF-1-induced VEGF upregulation occurs in both compartme

235 The TTF-derived iPSC clones exhibited similar morphology and

236 The TTF-TCNQ-ionic/ionic liquid gel was characterized by FT-

237 s presence of three short contacts among the TTF groups in the optimum geometry of the clip2(n+) dime

238 tive charge is equally distributed among the TTF groups, while a 1- au charge is located in the centr

239 n movements of the alpha-CD ring between the TTF and newly formed triazole ring systems have been elu

240 ansfer (CT) occurring in a dimer between the TTF residues and are rationalized based on a theoretical

241 The interactions between the TTF-C4P receptors and the fullerene guests are highly in

242 potential is applied, the ring encircles the TTF unit and displays a green color.

243 When the ring is encircling the TTF unit, this co-conformation of the rotaxane is the mo

244 value of 0.55 cm s(-1) was obtained for the TTF(*+/2+) process at a glassy carbon electrode and 2.7

245 s of thousands of packing structures for the TTF- and PMDI-based crystals are first generated based o

246 more effectively than the BIQ2+ guest in the TTF-C4P cavity, leads to back electron transfer, restori

247 e scaffolds and the electronic nature of the TTF arms: the highest binding efficiency is shown by rec

248 ase of the [2]catenane, the formation of the TTF hetero radical dimer is prevented sterically by the

249 show that the out-of-plane distortion of the TTF moiety in this macrocycle is responsible for the var

250 3]catenanes facilitates the formation of the TTF radical dimers under redox control, allowing an inve

251 h a butadiyne group, the distribution of the TTF radical-cation dimer can be changed from 60% to 100%

252 as also been shown that the stability of the TTF radical-cation dimers can be tuned by varying the se

253 hifts (i) in the oxidation potentials of the TTF unit in (a)-(c) and (ii) the reduction potentials of

254 The oxidation process of the TTF unit is dramatically hampered in the rotaxane, there

255 Upon oxidation of the TTF unit, the CBPQT(4+) ring moves to the DNP unit, prod

256 chi affects the oxidation potentials of the TTF units to the extent that switching can be subjected

257 ment of the quasi-reversible kinetics of the TTF(*+/2+) process.

258 rotaxane can be reversed on reduction of the TTF*+ or TTF2+ back to being a neutral TTF.

259 Coactivation of the TTF-1 and NKX2-8 pathways identified a cluster of patien

260 ancer cell lines showing coactivation of the TTF-1 and NKX2-8 pathways were shown to exhibit resistan

261 ther increase the secreted VEGF level of the TTF-1(+) lung cancer cells.

262 Subsequent cooling led to reformation of the TTF-based host-guest complexes in solution and contracti

263 salts serves to modulate the strength of the TTF-C4P-fullerene host-guest binding interactions and, i

264 e 1,3-alternate to the cone conformer of the TTF-C4Ps, thus acting as positive heterotropic allosteri

265 Solutions of the TTF-OXD and 9-(4,5-dimethyl-1,3-dithiol-2-ylidene)fluore

266 molecular crystals are designed based on the TTF and PMDI motifs and an extensive polymorph search.

267 t the alpha-CD ring prefers to reside on the TTF rather than on the triazole ring system by at least

268 It is found that the TTF addition does not obviously affect the discharge rea

269 ation of tetrathiafulvalene (TTF), where the TTF(0/*+) process is used as a reversible internal refer

270 understanding of the interactions within the TTF radical dimers.

271 ToRC comprises ISWI, Toutatis/TIP5 (TTF-I-interacting protein 5), and the transcriptional co

272 eyes were divided into 2 groups according to TTF in the first glaucomatous visual field: (1) eyes wit

273 On charging, TTF is oxidized to TTF(+) at the cathode surface; TTF(+) in turn oxidizes t

274 electronic affinity replaces the traditional TTF-bipyridinium interaction, which is over-ridden by st

275 arent donor to form the mixed-valence [TTF+*,TTF] cation-radical.

276 unique structure of the mixed-valence [TTF+*,TTF] dyad on (a) the electron-transfer mechanism for sel

277 ation of TTF+* to form the diamagnetic [TTF+,TTF+] dication and to also undergo the equally rapid cro

278 aximum of two long, multicenter [TTF](*+)...[TTF](*+) bonds when all TTF groups host a 1+ au of charg

279 ining two 1,5-dioxynaphthalene (DNP) and two TTF units) that is topologically isomeric with the doubl

280 on-rich macrocyclic polyether containing two TTF units of different constitutions, namely 4,4'-bis(hy

281 hthalene (DNP) containing macrocycles or two TTF-butadiyne-containing macrocycles as the pi-electron

282 8, we studied a subset of 1607 who underwent TTF probe analysis of 1 or more grafts during surgery.

283 is over-ridden by stabilizing mixed-valence (TTF)2*+ and radical-cation (TTF*+)2 states inside the 'm

284 its parent donor to form the mixed-valence [TTF+*,TTF] cation-radical.

285 f the unique structure of the mixed-valence [TTF+*,TTF] dyad on (a) the electron-transfer mechanism f

286 67, SOX11 expression was not associated with TTF, but patients with low SOX11 expression had shorter

287 ation of favorable prognosis associated with TTF-1(+) lung adenocarcinomas.

288 e lung epithelial cell (MLE15) extracts with TTF-1 and was identified by mass spectrometry.

289 munoprecipitated from the cell extracts with TTF-1.

290 there were no differences between eyes with TTF and eyes without TTF after adjusting for disparities

291 variate analysis demonstrated that eyes with TTF at presentation compared with eyes without TTF becam

292 rst glaucomatous visual field: (1) eyes with TTF, defined as visual field loss (VFL) including >/=1 o

293 PARP-2 and PARP-1 interact with TTF-1 and regulate the expression of surfactant protein

294 trated that the TAZ directly interacted with TTF-1.

295 tion, which results from an interaction with TTF-1.

296 ences between eyes with TTF and eyes without TTF after adjusting for disparities in disease severity

297 F at presentation compared with eyes without TTF became blind more often (56/182 [30.8%] vs. 22/127 [

298 -2 or 30-2 Humphrey fields; (2) eyes without TTF, defined as VFL only outside the 4 innermost points.

299 FM were superior to R-CVP in terms of 3-year TTF and PFS.

300 fter a median follow-up of 34 months, 3-year TTFs were 46%, 62%, and 59% for the respective treatment