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1 flammatory disease through the activation of toll-like receptors.
2 ell receptor (BCR), Fc receptors (FcRs), and toll-like receptors.
3 s a macrophage immunosuppressant by reducing Toll-like receptor 1/2 (TLR1/2) activation by bacterial
4                             Responses to all Toll-like receptor 1/2 (TLR1/2), TLR2/6, and TLR4 agonis
5 ls of pattern recognition receptors, such as Toll-like receptors 1 and 2, dectin 1, and dendritic cel
6 s trigger pro-inflammatory responses through Toll-like receptor 2 (TLR2) activation, and this whether
7  antibodies were used to dissect the role of Toll-like receptor 2 (TLR2) and programmed death-ligand
8 ed, albeit the recognition of lipoglycans by Toll-like receptor 2 (TLR2) appears to be important for
9         We examined the inflammatory role of Toll-like receptor 2 (TLR2) in age-related macular degen
10                         However, eliminating Toll-like receptor 2 (TLR2) permits bacterial replicatio
11                                              Toll-like receptor 2 (TLR2) plays a critical role in hos
12 es in the proinflammatory markers TNF-alpha, toll-like receptor 2 (TLR2), and TLR4.
13 l" anti-inflammatory phenotype by activating Toll-like receptor 2 (TLR2), which regulates the inducti
14 he Brucella effector protein TcpB suppresses Toll-like receptor 2 (TLR2)- and TLR4-mediated innate im
15 n of macrophages derived from MyD88-, TRIF-, Toll-like receptor 2 (TLR2)-, TLR4-, and TLR2/4-deficien
16 n induces pathogenic host inflammation via a Toll-like receptor 2 (TLR2)-dependent pathway, resulting
17 88 (MyD88), TANK binding kinase 1 (TBK1), or Toll-like receptor 2 (TLR2).
18  stimulates the innate immune system through Toll-like receptor 2 (TLR2); however, the pathogen-assoc
19                                              Toll-like receptor 2 agonism activated luminal endotheli
20 eneration and elevated surface expression of toll-like receptor 2 and CD11b on monocytes and neutroph
21  with broad specificity that is dependent on Toll-like receptor 2 and interleukin 1beta.
22  IFN-kappa was significantly increased after toll-like receptor 2 and UVB treatment in lupus keratino
23                                              Toll-like receptor 2 expression was unchanged.
24 e skin microbiome is a rich source of LTA, a Toll-like receptor 2 ligand, we mimicked the GF microbio
25 ation factor, and lipoteichoic acid (LTA), a Toll-like receptor 2 ligand.
26 bed flow and of neutrophils, hyaluronan, and Toll-like receptor 2 ligation in superficial intimal inj
27 implicate flow disturbance, neutrophils, and Toll-like receptor 2 signaling as mechanisms that contri
28 h healthy control subjects after exposure to toll-like receptor 2, 3, or 4 agonists or exposure to UV
29 r membrane of A. muciniphila, interacts with Toll-like receptor 2, is stable at temperatures used for
30               HMGB1 functions as a ligand of Toll-like receptors 2 and 4 on macrophages, leading to a
31 his article, we show that germ-free (GF) and Toll-like receptor-2 (Tlr2)-deficient mice have reduced
32 igh-iron conditions had reduced responses to Toll-like receptor-2, -3, and -4 agonists, which associa
33 olyinosine-polycytidylic acid (poly(I:C)), a Toll-like receptor 3 (TLR3) agonist used as a mimetic to
34                      Defects in genes of the Toll-like receptor 3 (TLR3) pathway are associated with
35 horylation, leading to inefficient RIG-I and Toll-like receptor 3 (TLR3) responses.
36 merous interferon-regulated genes, including Toll-like receptor 3 (Tlr3), which encodes an innate imm
37 mal organization to the selective control of toll-like receptor 3 (TLR3)- and TLR4-mediated proinflam
38     These interferon responses attributed to toll-like receptor 3 (TLR3)-activated Kupffer and liver
39                    Inhibition of BRD4 blocks Toll-like receptor 3 (TLR3)-dependent neutrophilia and R
40                                              Toll-like receptor 3 expression was analyzed in postmort
41                                              Toll-like receptor 3, an innate pattern recognition rece
42   We sought to evaluate an inhibitory mAb to Toll-like receptor 3, CNTO3157, on experimental HRV-16 i
43 ary and sufficient to stimulate WIHN through toll-like receptor 3.
44                                              Toll-like receptor-3 (TLR3), a member of the pathogen re
45 NA levels were assessed by real-time PCR and Toll like receptor 4 (TLR-4) protein expression by Weste
46              Additionally, the activation of toll like receptor 4 (TLR4) induced by LPS showed no alt
47               This activation is through the toll like receptor 4 (TLR4)/myeloid differentiation prim
48  increase in TJ permeability was mediated by toll-like receptor 4 (TLR-4)/MyD88 signal-transduction p
49                                              Toll-like receptor 4 (TLR4) activation by bacterial infe
50 e microbiota, hematopoietic cell deletion of Toll-like receptor 4 (TLR4) and inactivation of the IL-1
51 with a decrease in the expression of mucosal toll-like receptor 4 (TLR4) and its adaptor myeloid diff
52                 Here we identify endothelial Toll-like receptor 4 (TLR4) and the gut microbiome as cr
53  the combination of synthetic small-molecule Toll-like receptor 4 (TLR4) and TLR7 ligands is a potent
54 ntigens adjuvanted with ligands specific for Toll-like receptor 4 (TLR4) and TLR7/8 encapsulated in p
55  The results point to modified signaling via Toll-like receptor 4 (TLR4) as a possible mechanism for
56 further implicate the innate immune receptor toll-like receptor 4 (TLR4) as an underlying mechanism m
57                               Stimulation of Toll-like receptor 4 (TLR4) by bacterial lipopolysacchar
58                                   Similarly, Toll-like receptor 4 (TLR4) has been implicated in breas
59 Ps) induced NF-kappaB activation mediated by Toll-like receptor 4 (TLR4) in a glycoprotein (GP)-depen
60                   LPS-mediated activation of Toll-like receptor 4 (TLR4) in macrophages results in th
61                                              Toll-like receptor 4 (TLR4) is a critical component of i
62                      SIGNIFICANCE STATEMENT: Toll-like receptor 4 (TLR4) is a key mediator of innate
63                                              Toll-like receptor 4 (TLR4) is a pattern recognition mol
64          Innate immune system activation via Toll-like receptor 4 (TLR4) leads to inflammation and ox
65 morphine binds to the innate immune receptor toll-like receptor 4 (TLR4) localized primarily on micro
66                           Here, we show that Toll-like receptor 4 (TLR4) mediates cancer-induced musc
67                  To test the hypothesis that toll-like receptor 4 (TLR4) mediates proinflammatory pol
68 rs of innate immunity, via activation of the toll-like receptor 4 (TLR4) on myeloid cells.
69 rs of innate immunity, via activation of the toll-like receptor 4 (TLR4) on myeloid cells.
70 ow that GOS is recognized by and upregulates Toll-like receptor 4 (TLR4) on RAW264.7 macrophages, fol
71 entiation revealed that binding of S100A9 to Toll-like receptor 4 (TLR4) promotes activation of p38 m
72 emonstrate that hRetn binds the LPS receptor Toll-like receptor 4 (TLR4) through its N terminal and m
73 ent of Gram-negative bacteria that activates Toll-like receptor 4 (TLR4) to trigger proinflammatory r
74 300b, and its adaptor DAP12, associated with Toll-like receptor 4 (TLR4) upon LPS binding, thereby en
75                            Binding of MfP to Toll-like receptor 4 (TLR4) was determined by co-immunop
76                                       WD-fed Toll-like receptor 4 (TLR4)(-/-) mice did not exhibit my
77 -NS5A in liver up-regulate the expression of Toll-like receptor 4 (TLR4), and develop liver tumors co
78 o found to bind TRAM, an adaptor protein for Toll-like receptor 4 (TLR4), and thereby impact both vir
79           In the ALHS, we tested for a gene [Toll-like Receptor 4 (TLR4), encoding the endotoxin rece
80 ytoplasm and downregulated the expression of toll-like receptor 4 (TLR4), receptor for advanced glyca
81 gle study revealed a new complex composed of Toll-like receptor 4 (TLR4), TLR6, and CD36 induced by f
82 del of PHH, we demonstrate that IVH causes a Toll-like receptor 4 (TLR4)- and NF-kappaB-dependent inf
83                      Disease was linked to a Toll-like receptor 4 (TLR4)-dependent mechanism of IAP d
84 EL induced PTX3 expression by activating the Toll-like receptor 4 (TLR4)-dependent pathway via nuclea
85 ct ligations or sham surgeries on C57BL/6 or toll-like receptor 4 (TLR4)-knockout mice to induce live
86 ages involves multiple mechanisms, including Toll-like receptor 4 (TLR4)-mediated NADPH oxidase (NOX)
87 f opioids, and we recently demonstrated that Toll-like receptor 4 (TLR4)-mediated neuroinflammation i
88                                              Toll-like receptor 4 (TLR4)-mediated signaling was asses
89 ue damage and expressed by tumors, activates toll-like receptor 4 (TLR4)-mediated sterile inflammatio
90 ated predominantly via the membrane receptor Toll-like receptor 4 (TLR4).
91 s in cultured myenteric neurons that express Toll-like receptor 4 (TLR4).
92 ts demonstrate that the use of the synthetic Toll-like receptor 4 agonist glucopyranosyl lipid A in s
93 reduced lipopolysaccharide activation of the toll-like receptor 4 and increased survival times compar
94 evated hepatic stellate cell-derived TnC and Toll-like receptor 4 expression was observed in the diet
95 n end products (RAGE) messenger RNA, but not toll-like receptor 4 in hippocampal microglia.
96 amplified platelet response to the agonists; Toll-like receptor 4 inhibitor blunted this effect.
97  primes the cell for an enhanced response to toll-like receptor 4 ligands.
98 gen species production and activation of the Toll-like receptor 4 signaling pathway.
99 resistant to Fas-mediated apoptosis ex vivo, Toll-like receptor 4(TLR4)-ligation restored Fas-sensiti
100             In the brain, levels of RAGE and Toll-like receptor 4, glial fibrillary acidic protein an
101 also attenuated proinflammatory signaling by Toll-like receptor 4, which has a central role in Ad pat
102 nd SseK3 suppress TNF-alpha-induced, but not Toll-like receptor 4- or interleukin-induced, NF-kappaB
103 failed to induce depressive-like behavior in Toll-like receptor 4-deficient mice and in mice harborin
104 rass pollen or mite allergens to enhance the Toll-like receptor 4-mediated proallergic properties of
105 s, an event triggered by the innate receptor Toll-like receptor 4.
106 lowing binding of growth factor receptors or Toll-like receptor 4.
107 eta1 integrin and the innate immune receptor toll-like receptor 4.
108 ury triggering the macrophage activation via Toll-like receptor 4.
109 s blunted by incubation with an inhibitor of Toll-like receptor 4.
110         Additionally, we found a significant toll-like receptor 4/alpha4beta1-dependent enrichment in
111 livery system, and simultaneously, activated Toll-Like Receptor-4 (TLR-4) on APCs to release chemokin
112                          In this work, human Toll-Like Receptor-4 (TLR-4), a protein responsible for
113 innate immune signaling components including Toll-like receptor-4 and type I IFNs.
114                               The NMDA-R and Toll-like receptor-4 were not required for proinflammato
115 so improved the level of HA receptors (CD44, Toll-like receptor-4, and receptor for HA-mediated motil
116                                              Toll-like receptor 5 (TLR5) is considered an attractive
117                                              Toll-like receptor 5 (TLR5) recognition of flagellin ins
118 ellin:allergen fusion protein containing the Toll-like receptor 5 ligand flagellin A from Listeria mo
119            Fusion proteins incorporating the Toll-like receptor 5 ligand flagellin are currently unde
120 n of bacterial flagellins that interact with Toll-like receptor 5.
121                                              Toll-like receptor 7 (TLR7) mediates autoantigen and vir
122                                              Toll-like receptor 7 (TLR7) stimulation in the airways m
123                             The induction of toll-like receptor 7 (TLR7)-dependent type I interferons
124 cognition of intracellular Y. pestis by host Toll-like receptor 7 (TLR7).
125                        On treatment with the toll-like receptor 7 agonist imquimod (IMQ), Trim32 knoc
126 e isolated from whole blood, stimulated with Toll-like receptor 7 agonist, and analyzed by means of e
127 rge amounts of type 1 interferon (IFN) after Toll-like receptor 7 and 9 engagements.
128 it, colony stimulating factor 3 receptor and toll-like receptor 7 mRNA expression increases in the th
129  application of Aldara cream, containing the Toll-like receptor 7/8 agonist Imiquimod, is a widely us
130 ammatory conditions, exposure of APCs to the Toll-like receptor 7/8 agonist R848 increases nanocluste
131                                Activation of Toll-like receptor 7/8 by means of topical application o
132      We show that activation of pDCs through Toll-like receptor 7/8 suppresses ILC2-mediated AHR and
133 highly conserved cysteine residue (Cys98) on Toll-like receptor-7.
134 on by the pattern-recognition receptor (PRR) Toll-like receptor 8 (TLR8).
135 rich in unmethylated CG motifs (CpGs) engage Toll-Like Receptor 9 (TLR-9) in endosomes and are well d
136 re cultured with CD40 ligand (CD40L) and the Toll-like receptor 9 (TLR9) agonist cytidine-phosphate-g
137 thesized that a single drug molecule-a novel Toll-like receptor 9 (TLR9) agonist, MGN1703-could funct
138 ly, we identify parasite DNA-sensing through Toll-like receptor 9 (TLR9) along with inflammatory cyto
139                                              Toll-like receptor 9 (TLR9) enhances proinflammatory res
140 a key role in recognition, as highlighted by Toll-like receptor 9 (TLR9) in the endosomal compartment
141                                              Toll-like receptor 9 (TLR9) is an endosome bound, innate
142           We investigated the ability of the Toll-like receptor 9 (TLR9) ligand CpG to modulate estab
143 responsiveness of paired lung fibroblasts to Toll-like receptor 9 (TLR9) stimulation by CpG-oligodeox
144                                              Toll-like receptor 9 (TLR9)-deficient (TLR9(-/-)) mice a
145 FcvarepsilonRI receptor for IgE, followed by Toll-like receptor 9 (TLR9)-mediated sensing of DNA in p
146            In this study, we report that the Toll-like receptor 9 (TLR9)-MyD88 pattern-recognition re
147  a defective response to ligation of BCR and Toll-like receptor 9 (TLR9).
148 wed that stimulation of innate immunity with Toll-like receptor 9 agonist, class B CpG (cytosine-phos
149 hout co-administration or encapsulation of a Toll-Like Receptor 9 agonist.
150                       Breg cells obtained by Toll-like receptor 9 and CD40 activation of B cells prev
151                           Healthy neutrophil Toll-like receptor 9 expression increased upon stimulati
152 stem and microglia/macrophage activation via Toll-like receptor 9 using CpG (cytosine-phosphate-guani
153                                Intracellular Toll-like receptor 9 was induced by costimulation with i
154 -kappaB-->ZEB1 signaling represses the TLR9 (toll-like receptor 9), IFNG, MCP-1 (monocyte chemoattrac
155                             CD40 ligand- and Toll-like receptor 9-mediated signaling were less strong
156 or prostate cancer cells stably adorned with Toll-like receptor-9 ligand-loaded particles using strep
157                                              Toll-like receptor agonists are potent enhancers of inna
158  Western diet or by applying topical TLR7/8 (Toll-like receptor) agonists.
159 hypersensitivity accompanied by increases in Toll-like receptor and cytokine gene expression in the s
160 mmune response, leading to activation of the Toll-like receptor and NF-kappaB pathways.
161 her, it is critical for activating endosomal toll-like receptors and antiviral humoral immunity.
162                                Expression of Toll-like receptors and cellular adhesion molecules were
163  (MyD88) is an adaptor protein that mediates Toll-like receptors and interleukin-1 receptor signaling
164 tivated kinase 1 (TAK1) is a key mediator of toll-like receptors and pro-inflammatory cytokine signal
165  associated with its antagonistic effects on Toll-like receptors and suppression of RhoA GTPase signa
166                       These pathways include Toll-like receptors and the recently described cGAS-STIN
167 eceptors, including CD40, BAFF receptor, and Toll-like receptors, and also plays a critical role inhi
168  the innate immune system, via inhibition of Toll-like receptor- and complement receptor-mediated act
169 d by the induction of chemokines, cytokines, Toll-like receptors, antimicrobial peptides, monocytoid
170 thway activation in SS through activation of Toll-like receptor-dependent and -independent pathways.
171 nt work showing that a combinatorial code of Toll-like receptors downstream of pair-rule genes contri
172 ally upregulated by ligands of IL-1 receptor/Toll-like receptor family members via the activation of
173                                      Indeed, Toll-like receptors, G-protein-coupled receptors, integr
174 tivation of innate immune components such as Toll-like receptors, IL-1 receptor-associated kinase/tum
175 adhesion and signaling, including integrins, Toll-like receptors, immunoglobulins, and mutant myocili
176                                              Toll-like receptors in alveolar macrophages (AMPhi) reco
177         Although C-type lectin receptor- and Toll-like receptor-induced signaling pathways are key ac
178 osis factor secretion after stimulation with Toll-like receptor ligands and M. tuberculosis whole-cel
179 25 plus IL-33 (IL-25/IL-33), or a mixture of Toll-like receptor ligands to evaluate their ability to
180 s whereas exposure to innate stimuli such as Toll-like receptor ligands was not sufficient.
181           Activation of this reporter, using Toll-like receptor ligands, resulted in GFP expression,
182  cells migrate to lymph nodes and respond to toll-like receptor ligation; however, they differ marked
183 at wild-type pigs display both a basal and a Toll-like receptor-mediated ASL secretory response to th
184 atum orchestrates a molecular network of the Toll-like receptor, microRNAs, and autophagy to clinical
185 tection of circulating microbial products by Toll-like receptors on T cells, and this regulation is c
186 tor of interferon genes (STING), but not the Toll-like receptor or the mitochondrial antiviral-signal
187                  ICs co-localized with these toll-like receptor pathway proteins.
188                                              Toll-like receptors play a central role in the initiatio
189 R2-/- and TLR4-/- mice that are defective in toll like receptor signaling.
190                                Moreover, the toll-like receptor signaling adaptor protein TRIF (TIR-d
191                                     Impaired toll-like receptor signaling by the HBV surface antigen
192                     In contrast, stimulating Toll-like receptor signaling in medaka enhanced immune c
193 ve low-dose naltrexone influencing opioid or toll-like receptor signaling to improve calcium mobiliza
194 ession is not driven by adaptive immunity or toll-like receptor signaling, and that BabA may have oth
195 ways induced by the LPS stimulation included toll-like receptor signaling, IL-6 signaling, and Nrf2-m
196 regulates innate immune responses, including Toll-like receptor signaling, which initiate adaptive im
197 ccompanied by elevated activation of TCR and Toll-like receptor signaling-related proteins.
198 ation of the tumor-associated microbiota and toll-like receptor signaling.
199 RNAs are estrogen responsive and target TLR (toll-like receptor) signaling pathways.
200 o-inflammatory priming with phorbol ester or Toll-like receptor stimulation.
201 aling pathways initiated by IL-1, IL-18, and toll-like receptors, the precise contribution of MyD88 t
202                                              Toll-like receptor (Tlr) 13(-/-) BMDCs showed a weak res
203           In the present study, the roles of toll-like receptor (TLR) 2, TLR4 and MyD88, in exacerbat
204                       Therefore, the role of Toll-like receptor (TLR) 2, TLR4, myeloid differentiatio
205 ate molecules reportedly engage and activate Toll-like receptor (TLR) 4 and other TLRs, yet the inter
206 eversed the upregulation of calprotectin and Toll-like receptor (TLR) 4 in inflamed skin.
207 hma was induced in C57BL/6 J wild-type mice, Toll-like receptor (TLR) 4 knockout (Tlr4(-/-)) mice, an
208 sponse that was independent of both upstream Toll-like receptor (TLR) 4 signaling and downstream type
209 g distinct lung APC subsets or cell-specific Toll-like receptor (TLR) 4 signaling.
210                                    LPS binds Toll-like receptor (TLR) 4, which leads to the release o
211                                              Toll-like receptor (TLR) 5 binding was assessed with HEK
212 expression of two closely related receptors, Toll-like receptor (TLR) 7 and TLR9, is balanced to allo
213              Small-molecule imidazoquinoline Toll-like receptor (TLR) 8 agonists robustly activate ne
214                                              Toll-like receptor (TLR) activation contributes to prema
215                                              Toll-like receptor (TLR) activation stimulates antiviral
216  The TIR domain is able to interact with the Toll-like receptor (TLR) adaptors TIRAP and MyD88, as we
217 zation and activation of the inflammasome by Toll-like receptor (TLR) agonism with bacterial lipopoly
218                                              Toll-like receptor (TLR) agonist adjuvant formulations h
219  nitric oxide synthase (iNOS) mRNA following Toll-like receptor (TLR) agonist treatment.
220 acrophages submitted to oxidative stress and toll-like receptor (TLR) agonist.
221 reased inflammatory cytokines in response to Toll-like receptor (TLR) agonists and lipopolysaccharide
222 ons in combination with several Th1-inducing Toll-like receptor (TLR) agonists in vivo In mice, the T
223 atal innate immune response, for example, to Toll-like receptor (TLR) agonists, can reduce the effica
224  cells, IL-10 can be produced in response to Toll-like receptor (TLR) agonists.
225 other types of dangers through their role in Toll-like receptor (TLR) and interleukin 1 receptor (IL-
226                          Most members of the Toll-like receptor (TLR) and interleukin-1 receptor (IL-
227 onse gene 88 (MyD88), the common adaptor for toll-like receptor (TLR) and Interleukin-1 receptor sign
228 o insulin resistance and type 2 diabetes via Toll-like Receptor (TLR) and TNF-family cytokine recepto
229 s, acting as a negative regulator of ILR and Toll-like receptor (TLR) downstream signalling pathways
230              This study investigates whether Toll-like receptor (TLR) expression and signaling during
231 r immune cell composition by flow cytometry, Toll-like receptor (TLR) expression by quantitative PCR,
232 ith the latter reflected in changes in local Toll-like receptor (TLR) expression.
233 ctivate several members of the transmembrane Toll-like receptor (TLR) family.
234                                              TOLL-like receptor (TLR) ligands activate both innate an
235 blast response to self-antigens that contain Toll-like receptor (TLR) ligands is prominent in murine
236 cally relevant immune-agonists, specifically Toll-like receptor (TLR) ligands, using biodegradable, p
237 1-derived macrophages activated by different toll-like receptor (TLR) ligands.
238                          Cross-regulation of Toll-like receptor (TLR) responses by cytokines is essen
239         Accumulating evidence indicates that Toll-like receptor (TLR) signaling adapter protein inter
240 ed kinase 1 (TAK1) is critical for mediating Toll-like receptor (TLR) signaling and subsequent activa
241                                  The role of Toll-like receptor (TLR) signaling in processing of SLE
242                We determined the function of Toll-like receptor (TLR) signaling on the progression of
243 be a role for alphav integrins in regulating Toll-like receptor (TLR) signalling by modulating intrac
244 ptor protein TRAF6 has a central function in Toll-like receptor (TLR) signalling, yet the molecular m
245 ues are phosphorylated both before and after Toll-like receptor (TLR) stimulation.
246 ns involved in signaling pathways, including Toll-like receptor (TLR), mitogen-activated protein kina
247 HCC because it has critical roles in virus-, Toll-like receptor (TLR)-, and IFN-induced signaling pat
248                                 Dysregulated Toll-like receptor (TLR)-4 activation is involved in acu
249 Escherichia coli lipopolysaccharide (LPS), a Toll-like receptor (TLR)-4 agonist.
250 Extracted monocytes were stimulated with the toll-like receptor (TLR)-4 ligand, lipopolysaccharide (L
251 ne chest wall mammary cancers with a topical toll-like receptor (TLR)-7 agonist, imiquimod.
252 ses to T-cell-independent antigens through a Toll-like receptor (TLR)-amplified pathway involving tra
253                                              Toll-like receptor (TLR)-dependent pathways induce IFN-I
254                                              Toll-like receptor (TLR)-mediated sensing of the microbi
255 ctivation, a host response also critical for Toll-like receptor (TLR)-mediated signaling.
256 n activate CREBH in mouse liver tissues in a toll-like receptor (TLR)/MyD88-dependent manner.
257  of inflammation-associated genes, including toll-like receptor (TLR)2, the receptor for advanced gly
258                                Expression of Toll-like receptor (TLR)2, TLR4, and nuclear factor (NF)
259 ot suppressed by inhibitors of intracellular toll-like receptor (TLR)9.
260                                              Toll-like receptors (TLR) are conserved immune sensors m
261 nterpart, has been achieved using hybridized toll-like receptors (TLR) combining TLR1 and TLR2 onto a
262            Inflammatory mediators binding to Toll-Like receptors (TLR) induce an influx of superoxide
263  survival defect after engagement of CD40 or Toll-like receptors (TLR), despite paradoxically enhance
264 A unifying feature for the two components is Toll-like receptors (TLR), which are key regulators of t
265 -induced cytokine expression is abolished in Toll-like receptor (TLR2)(-/-) bone marrow-derived macro
266                Given the fundamental role of Toll-like receptors (TLRs) and complement in inflammatio
267 le of rescuing gp96 client proteins, such as Toll-like receptors (TLRs) and integrins, in a gp96-defi
268                   Recent work has identified Toll-like receptors (TLRs) and type I interferon (IFN) s
269 vital to rapidly responding to pathogens and Toll-like receptors (TLRs) are a critical component of t
270                                              Toll-like receptors (TLRs) are innate immune receptors f
271                                              Toll-like receptors (TLRs) are major players of the inna
272    In organisms from insects to vertebrates, Toll-like receptors (TLRs) are primary pathogen detector
273 RAK4 or MYD88, which mediate the function of Toll-like receptors (TLRs) except TLR3, contained VH4-34
274                                    Endosomal Toll-like receptors (TLRs) have been shown to be crucial
275 te innate immune activation through specific toll-like receptors (TLRs) in epidermal keratinocytes, a
276               The retention of intracellular Toll-like receptors (TLRs) in the endoplasmic reticulum
277    Therapies based on activation of multiple Toll-like receptors (TLRs) may offer superior therapeuti
278                                              Toll-like receptors (TLRs) play a role in innate immunit
279                                              Toll-like receptors (TLRs) play an important role in B c
280                                              Toll-like receptors (TLRs) play an important role in imm
281                            Ligand binding to Toll-like receptors (TLRs) results in dimerization of th
282 als are sensed and acted upon acutely by the Toll-like receptors (TLRs) to halt proliferation and act
283  critical role in innate immune signaling by Toll-like receptors (TLRs), and loss of IRAK4 activity i
284 tain nucleic acids that can be recognized by Toll-like receptors (TLRs), engulfment of ACs does not i
285 t al. (2016) report that TRIF, an adaptor of Toll-like receptors (TLRs), is essential for STING-media
286 e immunity, involved in signaling by several Toll-like receptors (TLRs), key pattern recognition rece
287                       When activated through toll-like receptors (TLRs), macrophages generate IL-33,
288 n recognition receptors (PRRs), specifically toll-like receptors (TLRs), sense and respond to pathoge
289                           Pathogen-activated Toll-like receptors (TLRs), such as TLR2 and TLR4, dimer
290 Several microglia-related molecules, such as Toll-like receptors (TLRs), the complement system, cytok
291 uction of the expression of Il12 and Il23 by Toll-like receptors (TLRs), which impaired the different
292                       One such family is the Toll-like receptors (TLRs).
293 oxidase pathway modulators and inhibitors of Toll-like receptors (TLRs).
294 production of membrane-associated mucins and Toll-like receptors (TLRs).
295 e 1/2 MAP kinase signaling pathway following Toll-like receptor, TNFR1, and IL-1R stimulation.
296 ficient mice, by which microbiota signal via Toll-like receptors to elicit IgD CSR.
297 ) mice) and mice defective in trafficking of Toll-like receptors to the endosome (Unc93b1(-/-) mice).
298 (myeloid differentiation factor 88) or TLRs (Toll-like receptors), we demonstrate that TLR2 and TLR6
299 es for proinflammatory cytokines and various toll-like receptors were overexpressed in SAH neutrophil
300 mma on stimulation of dectin-1, mannose, and Toll-like receptors with Candida albicans and Staphyloco

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