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1 e detection of the medically important genus Trichosporon.
2 Cryptococcus (32.9%), Saccharomyces (11.7%), Trichosporon (10.6%), and Rhodotorula (4.1%) were the mo
3 s one of six pathogenic species of the genus Trichosporon and the etiologic agent of pubic white pied
7 Candida famata/Candida guilliermondii), one Trichosporon beigelii (identified as Cryptococcus neofor
8 nding accounts for all of the enhancement in Trichosporon beigelii and Saccharomyces cerevisiae, but
10 ndida albicans, Cryptococcus neoformans, and Trichosporon beigelii) and two filamentous fungi (Asperg
11 ces cerevisiae, Sporobolomyces salmonicolor, Trichosporon beigelii, and 2 Prototheca spp. were evalua
14 ryptococcus albidus, Cryptococcus laurentii, Trichosporon beigelii/Trichosporon cutaneum, Rhodotorula
15 ryptococcus laurentii, Trichosporon beigelii/Trichosporon cutaneum, Rhodotorula spp., Rhodotorula rub
16 n organism recently transferred to the genus Trichosporon from Cryptococcus and now confirmed to be a
20 a case of sternal wound infection caused by Trichosporon inkin with a fatal outcome, in an immunocom
24 ; a case of disseminated infection caused by Trichosporon loubieri presented confirms its role as a h
27 the clinical course of four patients who had Trichosporon mycotoxinivorans recovered from multiple sp
28 ic morphologies, and biochemical features of Trichosporon mycotoxinivorans, a newly recognized pathog
31 ), defined as Aspergillus, Scedosporium, and Trichosporon species and Exophiala dermatitidis, in CF s
33 also the first report of disease caused by a Trichosporon species in a nontransplant patient with cys
34 ology provided a rapid means of detection of Trichosporon species with the flexibility to identify sp
35 es similar to those of rare Cryptococcus and Trichosporon species, and mixed cultures were often diff
36 iobolus, Rhodotorula, Zygosaccharomyces, and Trichosporon) were included, and 81.3% showed concordant
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