1 Thus, we conclude that 5-InsP7 regulates p53 expression, wher
2 Thus, we conclude that 6AP competitively occludes the protein
3 Thus, we conclude that a component of premature infant gut co
4 Thus, we conclude that additional contacts with protein subst
5 Thus, we conclude that ALB4 and STIC2 both participate in thy
6 Thus, we conclude that alternatively activated macrophages do
7 Thus, we conclude that an interaction between Abeta and alpha
8 Thus, we conclude that ATRA enhances both Arg-1 and iNOS expr
9 Thus, we conclude that BLIMP1 expression is both necessary an
10 Thus, we conclude that Bmi1 deficiency impairs the progressio
11 Thus, we conclude that CYP20-3 is a key effector protein that
12 Thus, we conclude that decorin prevented the development of h
13 Thus, we conclude that DHHC2-AKAP79/150 signaling is an essen
14 Thus, we conclude that ECM FN-dependent vasodilatation is med
15 Thus, we conclude that endothelial chimerism combined with va
16 Thus, we conclude that G1-dCT carries an essential role in Gb
17 Thus, we conclude that high humidity, a well-known disease-pr
18 Thus, we conclude that Hippo-Yap signaling suppresses cell po
19 Thus, we conclude that ICP27t2 and ICP27 are functionally ver
20 Thus, we conclude that IFN-gamma promotes melanoma cell survi
21 Thus, we conclude that Ikaros plays distinct roles at differe
22 Thus, we conclude that in a polyclonal CD4 T cell repertoire,
23 Thus, we conclude that individual reaction norms have followe
24 Thus, we conclude that it is not possible to distinguish betw
25 Thus, we conclude that micro-CT is highly suitable for target
26 Thus, we conclude that mtDNA mutations and impaired glucose u
27 Thus, we conclude that Nerfin-1 represses Notch activity in m
28 Thus, we conclude that NETs contribute to pathology in autoim
29 Thus, we conclude that Pgp3 is an effector virulence factor a
30 Thus, we conclude that PROPPIN membrane binding is curvature
31 Thus, we conclude that Rab GTPases are essential for membrane
32 Thus, we conclude that RASF contribute to the inflammatory pr
33 Thus, we conclude that '
rigidity' in the beta2-alpha2 loop re
34 Thus, we conclude that Ser5-001 is the predominant antiviral
35 Thus, we conclude that TaFROG encodes a new Sucrose Non-Ferme
36 Thus, we conclude that the advantage of the hydrolytic pathwa
37 Thus, we conclude that the association between hyperglycemia
38 Thus, we conclude that the FA pathway is required for the gro
39 Thus, we conclude that the Mediterranean invasion is the resu
40 Thus, we conclude that the NS3 linker is critical for mediati
41 Thus, we conclude that the spleen harbors a protective B-cell
42 Thus, we conclude that the type 1 pneumococcal pilus can acti
43 Thus, we conclude that there is no crystallographic support f
44 Thus, we conclude that tmTNF-alpha on the surface of highly p
45 Thus, we conclude that TRIM3 is a tumor suppressor mapping to
46 Thus, we conclude that VHH JM4, when targeted to the lipid ra
47 Thus, we conclude that XBP1s is an important regulator of adi
48 Thus, we conclude that Y. pestis YopJ and YopM can both exert
49 Thus, we conclude that YY2 is a major regulator of mESC self-
50 Thus, we conclude that Zeb2 regulates commitment to both the