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1 flammatory disease through the activation of toll-like receptors.
2 ell receptor (BCR), Fc receptors (FcRs), and toll-like receptors.
3 s a macrophage immunosuppressant by reducing Toll-like receptor 1/2 (TLR1/2) activation by bacterial
5 ls of pattern recognition receptors, such as Toll-like receptors 1 and 2, dectin 1, and dendritic cel
6 s trigger pro-inflammatory responses through Toll-like receptor 2 (TLR2) activation, and this whether
7 antibodies were used to dissect the role of Toll-like receptor 2 (TLR2) and programmed death-ligand
8 ed, albeit the recognition of lipoglycans by Toll-like receptor 2 (TLR2) appears to be important for
13 l" anti-inflammatory phenotype by activating Toll-like receptor 2 (TLR2), which regulates the inducti
14 he Brucella effector protein TcpB suppresses Toll-like receptor 2 (TLR2)- and TLR4-mediated innate im
15 n of macrophages derived from MyD88-, TRIF-, Toll-like receptor 2 (TLR2)-, TLR4-, and TLR2/4-deficien
16 n induces pathogenic host inflammation via a Toll-like receptor 2 (TLR2)-dependent pathway, resulting
18 stimulates the innate immune system through Toll-like receptor 2 (TLR2); however, the pathogen-assoc
20 eneration and elevated surface expression of toll-like receptor 2 and CD11b on monocytes and neutroph
22 IFN-kappa was significantly increased after toll-like receptor 2 and UVB treatment in lupus keratino
24 e skin microbiome is a rich source of LTA, a Toll-like receptor 2 ligand, we mimicked the GF microbio
26 bed flow and of neutrophils, hyaluronan, and Toll-like receptor 2 ligation in superficial intimal inj
27 implicate flow disturbance, neutrophils, and Toll-like receptor 2 signaling as mechanisms that contri
28 h healthy control subjects after exposure to toll-like receptor 2, 3, or 4 agonists or exposure to UV
29 r membrane of A. muciniphila, interacts with Toll-like receptor 2, is stable at temperatures used for
31 his article, we show that germ-free (GF) and Toll-like receptor-2 (Tlr2)-deficient mice have reduced
32 igh-iron conditions had reduced responses to Toll-like receptor-2, -3, and -4 agonists, which associa
33 olyinosine-polycytidylic acid (poly(I:C)), a Toll-like receptor 3 (TLR3) agonist used as a mimetic to
36 merous interferon-regulated genes, including Toll-like receptor 3 (Tlr3), which encodes an innate imm
37 mal organization to the selective control of toll-like receptor 3 (TLR3)- and TLR4-mediated proinflam
42 We sought to evaluate an inhibitory mAb to Toll-like receptor 3, CNTO3157, on experimental HRV-16 i
45 NA levels were assessed by real-time PCR and Toll like receptor 4 (TLR-4) protein expression by Weste
48 increase in TJ permeability was mediated by toll-like receptor 4 (TLR-4)/MyD88 signal-transduction p
50 e microbiota, hematopoietic cell deletion of Toll-like receptor 4 (TLR4) and inactivation of the IL-1
51 with a decrease in the expression of mucosal toll-like receptor 4 (TLR4) and its adaptor myeloid diff
53 the combination of synthetic small-molecule Toll-like receptor 4 (TLR4) and TLR7 ligands is a potent
54 ntigens adjuvanted with ligands specific for Toll-like receptor 4 (TLR4) and TLR7/8 encapsulated in p
55 The results point to modified signaling via Toll-like receptor 4 (TLR4) as a possible mechanism for
56 further implicate the innate immune receptor toll-like receptor 4 (TLR4) as an underlying mechanism m
59 Ps) induced NF-kappaB activation mediated by Toll-like receptor 4 (TLR4) in a glycoprotein (GP)-depen
65 morphine binds to the innate immune receptor toll-like receptor 4 (TLR4) localized primarily on micro
70 ow that GOS is recognized by and upregulates Toll-like receptor 4 (TLR4) on RAW264.7 macrophages, fol
71 entiation revealed that binding of S100A9 to Toll-like receptor 4 (TLR4) promotes activation of p38 m
72 emonstrate that hRetn binds the LPS receptor Toll-like receptor 4 (TLR4) through its N terminal and m
73 ent of Gram-negative bacteria that activates Toll-like receptor 4 (TLR4) to trigger proinflammatory r
74 300b, and its adaptor DAP12, associated with Toll-like receptor 4 (TLR4) upon LPS binding, thereby en
77 -NS5A in liver up-regulate the expression of Toll-like receptor 4 (TLR4), and develop liver tumors co
78 o found to bind TRAM, an adaptor protein for Toll-like receptor 4 (TLR4), and thereby impact both vir
80 ytoplasm and downregulated the expression of toll-like receptor 4 (TLR4), receptor for advanced glyca
81 gle study revealed a new complex composed of Toll-like receptor 4 (TLR4), TLR6, and CD36 induced by f
82 del of PHH, we demonstrate that IVH causes a Toll-like receptor 4 (TLR4)- and NF-kappaB-dependent inf
84 EL induced PTX3 expression by activating the Toll-like receptor 4 (TLR4)-dependent pathway via nuclea
85 ct ligations or sham surgeries on C57BL/6 or toll-like receptor 4 (TLR4)-knockout mice to induce live
86 ages involves multiple mechanisms, including Toll-like receptor 4 (TLR4)-mediated NADPH oxidase (NOX)
87 f opioids, and we recently demonstrated that Toll-like receptor 4 (TLR4)-mediated neuroinflammation i
89 ue damage and expressed by tumors, activates toll-like receptor 4 (TLR4)-mediated sterile inflammatio
92 ts demonstrate that the use of the synthetic Toll-like receptor 4 agonist glucopyranosyl lipid A in s
93 reduced lipopolysaccharide activation of the toll-like receptor 4 and increased survival times compar
94 evated hepatic stellate cell-derived TnC and Toll-like receptor 4 expression was observed in the diet
99 resistant to Fas-mediated apoptosis ex vivo, Toll-like receptor 4(TLR4)-ligation restored Fas-sensiti
101 also attenuated proinflammatory signaling by Toll-like receptor 4, which has a central role in Ad pat
102 nd SseK3 suppress TNF-alpha-induced, but not Toll-like receptor 4- or interleukin-induced, NF-kappaB
103 failed to induce depressive-like behavior in Toll-like receptor 4-deficient mice and in mice harborin
104 rass pollen or mite allergens to enhance the Toll-like receptor 4-mediated proallergic properties of
111 livery system, and simultaneously, activated Toll-Like Receptor-4 (TLR-4) on APCs to release chemokin
115 so improved the level of HA receptors (CD44, Toll-like receptor-4, and receptor for HA-mediated motil
118 ellin:allergen fusion protein containing the Toll-like receptor 5 ligand flagellin A from Listeria mo
126 e isolated from whole blood, stimulated with Toll-like receptor 7 agonist, and analyzed by means of e
128 it, colony stimulating factor 3 receptor and toll-like receptor 7 mRNA expression increases in the th
129 application of Aldara cream, containing the Toll-like receptor 7/8 agonist Imiquimod, is a widely us
130 ammatory conditions, exposure of APCs to the Toll-like receptor 7/8 agonist R848 increases nanocluste
135 rich in unmethylated CG motifs (CpGs) engage Toll-Like Receptor 9 (TLR-9) in endosomes and are well d
136 re cultured with CD40 ligand (CD40L) and the Toll-like receptor 9 (TLR9) agonist cytidine-phosphate-g
137 thesized that a single drug molecule-a novel Toll-like receptor 9 (TLR9) agonist, MGN1703-could funct
138 ly, we identify parasite DNA-sensing through Toll-like receptor 9 (TLR9) along with inflammatory cyto
140 a key role in recognition, as highlighted by Toll-like receptor 9 (TLR9) in the endosomal compartment
143 responsiveness of paired lung fibroblasts to Toll-like receptor 9 (TLR9) stimulation by CpG-oligodeox
145 FcvarepsilonRI receptor for IgE, followed by Toll-like receptor 9 (TLR9)-mediated sensing of DNA in p
148 wed that stimulation of innate immunity with Toll-like receptor 9 agonist, class B CpG (cytosine-phos
152 stem and microglia/macrophage activation via Toll-like receptor 9 using CpG (cytosine-phosphate-guani
154 -kappaB-->ZEB1 signaling represses the TLR9 (toll-like receptor 9), IFNG, MCP-1 (monocyte chemoattrac
156 or prostate cancer cells stably adorned with Toll-like receptor-9 ligand-loaded particles using strep
159 hypersensitivity accompanied by increases in Toll-like receptor and cytokine gene expression in the s
161 her, it is critical for activating endosomal toll-like receptors and antiviral humoral immunity.
163 (MyD88) is an adaptor protein that mediates Toll-like receptors and interleukin-1 receptor signaling
164 tivated kinase 1 (TAK1) is a key mediator of toll-like receptors and pro-inflammatory cytokine signal
165 associated with its antagonistic effects on Toll-like receptors and suppression of RhoA GTPase signa
167 eceptors, including CD40, BAFF receptor, and Toll-like receptors, and also plays a critical role inhi
168 the innate immune system, via inhibition of Toll-like receptor- and complement receptor-mediated act
169 d by the induction of chemokines, cytokines, Toll-like receptors, antimicrobial peptides, monocytoid
170 thway activation in SS through activation of Toll-like receptor-dependent and -independent pathways.
171 nt work showing that a combinatorial code of Toll-like receptors downstream of pair-rule genes contri
172 ally upregulated by ligands of IL-1 receptor/Toll-like receptor family members via the activation of
174 tivation of innate immune components such as Toll-like receptors, IL-1 receptor-associated kinase/tum
175 adhesion and signaling, including integrins, Toll-like receptors, immunoglobulins, and mutant myocili
178 osis factor secretion after stimulation with Toll-like receptor ligands and M. tuberculosis whole-cel
179 25 plus IL-33 (IL-25/IL-33), or a mixture of Toll-like receptor ligands to evaluate their ability to
182 cells migrate to lymph nodes and respond to toll-like receptor ligation; however, they differ marked
183 at wild-type pigs display both a basal and a Toll-like receptor-mediated ASL secretory response to th
184 atum orchestrates a molecular network of the Toll-like receptor, microRNAs, and autophagy to clinical
185 tection of circulating microbial products by Toll-like receptors on T cells, and this regulation is c
186 tor of interferon genes (STING), but not the Toll-like receptor or the mitochondrial antiviral-signal
193 ve low-dose naltrexone influencing opioid or toll-like receptor signaling to improve calcium mobiliza
194 ession is not driven by adaptive immunity or toll-like receptor signaling, and that BabA may have oth
195 ways induced by the LPS stimulation included toll-like receptor signaling, IL-6 signaling, and Nrf2-m
196 regulates innate immune responses, including Toll-like receptor signaling, which initiate adaptive im
201 aling pathways initiated by IL-1, IL-18, and toll-like receptors, the precise contribution of MyD88 t
205 ate molecules reportedly engage and activate Toll-like receptor (TLR) 4 and other TLRs, yet the inter
207 hma was induced in C57BL/6 J wild-type mice, Toll-like receptor (TLR) 4 knockout (Tlr4(-/-)) mice, an
208 sponse that was independent of both upstream Toll-like receptor (TLR) 4 signaling and downstream type
212 expression of two closely related receptors, Toll-like receptor (TLR) 7 and TLR9, is balanced to allo
216 The TIR domain is able to interact with the Toll-like receptor (TLR) adaptors TIRAP and MyD88, as we
217 zation and activation of the inflammasome by Toll-like receptor (TLR) agonism with bacterial lipopoly
221 reased inflammatory cytokines in response to Toll-like receptor (TLR) agonists and lipopolysaccharide
222 ons in combination with several Th1-inducing Toll-like receptor (TLR) agonists in vivo In mice, the T
223 atal innate immune response, for example, to Toll-like receptor (TLR) agonists, can reduce the effica
225 other types of dangers through their role in Toll-like receptor (TLR) and interleukin 1 receptor (IL-
227 onse gene 88 (MyD88), the common adaptor for toll-like receptor (TLR) and Interleukin-1 receptor sign
228 o insulin resistance and type 2 diabetes via Toll-like Receptor (TLR) and TNF-family cytokine recepto
229 s, acting as a negative regulator of ILR and Toll-like receptor (TLR) downstream signalling pathways
231 r immune cell composition by flow cytometry, Toll-like receptor (TLR) expression by quantitative PCR,
235 blast response to self-antigens that contain Toll-like receptor (TLR) ligands is prominent in murine
236 cally relevant immune-agonists, specifically Toll-like receptor (TLR) ligands, using biodegradable, p
240 ed kinase 1 (TAK1) is critical for mediating Toll-like receptor (TLR) signaling and subsequent activa
243 be a role for alphav integrins in regulating Toll-like receptor (TLR) signalling by modulating intrac
244 ptor protein TRAF6 has a central function in Toll-like receptor (TLR) signalling, yet the molecular m
246 ns involved in signaling pathways, including Toll-like receptor (TLR), mitogen-activated protein kina
247 HCC because it has critical roles in virus-, Toll-like receptor (TLR)-, and IFN-induced signaling pat
250 Extracted monocytes were stimulated with the toll-like receptor (TLR)-4 ligand, lipopolysaccharide (L
252 ses to T-cell-independent antigens through a Toll-like receptor (TLR)-amplified pathway involving tra
257 of inflammation-associated genes, including toll-like receptor (TLR)2, the receptor for advanced gly
261 nterpart, has been achieved using hybridized toll-like receptors (TLR) combining TLR1 and TLR2 onto a
263 survival defect after engagement of CD40 or Toll-like receptors (TLR), despite paradoxically enhance
264 A unifying feature for the two components is Toll-like receptors (TLR), which are key regulators of t
265 -induced cytokine expression is abolished in Toll-like receptor (TLR2)(-/-) bone marrow-derived macro
267 le of rescuing gp96 client proteins, such as Toll-like receptors (TLRs) and integrins, in a gp96-defi
269 vital to rapidly responding to pathogens and Toll-like receptors (TLRs) are a critical component of t
272 In organisms from insects to vertebrates, Toll-like receptors (TLRs) are primary pathogen detector
273 RAK4 or MYD88, which mediate the function of Toll-like receptors (TLRs) except TLR3, contained VH4-34
275 te innate immune activation through specific toll-like receptors (TLRs) in epidermal keratinocytes, a
277 Therapies based on activation of multiple Toll-like receptors (TLRs) may offer superior therapeuti
282 als are sensed and acted upon acutely by the Toll-like receptors (TLRs) to halt proliferation and act
283 critical role in innate immune signaling by Toll-like receptors (TLRs), and loss of IRAK4 activity i
284 tain nucleic acids that can be recognized by Toll-like receptors (TLRs), engulfment of ACs does not i
285 t al. (2016) report that TRIF, an adaptor of Toll-like receptors (TLRs), is essential for STING-media
286 e immunity, involved in signaling by several Toll-like receptors (TLRs), key pattern recognition rece
288 n recognition receptors (PRRs), specifically toll-like receptors (TLRs), sense and respond to pathoge
290 Several microglia-related molecules, such as Toll-like receptors (TLRs), the complement system, cytok
291 uction of the expression of Il12 and Il23 by Toll-like receptors (TLRs), which impaired the different
297 ) mice) and mice defective in trafficking of Toll-like receptors to the endosome (Unc93b1(-/-) mice).
298 (myeloid differentiation factor 88) or TLRs (Toll-like receptors), we demonstrate that TLR2 and TLR6
299 es for proinflammatory cytokines and various toll-like receptors were overexpressed in SAH neutrophil
300 mma on stimulation of dectin-1, mannose, and Toll-like receptors with Candida albicans and Staphyloco
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