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1 he expression of the innate immune receptor, Toll-like receptor 3.
2 ary and sufficient to stimulate WIHN through toll-like receptor 3.
3 nents to endosomal compartments that contain Toll-like receptor-3.
4 ch no signaling occurs via the intracellular Toll-like receptors 3, 7 and 9 (sensors for double-stran
5 RNA expression of protein kinase R (PKR) and Toll-like receptors 3, 7, and 9 was also measured from c
6 responses to RSV that correlated with lower Toll-like receptor 3/7 transcript and decreased expressi
10 ted, polyinosinic:polycytidylic acid (PIC, a Toll-like receptor 3 agonist) was highly effective as an
11 sion in SeV-infected cells demonstrated that Toll-like receptor 3, although essential for gene induct
15 incorporating FADD is largely independent of Toll-like receptor 3 and the dsRNA-dependent kinase PKR,
16 and IPS-1 adaptor cytosolic pathway and the Toll-like receptor 3 and TIR domain-containing adaptor-i
19 e disease viruses or after engagement of the Toll-like receptors 3 and 4 by double-stranded RNA and l
22 e innate pattern-recognition receptor TLR-3 (Toll-like receptor 3) and resulted in a marked inhibitio
24 re clearly defined with the observation that Toll-like receptor 3 can sense self RNA released from ne
25 We sought to evaluate an inhibitory mAb to Toll-like receptor 3, CNTO3157, on experimental HRV-16 i
26 gative lines were no longer able to induce a Toll-like receptor 3-dependent hyperinflammatory cytokin
29 double-stranded RNA from injured cells with toll-like receptor 3 drives the acute inflammatory respo
32 thimazole markedly decreased virally induced Toll-like receptor-3 expression and signaling and signif
33 otype and increased surface translocation of toll-like receptor 3 from the endosome to the surface.
34 in the toll-like receptor 2 gene (TLR2), the toll-like receptor 3 gene (TLR3), the toll-like receptor
37 ion of double-stranded RNA signaling through Toll-like receptor 3 is mediated by the viral protease N
39 elanoma differentiation associated gene 5 or toll-like receptor 3, is the cytoplasmic sensor for intr
45 on caused by poly(I:C)-induced activation of toll-like receptor 3 localized in intracellular vesicles
46 se (IKK)-epsilon) are critically involved in Toll-like receptor-3-mediated IFN-beta production throug
50 ation of type I IFN responses through either Toll-like receptor 3 or retinoic acid-inducible gene I/m
51 s caused by defects in the activation of the Toll-like receptor 3 pathway, overall contributed to the
54 on, or the binding of double-stranded RNA to Toll-like receptor 3, results in the coordinate activati
56 lication of the HGC, in which we generated a Toll-like receptor 3-specific connectome useful for the
57 ion, and macrophage apoptosis was reduced in toll-like receptor 3 (-/-), TIR-domain-containing adapte
58 Unilateral lung contusion was induced in toll-like receptor 3 (-/-), TIR-domain-containing adapte
60 model of brain inflammation by injecting the Toll-like receptor 3 (TLR-3) agonist polyinosinic:polycy
62 molecule that is critically involved in the Toll-like receptor 3 (TLR-3) and TLR-4 signaling pathway
66 (ISG15) E3 ligase, HERC5, in the context of Toll-like receptor 3 (TLR3) activation and IFN induction
68 -1 but did inhibit responses to poly(I.C), a Toll-like receptor 3 (TLR3) activator that does not sign
69 olyinosine-polycytidylic acid (poly(I:C)), a Toll-like receptor 3 (TLR3) agonist used as a mimetic to
71 are emerging; in particular, dsRNA receptors Toll-like receptor 3 (TLR3) and cytosolic helicases expr
72 of the mRNA to evade activation of endosomal Toll-like receptor 3 (TLR3) and downstream innate immune
73 ich is released from damaged skin, activates Toll-Like Receptor 3 (TLR3) and its downstream effectors
74 Poly(I:C) acts through two dsRNA sensors, Toll-like receptor 3 (TLR3) and melanoma differentiation
77 physical and functional relationship between Toll-like receptor 3 (TLR3) and the pattern recognition
80 UVB-damaged keratinocytes were dependent on Toll-like receptor 3 (TLR3) and Toll-like receptor adapt
81 anded RNA (dsRNA) induces phosphorylation of Toll-like receptor 3 (TLR3) at tyrosine 759 and subseque
82 ter activation in response to stimulation of Toll-like receptor 3 (TLR3) by extracellular double-stra
86 us 1 (HSV-1), children with inborn errors of toll-like receptor 3 (TLR3) immunity are prone to HSV-1
89 N) leads to a reduction in the expression of Toll-like receptor 3 (TLR3) in macrophages from young do
94 86, and CD40) expression and did not inhibit Toll-like receptor 3 (TLR3) ligand [poly(I:C)]-induced m
95 of innate immunity by viral infection or the toll-like receptor 3 (TLR3) ligand on liver regeneration
98 ants impaired IFN-beta production induced by Toll-like receptor 3 (TLR3) or TLR4 agonists but failed
100 ral dsRNA through two distinct pathways; the Toll-like receptor 3 (TLR3) pathway detects dsRNA phagoc
101 d loss-of-function studies, we find that the toll-like receptor 3 (TLR3) pathway enables efficient in
105 a (TNF-alpha) and has been implicated in the Toll-like receptor 3 (TLR3) response to double-stranded
107 tic properties, acts as a novel regulator of Toll-like receptor 3 (TLR3) signaling to interferon (IFN
111 y of RNA interference by directly activating Toll-like receptor 3 (TLR3), a double-stranded RNA immun
112 lock type I interferon signaling mediated by Toll-like receptor 3 (TLR3), a receptor we have previous
113 e 561 mRNA by exogenous dsRNA is mediated by Toll-like receptor 3 (TLR3), and it requires no new prot
114 tremendously when HEK293 cells overexpressed Toll-like receptor 3 (TLR3), and the increased secretion
115 sm through both its membrane-bound receptor, Toll-like receptor 3 (TLR3), as well as cytoplasmic rece
116 agonist that recognizes the cellular sensor Toll-like receptor 3 (TLR3), in regulating Valpha14iNKT
117 Differentiation-associated gene 5 (MDA5) and Toll-Like Receptor 3 (TLR3), induces the TANK-Binding Ki
118 d the modulatory effects of Th2 cytokines on Toll-like receptor 3 (TLR3), interferon-responsive facto
119 egfr1) siRNA suppressed CNV via cell-surface toll-like receptor 3 (TLR3), its adaptor TRIF, and induc
120 erleukin-1 (IL-1)/Toll receptor superfamily, Toll-like receptor 3 (TLR3), recognizes double-stranded
121 his family of pattern recognition receptors, toll-like receptor 3 (TLR3), recognizes viral double-str
122 se HCV dsRNAs also induced the expression of Toll-like receptor 3 (TLR3), retinoic acid-inducible gen
124 ors, respectively, and with the exception of Toll-like receptor 3 (TLR3), signal via the adaptor mole
126 eltaE3L is unaffected by genetic ablation of Toll-like receptor 3 (TLR3), TRIF, TLR9, and MyD88.
128 merous interferon-regulated genes, including Toll-like receptor 3 (Tlr3), which encodes an innate imm
129 mal organization to the selective control of toll-like receptor 3 (TLR3)- and TLR4-mediated proinflam
130 rmore, preexposure of LC to L3 did not alter Toll-like receptor 3 (TLR3)- or TLR4-mediated expression
131 These interferon responses attributed to toll-like receptor 3 (TLR3)-activated Kupffer and liver
134 hat West Nile virus (WNV) is able to inhibit Toll-like receptor 3 (TLR3)-mediated activation of inter
136 nse to viral infection is often triggered by Toll-like receptor 3 (TLR3)-mediated signaling by double
143 g the caspase-11 pathway in vivo with LPS or Toll-like receptor-3 (TLR3) agonist resulted in high mor
149 s well as following exposure to poly(I.C) (a Toll-like receptor 3 [TLR3] stimulus) and 5' poly(U) HCV
151 pothesized that small-molecule activators of toll-like receptor 3, together with external microenviro
152 kinase, 2'5'-oligoadenylate synthetase, and Toll-like receptor 3, transcripts that encode dsRNA-resp
153 is sensed by the human epithelial cells via Toll-like receptor 3, triggering Interferon Regulating F
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