ライフサイエンスコーパス: Trichomonas

1 mperature appears to affect the viability of Trichomonas.

2 In men with NGU, 19.9% were infected with trichomonas.

3 en were enrolled, and 16% were found to have Trichomonas (46/294).

4 nce of RNAs with a distinctive 5' DMG cap in Trichomonas and Giardia lineages that are absent in othe

5 eishmania, Neospora, Plasmodium, Toxoplasma, Trichomonas and Trypanosoma.

6 hol-PP-GlcNAc2Man5 (present in Entamoeba and Trichomonas) and dolichol-PP- and N-linked GlcNAc2 (pres

7 Black race, a new sex partner, a history of trichomonas, and the presence of symptoms were associate

8 rate (62%), followed by Candida with 18% and Trichomonas at 9%.

9 transmitted diseases clinic were tested for trichomonas, chlamydia, and gonorrhea.

10 Clinical isolates and mutant Trichomonas CPI-GC that had reduced affinity to galectin

11 Live Trichomonas depleted the extracellular levels of galecti

12 The prevalence of Trichomonas determined by culture was 5% (15 of 300 spec

13 a indicate that the thioredoxin reductase of Trichomonas differs fundamentally in structure from that

14 enzymes of Plasmodium spp., Trypanosomatida, Trichomonas, Entamoeba and Giardia, with special emphasi

15 s amplified with DNA from Trichomonas tenax, Trichomonas gallinae, Chlamydia trachomatis, Neisseria g

16 ly early branching eukaryotic lineages, like Trichomonas, Galpha is likely to function independently

17 For each of the three organisms, Trichomonas, Gardnerella, and Candida, positivity at eac

18 l four time points (0, 24, 48, and 72 h) for Trichomonas, Gardnerella, and Candida.

19 However, Trypanosoma and Trichomonas genomes predicted ER degradation-enhancing a

20 1 acquisition (HR, 1.9; 95% CI, 1.1-3.1) and Trichomonas infection (HR, 1.8; 95% CI, 1.3-2.4).

21 Trichomonas infection was more common in parous non-Hisp

22 e gynecologic examination were evaluated for Trichomonas infection.

23 s associated with a 4-fold-increased risk of Trichomonas infection.

24 ction of galectin-1 and -3 in the context of Trichomonas infection.

25 uire new gonorrhoea, syphilis, chlamydia, or trichomonas infections.

26 Trichomonas is a cause of nongonococcal urethritis (NGU)

27 Trichomonas is an amitochondriate parasitic protozoon sp

28 Thus via CPI-GC binding, Trichomonas is capable of regulating galectin bioavailab

29 lial clones, recombinant galectins, clinical Trichomonas isolates, and mutant protozoan derivatives t

30 ic Aptima Combo 2 (AC2) and Aptima TV (ATV), trichomonas microscopy, and culture.

31 galectin-1 suppressed chemokine responses to Trichomonas or CPI-GC/LPG.

32 The percent sensitivity versus control for Trichomonas ranged from 100% at time zero with and witho

33 The OSOM Trichomonas Rapid Test (Genzyme Diagnostics, Cambridge,

34 The OSOM Trichomonas Rapid Test is a simple, objective test that

35 3.2- and 4.2-fold after treating Candida and Trichomonas, respectively.

36 ment of a protocol for differentiating among Trichomonas species that commonly infect humans.

37 targeted product was amplified with DNA from Trichomonas tenax, Trichomonas gallinae, Chlamydia trach

38 chomonas hominis, Trichomonas vaginalis, and Trichomonas tenax.

39 specimen type routinely used for traditional trichomonas testing and the recommended specimen type fo

40 ction of chlamydia (CT), gonorrhea (GC), and trichomonas (TV).

41 terium Anabaena sp. (44%), and the protozoan Trichomonas vaginalis (46%), which targets its POR to an

42 there was some evidence of association with Trichomonas vaginalis (adjusted OR, 1.56; 95% CI, 1.00-2

43 es inoculated with Chlamydia trachomatis and Trichomonas vaginalis (n = 9) or medium (controls; n = 7

44 2 (HSV-2), syphilis, chlamydia, gonorrhoea, Trichomonas vaginalis (together defined as 'any STI') an

45 oth prevalent and persistent infections with Trichomonas vaginalis (TV) are common.

46 omatis (CT), Mycoplasma genitalium (MG), and Trichomonas vaginalis (TV) are sexually transmitted infe

47 Trichomonas vaginalis (TV) can be infected with double-s

48 n is affected by bacterial vaginosis (BV) or Trichomonas vaginalis (TV) infection has not been adequa

49 Trichomonas vaginalis (TV) is the most common nonviral s

50 Ureaplasma urealyticum biovar 2 (UU-2), and Trichomonas vaginalis (TV) using nucleic acid amplificat

51 sis (BV), vulvovaginal candidiasis (VVC), or Trichomonas vaginalis (TV), were randomly assigned to re

52 xidized form of the [2Fe-2S] ferredoxin from Trichomonas vaginalis (TvFd) are presented.

53 d Entamoeba histolytica, and the parabasalid Trichomonas vaginalis all belong to Class II of FBAs and

54 7,899 specimens submitted for live clinical Trichomonas vaginalis analyte-specific reagent (ASR) scr

55 o transcription-mediated amplification-based Trichomonas vaginalis analyte-specific-reagent (ASR) tes

56 s specific, since the related human parasite Trichomonas vaginalis and associated purified CPs did no

57 ption-mediated amplification (TMA) assay for Trichomonas vaginalis and BTUB FRET PCR, using self-obta

58 drial carrier family in the hydrogenosome of Trichomonas vaginalis and have shown that this protein,

59 icient in metronidazole-resistant strains of Trichomonas vaginalis and is thought to be necessary for

60 Additional Trichomonas vaginalis and Mycoplasma genitalium screenin

61 sence of a splicing apparatus in the protist Trichomonas vaginalis and show that RNA motifs found in

62 ent in the amitochondriate parasitic protist Trichomonas vaginalis and some but not all other trichom

63 Trichomonas vaginalis and viral pathogens (herpes simple

64 ing Giardia lamblia, Leishmania species, and Trichomonas vaginalis are persistently infected with dsR

65 omonas vaginalis prevalence using the Aptima Trichomonas vaginalis assay (ATV; Gen-Probe) and the pre

66 ts of a commercial NAA test (GenProbe Aptima Trichomonas vaginalis assay; ATV) for T. vaginalis were

67 clinic for a new complaint were screened for Trichomonas vaginalis by culture and by PCR analysis of

68 on as well as to determine the prevalence of Trichomonas vaginalis by culture in a large and diverse

69 amplification tests (NAATs) for detection of Trichomonas vaginalis by vaginal swabs; NAATs for detect

70 clinic for a new complaint were screened for Trichomonas vaginalis by wet-preparation (wet-prep) micr

71 The human-infective parasite Trichomonas vaginalis causes the most prevalent nonviral

72 The protozoan Trichomonas vaginalis colonizes the human urogenital tra

73 Trichomonas vaginalis colonizes the urogenital tract of

74 The extracellular parasite Trichomonas vaginalis contains a surface glycoconjugate

75 thral swabs were obtained at enrollment, for Trichomonas vaginalis culture; semen specimens were also

76 n of macaques with Chlamydia trachomatis and Trichomonas vaginalis decreases the prophylactic efficac

77 determine if secreted cysteine proteases of Trichomonas vaginalis degrade SLPI and render it nonfunc

78 condary test in improving the sensitivity of Trichomonas vaginalis detection in young women over that

79 This study compared two assays for Trichomonas vaginalis detection, Gen-Probe's transcripti

80 obe was designed and evaluated for detecting Trichomonas vaginalis DNA in a 5' nuclease (TaqMan) assa

81 polymerase chain reaction (PCR) analysis for Trichomonas vaginalis DNA.

82 Infection with Trichomonas vaginalis during pregnancy has been associat

83 The crystal structure of the oxidized Trichomonas vaginalis ferredoxin (Tvfd) showed a unique

84 odified medium to InPouch for the culture of Trichomonas vaginalis from pooled vaginal secretions.

85 Two genes coding for Trichomonas vaginalis glucokinase were isolated and sequ

86 ase (PPi-PFK) of the amitochondriate protist Trichomonas vaginalis has been purified.

87 The utility of TMA for detection of Trichomonas vaginalis has recently been described.

88 tica, Leishmania spp., Trypanosoma cruzi and Trichomonas vaginalis have genes encoding homologues of

89 Nucleic acid amplification tests for Trichomonas vaginalis have improved sensitivity for dete

90 plasma genitalium, Chlamydia trachomatis, or Trichomonas vaginalis in 10 patients (8%).

91 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis in liquid-based cytology specimens

92 could also detect Mycoplasma genitalium and Trichomonas vaginalis in men and women reporting a histo

93 The impact of the viability of Trichomonas vaginalis in urine on wet mount, culture, an

94 Trichomonas vaginalis infected with a double-stranded RN

95 l SLPI levels have been correlated with both Trichomonas vaginalis infection and poor reproductive he

96 Trichomonas vaginalis infection in males has been largel

97 Trichomonas vaginalis infection in men is an important c

98 ed to wet mount and PCR for the diagnosis of Trichomonas vaginalis infection in women.

99 Trichomonas vaginalis infection is a sexually transmitte

100 Trichomonas vaginalis infection is estimated to be the m

101 The prevalence of Trichomonas vaginalis infection is highest in women with

102 Trichomonas vaginalis infection is highly prevalent worl

103 Trichomonas vaginalis infection is highly prevalent, may

104 Trichomonas vaginalis infection is the most prevalent no

105 omen in Mombasa, Kenya, to determine whether Trichomonas vaginalis infection was associated with an i

106 = 756 men), we identified 150 men (20%) with Trichomonas vaginalis infection, 358 men (47%) with HIV

107 ns related to the diagnosis and treatment of Trichomonas vaginalis infection, as well as the associat

108 esented regarding conditions associated with Trichomonas vaginalis infection, including human immunod

109 k women, being 30 to 40 years of age, recent Trichomonas vaginalis infection, primary or recurrent ge

110 infection, Chlamydia trachomatis infection, Trichomonas vaginalis infection, vulvovaginal candidiasi

111 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis infection.

112 eria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis infection.

113 Trichomonas vaginalis infections are usually asymptomati

114 eria gonorrhoeae, Mycoplasma genitalium, and Trichomonas vaginalis infections as well as the characte

115 tudy was to evaluate potential mechanisms of Trichomonas vaginalis involvement in human immunodeficie

116 Trichomonas vaginalis is a common sexually transmitted i

117 Trichomonas vaginalis is a highly divergent, unicellular

118 Trichomonas vaginalis is a parasite of the urogenital tr

119 Trichomonas vaginalis is a parasitic protozoan and the c

120 Trichomonas vaginalis is a parasitic protozoan purine au

121 Trichomonas vaginalis is a protist that causes the most

122 Trichomonas vaginalis is a protozoan parasite of humans

123 Trichomonas vaginalis is a unicellular microaerophilic e

124 Trichomonas vaginalis is an anaerobic protozoan parasite

125 Trichomonas vaginalis is an extracellular protozoan para

126 Trichomonas vaginalis is an important pathogen in both m

127 Trichomonas vaginalis is an underestimated sexually tran

128 Host parasitism by Trichomonas vaginalis is complex and in part mediated by

129 Host parasitism by Trichomonas vaginalis is complex, and the adhesion to va

130 from the sexually transmitted human parasite Trichomonas vaginalis is described.

131 The protist Trichomonas vaginalis is one of the most common human se

132 Trichomonas vaginalis is one of the most common nonviral

133 ecreted cysteine protease (CP) fraction from Trichomonas vaginalis is shown here to induce apoptosis

134 Trichomonas vaginalis is the agent of a highly prevalent

135 Trichomonas vaginalis is the most prevalent nonviral sex

136 Trichomonas vaginalis is the most prevalent nonviral sex

137 With gap1 from Trichomonas vaginalis obtained earlier, the data include

138 unusual case of extragenital infection with Trichomonas vaginalis of the conjunctiva of a 32-year-ol

139 rican women who used drugs were screened for Trichomonas vaginalis on > or =2 occasions between March

140 230) performed rapid point-of-care tests for Trichomonas vaginalis on self-collected vaginal swabs.

141 ated in patients concomitantly infected with Trichomonas vaginalis or Chlamydia trachomatis in additi

142 ins AP65, AP51, AP33 and AP23 synthesized by Trichomonas vaginalis organisms in high iron play a role

143 culture and evaluated their interaction with Trichomonas vaginalis parasites to complement previous s

144 Trichomonas vaginalis PCR using reagents from a commerci

145 Our aim was to determine Trichomonas vaginalis prevalence using the Aptima Tricho

146 Trichomonas vaginalis produces soluble factors that have

147 binding protein from the primitive eukaryote Trichomonas vaginalis reveals how a single protein can o

148 s the gold standard for clinical culture for Trichomonas vaginalis screening.

149 Our analyses of the Trichomonas vaginalis SCS sequences also confirmed the m

150 Trichomonas vaginalis secretes putrescine that is readil

151 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis Sequencing was used to assess macr

152 the amitochondrial Entamoeba histolytica and Trichomonas vaginalis species were analyzed.

153 The InPouch Trichomonas vaginalis test is the gold standard for clin

154 aginalis were compared with the Affirm VPIII Trichomonas vaginalis test.

155 iterature has reported increased accuracy of Trichomonas vaginalis transcription-mediated amplificati

156 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis via commercial transcription-media

157 LV was found to be more thermoresistant than Trichomonas vaginalis virus 1, but no specific protein m

158 iruses, including Leishmania RNA viruses and Trichomonas vaginalis viruses.

159 GPI of the parabasalid Trichomonas vaginalis was also sequenced.

160 ransport medium to maintain the viability of Trichomonas vaginalis was determined by comparing transp

161 DNA detection, but Chlamydia trachomatis and Trichomonas vaginalis were not.

162 eria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis were performed.

163 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis) and the E6/E7 mRNA of human papil

164 lastida (Leishmania major), the Parabasalia (Trichomonas vaginalis), and the Microsporidia (Vairimorp

165 ) and three partial draft genomes (including Trichomonas vaginalis).

166 (zero for bacterial vaginosis, candida, and Trichomonas vaginalis).

167 Trichomonas vaginalis, a flagellated protozoan, is the a

168 In Trichomonas vaginalis, a Myb1 protein was previously dem

169 In Trichomonas vaginalis, a parabasalian flagellate thought

170 Among them, Trichomonas vaginalis, a parasite adapted to the human g

171 describe the genome sequence of the protist Trichomonas vaginalis, a sexually transmitted human path

172 ribed in the divergent unicellular eukaryote Trichomonas vaginalis, although genome analyses reveal t

173 The discovery that Trichomonas vaginalis, an early diverging protist that l

174 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis, and bacterial vaginosis testing w

175 the pathogenic protozoa Giardia lamblia and Trichomonas vaginalis, and the bacterial pathogens Helic

176 the slime-mold Dictyostelium, the protozoan Trichomonas vaginalis, and the bacterium Burkholderia ps

177 ntamoeba fragilis, Pentatrichomonas hominis, Trichomonas vaginalis, and Trichomonas tenax.

178 he three common organisms causing vaginitis: Trichomonas vaginalis, Candida species, and Gardnerella

179 om Trypanosoma cruzi, Entamoeba histolytica, Trichomonas vaginalis, Cryptococcus neoformans, and Sacc

180 Women infected with Neisseria gonorrhoeae, Trichomonas vaginalis, or Chlamydia trachomatis had high

181 en, infections with Neisseria gonorrhoeae or Trichomonas vaginalis, or long-term follow-up.

182 fungal pathogens, as well as protozoa, e.g., Trichomonas vaginalis, Plasmodium berghei, and sporozoit

183 the genome databases for Giardia lamblia and Trichomonas vaginalis, respectively, and represent the l

184 e (rSAHH) cloned from Pseudomonas putida and Trichomonas vaginalis, respectively.

185 A related human trichomonad, Trichomonas vaginalis, showed no cytotoxic effects, indi

186 hydrogenosome-bearing, unicellular eukaryote Trichomonas vaginalis, that contains the active site mot

187 Trichomonas vaginalis, the causative agent for human tri

188 e medically important parasites: the protist Trichomonas vaginalis, the hard tick Ixodes ricinus, and

189 s simplex virus 2 infection, genital ulcers, Trichomonas vaginalis, vaginitis or cervicitis, and male

190 CR assay, using primers against pfoB gene of Trichomonas vaginalis, was developed and evaluated using

191 Trichomonas vaginalis, which causes the most common nonv

192 equences in the protists Giardia lamblia and Trichomonas vaginalis, which may represent the deepest k

193 tein-encoding genes in the parasitic protist Trichomonas vaginalis, which represents one of the deepe

194 aginotropic extracellular protozoan parasite Trichomonas vaginalis.

195 drial protist pathogens: Giardia lamblia and Trichomonas vaginalis.

196 , Entamoeba histolytica, Giardia lamblia and Trichomonas vaginalis.

197 t preparation and a culture method to detect Trichomonas vaginalis.

198 Trypanosoma brucei, Leishmania donovani, and Trichomonas vaginalis.

199 in one of the earliest diverging protozoans, Trichomonas vaginalis.

200 is specifically related to a homologue from Trichomonas vaginalis.

201 a, that represented by the parasitic protist Trichomonas vaginalis.

202 ctably transform the human-infective protist Trichomonas vaginalis.

203 ption of protein-coding genes of the protist Trichomonas vaginalis.

204 nscription factor in the protozoan parasite, Trichomonas vaginalis.

205 for Candida spp., Gardnerella vaginalis, and Trichomonas vaginalis.

206 ally transmitted infections (STIs) caused by Trichomonas vaginalis.

207 Women were also tested for Trichomonas vaginalis.

208 sekeeping genes, to genetically characterize Trichomonas vaginalis.

209 bation of culture media for the detection of Trichomonas vaginalis.

210 The parasite he discovered was Trichomonas vaginalis; and, in collaboration with Foucau

211 For Trichomonas vaginitis (n=55), HIV-1 RNA decreased from 3

212 d after treatment of Candida vulvovaginitis, Trichomonas vaginitis, and bacterial vaginosis.

213 Leukorrhea on microscopy was associated with Trichomonas vaginitis.

214 r gonorrhea, chlamydia, bacterial vaginosis, trichomonas, vulvovaginal candidiasis, pelvic inflammato

215 the major antioxidant defense mechanisms in Trichomonas was confirmed by showing that the parasite r