ライフサイエンスコーパス: Trichinella spiralis

1 ctor phase of a secondary immune response to Trichinella spiralis.

2 zing hepatitis following oral infection with Trichinella spiralis.

3 racterize cellular responses to muscle-stage Trichinella spiralis.

4 yperplasia in the mouse after infection with Trichinella spiralis.

5 the course (0 to 289 h) of an infection with Trichinella spiralis.

6 We report a draft genome sequence of Trichinella spiralis, a food-borne zoonotic parasite, wh

7 The mouse is a natural host for Trichinella spiralis, a worm that establishes chronic in

8 polygyrus, Nippostrongylus brasiliensis, and Trichinella spiralis) alters intestinal epithelial cell

9 ated with significantly delayed expulsion of Trichinella spiralis and increased deposition of muscle

10 pulsion of two other gut-dwelling nematodes (Trichinella spiralis and Nippostrongylus brasiliensis).

11 due were detected in the parasitic nematodes Trichinella spiralis and Trichinella pseudospiralis.

12 p necrotizing hepatitis after infection with Trichinella spiralis, and inflammation is dependent on t

13 We used a natural mouse parasite, Trichinella spiralis, and multipoint intravital time-lap

14 MMC) recruitment coincides with expulsion of Trichinella spiralis, at a time when the majority of the

15 is provided here that the parasitic nematode Trichinella spiralis can catalyze the conversion and thu

16 Although the forthcoming genome sequence of Trichinella spiralis can provide invaluable comparative

17 ection of mammalian skeletal muscle cells by Trichinella spiralis causes host nuclei to become polypl

18 Trichinella spiralis creates a unique intracellular habi

19 The L1 stage of the parasitic nematode Trichinella spiralis displays on its surface glycoprotei

20 pond vigorously to intestinal infection with Trichinella spiralis, eliminating a role for T-bet in MC

21 rimary infection with the parasitic nematode Trichinella spiralis, eosinophils play an important immu

22 Because mice infected with Trichinella spiralis experience a pronounced, but transi

23 hown to have a critical role in clearance of Trichinella spiralis from the intestinal tract.

24 le for the expulsion of the related nematode Trichinella spiralis from these animals.

25 hanced expulsion of the intestinal nematode, Trichinella spiralis, from the small intestine.

26 at wild boar meat products contaminated with Trichinella spiralis had entered the food chain in Germa

27 te mitochondrial DNA (mtDNA) of the nematode Trichinella spiralis has been amplified in four overlapp

28 The antibody response to the L1 stage of Trichinella spiralis has been described as biphasic.

29 Studies with rodents infected with Trichinella spiralis, Heligmosomoides polygyrus, Nippost

30 ed in the survival of the parasitic nematode Trichinella spiralis in an intracellular environment are

31 on triggered by the intraepithelial nematode Trichinella spiralis in jejunal epithelium from BALB/c m

32 of acute inflammation during development of Trichinella spiralis in the muscle.

33 Infection of mice with the nematode parasite Trichinella spiralis induces changes in the proteome of

34 tinal infection with the parasitic nematode, Trichinella spiralis, induces a pronounced eosinophilia

35 in were observed in the jejunal submucosa of Trichinella spiralis-infected BALB/c mice.

36 Jejunal segments isolated from Trichinella spiralis-infected mice were used to assess 5

37 mice and in mesenteric lymph nodes (mLN) of Trichinella spiralis-infected mice.

38 Previous work has shown that Trichinella spiralis-infected rats transport IgE from pl

39 Trichinella spiralis infection elicits a vigorous IgE re

40 hat robust induction of IL4 responses during Trichinella spiralis infection enhance the presence of n

41 Trichinella spiralis infection induces a strong type 2 c

42 We previously demonstrated that Trichinella spiralis infection inhibits host inducible N

43 After Trichinella spiralis infection of mice, we show that bas

44 e mast cell responses and inflammation after Trichinella spiralis infection or the induction of food

45 Trichinella spiralis is a highly destructive parasitic n

46 Trichinella spiralis is an obligate parasite of animals

47 Expulsion of the gastrointestinal nematode Trichinella spiralis is associated with pronounced masto

48 Infection with the parasitic nematode Trichinella spiralis is initiated when the L1 larva inva

49 parasites, Nippostrongylus brasiliensis and Trichinella spiralis, is similar in that both require IL

50 inophils protect intracellular, muscle-stage Trichinella spiralis larvae against NO-mediated killing.

51 Trichinella spiralis larvae were identified in wild boar

52 ccompanying the expulsion of the GI parasite Trichinella spiralis may be dependent on IL-4 and mediat

53 Infection with Trichinella spiralis rarely leads to significant morbidi

54 olate-induced peritonitis and infection with Trichinella spiralis, stimulated similar responses in Ga

55 metabolizing enzymes in secreted products of Trichinella spiralis suggests that endoparasites use sim

56 In parasitic infection with Trichinella spiralis, the immune response incorporates b

57 We have used the parasite helminth Trichinella spiralis to study the generation and differe

58 y response caused by an intestinal parasite, Trichinella spiralis, to study the relationship between

59 We have surveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis

60 Infection of mice with the nematode Trichinella spiralis triggers recruitment and differenti

61 ulsion of another gastrointestinal nematode, Trichinella spiralis, unlike N. brasiliensis expulsion,

62 binant human chymase efficiently degrade the Trichinella spiralis virulence factor heat shock protein

63 n the larval stage of the parasitic nematode Trichinella spiralis was shown to invade epithelial cell

64 MC in the jejunum of BALB/c mice exposed to Trichinella spiralis were assessed during the course of

65 t it does not contribute to immunity against Trichinella spiralis, which lives within IEC.