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1 minths (Heligmosomoides polygyrus bakeri and Trichuris muris).
2 sing the murine model of whipworm infection, Trichuris muris.
3 phiregulin delayed expulsion of the nematode Trichuris muris.
4 sponse against the gastrointestinal helminth Trichuris muris.
5 impaired immunity to the intestinal helminth Trichuris muris.
6 o infection by the gastrointestinal nematode Trichuris muris.
7 and mice infected with the enteric parasite Trichuris muris.
8 tion with the gut-dwelling helminth parasite Trichuris muris.
9 ris trichiura and the mouse laboratory model Trichuris muris.
10 with the gastrointestinal nematode parasite Trichuris muris.
11 e large intestine dwelling helminth parasite Trichuris muris.
12 atory conditions in intestines infected with Trichuris muris and within the tumor microenvironment (B
13 urveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis culicivorax and identi
16 infected with the gastrointestinal helminth Trichuris muris displayed accelerated expulsion of paras
17 polygyrus, Nippostronglyus brasiliensis, and Trichuris muris have provided considerable information a
18 infection with the gastrointestinal helminth Trichuris muris, immunity to which is critically depende
19 c infection by the gastrointestinal nematode Trichuris muris in susceptible AKR mice, which mount a T
20 ngruent infection with the nematode parasite Trichuris muris in the large intestine around the time o
21 the chronically infecting parasitic nematode Trichuris muris in the large intestine of mice is depend
26 infection with the caecum-dwelling helminth Trichuris muris is dependent on interleukin (IL)-4 and I
27 Expulsion of the gastrointestinal nematode Trichuris muris is mediated by a T helper (Th) 2 type re
28 infection with the gastrointestinal parasite Trichuris muris, memory CD4+ T cells persist in the drai
31 okines in immunity to the parasitic helminth Trichuris muris, the local effector mechanism culminatin
32 n infection with the cecum-dwelling nematode Trichuris muris, the majority of inbred strains of mice
33 IL-12-, IL-4-, and IL-12-deficient mice with Trichuris muris to determine whether IL-10 contributes t
35 nic infection with gastrointestinal helminth Trichuris muris, we identify dual functions for RELMbeta
36 le to infection with the intestinal nematode Trichuris muris, whereas their wild-type littermates wer
37 ection with the intestinal helminth parasite Trichuris muris, which yields a chronic infection becaus
38 e show that intestinal Th2 responses against Trichuris muris worms and Schistosoma mansoni eggs do no
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