ライフサイエンスコーパス: Th

1 Th cell subsets develop in response to multiple activati

2 Th cells sensitized against autoantigens acquire pathoge

3 Th cells show an impaired response to chemotactic stimul

4 Th subsets Th1 (CXCR3(+)), Th17 (CCR6(+)), and particula

5 matory response, cytokine profiles and Th-1, Th-2 and Th-17 cells numbers in each mating type treated

6 and Tr1 cell populations, for a total of 11 Th cell, 4 Treg, and 1 Tr1 cell subsets.

7 The reaction of (C5 Me5 )2 Th(CH3 )2 with the phosphonium salts [CH3 PPh3 ]X (X=Cl,

8 f methane are liberated to afford (C5 Me5 )2 Th[CHPPh3 ]X, rare terminal phosphorano-stabilized carbe

9 sphorus(V) intermediate, yielding (C5 Me5 )2 Th[kappa(2) -(C,C')-(CH2 )(CH2 )PPh2 ]Cl.

10 a ((235)U, (238)U, (210)Po, (232)Th and (228)Th) and gamma spectrometry ((137)Cs, (40)K, (226)Ra and

11 and <0.57Bqkg(-1) respectively, (235)U, (228)Th and (232)Th were always <0.007Bqkg(-1).

12 (230)Th/U radiometric analysis of multiple bone specimens usi

13 A (230)Th-dated stalagmite delta(18)O record between 88 and 22

14 s followed by measuring the accumulated (230)Th daughter product relative to its parent (234)U nuclid

15 , (143)Nd/(144)Nd, (176)Hf/(177)Hf, and (230)Th/(232)Th.

16 he required gradient of thorium isotope (230)Th over 3.6 meters for 1000 years, much less 10,000 year

17 se from the ocean islands have moderate (230)Th and (226)Ra excesses, reflecting mantle melting in th

18 ioactive decay equations based on the n((230)Th)/n((234)U) amount ratio.

19 nkton productivity (using opal, (231)Pa/(230)Th and excess Ba), and the degree of nitrate consumption

20 The ingrowth of the (230)Th daughter product in the material was followed by meas

21 We present new (238)U-(230)Th-(226)Ra-(210)Pb and supporting data for young lavas f

22 nd levels of natural ((40)K, (238)U and (232)Th and their progeny) and artificial radionuclides ((137

23 g(-1) respectively, (235)U, (228)Th and (232)Th were always <0.007Bqkg(-1).

24 powder contained 6 pg/g and 2 pg/g for (232)Th and (238)U, respectively.

25 powder were typically below 1 pg/g for (232)Th and 2 pg/g for (238)U, corresponding to 4 and 25 muBq

26 ld and purity, from a proton irradiated (232)Th matrix.

27 ined by alpha ((235)U, (238)U, (210)Po, (232)Th and (228)Th) and gamma spectrometry ((137)Cs, (40)K,

28 d/(144)Nd, (176)Hf/(177)Hf, and (230)Th/(232)Th.

29 nal transects of dust (derived from the (232)Th proxy), phytoplankton productivity (using opal, (231)

30 Based on flux assessments from (238)U:(234)Th disequilibrium and sediment traps, we found 2 to 3 ti

31 luride complexes of thorium, [K(18-crown-6)][Th(E)(NR2)3] (E = Se, 4; E = Te, 5), respectively.

32 rmined for the (solely) tetravalent actinide Th on calcite, suggesting reduction of Np(V) to Np(IV) b

33 cells) that binds to the ARRE-2 in activated Th cells.

34 nhanced the transport of previously adsorbed Th(IV).

35 Cl-amidine had a pro-apoptotic effect on all Th subsets in vitro with Th17 cells appearing to be the

36 rotein was expressed from a knock-in allele (Th-MYCN/Trp53(KI)).

37 An[kappa(2)-(N,C)-CH2Si(CH3)2N(SiMe3)] (An = Th or U), alcohol additions to unsaturated carbon-nitrog

38 NSiMe2 Bu(t) )3 , An=U, Pn=P, As, Sb, Bi; An=Th, Pn=P, As; Tren(TIPS) =N(CH2 CH2 NSiPr(i)3 )3 , An=U,

39 H2 CH2 NSiPr(i)3 )3 , An=U, Pn=P, As, Sb; An=Th, Pn=P, As, Sb].

40 ice heterozygous for Trp53(KI) (n = 188) and Th-MYCN mice with wild-type p53 (n = 101).

41 sponse, cytokine profiles and Th-1, Th-2 and Th-17 cells numbers in each mating type treated mice sho

42 nce of 3, which formally contains Th(3+) and Th(4+), suggested that KC8 could reduce [(C5Me5)2ThH2]2.

43 The expression patterns of Cgrpalpha and Th formed opposing gradients, with Th being preferential

44 pharmacologic IKK2 inhibition reduced DC and Th cell activation and ameliorated nephrotoxic serum-ind

45 4(+) T cell marker expression, lifespan, and Th/regulatory T cell function.

46 valent, as essentially isostructural U=P and Th=P analogues.

47 inflammatory response, cytokine profiles and Th-1, Th-2 and Th-17 cells numbers in each mating type t

48 The U-Sb and Th-Sb moieties are unprecedented examples of any kind of

49 ound to feature the shortest known Th-Se and Th-Te bond distances.

50 In cocultures of SF and Th cells, tryptophan was completely depleted within a fe

51 Co, Fe, K, Mg, Mn, Mo, Na, Ni, Se, Sb, U and Th (p<0.05, all) among honeys.

52 A rapid new method for determining the U and Th mass concentrations in high radiopurity plastics is d

53 ssociate with less hydrated and more anionic Th-nitrato complexes.

54 ounds obtained by evaporating acidic aqueous Th-nitrate solutions in the presence of A(+) counterions

55 athological interaction between autoreactive Th cells and mononuclear phagocytes in the CNS drives in

56 ease of the CNS, is mediated by autoreactive Th cells.

57 hile still partially conjugated with the BDT-Th core.

58 t differences in tumor-free survival between Th-MYCN mice heterozygous for Trp53(KI) (n = 188) and Th

59 As, Se, Sr, Mo, Cd, Sn, Sb, Ba, Hg, Pb, Bi, Th, and U) in green coffee samples and their infusions w

60 was required for full Bhlhe40 expression by Th cells after immunization.

61 Out of 20 experimental lines, 10 carried Th. intermedium chromatin as T4DL*4Ai#2S translocations,

62 cytidylic acid) that efficiently induces CD4 Th cells, as well as cross-primes CD8 CTL responses.

63 The IL-9-secreting Th9 subset of CD4 Th cells develop in response to an environment containin

64 ion of adaptive immunity by depletion of CD4 Th cells most likely contributes to loss of immune contr

65 Ox40 was highly expressed on several CD4 Th cell subsets in the spleen and kidney of diseased mic

66 CD4(+) Th cells activated by a microbial biofilm are thought to

67 Human CD3(+)CD4(+) Th cells, FOXP3(+) T regulatory (Treg) cells, and T regu

68 We characterize the CD3(+)CD4(+) Th, Treg, and Tr1 cell populations simultaneously across

69 addition to the already defined CD3(+)CD4(+) Th, Treg, and Tr1 cell populations, for a total of 11 Th

70 anced CD4 response, in which distinct CD4(+) Th subsets act in synergy to control the infection.

71 Follicular CD4(+) Th (Tfh) cells provide B cell help in germinal center re

72 that increased expression of Bcl-6 in CD4(+) Th cell populations correlated with enhanced enrichment

73 ed the capacity of ILT3.Fc to inhibit CD4(+) Th cell proliferation and to induce the generation of CD

74 Ex vivo analysis of CD4(+) Th cell populations revealed an increased amount of Th1

75 are in contact with large numbers of CD4(+) Th cells.

76 diversity of M. tuberculosis-specific CD4(+) Th subsets and determine whether HIV infection impacts s

77 uilibrium of M. tuberculosis-specific CD4(+) Th subsets.

78 response against IgG2a(a) 3F7.A10 was CD4(+) Th cell-dependent, dominated by the IgG1 subclass, and I

79 ls (Treg cells) and effector T helper cells (Th cells), and recently identified innate lymphoid cells

80 ncy allowed the retrovirus to induce central Th cell tolerance in the infected offspring.

81 and few crystallographically characterizable Th(3+) complexes are known due to their highly reducing

82 on glassy carbon electrode (GCE/f-MWCNT-Chit@Th) for quick and sensitive detection of UPEC in aqueous

83 antibody on the surface of GCE/f-MWCNT-Chit@Th.

84 , 7 lines were positive for alien chromatin (Th. intermedium or rye) on chromosome 1B.

85 CMV-specific CD8+ (CMV-Tc), CD4+ (CMV-Th) T cell activity, and natural killer (NK) cell number

86 insertion, resulting in the alkoxide complex Th(OCH2NMe2)(L3) (4).

87 A new thorium monoalkyl complex, Th(CH2SiMe3)(L3) (L = MeC(N(i)Pr)2) (2), undergoes inser

88 series of thorium chalcogenolate complexes, Th(ECH2SiMe3)(L3) (E = S, SS, Se, Te; 5-8).

89 -phosphido complex under ambient conditions (Th=P=Th).

90 The existence of 3, which formally contains Th(3+) and Th(4+), suggested that KC8 could reduce [(C5M

91 ne volume fraction (Ct.BV/TV), thickness (Ct.Th) and porosity.

92 CCR6(+) and CXCR3(+) Th cells accumulate in the blood of aviremic HIV-1-infec

93 oward IL-6-independent Th1 and CD4 cytotoxic Th cell responses.

94 quency in NOD mice correlates with defective Th-POK expression by thymic Valpha14iNKT cells.

95 store differentiation of TGF-beta1-dependent Th cell lineages, including Th17, Th9, and induced regul

96 and promotes development of IL-17-dependent, Th cell-transferable protective immunity.

97 specific for autoantigens, and we described Th as well as CD8(+) T cells specific for the autoallerg

98 r RA patients on total Th cells or different Th cell subsets of healthy donors was analyzed in vitro.

99 f mucosal Ag presentation cells in directing Th cell immune responses against oral pathogens and thei

100 The reactivity of the recently discovered Th(2+) complex [K(18-crown-6)(THF)2][Cp''3Th], 1 [Cp'' =

101 deling occurs in naive CD4(+) T cells during Th cell differentiation using a type-2-infection model.

102 chemical immunosensor based on thionine dye (Th) immobilized on functionalized-multiwalled carbon nan

103 pared with that observed in non-TFH effector Th cells generated in response to influenza infection.

104 ulated relative to the corresponding element/Th ratio of the Upper Continental Crust) reveal maximum

105 ammatory phenotype characterized by elevated Th-17 lymphocytes and, conversely, a blunted alveolar ma

106 lhe40 expression identifies encephalitogenic Th cells and defines a PTX-IL-1-Bhlhe40 pathway active i

107 ng that the zinc finger transcription factor Th-POK is a key negative regulator of thymic NKT17 cell

108 Follicular Th (Tfh) cells orchestrate physiological germinal center

109 portant for CD4(+) Th17, Th9, and follicular Th cell development.

110 within IL-17-secreting T cell and follicular Th cell paradigms to generate IL-21 and IL-17A, which dr

111 c germinal center (GC) B cell and follicular Th cell responses to compare the induction of these resp

112 (-/-) mice are unable to generate follicular Th and Th2 cells.

113 otein ICOS on T cells and induced follicular Th cell differentiation.

114 ion and promotes the induction of follicular Th (TFH) cells, CD4(+) T cells that support B cell affin

115 lower frequency of virus-specific follicular Th (Tfh) cells and increased the Th1 to Tfh ratio.

116 Ag-specific follicular Th cell responses to adenovirus vectored vaccines exceed

117 n immunophenotype capable of Th1, follicular Th, and CTL functionalities, yet they are unable to be i

118 e cells expressed markers characteristic for Th cells (CD154) and produced the cytokine TNF-alpha or

119 target of rapamycin (mTOR) is essential for Th cell proliferation and effector differentiation, maki

120 ts show that Smads are directly required for Th cell differentiation independent of Runx induction bu

121 C subset diverges as a distinct lineage from Th and circulating natural killer cells but shares circu

122 cells, opposite to activated IFN-gamma(-/-) Th cells, partially reconstituted CF and HF in TCR-alpha

123 The GC-GNs-Th electrode is subjected to further modifications to fa

124 ite-like nanostructures (DGNs) on the GC-GNs-Th surface, constructing GC-GNs-Th-GOx and GC-GNs-Th-DGN

125 ctrode via graphene nanosheets (GNs) (GC-GNs-Th).

126 rface, constructing GC-GNs-Th-GOx and GC-GNs-Th-DGNs modified electrodes, respectively.

127 Also, the GC-GNs-Th-DGNs sensor showed a wide dynamic response range for

128 n the GC-GNs-Th surface, constructing GC-GNs-Th-GOx and GC-GNs-Th-DGNs modified electrodes, respectiv

129 (ThAsH2), -arsinidiides (ThAs(H)K and ThAs(H)Th) and arsenido (ThAsTh) linkages stabilized by a bulky

130 parent dithorium(IV)-phosphinidiide (Th-P(H)-Th) and a discrete actinide-phosphido complex under ambi

131 (+)CD8(+) cytotoxic and CD3(+)CD4(+) helper (Th) T lymphocytes, together with increased Th1, Th2, Th1

132 sterols and stanols can shift the T helper (Th) 1/Th2 balance toward a Th1-type immune response, whi

133 and lower frequency of pathogenic T helper (Th) 17 cells.

134 Interleukin (IL)-23-induced T helper (Th) 17 pathway is involved in the pathogenesis of period

135 nt up-regulation of genes encoding T helper (Th) 2 (e.g., IL4, IL5, IL13, IL31, and IL33), Th9 (IL9),

136 Interleukin-4 (IL-4)-induced T helper (Th) 2 cells promote susceptibility to the protozoan para

137 the precise mechanisms involved in T helper (Th) 2 polarization by dendritic cells (DCs) are currentl

138 nal kinships to cytokine-secreting T helper (Th) cell counterparts.

139 features that define autoreactive T helper (Th) cell pathogenicity remain obscure.

140 ostasis and mirror adaptive CD4(+) T helper (Th) cell subtypes in both usage of effector molecules an

141 first cytokine produced when naive T helper (Th) cells are activated and differentiate into dividing

142 ; however, the potential of CD4(+) T helper (Th) cells for ACT is gaining interest.

143 CD4(+) T helper (Th) cells play a central role in the adaptive immune res

144 cell receptor stimulation, CD4(+) T helper (Th) lymphocytes release extracellular vesicles (EVs) con

145 dritic cells secrete low levels of T helper (Th)-1 polarization associated cytokines.

146 gramming, and (3) skewing toward a T helper (Th)/T cytotoxic (Tc)17 transcriptional program.

147 Interleukin (IL)-33 is involved in T helper (Th)2-biased immune responses in mice infected with Schis

148 d overlapping lineages, defined as T helper (Th)22 and IL-22(+) Th17 cells.

149 KT, as well as their switch from a T helper (Th-2; ie IL-4, IL-13) to Th-1 (ie IFN-gamma) cytokine pr

150 es IL-12 and IL-23 are involved in T-helper (Th) 1 and Th17 immunity, respectively.

151 evels of interleukin 23 (IL23) and T-helper (Th) 17 cell pathway molecules are increased in inflamed

152 T-helper (Th) 17 cells are important in the control of Streptococc

153 coidosis is classically defined by T-helper (Th) cell type 1 inflammation (e.g., IFN-gamma production

154 ensively studied in the context of T-helper (Th)1 and Th2 responses.

155 c for FlaX, A4-fla2, or YidX had a T-helper (Th)1 phenotype; a larger proportion of CD4(+) T cells sp

156 t Grp94 promoted alphaSyn-specific T-helper (Th)1/Th17 and IgG1 antibody responses (up to a 3-fold in

157 PHD proteins limit pulmonary type helper (Th)-1 responses, promote CD4(+)-regulatory T (Treg) cell

158 d Pb, LREE then La-Ce-Nd-Sm, Lu(Yb), and Hf, Th, and U, respectively) along with an additional, in-ho

159 llation, we were able to achieve the highest Th wt % in mono- and biactinide frameworks with minimal

160 ytokine production by naive and memory human Th cells.

161 -sensing C-fibers, and tyrosine hydroxylase (Th), specific to low-threshold mechanoreceptive C-fibers

162 ne regulatory circuitries for four human ILC-Th counterparts derived from mucosal environments, revea

163 toimmune disease SNP associations within ILC-Th subsets.

164 In Th cell subsets, Th2 cells produce considerable amounts

165 Our data demonstrate an important balance in Th subset diversity defined by lineage-defining transcri

166 To test whether these changes in Th cell differentiation potential rendered Grb2(fl/fl) C

167 uld lead to a disturbance of later events in Th cell plasticity, leading to autoimmune diseases or ot

168 a strong stimulus for Bhlhe40 expression in Th cells.

169 n and functionality of E-selectin ligands in Th type 17 lymphocytes (Th17 cells) and report that CD43

170 counts (TFPI/TF) was significantly lower in Th+ versus Th- BS patients (p = 0.0002), and no patient

171 Here, we used optogenetics in Th::Cre rats to selectively stimulate VTA dopamine neuro

172 n important role in preventing inappropriate Th cell responses under normal conditions.

173 ns associated with CD4(+) T cells (including Th follicular functionality in lymphoid tissues and Th2

174 f CD4(+) T cells can be recruited, including Th cells (Th1, Th2, and Th17), T follicular helper cells

175 The redifferentiation of T reg cells into Th cells has been identified in hyperinflammatory diseas

176 ntly higher than that of nonfluorinated ITIC-Th (8.88%).

177 ectron mobility than the nonfluorinated ITIC-Th.

178 and were found to feature the shortest known Th-Se and Th-Te bond distances.

179 l-Th biochemically modulates various anti-neoplastic agent

180 l-Th enhances the umami taste but its use is limited due t

181 l-Th has become choice ingredient in CNS active products d

182 effort to condense the recent research on l-Th highlighting its biological resource, plausible role

183 l-theanine (l-Th), a non-protein amino acid present in tea, is a valua

184 otes robust T cell proliferation, but limits Th cells polarization and production of IL-1beta and oth

185 with some REEs (i.e. Sc, La, Ce, Pr, Nd, Lu, Th).

186 d for the differentiation of the other major Th subsets are well defined, those responsible for Tfh c

187 e PRMT5 in the regulation of adaptive memory Th cell responses and suggest that PRMT5 inhibitors may

188 ule PRMT5 inhibitors severely blunted memory Th expansion, with preferential suppression of Th1 cells

189 imal proliferation of mouse and human memory Th cells.

190 r in naive compared with activated or memory Th cells; in the latter, Ets-2 participates in a change

191 ocked in naive, but not activated or memory, Th cells by the transcription factor Ets-2 that binds to

192 hanisms by which noninflammatory SF modulate Th cell responses and to determine the immunosuppressive

193 oblasts play an important role in modulating Th cell responses to NTHi.

194 without EP) shifted responses towards a more Th-17 dominated phenotype, associated with mucosal and e

195 independent preinduction repressor in naive Th cells and does not interact physically with the trans

196 In naive Th cells, Ets-2 mRNA expression, Ets-2 protein levels, a

197 sults in a physiological transition of naive Th cells to Th0 cells upon antigenic stimulation.

198 stimulated DCs is altered, an MLR with naive Th cells was performed.

199 reinstated sensitivity to IR in only 50% of Th-MYCN/Trp53(KI/KI) tumors, indicating the acquisition

200 In the absence of Th(IV), changes in ionic strength were ineffective at re

201 Ag-experienced B cells rely on the action of Th cells to produce these pathogenic Abs.

202 In contrast, the adsorption of Th(IV) had a marked impact on the surface charge of the

203 Here we report that the deletion of Th in hematopoietic cells of adult mice neither alters e

204 marily due to the pH-dependent desorption of Th(IV) caused by the change in ionic strength.

205 ytokine that promotes the differentiation of Th cell subsets, including Th1, Th2, and Th9 cells, but

206 nce of T1D, modulated the differentiation of Th cells and Tregs, and decreased the levels of IFN-gamm

207 nes required for terminal differentiation of Th cells, decreased in the CNS of NPC-treated mice, cons

208 response and tolerance, but the diversity of Th, Treg, and Tr1 cell subsets has not been fully charac

209 The expression of Th and Cgrpalpha was not mutually exclusive and co-expre

210 We studied the transport and mobility of Th(IV), as an analogue for Pu(IV) and other tetravalent

211 activating signals influence the outcome of Th cell differentiation via differential regulation of m

212 releasing colloids while in the presence of Th(IV), decreases in ionic strength liberated significan

213 SF strongly suppressed the proliferation of Th cells and the secretion of IFN-gamma in a cell contac

214 e the ability to exhibit a broad spectrum of Th subsets, defined by specific patterns of transcriptio

215 We postulate that the suppression of Th cell growth by SF through tryptophan catabolism may p

216 Instead, the enhanced transport of Th(IV) was primarily due to the pH-dependent desorption

217 y a minor role in enhancing the transport of Th(IV).

218 unctions are performed by different types of Th cells endowed with distinct migratory capacities and

219 microbes have advanced our understanding of Th cell functional heterogeneity, in particular with the

220 f wild-type animals were highly dependent on Th cells or IL-5.

221 ct on colloid transport than colloids had on Th(IV) transport.

222 CMV-Tc and/or Th became (-) in 50% to 70% of these sero (+) patients a

223 sero (+) patients were (+) for CMV-Tc and/or Th predesensitization, while 3 sero (-) patients showed

224 miR-23 cluster also negatively impacts other Th lineages, enforced expression of miR-24, in contrast

225 though whether this paradigm exists in other Th subsets is not clear.

226 trinsically limited in comparison with other Th effector cells, as the biological role of a GC Tfh ce

227 phido complex under ambient conditions (Th=P=Th).

228 he kinematic tracer protactinium/thorium (Pa/Th) with the deep water-mass tracer, epibenthic delta(13

229 ized by a dramatic suppression of pathogenic Th responses as well as induction of IL-10-producing reg

230 the induction of GM-CSF-producing pathogenic Th cells.

231 xamples of a parent thorium(IV)-phosphanide (Th-PH2), a terminal thorium(IV)-phosphinidene (Th=PH), a

232 -PH2), a terminal thorium(IV)-phosphinidene (Th=PH), a parent dithorium(IV)-phosphinidiide (Th-P(H)-T

233 =PH), a parent dithorium(IV)-phosphinidiide (Th-P(H)-Th) and a discrete actinide-phosphido complex un

234 nic strength groundwater contaminant plumes, Th(IV) had a much greater effect on colloid transport th

235 Although IL-2 is produced by all polarized Th subsets to some level, how it impacts cytokine produc

236 r, TIM-4(+) B cells promoted proinflammatory Th differentiation in vivo, increasing IFN-gamma while d

237 gnals induce high Akt activity that promotes Th cell induction.

238 he offspring's ability to mount a protective Th cell-dependent B cell response.

239 roved morphology of the donor-acceptor (PTB7-Th:NDP-V) blend, which is evidenced by the enhanced hole

240 sed low bandgap donor polymers, such as PTB7-Th.

241 complementary absorption of low-bangap PTB7-Th and small-bandgap ATT-2 in NIR region, the proof-of-c

242 with those of the corresponding PC71BM/PTB7-Th-based solar cells.

243 The blend TPB:PTB7-Th films show favorable morphology and efficient charge

244 By combining with PTB7-Th, the as-cast OPVs yield PCEs of up to 9.58% with a fi

245 ments support a mechanism involving reactive Th-NHC metallacycle intermediates (Int and 2).

246 helper 9 (Th9) cells, a recently recognized Th cell subset, are involved in autoimmune diseases.

247 c regulation through microRNA-133b-regulated Th-POK expression and signals provided by DCs are fundam

248 Blocking IDO1 activity completely restored Th cell proliferation, but not IFN-gamma production.

249 and the reduced capacity of RASF to restrict Th cell proliferation through tryptophan metabolism may

250 l honey (Al, As, Be, Ca, Cr, Mn, Mo, Ni, Se, Th and U), common heather (Co, K, Mg, Na, V), sage (Ag,

251 n-Snca expression and no effect on rat-Snca, Th, Bdnf or Trk2.

252 o clonal expansion of P. gingivalis-specific Th cells and induced regulatory T cells does not depend

253 ubsets among other lymphocytes, specifically Th cells, innate lymphoid cells (ILC), and gammadelta T

254 ts of T cell biology, particularly helper T (Th) 2 immunity.

255 quires the specification of CD4(+) helper T (Th) cells into distinct fates, including Th1 cells that

256 bone volume fraction (BV/TV), thickness (Tb.Th), number (Tb.N), connectivity density (Conn.Dn), and

257 acts with [Et3NH][BPh4] to form the terminal Th(4+) hydride complex Cp''3ThH, 2, a reaction that form

258 We found that Th-MYCN/Trp53(KI/KI) tumors were resistant to ionizing r

259 Moreover, we found that Th-POK expression is under epigenetic regulation mediate

260 We have previously shown that Th cells require the transcription factor Bhlhe40 to med

261 The Th [Formula: see text] occupancy in the ground state was

262 The Th(3+) complex, (C5Me5)3Th, has been isolated despite th

263 The Th-Bi combination was too unstable to isolate, underscor

264 ing ThO2 as an example, data measured at the Th 3d edge were interpreted within the framework of the

265 state was estimated to be twice that of the Th [Formula: see text] states.

266 fects the overall solid-state packing of the Th-nitrato complexes but also influences the composition

267 s but also influences the composition of the Th-nitrato monomeric anions themselves.

268 Amongst BS patients, the Th+ group had increased total and TF positive MP numbers

269 EG ameliorates autoimmunity by targeting the Th 17-Tregs axis, making it a promising candidate drug f

270 numbers (both p </= 0.0002) compared to the Th- group, but had a lower proportion of TFPI positive M

271 7s, 6d and 5f orbital contributions to the Th-As bonds are suggested by quantum chemical calculatio

272 wed some resistance, while together with the Th. intermedium 4Ai#2S offered superior resistance to th

273 e relationship between ILC subsets and their Th cell counterparts, we measured genome-wide chromatin

274 evoted to functional polarization with their Th counterparts.

275 Thionine (Th) diazonium cation is covalently attached onto the gla

276 ies found for two related series of thorium (Th)-nitrate molecular compounds obtained by evaporating

277 S patients (21 with a history of thrombosis (Th+) and 67 without (Th-).

278 ch from a T helper (Th-2; ie IL-4, IL-13) to Th-1 (ie IFN-gamma) cytokine profile.

279 Th2 cell levels) were performed according to Th cell responses in gingival tissues.

280 from osteoarthritis or RA patients on total Th cells or different Th cell subsets of healthy donors

281 nsgenic mouse system in which TCR-transgenic Th cells specific to hen egg lysozyme (HEL) are adoptive

282 , Zr, Ba, Cs, Ba, La, Ce, Nd, Sm, Dy, Lu, U, Th) in glassy fallout from the first nuclear test, Trini

283 per, we used Li concentration profiles and U-Th ages to constrain the thermal conditions of magma sto

284 by ion microprobe and uranium isotopes and U-Th dating by laser ablation inductively coupled plasma m

285 ew (14)C, zircon U-Th crystallization and (U-Th)/He ages show resurgence commenced at 69.7+/-4.5 ka a

286 A major issue in thermochronology and U-Th-Pb dating is the effect of radiation damage, created

287 More than 350 dates (by (14)C, U-Th, TL and (36)Cl) were obtained over the last 15 y.

288 We present the results of U-Th dating of calcite veins in the Loma Blanca normal fau

289 estimate obtained from the teeth, with the U-Th age for the oldest flowstone overlying Homo naledi fo

290 ated luminescence dating of sediments with U-Th and palaeomagnetic analyses of flowstones to establis

291 In concert with U-Th dates recording decreased recurrence intervals, we in

292 Here we reveal that new (14)C, zircon U-Th crystallization and (U-Th)/He ages show resurgence co

293 Elevated U/Th ratios in these BIFs relative to the contemporary cru

294 PI/TF) was significantly lower in Th+ versus Th- BS patients (p = 0.0002), and no patient with a TFPI

295 of Akt signaling networks during Treg versus Th induction demonstrates that Akt differentially regula

296 the capacity for two-electron reduction via Th(3+) and sterically induced reduction.

297 ificantly lower IDO1 expression and a weaker Th cell suppressive capacity compared with osteoarthriti

298 alpha and Th formed opposing gradients, with Th being preferentially expressed in apical and Cgrpalph

299 Surprisingly, infected mice with Th cell impairment experienced a compensatory neutrophil

300 history of thrombosis (Th+) and 67 without (Th-).