ライフサイエンスコーパス: Trichuris

1 ical for immunity to the intestinal pathogen Trichuris.

2 ed type 2 cytokine responses and immunity to Trichuris.

3 acts like an "epithelial escalator" to expel Trichuris and that the rate of epithelial cell movement

4 e gut of mouse strains that are resistant to Trichuris, and IL-25-deficient mice on a genetically res

5 ter infection with the gut-dwelling parasite Trichuris, fail to develop a pathogen-specific CD4+ T he

6 .7% had one or more nematodes, most commonly trichuris (in 3.9%).

7 011 African refugees were giardia (in 5.7%), trichuris (in 5.0%), and schistosoma (in 1.8%); among 55

8 ively expressed in the colon and exposure to Trichuris induced the expression of IL-31 in CD4(+) T ce

9 FN-gamma and TNF-alpha and failed to develop Trichuris-induced intestinal inflammation.

10 Critically, neutralization of IFN-gamma in Trichuris-infected TSLPR(-/-) mice restored Th2 cytokine

11 Blockade of proinflammatory cytokines during Trichuris infection ablates the requirement for IKK-beta

12 In response to Trichuris infection, IL-31Ralpha(-/-) mice exhibited inc

13 atory conditions in intestines infected with Trichuris muris and within the tumor microenvironment (B

14 infected with the gastrointestinal helminth Trichuris muris displayed accelerated expulsion of paras

15 polygyrus, Nippostronglyus brasiliensis, and Trichuris muris have provided considerable information a

16 c infection by the gastrointestinal nematode Trichuris muris in susceptible AKR mice, which mount a T

17 ngruent infection with the nematode parasite Trichuris muris in the large intestine around the time o

18 the chronically infecting parasitic nematode Trichuris muris in the large intestine of mice is depend

19 We used the well-established model of Trichuris muris infection to investigate the innate resp

20 is induced in the cecum of mice resistant to Trichuris muris infection.

21 with the gastrointestinal toxin piroxicam or Trichuris muris infection.

22 ation in MLN and mount Th1 cell responses to Trichuris muris infection.

23 infection with the caecum-dwelling helminth Trichuris muris is dependent on interleukin (IL)-4 and I

24 Expulsion of the gastrointestinal nematode Trichuris muris is mediated by a T helper (Th) 2 type re

25 Th cells, ICOS(-/-) mice were infected with Trichuris muris or Toxoplasma gondii.

26 Trichuris muris resides in intimate contact with its hos

27 IL-12-, IL-4-, and IL-12-deficient mice with Trichuris muris to determine whether IL-10 contributes t

28 The intestinal nematode Trichuris muris was recently demonstrated to utilise mic

29 e show that intestinal Th2 responses against Trichuris muris worms and Schistosoma mansoni eggs do no

30 minths (Heligmosomoides polygyrus bakeri and Trichuris muris).

31 urveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis culicivorax and identi

32 lminth parasites, Heligmosomoides polygyrus, Trichuris muris, and Schistosoma mansoni.

33 In studies of mice infected with Trichuris muris, blocking B7 ligand interactions inhibit

34 infection with the gastrointestinal helminth Trichuris muris, immunity to which is critically depende

35 infection with the gastrointestinal parasite Trichuris muris, memory CD4+ T cells persist in the drai

36 okines in immunity to the parasitic helminth Trichuris muris, the local effector mechanism culminatin

37 n infection with the cecum-dwelling nematode Trichuris muris, the majority of inbred strains of mice

38 nic infection with gastrointestinal helminth Trichuris muris, we identify dual functions for RELMbeta

39 le to infection with the intestinal nematode Trichuris muris, whereas their wild-type littermates wer

40 ection with the intestinal helminth parasite Trichuris muris, which yields a chronic infection becaus

41 and mice infected with the enteric parasite Trichuris muris.

42 ris trichiura and the mouse laboratory model Trichuris muris.

43 with the gastrointestinal nematode parasite Trichuris muris.

44 e large intestine dwelling helminth parasite Trichuris muris.

45 sing the murine model of whipworm infection, Trichuris muris.

46 phiregulin delayed expulsion of the nematode Trichuris muris.

47 sponse against the gastrointestinal helminth Trichuris muris.

48 impaired immunity to the intestinal helminth Trichuris muris.

49 o infection by the gastrointestinal nematode Trichuris muris.

50 tion with the gut-dwelling helminth parasite Trichuris muris.

51 Paradoxically, Trichuris of pigs has shown substantial promise as a tre

52 between infection with Schistosoma mansoni, Trichuris, or Strongyloides species and P. falciparum in

53 ry CD4+ T cells develop after infection with Trichuris, persist in the GALT, and mediate protective i

54 0.06), hookworm (prevalence ratio, 0.07), or trichuris (prevalence ratio, 0.27) but were not less lik

55 tory leucocyte proteinase-1 (SLP-1), but the Trichuris protein family differs in being composed of mu

56 nsity, types of worms (ascaris, hookworm, or trichuris), risk of bias, cluster versus individual tria

57 administration of ova from the pig whipworm Trichuris suis (T. suis; TSO) has been proposed for the

58 found that soluble products derived from the Trichuris suis (TsSP) significantly affect the different

59 in the porcine proximal colon in response to Trichuris suis (whipworm) infection using 16S rRNA gene-

60 whether infection with the nematode parasite Trichuris suis alters systemic cytokine levels, cellular

61 Trichuris suis and Oesophagostomum dentatum larvae were

62 ve shown that administration of the nematode Trichuris suis can be beneficial in treating various imm

63 ined the efficacy and safety of the helminth Trichuris suis in therapy of ulcerative colitis.

64 rhinitis received three weekly doses of 2500 Trichuris suis ova (n = 45) or placebo (n = 44) over 6 m

65 Trichuris suis ova (TSO) have been tested for therapeuti

66 example, to assess the safety or efficacy of Trichuris suis ova in allergies, inflammatory bowel dise

67 Patients received 2500 Trichuris suis ova or placebo orally at 2-week intervals

68 his study suggests that cytokines induced by Trichuris suis ova treatment do not alter allergic react

69 Trichuris suis ova were obtained from the US Department

70 Eggs of the pig whipworm (Trichuris suis ova) have been shown to be safe in multip

71 tory bowel disease, exposure to the helminth Trichuris suis reduces disease activity.

72 Treatment with Trichuris suis soluble products during monocyte-to-macro

73 The whipworm (Trichuris suis) secretes prostaglandin E2 to suppress pr

74 rm (Necator americanus) or porcine whipworm (Trichuris suis) show that they are safe and may be effec

75 hole-genome sequences of the human-infective Trichuris trichiura and the mouse laboratory model Trich

76 The cytokine and antibody response to Trichuris trichiura infection was determined for 96 pers

77 Wheezing status, Ascaris lumbricoides and Trichuris trichiura infection, IL-10 production by perip

78 iardia lamblia, Enterocytozoon bieneusi, and Trichuris trichiura was found in all animals regardless

79 The percentage of children infected with Trichuris trichiura was highest among children who did n

80 lminths (hookworm, Ascaris lumbricoides, and Trichuris trichiura) are the most widespread NTDs, but b

81 lminths (Ascaris lumbricoides, hookworm, and Trichuris trichiura) are widespread and often occur conc

82 Whipworm (Trichuris trichiura) infection is a soil-transmitted hel

83 ections (ie, hookworm, Ascaris lumbricoides, Trichuris trichiura) with the Kato-Katz method.

84 ca), and helminths (Ascaris lumbricoides and Trichuris trichiura), as well as two extrinsic controls

85 cylostoma duodenale and Necator americanus), Trichuris trichiura, and Strongyloides stercoralis.

86 Infection with malaria, Trichuris trichiura, Ascaris lumbricoides, and hookworms

87 on infection with STH (Ascaris lumbricoides, Trichuris trichiura, hookworm [Ancylostoma duodenale and

88 chlii, Endolimax nana, Ascaris lumbricoides, Trichuris trichiura, Strongyloides stercoralis, and Neca

89 The major E/S protein of Trichuris trichiura, the human whipworm, is a highly imm

90 f human infection with the whipworm of dogs, Trichuris vulpis, in a woman with duodenal ulcer disease

91 Trichuris (whipworm) infects 1 billion people worldwide

92 Following exposure to low-dose Trichuris, wild-type C57BL/6 mice exhibit persistent inf

93 -/-) mice exhibited accelerated expulsion of Trichuris with significantly decreased worm burdens comp