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1 dney disease-associated ApoL1 variants lysed Trypanosoma brucei rhodesiense.
2 in cytokine gene knockout mice infected with Trypanosoma brucei rhodesiense.
3 roove binders was evaluated in vitro against Trypanosoma brucei rhodesiense (8 compounds) and Plasmod
4 n the host immune response to infection with Trypanosoma brucei rhodesiense, a protozoan parasite res
5 ural product displayed high activity against Trypanosoma brucei rhodesiense, a recalcitrant parasite
6 ei isolate, and several clinical isolates of Trypanosoma brucei rhodesiense (agent of east African sl
7 ition of the cysteine proteases rhodesain of Trypanosoma brucei rhodesiense and falcipain-2 of Plasmo
8 .045 muM against the human pathogenic strain Trypanosoma brucei rhodesiense and is more than 4000 tim
9 brucei , Trypanosoma brucei gambiense , and Trypanosoma brucei rhodesiense and is one of Africa's ol
10 tent inhibitory effect against the parasites Trypanosoma brucei rhodesiense and Leishmania donovani w
11 wed promising in vitro activity against both Trypanosoma brucei rhodesiense and Plasmodium falciparum
14 ican human African trypanosomiasis caused by Trypanosoma brucei rhodesiense and the West African form
15 t cause human African Trypanosomiasis (HAT), Trypanosoma brucei rhodesiense and Trypanosoma brucei ga
17 The expansion of sleeping sickness caused by Trypanosoma brucei rhodesiense beyond its traditional fo
19 ainst cultured Trypanosoma brucei brucei and Trypanosoma brucei rhodesiense cells with ED(50) values
21 r proteins that contribute to virulence, and Trypanosoma brucei rhodesiense EVs contain the serum res
22 The bloodstream trypanosome clone MVAT4 of Trypanosoma brucei rhodesiense expresses a metacyclic vs
24 ch as Plasmodium falciparum, Lassa Virus and Trypanosoma brucei rhodesiense, has resulted in elevated
25 iprotozoal assays, ten of the oils inhibited Trypanosoma brucei rhodesiense (IC(50) 15.9-64.5 mug/mL)
26 furan were synthesized and evaluated against Trypanosoma brucei rhodesiense in the STIB900 mouse mode
27 rface glycoprotein (VSG) gene in bloodstream Trypanosoma brucei rhodesiense involves a duplicative tr
29 subspecies, Trypanosoma brucei gambiense and Trypanosoma brucei rhodesiense, is a parasitic disease t
30 -stage HAT, and the only drug for late-stage Trypanosoma brucei rhodesiense, is intravenous melarsopr
31 s on the proliferation of Trypanosoma cruzi, Trypanosoma brucei rhodesiense, Leishmania donovani, Tox
32 To address this hypothesis, we transformed Trypanosoma brucei rhodesiense LouTat 1 with the 117 VSG
33 stance to most African trypanosomes, but not Trypanosoma brucei rhodesiense or T.b. gambiense, which
34 ckness, is caused by the protozoan parasites Trypanosoma brucei rhodesiense or Trypanosoma brucei gam
35 or in vitro antiprotozoal activities against Trypanosoma brucei rhodesiense, Plasmodium falciparum, a
36 otozoal activities of compounds 1-60 against Trypanosoma brucei rhodesiense, Plasmodium falciparum, a
38 d and tested for in vitro activities against Trypanosoma brucei rhodesiense, Plasmodium falciparum, a
39 ir in vitro antiprotozoal properties against Trypanosoma brucei rhodesiense, Plasmodium falciparum, a
40 otozoal activities of compounds 1-43 against Trypanosoma brucei rhodesiense, Plasmodium falciparum, a
41 ve strategies for the sustainable control of Trypanosoma brucei rhodesiense (Rhodesian) sleeping sick
43 layed in vitro nanomolar IC50 values against Trypanosoma brucei rhodesiense STIB900 with selectivity
44 18b show IC50 values of 5 nM or less against Trypanosoma brucei rhodesiense (T. b. r.) and 10a, 10b,
45 3 isoxazole derivatives were assayed against Trypanosoma brucei rhodesiense (T. brucei rhodesiense) S
46 cribed, followed by their evaluation against Trypanosoma brucei rhodesiense, T. cruzi, Leishmania don
47 ave additional trypanolytic activity against Trypanosoma brucei rhodesiense, the cause of acute Afric
48 rtant for early resistance to infection with Trypanosoma brucei rhodesiense, the extracellular protoz
49 orrelation with the human infective parasite Trypanosoma brucei rhodesiense, the most potent compound
50 ting against four other protozoan parasites: Trypanosoma brucei rhodesiense , Trypanosoma cruzi , Lei
51 st important human trypanosomatid pathogens (Trypanosoma brucei rhodesiense, Trypanosoma cruzi, and L
52 st important human trypanosomatid pathogens (Trypanosoma brucei rhodesiense, Trypanosoma cruzi, and L
53 growth inhibition of the bloodstream form of Trypanosoma brucei rhodesiense trypomastigotes by bispho
54 ctive (0.5 muM </= IC(50) < 6.0 muM) against Trypanosoma brucei rhodesiense trypomastigotes were 5-31
55 c markers in CSF from patients infected with Trypanosoma brucei rhodesiense, using 1H nuclear magneti
56 ite concentrations in patients infected with Trypanosoma brucei rhodesiense, using liquid chromatogra
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