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1 ia guyanensis , Leishmania amazonensis , and Trypanosoma cruzi .
2 ureus), a virus (influenza), and a parasite (Trypanosoma cruzi).
3 potentially foodborne parasites (for example Trypanosoma cruzi).
4 y infection caused by the protozoan parasite Trypanosoma cruzi.
5 cal disease caused by the protozoan parasite Trypanosoma cruzi.
6 tes of Leishmania major and epimastigotes of Trypanosoma cruzi.
7 atin America that is caused by the protozoan Trypanosoma cruzi.
8 Rs, are highly susceptible to infection with Trypanosoma cruzi.
9 S) and inflammatory cytokines in response to Trypanosoma cruzi.
10 sed by infection with the protozoan parasite Trypanosoma cruzi.
11 d as a regulator of stage differentiation in Trypanosoma cruzi.
12 has been implemented for West Nile virus and Trypanosoma cruzi.
13 ase is caused by infection with the parasite Trypanosoma cruzi.
14 rucial for control of the protozoan parasite Trypanosoma cruzi.
15 the kinetoplastidea including Leishmania and Trypanosoma cruzi.
16 asitemia and mortality in mice infected with Trypanosoma cruzi.
17 ed from endothelial cells were infected with Trypanosoma cruzi.
18 used by the kinetoplastid protozoan parasite Trypanosoma cruzi.
19 ic analysis of the four life-cycle stages of Trypanosoma cruzi.
20 ic protection against the protozoan pathogen Trypanosoma cruzi.
21 ma brucei, 27 in Leishmania major, and 24 in Trypanosoma cruzi.
22 obe directed at a kinetoplast DNA segment of Trypanosoma cruzi.
23 CI 2.2-6.8) were estimated to be infected by Trypanosoma cruzi.
24 viduals infected with the protozoan parasite Trypanosoma cruzi.
25 mmunity following intradermal infection with Trypanosoma cruzi.
26 ection caused by the intracellular protozoan Trypanosoma cruzi.
27 s' disease, caused by the protozoan parasite Trypanosoma cruzi.
28 the authors present a 2.5-A structure of the Trypanosoma cruzi 60S ribosomal subunit and propose a mo
33 Chagas disease is caused by the protozoan Trypanosoma cruzi, affecting millions of people worldwid
34 as disease, caused by the protozoan parasite Trypanosoma cruzi, affects 7 million people in Latin Ame
35 ease, caused by the hemoflagellate protozoan Trypanosoma cruzi, affects millions of people in South a
36 gas disease is a chronic infection caused by Trypanosoma cruzi, an intracellular protozoan parasite.
37 disease is caused by the protozoan parasite Trypanosoma cruzi and affects 5-8 million people in Lati
38 in the infective and intracellular stages of Trypanosoma cruzi and are recognized as antigenic target
39 Chagas disease is caused by the parasite Trypanosoma cruzi and is an important cause of morbidity
40 ago, is caused by the intracellular parasite Trypanosoma cruzi and is most frequently associated with
41 f Kinetoplastida -Trypanosoma brucei brucei, Trypanosoma cruzi and Leishmania major - are now complet
44 lular protozoan parasites Toxoplasma gondii, Trypanosoma cruzi and Leishmania spp. target macrophages
45 have been considered as virulence factors of Trypanosoma cruzi and Leishmania spp., and have been dem
46 interactions between the protozoan parasite Trypanosoma cruzi and mammalian hosts at primary sites o
47 e is a chronic infection in humans caused by Trypanosoma cruzi and manifested in progressive cardiomy
48 rified Complex II from the parasitic protist Trypanosoma cruzi and obtained the unexpected result tha
53 ng the etiological agents of Chagas disease (Trypanosoma cruzi) and African sleeping sickness (Trypan
54 disease is caused by the protozoan parasite Trypanosoma cruzi, and activation of CD8(+) T cells is c
55 in the developing world, rheumatic carditis, Trypanosoma cruzi, and bacterial infections such as diph
56 strate binding cavities (Trypanosoma brucei, Trypanosoma cruzi, and L. infantum) suggests that substr
57 d pathogens (Trypanosoma brucei rhodesiense, Trypanosoma cruzi, and Leishmania donovani), and several
58 d pathogens (Trypanosoma brucei rhodesiense, Trypanosoma cruzi, and Leishmania donovani), and two com
59 equence of the pathogens Trypanosoma brucei, Trypanosoma cruzi, and Leishmania major provides an oppo
60 logues in the genomes of Trypanosoma brucei, Trypanosoma cruzi, and Leishmania major were identified.
61 d genome architecture of Trypanosoma brucei, Trypanosoma cruzi, and Leishmania major, three related p
64 cellular human parasites Trypanosoma brucei, Trypanosoma cruzi, and Leishmania spp., the spliced-lead
65 lude the human pathogens Trypanosoma brucei, Trypanosoma cruzi, and Leishmania spp., which in humans
69 ther eukaryotes, the protein-coding genes of Trypanosoma cruzi are arranged in large polycistronic ge
71 igotes, the highly motile infective forms of Trypanosoma cruzi, are capable of infecting several cell
72 ng infection with the intracellular parasite Trypanosoma cruzi, as evidenced by transcriptome and cyt
74 protein (FCaBP) of the flagellated protozoan Trypanosoma cruzi associates with the flagellar membrane
76 NF) produced by the Chagas' disease parasite Trypanosoma cruzi binds nerve growth factor (NGF) recept
77 bolically challenged and upon infection with Trypanosoma cruzi (Brazil strain) suffer high mortality.
78 linical trials, was previously found to kill Trypanosoma cruzi by blocking sterol 14 alpha-demethylas
79 tually enhances infection with the protozoan Trypanosoma cruzi, by a mechanism that may involve facil
80 kills the causative agent of Chagas disease, Trypanosoma cruzi, by blocking ergosterol biosynthesis a
84 examined the Leishmania major (Friedlin) and Trypanosoma cruzi (CL Brener) genome projects for SL RNA
86 mania species, unlike Trypanosoma brucei and Trypanosoma cruzi, contain genes encoding MTHFR and two
87 aminopyridyl-based lead inhibitors targeting Trypanosoma cruzi CYP51 (TcCYP51) has been developed usi
89 rom the parasite that causes Chagas disease, Trypanosoma cruzi, directly in whole, unprocessed human
90 he pathogenic species Trypanosoma brucei and Trypanosoma cruzi, edit their post-transcriptional mitoc
93 ovel finding that the intracellular pathogen Trypanosoma cruzi elicits immediate and sustained repres
95 by the oligosaccharyltransferase (OST) from Trypanosoma cruzi, Entamoeba histolytica, Trichomonas va
98 PDNF, produced by the Chagas' disease agent Trypanosoma cruzi, functionally mimics mammalian neurotr
99 s a putative nucleoside phosphorylase in the Trypanosoma cruzi genome was overexpressed in Escherichi
101 e extreme genetic diversity of the protozoan Trypanosoma cruzi has been proposed to be associated wit
106 nosoma brucei, and the American trypanosome, Trypanosoma cruzi, have the capacity to synthesize vitam
108 presumably by keeping the etiological agent Trypanosoma cruzi in check through protective immunity a
109 The journey of the Chagas' disease parasite Trypanosoma cruzi in the human body usually starts in th
112 ar why only a proportion of children born to Trypanosoma cruzi-infected mothers acquire the infection
113 ivities (39 to 58%) and ATP (38%) content in Trypanosoma cruzi-infected murine hearts compared with n
119 nic epithelial model HCT116 cells respond to Trypanosoma cruzi infection by secreting defensin alpha-
123 at immune responses in subjects with chronic Trypanosoma cruzi infection display features common to o
125 Increasingly during the past few decades, Trypanosoma cruzi infection has been detected in North A
126 mmunochromatographic dipstick test to detect Trypanosoma cruzi infection in 366 human serum samples w
127 or T-bet (Tbx21) regulates Th17 responses to Trypanosoma cruzi infection in an IFN-gamma-independent
129 e required for host control of intracellular Trypanosoma cruzi infection in mice, although the basis
132 loss of certain functional activities during Trypanosoma cruzi infection might result in the inabilit
133 to reach cardiac tissue in vivo, even after Trypanosoma cruzi infection, a known inducer of lymphoid
134 ion of Th17 cells, we used a murine model of Trypanosoma cruzi infection, a protozoan parasite that c
136 nic Chagas disease cardiomyopathy, caused by Trypanosoma cruzi infection, is a major cause of heart f
138 tablishing a system for drug cure of chronic Trypanosoma cruzi infection, we present the first extens
147 es to lymphocytic choriomeningitis virus and Trypanosoma cruzi infections, and a specific defect in C
151 oles) displayed significant activity against Trypanosoma cruzi intracellular amastigotes (IC(50) rang
152 sly implicated in plasma membrane repair and Trypanosoma cruzi invasion, events which are mediated by
159 om basic amino acids, the protozoan parasite Trypanosoma cruzi is incapable of polyamine biosynthesis
161 flagellar calcium-binding protein (FCaBP) of Trypanosoma cruzi is localized to the flagellar membrane
162 ium-binding protein (FCaBP) of the protozoan Trypanosoma cruzi is targeted to the flagellar membrane
164 The insect-transmitted protozoan parasite Trypanosoma cruzi is the causative agent of Chagas disea
171 sed by infection with the protozoan parasite Trypanosoma cruzi, is a leading cause of heart disease (
173 aused by the eukaryotic (protozoan) parasite Trypanosoma cruzi, is an alarming emerging global health
175 Chagas disease, caused by the protozoan Trypanosoma cruzi, is endemic in Latin America and affec
177 me found in parasitic Leishmania species and Trypanosoma cruzi, is implicated in deglutathionylation
178 l of many intracellular pathogens, including Trypanosoma cruzi, is reported to be dependent on the pr
179 s' disease, caused by the protozoan parasite Trypanosoma cruzi, is the most common cause of cardiac-r
180 heart disease (CHD), caused by the parasite Trypanosoma cruzi, is the most common form of myocarditi
181 are highly conserved in clinically relevant Trypanosoma cruzi isolates and are recognized by B and T
182 umber of dialkyl imidazoles as inhibitors of Trypanosoma cruzi lanosterol-14alpha-demethylase (L14DM)
183 of intracellular infection by the protozoan Trypanosoma cruzi leads to the development of human Chag
184 parasites: Trypanosoma brucei rhodesiense , Trypanosoma cruzi , Leishmania donovani , and P. falcipa
185 y infection with the kinetoplastid parasites Trypanosoma cruzi, Leishmania spp. and Trypanosoma bruce
186 cellular effects at different stages of the Trypanosoma cruzi life cycle, the structural data provid
187 or Chagas' disease, we evaluated against all Trypanosoma cruzi life stages the in vitro trypanocidal
188 hmania-like flagellum attachment zone, and a Trypanosoma cruzi-like cytostome are ancestral features,
189 a novel mechanism used by diverse pathogens (Trypanosoma cruzi, Listeria monocytogenes, and adenoviru
191 ence for a novel immune evasion mechanism of Trypanosoma cruzi, mediated by host cell plasma membrane
195 rom MIIG mice were unable produce NO or kill Trypanosoma cruzi or Leishmania major after priming with
200 mice with the myotropic Colombiana strain of Trypanosoma cruzi, parasites persisted in tissue at low
202 and virulence of Trypanosoma brucei and, in Trypanosoma cruzi, PDEC2 was shown to be required for no
207 ro against T rypanosoma brucei rhodesiense , Trypanosoma cruzi , Plasmodium falciparum , and Leishman
208 e parasite sources of these three proteases, Trypanosoma cruzi, Plasmodium falciparum, and Trypanosom
209 ork was to analyze the predictive value of a Trypanosoma cruzi-positive polymerase chain reaction (PC
210 EbS was more toxic for T. brucei than for Trypanosoma cruzi, probably due to lower levels of TryR
211 e infectious stage of the protozoan parasite Trypanosoma cruzi produces a surface-anchored complement
212 eukaryotic parasites Trypanosoma brucei and Trypanosoma cruzi provide a first structural view of a e
214 Immune control of the protozoan parasite Trypanosoma cruzi requires the activation of both CD4+ a
217 sceptible mice with the Colombiana strain of Trypanosoma cruzi results in an orchestrated expression
218 -genome sequencing of the protozoan pathogen Trypanosoma cruzi revealed that the diploid genome conta
219 comparison of the dUTPases from C.jejuni and Trypanosoma cruzi reveals a common fold with certain dis
220 es, we determined the 2.5-A structure of the Trypanosoma cruzi ribosome large subunit by single-parti
224 rystal structures of the drug target enzyme, Trypanosoma cruzi sterol 14alpha-demethylase (CYP51), co
226 expressed in the heart after infection with Trypanosoma cruzi, suggesting that they play an importan
228 c fungicide fenarimol (1) as an inhibitor of Trypanosoma cruzi ( T. cruzi ), the causative agent of C
230 as disease, caused by the protozoan parasite Trypanosoma cruzi (T. cruzi), is an increasing threat to
231 hibited the growth of the parasitic protozoa Trypanosoma cruzi, T. brucei, and Leishmania donovani.
233 om the enzymes of Homo sapiens (Hs-SAHH) and Trypanosoma cruzi (Tc-SAHH) are qualitatively similar bu
234 Homo sapiens (Hs-SAHH) and from the parasite Trypanosoma cruzi (Tc-SAHH) are very similar in structur
237 gas disease, sterol 14alpha-demethylase from Trypanosoma cruzi (TCCYP51), was found to be catalytical
239 vector-borne pathogens, with the example of Trypanosoma cruzi (the etiological agent of Chagas disea
240 re effective agents against proliferation of Trypanosoma cruzi , the etiologic agent of American tryp
245 f a number of protozoan pathogens, including Trypanosoma cruzi, the agent of human Chagas disease.
247 lled intestinal microbiota, neither affected Trypanosoma cruzi, the causal agent of Chagas disease, i
249 characterized from the unicellular protozoan Trypanosoma cruzi, the causative agent of Chagas disease
250 the major cysteine protease of the protozoan Trypanosoma cruzi, the causative agent of Chagas disease
251 znidazole is the frontline drug used against Trypanosoma cruzi, the causative agent of Chagas disease
253 ted with AT during part of their life cycle: Trypanosoma cruzi, the causative agent of Chagas disease
254 and biochemical features of telomerase from Trypanosoma cruzi, the causative agent of Chagas disease
256 gi, and more recently the protozoan parasite Trypanosoma cruzi, the causative agent of Chagas disease
257 st resistance to the intracellular protozoan Trypanosoma cruzi, the causative agent of Chagas' diseas
258 trans-Sialidase is an essential enzyme for Trypanosoma cruzi, the causative agent of Chagas' diseas
259 However, the extent of the repeats within Trypanosoma cruzi, the causative agent of Chagas' diseas
260 alpha-1 displays a trypanocidal role against Trypanosoma cruzi, the causative agent of Chagas' diseas
261 casein kinase II (CKII) substrate (Tc-1) of Trypanosoma cruzi, the causative agent of Chagas' diseas
262 (NMT), an essential and druggable target in Trypanosoma cruzi, the causative agent of Chagas' diseas
266 of the contractile vacuole complex (CVC) of Trypanosoma cruzi, the etiologic agent of Chagas disease
269 critical events throughout the life cycle of Trypanosoma cruzi, the etiological agent of Chagas disea
270 ed to date including heteroxenous members of Trypanosoma cruzi, the extracellular Trypanosoma brucei,
272 n, of the structure of the 80S ribosome from Trypanosoma cruzi, the kinetoplastid protozoan pathogen
273 ves that inhibit TIM's catalytic activity in Trypanosoma cruzi, the parasite that causes Chagas disea
277 idate clathrin-associated proteins (CAPs) in Trypanosoma cruzi; the cohort includes orthologs of many
278 Cryptosporidium parvum, Leishmania spp., Trypanosoma cruzi, Theileria spp., Toxoplasma gondii and
279 elivers Gzms into three protozoan parasites (Trypanosoma cruzi, Toxoplasma gondii and Leishmania majo
285 on host cell contact, the protozoan parasite Trypanosoma cruzi triggers cytosolic Ca(2+) transients t
286 ors in the L. major, Trypanosoma brucei, and Trypanosoma cruzi (Tritryp) genomes suggest that the mec
287 tandem-repeated genes display codon bias in Trypanosoma cruzi, Trypanosoma brucei and Leishmania maj
288 Human galectin-3 binds to the surface of Trypanosoma cruzi trypomastigotes and human coronary art
289 pest insect that has the ability to transmit Trypanosoma cruzi under experimental laboratory conditio
291 sceptible mice with the Colombiana strain of Trypanosoma cruzi was characterized in an attempt to det
293 telomeres in the South American Trypanosome, Trypanosoma cruzi, we became interested in the telomeric
295 es that the intracellular protozoan parasite Trypanosoma cruzi, which causes heart disease in chronic
296 disease is caused by the protozoan parasite Trypanosoma cruzi, which depends on the production of en
298 Cruzain is the major cysteine protease of Trypanosoma cruzi, which is the causative agent of Chaga
300 oride inhibited trypanothione reductase from Trypanosoma cruzi with a linear competitive Ki value of
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