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1 Male C57BL/6, wild-type, toll-like receptor 2-/-.
2 as pathogen-associated molecular patterns by Toll-like receptor 2.
3 C. glutamicumDeltamptD mutants, to activate Toll-like receptor 2.
4 feri was mediated primarily by activation of Toll-like receptor 2.
5 bs specific for the innate immunity receptor Toll-like receptor 2.
6 tivation of antigen-presenting cells through Toll-like receptor 2.
7 , heat shock protein 70 also signals through Toll-like receptor 2.
8 PILAM, mediates its biological activity via Toll-like receptor 2.
9 receptors expressed by macrophages, CD36 and toll-like receptor 2.
10 wide variety of pathogens likely detected by Toll-like receptor 2.
11 l stimulated HEK-293 cells expressing either Toll-like receptor 2/1 (TLR2/1) or TLR2/6, but only HspX
14 r or father, IL-10 production in response to Toll-like receptor 2, 3, and 4 agonists, ovalbumin, salm
15 h healthy control subjects after exposure to toll-like receptor 2, 3, or 4 agonists or exposure to UV
16 igh-iron conditions had reduced responses to Toll-like receptor-2, -3, and -4 agonists, which associa
19 Histone toxicity on glomeruli ex vivo was Toll-like receptor 2/4 dependent, and lack of TLR2/4 att
22 treatment with a synergistic combination of Toll-like receptor 2/6 (TLR 2/6) and TLR9 agonists (Pam2
27 Interestingly, maturation of MDLC with a toll-like receptor 2 agonist or transforming growth fact
29 2, and IFNgamma upon stimulation with Con A, Toll-like receptor 2 agonist, or anti-CD3 antibodies.
32 nting altered surface exposure or release of Toll-like receptor 2 agonists during rapid growth of Fra
33 or necrosis factor-alpha) following specific Toll-like receptor 2 and 4 (TLR-2/TLR-4) agonist challen
34 eneration and elevated surface expression of toll-like receptor 2 and CD11b on monocytes and neutroph
38 s with previously identified risk alleles in Toll-like receptor 2 and TIRAP was associated with an ad
39 as compared to activation via heterodimeric Toll-like receptor 2 and Toll-like receptor 1 (TLR2/1) b
40 IFN-kappa was significantly increased after toll-like receptor 2 and UVB treatment in lupus keratino
41 s, LPS-induced surface expression of MHC II, Toll-like receptor-2 and -4 were markedly lower (80%, P
44 rance, as evidenced by the downregulation of Toll-like receptors 2 and 4 and of inflammatory cytokine
45 pneumophila resulted in the up-regulation of Toll-like receptors 2 and 4 and the activation of CD40,
46 nate immune activation and signaling through Toll-like receptors 2 and 4 at early times postinfection
48 on of different forms of Candida albicans by Toll-like receptors 2 and 4 on mononuclear leukocytes ha
50 HIF IL-6 and IL-8 production independent of Toll-like receptors 2 and 4, receptor for advanced glyca
52 and OX40L expression on DCs, independent of Toll-like receptors 2 and 4, the NLRP3 inflammasome, and
55 e inflammatory condition that is mediated by Toll-like receptors 2 and 6 (TLR2 and TLR6) and which in
56 ia activation of the innate immune receptor, Toll-like receptor 2, and subsequent inflammatory cell i
59 en receptors; peptidoglycan, which activates Toll-like receptor 2; and lipopolysaccharide (LPS), whic
61 mmasome and the bacterial lipoprotein sensor Toll-like receptor 2, but not in single knockout mice, d
63 AP)-containing) bacteria to demonstrate that Toll-like receptor 2 can recognize peptidoglycan (PGN).
65 e, the increased arthritis severity of TLR2 (Toll-like receptor 2)-deficient mice was reduced by the
66 e in response is not because of variation in Toll-like receptor 2-dependent activation of the signali
71 ensis infection induces an early increase in Toll-like receptor 2 expression, which facilitates paras
72 ingle-nucleotide polymorphisms (SNPs) in the toll-like receptor 2 gene (TLR2), the toll-like receptor
75 ciently inhibited the expression of IL-8 and Toll-like receptor 2 in M. furfur-infected human keratin
76 s induce the activation of NF-kappaB through Toll-like receptor 2, independent of enzymatic activity.
77 e are capable of inducing interleukin-6 in a Toll-like receptor 2-independent manner, whereas the dat
80 mmensal bacteria by dendritic cells (through toll-like receptor 2), innate immune responses facilitat
83 r membrane of A. muciniphila, interacts with Toll-like receptor 2, is stable at temperatures used for
84 nterferon knockout (IFN-gamma(-/-)), BALB/c, Toll-like receptor 2 knockout (TLR2(-/-)), and C57BL/6 m
85 (C3H/HeJ) or toll-like receptor-2 signaling (toll-like receptor-2 knockouts) had similar effects on p
86 r advanced glycation end products (RAGE) and Toll-like receptor 2, leading to local delivery of monoc
87 e skin microbiome is a rich source of LTA, a Toll-like receptor 2 ligand, we mimicked the GF microbio
89 ity of Mtb-derived membrane vesicles bearing Toll-like receptor 2 ligands, including the lipoproteins
90 bed flow and of neutrophils, hyaluronan, and Toll-like receptor 2 ligation in superficial intimal inj
91 Crohn disease-like Card15 mutation increased Toll-like receptor 2-mediated activation of NF-kappa B-c
92 he ability of the whole organism to activate Toll-like receptor 2-mediated MyD88 signaling in macroph
93 ed production of nitric oxide synthase 2 via Toll-like receptor 2-mediated nuclear factor-kappaB acti
95 e was also a significant survival benefit in toll-like receptor 2-/- mice compared to wild-type mice.
99 ngivalis increased mRNA expressions of NOD2, Toll-like receptor 2, myeloid differentiation primary re
100 d enhance innate immune function and neither toll-like receptor-2 nor toll-like receptor-4 are necess
101 ession of key pattern recognition receptors (Toll-like receptor 2, Nucleotide-binding oligomerization
104 acrophages deficient in expression of either Toll-like receptor 2 or the Toll-like receptor accessory
105 PBMC cytolysis of leukemic cells, partly via Toll-like receptor-2/protein kinase C/nuclear factor-kap
106 ells, and CD14(+) monocytes that coexpressed Toll-like receptor-2, receptor-3, receptor-4, receptor-5
107 Furthermore, dectin-1 receptors rather than Toll-like receptor 2 receptors were shown to be necessar
110 implicate flow disturbance, neutrophils, and Toll-like receptor 2 signaling as mechanisms that contri
111 d by PSA during intestinal inflammation, and Toll-like receptor 2 signaling is required for both Treg
112 as not required for IL-8 production and that Toll-like receptor 2 signaling might be affected in the
114 toll-like receptor-4 signaling (C3H/HeJ) or toll-like receptor-2 signaling (toll-like receptor-2 kno
117 recognition with monoclonal antibody against Toll-like receptor 2 suggests that induction of innate i
118 side a universal T cell helper epitope and a Toll-like receptor 2 targeting lipid moiety to form lipo
119 mune system, the mannose-binding lectin, and Toll-like receptor 2 that both specifies and amplifies t
120 ponse (e.g., tumor necrosis factor alpha and Toll-like receptor 2), the complement system, the respon
122 nts expressed significantly higher levels of Toll-like receptor 2 (TLR-2) and exhibited significantly
123 d to explore the potential interplay between Toll-like receptor 2 (TLR-2) and NOD-2 in joint inflamma
124 er, BDCA1(+) mDCs expressed higher levels of Toll-like receptor 2 (TLR-2) and scavenger receptor A (S
125 s in order to characterize the expression of Toll-like receptor 2 (TLR-2) and TLR-4 and the responses
126 macrophages isolated from mice deficient in Toll-like receptor 2 (TLR-2) but not TLR-4 are resistant
128 ity and was completely inhibited by blocking Toll-like receptor 2 (TLR-2) or depleting its mediator M
130 In this study, we delivered signals through Toll-like receptor 2 (TLR-2) to reinvigorate functionali
131 ls, we conducted a study to test the role of Toll-like receptor 2 (TLR-2), TLR-4, and the cytosolic T
132 duce cell death in peritoneal B1a cells from Toll-like receptor 2 (TLR-2)- or NLRP3 inflammasome-defi
135 f cultured bone marrow cells with ligands to Toll-like receptor 2 ([TLR-2] zymosan) and TLR-9 (ODN M3
140 s trigger pro-inflammatory responses through Toll-like receptor 2 (TLR2) activation, and this whether
142 have reported that it can be an agonist for Toll-like receptor 2 (TLR2) and an antagonist or agonist
143 activate innate immune responses by engaging Toll-like receptor 2 (TLR2) and are highly immunostimula
144 reviously, we have reported that ligation of Toll-like receptor 2 (TLR2) and Dectin 1 on antigen-pres
145 how that two pathogen recognition receptors, Toll-like receptor 2 (TLR2) and dectin-1, recognizing th
147 s and astrocytes through a pathway involving Toll-like receptor 2 (TLR2) and poly(ADP-ribose) polymer
148 antibodies were used to dissect the role of Toll-like receptor 2 (TLR2) and programmed death-ligand
149 e the ability of the innate sensing molecule Toll-like receptor 2 (TLR2) and the signaling molecule M
150 tion was inhibited by blocking antibodies to Toll-like receptor 2 (TLR2) and TLR1, indicating that hu
153 minor reduction in mice doubly deficient in Toll-like receptor 2 (Tlr2) and Tlr4 and normal response
154 naling was TLR-dependent as the knockdown of Toll-like receptor 2 (TLR2) and TLR4 blocked NFkappaB an
157 ratory infections to investigate the role of Toll-like receptor 2 (TLR2) and TLR4 in the host respons
159 Here we show that miR-302b is induced by Toll-like receptor 2 (TLR2) and TLR4 through ERK-p38-NF-
160 lis, a gram-negative bacterium recognized by Toll-like receptor 2 (TLR2) and TLR4, which are expresse
165 ed, albeit the recognition of lipoglycans by Toll-like receptor 2 (TLR2) appears to be important for
168 thelial cell lines, we previously identified Toll-like receptor 2 (TLR2) as the principal epithelial
169 have reported that it can be an agonist for Toll-like receptor 2 (TLR2) as well as an antagonist or
170 t VCAN exerts tolerogenic activities through Toll-like receptor 2 (TLR2) binding, the immunoregulator
172 rlying mechanism revealed that activation of Toll-like receptor 2 (TLR2) by curli fibers is critical
173 mplicated in preventing inflammation through Toll-like receptor 2 (TLR2) by inducing expression of th
175 e found that OMPC and Hib-OMPC engaged human Toll-like receptor 2 (TLR2) expressed in human embryonic
176 ation in HeLa cells, likely due to a lack of Toll-like receptor 2 (TLR2) expression in these cells.
177 neumococcal clearance was not dependent upon Toll-like receptor 2 (TLR2) expression, oxidative stress
178 tantly, this colitis was highly dependent on Toll-like receptor 2 (TLR2) function since it was suppre
182 morphism in the Toll-IL-1 receptor domain of Toll-like receptor 2 (TLR2) has been linked to increased
184 en gram-positive bacterial superantigens and toll-like receptor 2 (TLR2) in health and critical illne
186 ng cluster for the interaction of LipL32 and Toll-like receptor 2 (TLR2) in induced inflammatory resp
187 ycobacterium tuberculosis, to synergize with Toll-like receptor 2 (TLR2) in mediating these responses
197 Although the pattern recognition receptor Toll-like receptor 2 (TLR2) is typically thought to reco
199 l-(l ysyl)3-lysine (Pam3CysSK4), a synthetic Toll-like receptor 2 (TLR2) ligand, that was given at im
203 creened for their ability to either activate Toll-like receptor 2 (TLR2) or TLR4 and to antagonize TL
209 he present study was to assess the effect of Toll-like receptor 2 (TLR2) polymorphisms on susceptibil
210 study continues to explore the plasticity of Toll-like receptor 2 (TLR2) previously described in immu
212 athogen Porphyromonas gingivalis activates a Toll-like receptor 2 (TLR2) response that triggers infla
214 Both interleukin-1 receptor (IL-1R) and toll-like receptor 2 (TLR2) signal via the adapter molec
216 e the most potent microbial agonists for the Toll-like receptor 2 (TLR2) subfamily, and this pattern
218 shed work indicates that the contribution of Toll-like receptor 2 (TLR2) to host resistance during ac
219 ingivalis activate cross talk signaling from Toll-like receptor 2 (TLR2) to the beta2 integrin CD11b/
221 e ability of peptidoglycan (PGN) to activate Toll-like receptor 2 (TLR2) was recently questioned, we
222 olonization are in part due to activation of Toll-like receptor 2 (TLR2), a receptor for lipoproteins
223 crophages to produce TNF-alpha primarily via Toll-like receptor 2 (TLR2), although late pneumococcal
225 es of the putative GXM receptors CD14, CD18, Toll-like receptor 2 (TLR2), and TLR4 in GXM clearance f
227 ns of carboxyalkylpyrroles are recognized by Toll-like receptor 2 (TLR2), but not TLR4 or scavenger r
228 , the microbial pattern recognition receptor toll-like receptor 2 (TLR2), but not TLR4, was necessary
229 ipoproteins activate innate immune cells via Toll-like receptor 2 (TLR2), but TLR2-deficient mice are
230 tool in vivo by analysing the expression of toll-like receptor 2 (TLR2), corresponding to the microg
231 that the innate immune recognition receptor, Toll-like receptor 2 (TLR2), is crucial for inflammatory
232 ond to Pneumocystis murina organisms through Toll-like receptor 2 (TLR2), leading to the nuclear tran
234 osteoclast modulation through engagement of Toll-like receptor 2 (TLR2), though the factors responsi
236 i-SBR immune response in mice and to require Toll-like receptor 2 (TLR2), TLR4, and MyD88 signaling f
237 ggering of mouse peritoneal neutrophils with Toll-like receptor 2 (TLR2), TLR4, and TLR9 ligands, but
238 ional type I IFN receptor but independent of Toll-like receptor 2 (TLR2), TLR4, TLR9, and the adapter
239 connect a key pattern recognition receptor, Toll-like receptor 2 (TLR2), to hyperbilirubinemia-induc
241 lipopeptide Pam3CysSK4, a popular agonist of Toll-like receptor 2 (TLR2), were designed making use of
244 l" anti-inflammatory phenotype by activating Toll-like receptor 2 (TLR2), which regulates the inducti
247 he Brucella effector protein TcpB suppresses Toll-like receptor 2 (TLR2)- and TLR4-mediated innate im
248 n of macrophages derived from MyD88-, TRIF-, Toll-like receptor 2 (TLR2)-, TLR4-, and TLR2/4-deficien
249 and bacterial translocation were assessed in Toll-like receptor 2 (TLR2)-deficient and tumor necrosis
250 ural killer T cells, fails to enhance EAE in Toll-like receptor 2 (TLR2)-deficient mice and, in vitro
251 induces MCP-1 up-regulation in the SLFs via Toll-like receptor 2 (TLR2)-dependent activation of NF-k
252 es in infected macrophages are all driven by Toll-like receptor 2 (TLR2)-dependent activation of the
253 ral pathogen Porphyromonas gingivalis induce Toll-like receptor 2 (TLR2)-dependent macrophage activat
254 d COX2 expression, which were augmented in a Toll-like receptor 2 (TLR2)-dependent manner by macropha
257 n induces pathogenic host inflammation via a Toll-like receptor 2 (TLR2)-dependent pathway, resulting
258 nfected with LASV or with LCMV-WE suppressed Toll-like receptor 2 (TLR2)-dependent proinflammatory cy
259 matory reactions in gingival fibroblasts and Toll-like receptor 2 (TLR2)-dependent secretion of inter
260 ose-dependent NF-kappaB reporter activity in Toll-like receptor 2 (TLR2)-expressing HEK293T cells and
282 own that curli fibrils are recognized by the Toll-like receptor 2 (TLR2)/TLR1 heterodimer complex.
284 ncoding the anti-apoptotic protein BIRC3 and Toll-like receptor 2 (TLR2); and the chromatin modifiers
285 ediated by the pathogen recognition receptor Toll-like receptor 2 (TLR2); furthermore, Legionella ind
286 stimulates the innate immune system through Toll-like receptor 2 (TLR2); however, the pathogen-assoc
288 scle (ASM) cells, we show that activation of toll-like receptor 2 (TLR2; mimicking bacterial infectio
290 ty by increasing pattern recognition through Toll-like receptor-2 (TLR2), and increasing the expressi
291 ast, when we transferred neutrophils lacking Toll-like receptor-2 (TLR2), TLR3, TLR4, TLR7 and TLR9,
292 attern recognition receptor dectin-1 but not Toll-like receptor-2 (TLR2), zymosan-mediated RGC regene
293 his article, we show that germ-free (GF) and Toll-like receptor-2 (Tlr2)-deficient mice have reduced
294 Emerging reports reveal that activating Toll-like receptor-2 (TLR2)-MyD88 signals in CD8 T lymph
296 ced early during IAPP aggregation provided a Toll-like-receptor-2- (TLR2-) dependent stimulus for NF-
297 e I interferon (IFN) signaling downstream of Toll-like receptor 2/Toll-like receptor 4 activation in
298 mphoblastoid cell lines (LCLs) partially via Toll-like receptor 2 triggering, as did purified GAS pep
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