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1                         In addition, a novel Trichophyton allergen, encoding 412 amino acids, was ide
2 n test responses to Candida albicans, mumps, Trichophyton, and a bacterial antigen made from lysed St
3  conclude that different immune responses to Trichophyton are mediated by distinct T cell repertoires
4                             Unlike most Ags, Trichophyton can elicit either immediate (IH) or delayed
5                   Dermatophytes of the genus Trichophyton cause infections of human skin, nails, and
6 es induced by a representative dermatophyte, Trichophyton equinum.
7                      The dermatophyte fungus Trichophyton exhibits unique immunologic properties by i
8 te hypersensitivity skin test reactions to a Trichophyton extract exhibited IgE antibody binding to a
9 d in 59 adults who had been skin tested with Trichophyton extract.
10 cobacterium avium, and a filamentous fungus, Trichophyton fischeri.
11  of Eurotiomycetes including Aspergillus and Trichophyton from within a lichen-forming ancestral grou
12 tes, including Trichophyton rubrum (n = 15), Trichophyton mentagrophytes (n = 7), Trichophyton tonsur
13 ogenic dermatophytes Trichophyton rubrum and Trichophyton mentagrophytes as well as against the breas
14 o have elevated MICs to terbinafine and five Trichophyton mentagrophytes strains.
15 d individual responses to tetanus toxoid and Trichophyton mentagrophytes were significantly higher th
16 linical strains each of Trichophyton rubrum, Trichophyton mentagrophytes, and Epidermophyton floccosu
17 ncountered in a clinical mycology laboratory-Trichophyton mentagrophytes, Trichophyton rubrum, Tricho
18 ophytes tested included Trichophyton rubrum, Trichophyton mentagrophytes, Trichophyton tonsurans, Epi
19 r forms of severe disease caused by Candida, Trichophyton, Phialophora, and Exophiala species, includ
20 Forty-eight dermatophyte isolates, including Trichophyton rubrum (n = 15), Trichophyton mentagrophyte
21   Distinct immune responses in humans to the Trichophyton rubrum Ag, Tri r 2, are associated with dif
22 recombinant DC-HIL to bind the dermatophytes Trichophyton rubrum and Microsporum audouinii, but not s
23  tested against the pathogenic dermatophytes Trichophyton rubrum and Trichophyton mentagrophytes as w
24 mologous protein, Tri r 4, was cloned from a Trichophyton rubrum cDNA library, and the recombinant pr
25                The three structural genes of Trichophyton rubrum encoding actin (3,429 bp) and two an
26       This patient had extensive superficial Trichophyton rubrum infection of the skin and nails with
27        These candidate strains included five Trichophyton rubrum strains known to have elevated MICs
28 ri r 2, derived from the dermatophyte fungus Trichophyton rubrum, exhibits unique immunologic charact
29 parators against 25 clinical strains each of Trichophyton rubrum, T. mentagrophytes, and Epidermophyt
30 ty study, 5 blinded clinical strains each of Trichophyton rubrum, Trichophyton mentagrophytes, and Ep
31            The dermatophytes tested included Trichophyton rubrum, Trichophyton mentagrophytes, Tricho
32 logy laboratory-Trichophyton mentagrophytes, Trichophyton rubrum, Trichophyton tonsurans, and M. cani
33 Candida albicans, Microsporum audouinii, and Trichophyton rubrum.
34  agar supported the production of conidia by Trichophyton rubrum.
35 enic fungus which resembles the dermatophyte Trichophyton rubrum.
36                   Trichophyton violaceum and Trichophyton soudanense, which are common causes of tine
37 nation of slide cultures, and, for suspected Trichophyton species, use of additional media).
38 tinct antigens derived from the dermatophyte Trichophyton that serve as targets for diverse immune re
39  = 15), Trichophyton mentagrophytes (n = 7), Trichophyton tonsurans (n = 11), and Epidermophyton floc
40                                    An 83-kDa Trichophyton tonsurans allergen (Tri t 4) was previously
41 with an extract from the dermatophyte fungus Trichophyton tonsurans can result in either immediate (I
42             This communication details mixed Trichophyton tonsurans infections and highlights the nee
43                                              Trichophyton tonsurans is now the most common cause of t
44                                              Trichophyton tonsurans is the major pediatric pathogen i
45                      Tinea capitis caused by Trichophyton tonsurans remains to be a very common infec
46 Neosartorya fischeri, Microsporum canis, and Trichophyton tonsurans were shown to be able to catalyze
47 ophyton mentagrophytes, Trichophyton rubrum, Trichophyton tonsurans, and M. canis-was excellent.
48 ophyton rubrum, Trichophyton mentagrophytes, Trichophyton tonsurans, Epidermophyton floccosum, and Mi
49 ition over time was examined in responses to Trichophyton using primary PBMC cultures established fro
50 re only observed with occasional isolates of Trichophyton verrucosum.
51                                              Trichophyton violaceum and Trichophyton soudanense, whic

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