ライフサイエンスコーパス: Toxoplasma gondii

1 or the lytic cycle of the protozoan parasite Toxoplasma gondii.

2 long immunity to virulent acute infection by Toxoplasma gondii.

3 omposition and function in the centrosome of Toxoplasma gondii.

4 a parasite Plasmodium falciparum and related Toxoplasma gondii.

5 obligate intracellular apicomplexan parasite Toxoplasma gondii.

6 es (ROPs) are essential virulence factors of Toxoplasma gondii.

7 onmentally driven infectious disease such as Toxoplasma gondii.

8 autophagy related 5 (Atg5) in resistance to Toxoplasma gondii.

9 s gliding motility and host cell invasion by Toxoplasma gondii.

10 es of this cytokine following challenge with Toxoplasma gondii.

11 the immune system responds to infection with Toxoplasma gondii.

12 (ROP5) expressed by the protozoan parasite, Toxoplasma gondii.

13 we identified candidates for APT activity in Toxoplasma gondii.

14 tion by the pervasive intracellular parasite Toxoplasma gondii.

15 y with the SRS-domain containing proteins of Toxoplasma gondii.

16 roiditis manifests in patients infected with Toxoplasma gondii.

17 nfecting them with the Th1-inducing parasite Toxoplasma gondii.

18 ng retinal infection caused by the parasite, Toxoplasma gondii.

19 n the absence of TLR-mediated recognition of Toxoplasma gondii.

20 d recombination in the apicomplexan parasite Toxoplasma gondii.

21 omote invasion or egress during infection by Toxoplasma gondii.

22 (GBPs) in this process during infection with Toxoplasma gondii.

23 ern that is caused by the protozoan parasite Toxoplasma gondii.

24 horous vacuole of the intracellular parasite Toxoplasma gondii.

25 t protection against the parasitic protozoan Toxoplasma gondii.

26 the SAG family of surface antigens found in Toxoplasma gondii.

27 arasitic pathogens Plasmodium falciparum and Toxoplasma gondii.

28 ution of fatty acid biosynthetic pathways in Toxoplasma gondii.

29 st defense functions of mGBP2 in response to Toxoplasma gondii.

30 cal but distinct roles during infection with Toxoplasma gondii.

31 integral component of the immune response to Toxoplasma gondii.

32 niche for apicomplexan parasites, including Toxoplasma gondii.

33 cycle of the eukaryotic single-cell parasite Toxoplasma gondii.

34 with the brain-dwelling, protozoan parasite, Toxoplasma gondii.

35 parasitic infections in the world, caused by Toxoplasma gondii.

36 s a disease caused by the protozoan parasite Toxoplasma gondii.

37 pathogens, including the protozoan parasite Toxoplasma gondii.

38 sential enzyme in the opportunistic pathogen Toxoplasma gondii.

39 ration of TH1 cells following infection with Toxoplasma gondii.

40 vel tool for visualising F-actin dynamics in Toxoplasma gondii.

41 re infected with the intracellular protozoan Toxoplasma gondii(1).

42 Intriguingly, Toxoplasma gondii, a highly successful apicomplexan para

43 in CNS infections, including infection with Toxoplasma gondii, a protozoan parasite that naturally e

44 During infection with Toxoplasma gondii, a similar population emerged that lim

45 enotypic variation characterizes isolates of Toxoplasma gondii, a ubiquitous zoonotic parasite that s

46 e previously shown that the single allele of Toxoplasma gondii actin depolymerizing factor (TgADF) ha

47 The obligate intracellular protozoan Toxoplasma gondii actively invades mammalian cells and,

48 al pH, was cryotrapped in the active site of Toxoplasma gondii aldolase crystals to high resolution.

49 Toxoplasma gondii AMA1 (TgAMA1) is secreted onto the par

50 Toxoplasma gondii, an apicomplexan parasite prevalent in

51 The cytoskeleton of Toxoplasma gondii, an important human parasite, contains

52 In contrast to L3, live tachyzoites of Toxoplasma gondii, an intracellular parasite, induced pr

53 Toxoplasma gondii, an obligate intracellular parasite of

54 Toxoplasma gondii, an obligate intracellular protozoan p

55 dence of primary gestational infections with Toxoplasma gondii and congenital toxoplasmosis in Austri

56 seases caused by the apicomplexan protozoans Toxoplasma gondii and Cryptosporidium parvum are a major

57 hree protozoan parasites (Trypanosoma cruzi, Toxoplasma gondii and Leishmania major), in which the Gz

58 We compared immune responses to Toxoplasma gondii and MO4 melanoma in DT-treated zDC- an

59 a-mediated elimination of pathogens, such as Toxoplasma gondii and murine norovirus.

60 ctor, restricting tryptophan availability to Toxoplasma gondii and other pathogens in vitro.

61 zes to the apical pole in invasive stages of Toxoplasma gondii and Plasmodium berghei, and apical pos

62 comparative activities of ADF proteins from Toxoplasma gondii and Plasmodium falciparum, the human m

63 e apicoplasts of the apicomplexan parasites, Toxoplasma gondii and Plasmodium falciparum.

64 e intracellular apicomplexan parasites, e.g. Toxoplasma gondii and Plasmodium species, induce an IFNg

65 Apicomplexan parasites Toxoplasma gondii and Plasmodium spp. use latent stages

66 e profilin protein of the protozoan parasite Toxoplasma gondii and regulates IL-12 production by DCs

67 How the protozoan pathogen Toxoplasma gondii and related parasites shuttle proteins

68 The apical complex of Toxoplasma gondii and some other apicomplexans includes

69 dant in inducing T-bet in mice infected with Toxoplasma gondii and that T-bet did not contribute to i

70 Upon infections with Toxoplasma gondii and vaccinia virus, mice with stabiliz

71 morbillivirus, herpesvirus, West Nile virus, Toxoplasma gondii, and Brucella spp.

72 Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter rodentium infections.

73 thogen challenges, including H1N1 influenza, Toxoplasma gondii, and Ebola virus.

74 esistance against a prototypic Th1 pathogen, Toxoplasma gondii, and for the generation of pathogen-sp

75 c cells (DCs) are critical for resistance to Toxoplasma gondii, and infection with this pathogen lead

76 The growth rates of Arabidopsis thaliana, Toxoplasma gondii, and Plasmodium falciparum were inhibi

77 estinalis, Leishmania spp., Plasmodium spp., Toxoplasma gondii, and Trypanosoma spp.

78 ovirus, herpes simplex virus, rubella virus, Toxoplasma gondii, and Zika virus.

79 ggests a role for these cells in response to Toxoplasma gondii antigens, although it remains unclear

80 Here we tested this model using the Toxoplasma gondii apical membrane protein 1 (TgAMA1), wh

81 Plasmodium falciparum and Toxoplasma gondii are widely studied parasites in phylum

82 Using Toxoplasma gondii as a genetic model, we show that a key

83 Here, we use Toxoplasma gondii as a model system to functionally char

84 Here we establish that Toxoplasma gondii aspartyl protease 3 (ASP3) resides in

85 After acute oral infection with Toxoplasma gondii, both WT and Crtam(-/-) mice mounted a

86 rmation of naturally occurring peptides from Toxoplasma gondii bound by HLA-A*02:01.

87 t defense against the intracellular pathogen Toxoplasma gondii, but prior infection with Toxoplasma c

88 function of the motility apparatus of living Toxoplasma gondii by adhering a microsphere to the surfa

89 tance to the intracellular vacuolar pathogen Toxoplasma gondii by inducing the destruction of the par

90 tent and selective inhibitor (compound 1) of Toxoplasma gondii calcium-dependent protein kinase 1 (Tg

91 Reactivation of chronic infection with Toxoplasma gondii can cause life-threatening toxoplasmic

92 The apicomplexan parasite Toxoplasma gondii can cause severe disease in immunocomp

93 It has been proposed that Toxoplasma gondii can cross biological barriers as a mot

94 omplexan parasites Plasmodium falciparum and Toxoplasma gondii cause host cell cytolysis to facilitat

95 hronic infection with the protozoan parasite Toxoplasma gondii causes a nonresolving Th1 myositis wit

96 oplasmosis, pyrazolopyrimidine inhibitors of Toxoplasma gondii CDPK1 demonstrated in vitro and in viv

97 at removal of the autoinhibitory domain from Toxoplasma gondii CDPK1 is not sufficient for kinase act

98 Upon infection, the intracellular parasite Toxoplasma gondii co-opts critical functions of its host

99 is and Acanthamoeba amebic encephalitis with Toxoplasma gondii coinfection.

100 Here, the phenotype of a Toxoplasma gondii conditional mutant impaired in host ce

101 cally raised meat could increase the risk of Toxoplasma gondii contamination of meat.

102 inst intracellular pathogens, especially for Toxoplasma gondii control.

103 The intracellular parasite Toxoplasma gondii converts from a rapidly replicating ta

104 ior exposure to the pathogens Brucella spp., Toxoplasma gondii, Coxiella burnetii, Francisella tulare

105 tumoral injection of an attenuated strain of Toxoplasma gondii (cps) that cannot replicate in vivo an

106 The obligate intracellular parasite Toxoplasma gondii critically relies on host cell invasio

107 The Toxoplasma gondii cyst stage is resistant to drug therap

108 ed that after intraperitoneal infection with Toxoplasma gondii cysts, resident mononuclear phagocytes

109 Genetic experiments in Toxoplasma gondii demonstrate two essential components o

110 ssion of the obligate intracellular parasite Toxoplasma gondii depends on its ability to switch betwe

111 The protozoan parasite Toxoplasma gondii develops within a parasitophorous vacu

112 In this study, we document that Toxoplasma gondii differentiation and reactivation are m

113 In Toxoplasma gondii, disruption of the first, fifth, or si

114 ng fluorescent proteins or targeted genes of Toxoplasma gondii, driven by N. caninum promoters, have

115 contribution of each gene from the parasite Toxoplasma gondii during infection of human fibroblasts.

116 When Toxoplasma gondii egresses from the host cell, glycerald

117 Here, we show that the protozoan pathogen Toxoplasma gondii elicits the production of NETs from hu

118 cus identified in the intracellular parasite Toxoplasma gondii encodes a family of related catalytica

119 Toxoplasma gondii encodes three protein kinase A catalyt

120 The genome of Toxoplasma gondii encodes ubiquitination machinery, but

121 The intracellular parasite Toxoplasma gondii enjoys a wide host range and is adept

122 A Toxoplasma gondii enzyme-linked immunoassay that detects

123 Toxoplasma gondii establishes a chronic infection by for

124 cellular pathogen, the apicomplexan parasite Toxoplasma gondii evades immune system-mediated clearanc

125 d host cell egress by the protozoan parasite Toxoplasma gondii expanded the functional diversity of p

126 In Toxoplasma gondii, extracellular potassium levels and ot

127 In Toxoplasma gondii, fatty acid synthesis via the apicopla

128 ce in the ubiquitous intracellular protozoon Toxoplasma gondii for its natural intermediate host, the

129 Here we show that following acute Toxoplasma gondii gastrointestinal infection of mice, co

130 uous supply of potent killer T cells to curb Toxoplasma gondii growth during latency.

131 Here, we demonstrate that the apicomplexan Toxoplasma gondii harbors homologues of proteins from al

132 The intracellular parasite Toxoplasma gondii has multiple strategies to alter host

133 A chronic infection with the parasite Toxoplasma gondii has previously been shown to protect m

134 Waterborne outbreaks of Toxoplasma gondii have focused attention on the importan

135 ell responses are required for resistance to Toxoplasma gondii; however, the events that lead to prod

136 ROP16 is a protein kinase of Toxoplasma gondii identified in the mouse model as a vir

137 sive neurocognitive assessments and had anti-Toxoplasma gondii immunoglobulin G (anti-Toxo IgG) measu

138 Using the zoonotic parasite Toxoplasma gondii in California, USA as a model for coas

139 e Ca(2+) signaling in the model apicomplexan Toxoplasma gondii In doing so, we took advantage of the

140 lator of autophagy and autophagic killing of Toxoplasma gondii in host cells.

141 elements for detection and immune control of Toxoplasma gondii in mice, but not in humans.

142 Studies in Plasmodium spp. and Toxoplasma gondii in particular have revealed that these

143 ter, cytomegalovirus, Epstein-Barr virus and Toxoplasma gondii in patients with uveitis.

144 intain the latency of chronic infection with Toxoplasma gondii in the brain.

145 lity of CD8+ T cells to control the pathogen Toxoplasma gondii in the brains of chronically infected

146 The population structure of Toxoplasma gondii includes three highly prevalent clonal

147 nd that the intracellular protozoan parasite Toxoplasma gondii induced an early IL-1beta response (wi

148 Chronic infection with Toxoplasma gondii induces a potent resistance against re

149 s during the chronic stage of infection with Toxoplasma gondii induces production of IgG antibodies t

150 is essential for CD8+ T-cell recognition of Toxoplasma gondii infected cells.

151 f immune responses that limit cyst burden in Toxoplasma gondii-infected mice.

152 Treatment of Toxoplasma gondii infection acquired during pregnancy di

153 re we show that GCs are induced during acute Toxoplasma gondii infection and directly control the T c

154 Toxoplasma gondii infection causes substantial morbidity

155 Toxoplasma gondii infection has been described previousl

156 Tfh-like cells were rapidly generated after Toxoplasma gondii infection in mice, but T-bet constrain

157 The course of Toxoplasma gondii infection in rats closely resembles th

158 n, although their role in protection against Toxoplasma gondii infection is not thoroughly understood

159 Intrinsic to Toxoplasma gondii infection is the parasite-induced modu

160 An early hallmark of Toxoplasma gondii infection is the rapid control of the

161 Toxoplasma gondii infection occurs through the oral rout

162 the epigenomic and transcriptomic effects of Toxoplasma gondii infection on human host cells and demo

163 rns of hydrocephalus secondary to congenital Toxoplasma gondii infection were identified and characte

164 immunity exacerbates ileitis induced by oral Toxoplasma gondii infection.

165 osis Study (NCCCTS) have a high incidence of Toxoplasma gondii infection.

166 August 2010 for serologic evidence of recent Toxoplasma gondii infection.

167 3, TLR7, and TLR9, are highly susceptible to Toxoplasma gondii infection.

168 as in response to Listeria monocytogenes and Toxoplasma gondii infection.

169 xpression by CNS-infiltrating T cells during Toxoplasma gondii infection.

170 new insights into the roles of CDPKs during Toxoplasma gondii infection.

171 responses using a mouse model for persistent Toxoplasma gondii infection.

172 The obligate intracellular parasite Toxoplasma gondii infects and persists within neurons of

173 Toxoplasma gondii infects both hematopoietic and nonhema

174 during acute mucosal infection of mice with Toxoplasma gondii, inflammatory monocytes acquire a tiss

175 into host cells through endocytic pathways, Toxoplasma gondii initiates infection at the cell surfac

176 Toxoplasma gondii is a classic model for studying obliga

177 Toxoplasma gondii is a common parasite of humans and ani

178 Toxoplasma gondii is a globally distributed parasite pat

179 Coastal habitat contamination with Toxoplasma gondii is a health risk to humans and marine

180 Toxoplasma gondii is a highly prevalent obligate intrace

181 Toxoplasma gondii is a highly successful protozoan paras

182 d control of HIV type 1 replication, whereas Toxoplasma gondii is a life-threatening opportunistic in

183 The intracellular protozoan parasite Toxoplasma gondii is a major food-borne illness and oppo

184 Toxoplasma gondii is a member of the phylum Apicomplexa

185 Toxoplasma gondii is a protist parasite of warm-blooded

186 Toxoplasma gondii is a protozoan pathogen in the phylum

187 Toxoplasma gondii is a ubiquitous protozoan parasite cap

188 Toxoplasma gondii is a ubiquitous, obligate intracellula

189 Toxoplasma gondii is a widely distributed protozoan path

190 Toxoplasma gondii is a widespread parasite of warm-blood

191 Toxoplasma gondii is a widespread parasitic pathogen tha

192 Toxoplasma gondii is a widespread protozoan parasite inf

193 The obligate intracellular parasite Toxoplasma gondii is able to infect a broad range of hos

194 The obligate intracellular parasite Toxoplasma gondii is able to infect nearly all nucleated

195 Infection with Toxoplasma gondii is acquired through consumption of und

196 Toxoplasma gondii is among the most prevalent parasites

197 Toxoplasma gondii is an apicomplexan parasite that cause

198 The parasite Toxoplasma gondii is an environmentally persistent patho

199 Toxoplasma gondii is an intracellular parasite that caus

200 Toxoplasma gondii is an intracellular parasite that has

201 Toxoplasma gondii is an obligate intracellular parasite

202 Toxoplasma gondii is an obligate intracellular parasite

203 Toxoplasma gondii is an obligate intracellular parasite

204 Toxoplasma gondii is an obligate intracellular parasite

205 Toxoplasma gondii is an obligate intracellular parasite

206 Toxoplasma gondii is an obligate intracellular parasite

207 Toxoplasma gondii is an obligate intracellular parasite

208 Toxoplasma gondii is an obligate intracellular protozoan

209 Toxoplasma gondii is an obligate intracellular protozoan

210 Toxoplasma gondii is an obligate intracellular protozoan

211 Toxoplasma gondii is an obligate, intracellular eukaryot

212 The life cycle of Toxoplasma gondii is complex, with multiple proliferatio

213 Toxoplasma gondii is considered to be one of the most su

214 Cerebral infection with the parasite Toxoplasma gondii is followed by activation of resident

215 ted target of actinonin in P. falciparum and Toxoplasma gondii is FtsH1, a homolog of a bacterial mem

216 Unlike its animal counterparts, Skp1 from Toxoplasma gondii is hydroxylated by an O2-dependent pro

217 The intracellular parasite Toxoplasma gondii is incapable of sterol synthesis and u

218 CD40-mediated autophagic killing of Toxoplasma gondii is known to require TNF-alpha.

219 As an intracellular protozoan parasite, Toxoplasma gondii is likely to exploit proteases for hos

220 Toxoplasma gondii is sensitive to bulky pyrazolo [3,4-d]

221 Toxoplasma gondii is the causative agent of toxoplasmosi

222 Toxoplasma gondii is the model parasite of the phylum Ap

223 The intracellular protozoan Toxoplasma gondii is well known for its skill at invadin

224 gMyoA, a class XIVa myosin from the parasite Toxoplasma gondii, is required for the parasite to effic

225 In mice, infection with Toxoplasma gondii leads to a Th1-polarized parasite-spec

226 Infection by Toxoplasma gondii leads to massive changes to the host c

227 Toxoplasma gondii, like all apicomplexan parasites, uses

228 defense against the intracellular pathogens Toxoplasma gondii, Listeria monocytogenes, and Mycobacte

229 in the context of three distinct infections: Toxoplasma gondii, Listeria monocytogenes, and vaccinia

230 nfections with pathogens such as Zika virus, Toxoplasma gondii, Listeria monocytogenes, Treponema pal

231 in, we produced recombinant histidine-tagged Toxoplasma gondii MIF (TgMIF), a 12-kDa protein that lac

232 displayed potent antiparasitic activity in a Toxoplasma gondii model.

233 Toxoplasma gondii modifies its host cell to suppress its

234 , including Bacillus anthracis lethal toxin, Toxoplasma gondii, muramyl dipeptide, and host intracell

235 he founding member of the class XIVa family, Toxoplasma gondii myosin A (TgMyoA), is a monomeric unco

236 ases of invasive protozoal infections due to Toxoplasma gondii (n = 3), Trypanosoma cruzi, and Leishm

237 acellular and promiscuous protozoan parasite Toxoplasma gondii needs an extensive membrane biogenesis

238 ed to functions within pathways/networks for Toxoplasma gondii, Neospora caninum, Cryptosporidium and

239 Indeed, in mice challenged with the parasite Toxoplasma gondii, NK and T cell responses are character

240 Natural infection by Toxoplasma gondii occurs via oral ingestion of tissue cy

241 deliver a double punch that can destroy the Toxoplasma gondii parasite and its niche inside cells.

242 ed immunosorbent assay (ELISA) distinguishes Toxoplasma gondii parasite types (II and not exclusively

243 Toxoplasma gondii parasites must actively invade host ce

244 Bradyzoite forms of Toxoplasma gondii persist in tissue cysts for the lifeti

245 Toxoplasma gondii persistently infects over two billion

246 medical and veterinary importance, including Toxoplasma gondii, Plasmodium falciparum, and C. parvum

247 aled that T cell responses generated against Toxoplasma gondii, Plasmodium sp., and Leishmania sp. ca

248 Toxoplasma gondii possesses a bifunctional farnesyl diph

249 nfecting mice with the Th1-inducing parasite Toxoplasma gondii prior to highly pathogenic avian H5N1

250 The intracellular parasite Toxoplasma gondii produces two nucleoside triphosphate d

251 Apicomplexan parasites such as Toxoplasma gondii rely on a unique form of locomotion kn

252 Toxoplasma gondii replicates asexually by a unique inter

253 TRIM21 as a previously unknown modulator of Toxoplasma gondii resistance in vivo thereby extending h

254 smodium falciparum, the agent of malaria, or Toxoplasma gondii, responsible for human toxoplasmosis,

255 mucosal responses to the protozoan parasite Toxoplasma gondii resulted in dysbiosis and the eliminat

256 ted in a 100% reduction in the recurrence of Toxoplasma gondii retinochoroiditis over 1 year of treat

257 from Campinas, Brazil, with active recurrent Toxoplasma gondii retinochoroiditis were included.

258 A recent study in Toxoplasma gondii revealed a unique bipartite structure

259 The reduction in invasiveness of the Toxoplasma gondii RH-AMA1 knockout (RH-AMA1(KO)) tachyzo

260 Toxoplasma gondii serology demonstrated exposure.

261 Primary infection with Toxoplasma gondii stimulates production of high levels o

262 Transmissible stages of Toxoplasma gondii store energy in the form of the carboh

263 displayed superb antiparasitic activity in a Toxoplasma gondii strain that relies on CpIMPDH (EC50 =

264 iments performed on the rat pineal gland and Toxoplasma gondii, successfully detecting known and prev

265 Toxoplasma gondii (T. gondii) is a neurotropic protozoan

266 Toxoplasma gondii (T. gondii) is an apicomplexan parasit

267 ting uracil auxotroph strain of the parasite Toxoplasma gondii (T. gondii), which preferentially inva

268 oites of the apicomplexan protozoan parasite Toxoplasma gondii (T. gondii).

269 y Pneumocystis jirovecii (P. jirovecii, pj), Toxoplasma gondii (T. gondii, tg), and Mycobacterium avi

270 ugs, a compound library was screened against Toxoplasma gondii tachyzoites with activity discovered f

271 3 in the parasites of this phylum, including Toxoplasma gondii (TgElp3), possesses a unique C-termina

272 is, but the enzyme of the protozoan parasite Toxoplasma gondii (TgPBGS) differs from that of its huma

273 olar proton pyrophosphatase (H(+) -PPase) of Toxoplasma gondii (TgVP1), a membrane proton pump, local

274 n infection caused by the protozoan parasite Toxoplasma gondii that can lead to severe sequelae in th

275 s to construct a comprehensive genealogy for Toxoplasma gondii that incorporates sexual recombination

276 In Dictyostelium (a social amoeba), Toxoplasma gondii (the agent for human toxoplasmosis), a

277 In apicomplexan parasites such as Toxoplasma gondii, the apical complex includes a spiral

278 Toxoplasma gondii, the causative agent of toxoplasmosis,

279 Toxoplasma gondii, the causative agent of toxoplasmosis,

280 Toxoplasma gondii, the most common parasitic infection o

281 As a major natural host for Toxoplasma gondii, the mouse is widely used for the stud

282 In the ubiquitous protozoan parasite Toxoplasma gondii, the oocyst stage possesses a bilayere

283 use pathogens, Heligmosomoides polygyrus and Toxoplasma gondii, to investigate the negative impact of

284 use pathogens, Heligsomosoides polygyrus and Toxoplasma gondii, to investigate the negative impact of

285 Toxoplasma gondii transmission between intermediate host

286 It is believed that infection by Toxoplasma gondii triggers a lifelong protective immunit

287 d that systemic infection with the protozoan Toxoplasma gondii triggers not only a transient increase

288 complexa phylum, such as Plasmodium spp. and Toxoplasma gondii, undergo complex life cycles involving

289 ne the polymerization properties of actin in Toxoplasma gondii, unexpectedly finding that it exhibits

290 e to reactivation of cerebral infection with Toxoplasma gondii using a murine model.

291 pes 1 and 2, Mycobacterium tuberculosis, and Toxoplasma gondii via real-time polymerase chain reactio

292 The Toxoplasma gondii virulence factors ROP5 and ROP18 both

293 Toxoplasma gondii was not detected in field-collected sn

294 ng pathways that regulate these processes in Toxoplasma gondii We found that microneme secretion was

295 Using Toxoplasma gondii, we establish that the pathway is esse

296 Salmonella typhimurium or Toxoplasma gondii were administered to knockout (KO) mic

297 Negative results for Toxoplasma gondii were associated with a higher prevalen

298 ded in apicomplexan parasites, especially in Toxoplasma gondii where 14 separate genes encoding these

299 is infected with the intracellular parasite Toxoplasma gondii, which contributes significantly to th

300 The survival of Toxoplasma gondii within its host cell requires protein