ライフサイエンスコーパス: U937 cell

1 mal hyaluronan matrix that binds leukocytes (U937 cells).

2 2 inhibition for their effects in NB4 and in U937 cells.

3 ression of TNF-alpha in U1 cells compared to U937 cells.

4 e and two different ascorbate derivatives in U937 cells.

5 on 1 5'-UTR variant in HL-60, MonoMac 6, and U937 cells.

6 ompromised in Leishmania-infected J774G8 and U937 cells.

7 cAMP at the concentrations < 0.05 microM in U937 cells.

8 to increase cAMP via beta2-adrenoceptors in U937 cells.

9 for soluble rhVCAM-1 ligand and monoblastoid U937 cells.

10 alpha.TNFR complexes in human myelomonocytic U937 cells.

11 e) or PLZF/RAR alpha (retinoid-resistant) in U937 cells.

12 fragment showed strong promoter activity in U937 cells.

13 alpha, but not in PLZF/RAR alpha expressing U937 cells.

14 which also decreased S1P levels in cultured U937 cells.

15 romatinized protein kinase Cbeta promoter of U937 cells.

16 ession of differentiation markers (CD11b) in U937 cells.

17 ndogenous cell surface levels of CD13/APN in U937 cells.

18 dicals production, and lowers cAMP levels in U937 cells.

19 also with cell membrane-anchored testisin on U937 cells.

20 molecules found in transfected and wild-type U937 cells.

21 lowering of cAMP levels in H(2)O(2)-treated U937 cells.

22 gonists inhibited HIV-1 promoter activity in U937 cells.

23 the nucleus of CD4(+) T cells and Jurkat and U937 cells.

24 a secretion or oxygen radicals production in U937 cells.

25 ective than the type II secretion mutants in U937 cells.

26 ly, and arrests the cell cycle in mitosis in U937 cells.

27 rbol 12-myristate 13-acetate (PMA)-activated U937 cells.

28 nding protein which interacted with Box B in U937 cells.

29 axis in human peripheral blood monocytes and U937 cells.

30 petitive binding studies to the FcgammaRI of U937 cells.

31 an infectivity defect in the macrophage-like U937 cells.

32 reased TNFalpha production in differentiated U937 cells.

33 inding to this site in nuclear extracts from U937 cells.

34 were isolated and perfused with monocytes or U937 cells.

35 promoter before and after differentiation of U937 cells.

36 binding and regulating the FUTIV promoter in U937 cells.

37 hidonate incorporation into nor release from U937 cells.

38 cellular H(2)O(2) content in NB4 but not in U937 cells.

39 formis amoebae and the human macrophage-like U937 cells.

40 MKN45 and AZ-521 but also in human monocytic U937 cells.

41 omeric DNA loss, which induces gamma-H2AX in U937 cells.

42 an nonphagocytic cells but not to phagocytic U937 cells.

43 MKP-1 promoter in the presence of GM-CSF in U937 cells.

44 NQO) and tert-butylhydroperoxide (t-BOOH) in U937 cells.

45 ell-killing activity in Mcl-1-overexpressing U937 cells.

46 transactivator activity in PAI-2-expressing U937 cells.

47 lize IFNalpha activity were determined using U937 cells.

48 ne bone marrow-derived macrophages and human U937 cells.

49 dent induction of apoptosis in proliferating U937 cells.

50 ctor may stimulate osteopontin expression by U937 cells.

51 ith that released by independent cultures of U937 cells.

52 poptosis-inducing factor in nonproliferating U937 cells.

53 rug library for its cytotoxic effect against U937 cells.

54 uced tissue factor pro-coagulant activity of U937 cells.

55 culture-stimulated osteopontin expression by U937 cells.

56 ecreases S1P levels in histiocytic lymphoma (U937) cells.

57 7(-) (23.89 + 2.78 microm, n = 3) than in MI-U937(+) cells (50.77 + 4.11 microm, n = 3).

58 interleukin-6 [IL-6]) following infection of U937 cells, a human macrophage-like cell line.

59 of either PML-RAR alpha or PLZF-RAR alpha in U937 cells, a non-APL myeloid cell line, led to a dramat

60 Monocytic U937 cells adhered at 4 degrees C to the apical surface

61 he acquisition of mitoxantrone resistance in U937 cells adhered to fibronectin versus cells selected

62 Furthermore, U937 cell adhesion per hyaluronan content is higher in t

63 Furthermore, we demonstrated that myeloid U937 cell adhesion to both OA ST and RA ST fibroblasts a

64 tunicamycin and poly(I,C) at the same time, U937 cell adhesion was partially additive, implying that

65 icantly reduces TNF-alpha-induced monocytic (U937) cell adhesion to HAEC under in vitro flow conditio

66 al time PCR studies showed that coculture of U937 cells and adipocytes increased osteopontin mRNA in

67 Both binding of ANX1 to U937 cells and ANX1-mediated inhibition of cell adhesion

68 blocked PMA/FP-induced TNFalpha secretion in U937 cells and attenuated apoptosis.

69 FPR mutants were expressed in human myeloid U937 cells and characterized for functions in addition t

70 and H2 histamine receptors (H1R and H2R) in U937 cells and Chinese hamster ovary-transfected cells.

71 on of histone H2B was observed on stimulated U937 cells and cultured neutrophils by confocal microsco

72 pplied a genome-wide CRISPR library to human U937 cells and exposed to them to Ara-C.

73 The map mutants grew within macrophage-like U937 cells and Hartmannella amoebae to the same degree a

74 concentration of the enzyme GAPDH in single U937 cells and HEK 293 cells, and found amounts within a

75 urther confirmed by coimmunoprecipitation in U937 cells and HEK293 cells.

76 s, but naturally produced by human monocytic U937 cells and HT-1080 fibrosarcoma cells, did not stimu

77 In addition, U937 cells and human keratinocyte cells were also stimul

78 romoters by chromatin immunoprecipitation in U937 cells and human PBMCs indicated that although the p

79 crease in MMP9 expression in human monocytic U937 cells and in primary sputum macrophages, which was

80 MLL-AF9 arrested growth of U937 cells and induced these cells to differentiate into

81 l ETS domain induced both differentiation of U937 cells and inhibited their growth in vitro and in vi

82 stimulated chemotaxis of monocytic THP-1 and U937 cells and primary monocytes and macrophages.

83 Stimulation of myeolomonocytic U937 cells and purified neutrophils with C5a resulted in

84 moter activity in cells that express PTP-oc (U937 cells and RAW264.7 cells) but not in cells that do

85 growth inhibitory effect on human leukaemic U937 cells and sufficient toxicological safety on normal

86 live imaging of this interaction between the U937 cells and the hyaluronan matrix and their subsequen

87 nt with high levels of glucose in Jurkat and U937 cells and was not due to transcriptional regulation

88 h a leukocyte endothelial step essential for U937 cell, and possibly monocyte, transmigration both in

89 release from differentiated but not control U937 cells, and electrospray ionization mass spectrometr

90 termined both in vivo, in stably transfected U937 cells, and in vitro, using a novel reconstitution s

91 to increase cAMP via beta2-adrenoceptors in U937 cells, and may have potential effects on human heal

92 ted apoptosis of L929 fibroblasts, monocytic U937 cells, and other cell types.

93 LT beta R complex was affinity-purified from U937 cells, and proteins associated with the complex wer

94 f U937 cells as well as apoptotic effects in U937 cells, and that these effects may be through the in

95 prevent apoptosis during differentiation of U937 cells, and the constitutive expression of Bcl-2 is

96 When myelomonoblastic U937 cells are treated with vitamin D(3) and TGF-beta, t

97 By using the expression levels of pure U937 cells as a control, it was shown that the gene expr

98 amoeba castellannii or human macrophage-like U937 cells as host cells.

99 xhibited inhibitory effects on the growth of U937 cells as well as apoptotic effects in U937 cells, a

100 Ultrastructurally, in U937 cells as well as human neutrophils and eosinophils,

101 CD33 by 80% in phorbol-ester differentiated U937 cells, at concentrations as low as 10 ng/ml.

102 H2 they elicited superoxide production, from U937 cells, at levels of 35-45% relative to that obtaine

103 behavior, WN-1 displays PMA-like behavior in U937 cell attachment and proliferation assays, as well a

104 the ability of SAMHD1 to restrict HIV-1 in a U937 cell-based restriction assay.

105 In the presence of U937 cells, both the AHP-anti-dsDNA and C3b-opsonized IC

106 ollectively, these findings indicate that in U937 cells, bryostatin 1 promotes paclitaxel-mediated mi

107 specifically binds FosB in PAI-2-expressing U937 cells but not in HeLa cells that do not express PAI

108 hibitors also lowered cyclin E expression in U937 cells but not in PC-3 cells, indicating underlying

109 ully with wild type IFN-gamma for binding to U937 cells but only at a greater than 100-fold higher co

110 and adipocytes increased osteopontin mRNA in U937 cells, but not adipocytes, suggesting that adipocyt

111 alysis revealed that expression of PB1-F2 in U937 cells, but not in A549 cells, results in the presen

112 tion increased the rate of 55Fe release from U937 cells by about 250%.

113 te Lyme disease and were also upregulated in U937 cells by B. burgdorferi in a time- and concentratio

114 gnaling, PI 3-kinase activities in HepG2 and U937 cells can be stimulated by TNF in a rapid but trans

115 enous addition of Sph or N,N-dimethyl-Sph to U937 cells causes caspase 3 activation and release of PK

116 ause Cdk9 function is induced in PMA-treated U937 cells, Cdk9 may play an antiapoptotic role during m

117 Cp protein synthesis in U937 cells ceased after 16 h even in the presence of abu

118 In U937 cells, coadministration of FP blocked PMA-induced e

119 human macrophages and 2- to 3-fold higher in U937 cells compared to the serovar Typhimurium and Enter

120 ed in p53-knockdown OCI-AML3 and p53-mutated U937 cells, confirming a role for p53 in the regulation

121 U937 cells constitutively express the antiapoptotic prot

122 tor alpha (RARalpha) chimeric protein in the U937 cells containing a Zn2+-inducible expression vector

123 In U937 cells, CSE stimulated mTOR activity and c-Jun expre

124 unoaffinity column binds a 70-kDa protein in U937 cell culture supernatant.

125 ebrafish and short hairpin RNA knockdowns in U937 cells cultured with human dermal endothelial cells.

126 Inhibition of U937 cell cytokine production correlated with CD200R exp

127 In vitro studies using human leukemia U937 cells demonstrated that K145 accumulates in U937 ce

128 nalyses of LBs purified from human monocytic U937 cells detected, common to LBs in other cells, prote

129 LAQ824-induced lethality in U937 cells did not involve the extrinsic apoptotic pathw

130 terns typical of known E2F target genes in a U937 cell differentiation system.

131 15S-transfected U937 cells displayed a reduced (50%) degree of trans-end

132 nt Vpr can potentiate virion production from U937 cells, downregulate NF-kappaB induction, and enhanc

133 Conditional expression of RUNX1-CBF2T1 in U937 cells downregulated CEBPA mRNA, protein and DNA bin

134 However, U937 cells ectopically expressing CrmA, dominant-negativ

135 in HL-60 promyelocytic leukemia cells and in U937 cells ectopically expressing the Bcl-2 protein.

136 uiescent NIH 3T3 cells and in differentiated U937 cells, even though the promoter is inactive.

137 king control subjects, or of human monocytic U937 cells exposed to cigarette smoke extract (CSE), was

138 U937 cells express group IV phospholipase A(2) (cPLA(2))

139 U937 cells express iPLA(2) mRNA and activity, but iPLA(2

140 ipids in others, but it is not known whether U937 cells express iPLA(2).

141 a1 and biologically activate human monocytic U937 cells expressing both receptors.

142 of INK4b RNA as shown in vitamin D3-treated U937 cells expressing CBFbeta-SMMHC.

143 ene delivery, we generated stably transduced U937 cells expressing either Fcgr3-rs or Fcgr3.

144 rthermore, the lipid membrane composition of U937 cells expressing PB1-F2 was also altered in a cell

145 In addition, U937 cells expressing R77H-CD11b display increased IL-6

146 cell migration was studied using transfected U937 cells expressing variable protein levels.

147 n molecule-1 (VCAM-1) and human monoblastoid U937 cells expressing Very Late Antigen-4 (VLA-4).

148 Recently, we reported that in U937 cells, expression of the CD11c gene is controlled b

149 In this study, we report that in U937 cells, expression of the CD11c gene is mediated by

150 istate 13-acetate-induced differentiation of U937 cells, fatty acid synthesis and its metabolic proce

151 When overexpressed in pervanadate-treated U937 cells, FDF03 was tyrosine-phosphorylated and recrui

152 of DNA methylation in human macrophage-like U937 cells following infection with Burkholderia pseudom

153 cell proliferation and induced apoptosis in U937 cells following treatment of 48 hours.

154 Coexposure of U937 cells for 24 h to marginally toxic concentrations o

155 d a dominant-negative form of RUNX1 protects U937 cells from apoptotic stimuli previously shown to be

156 means of beta1 integrins appears to protect U937 cells from initial drug-induced DNA damage by reduc

157 (2) reduced SIRT1 expression and activity in U937 cells; furthermore, cigarette smoke exposure also c

158 that full-length CD163 cDNAs from PAM, human U937 cells (histiocytic lymphoma), African green monkey

159 Enhanced Raman Spectroscopy (SERS) probe for U937 cells identification in vitro.

160 oid leukemia (AML) cell lines, as well as in U937 cells in combination with doxorubicin, 3i showed hi

161 s in stably transfected human myelomonocytic U937 cells in response to other TLR agonists.

162 ntial for proper differentiation of leukemic U937 cells in response to retinoic acid.

163 ne were obtained and compared with wild-type U937 cells in various models of cell migration in vitro

164 d melanoma cells by IFN-gamma-differentiated U937 cells in vitro via release of reactive oxygen speci

165 lic enzyme (PAF acetylhydrolase; PAF-AH), on U937 cells, in cell free and in intact cell experiments.

166 ary macrophages and differentiated monocytic U937 cells, in which occludin silencing resulted in 75 a

167 Expression of eNOS in transfected U937 cells increased phorbol 12-myristate 13-acetate-ind

168 rthermore, overexpression of C/EBPepsilon in U937 cells increased the activity of the wild-type lacto

169 f Puralpha to activate the CD11c promoter in U937 cells increases, as does that of Sp1.

170 Monocytes and in vitro differentiated U937 cells induce focal loss in the staining of VE-cadhe

171 UV irradiation or camptothecin treatment of U937 cells induced apoptosis and caused a significant ch

172 f constitutively active Fes into bipotential U937 cells induced the appearance of fully differentiate

173 , we discovered the novel finding that human U937 cells infected with 2 different concentrations of S

174 Overexpression of TEL2 in U937 cells inhibited differentiation induced by vitamin

175 ion, stable, inducible expression of PLZF in U937 cells inhibited the ability of 1,25(OH)(2)D(3) to i

176 mined by the number of fluorescently labeled U937 cells (injected intravenously) detected in grafts b

177 ining the localization of pro-myelomonocytic U937 cells into synovial tissue transplanted into SCID m

178 MP-1-dependent transcriptional repression in U937 cells is c-myc, providing an explanation for cessat

179 ell death induced by 60- or 300-ns pulses in U937 cells is the loss of the plasma membrane integrity

180 necessary for macrophage differentiation of U937 cells, it is not sufficient, based on the inability

181 this paper that CD23-initiated signaling in U937 cells leads to hyperphosphorylation of I kappa B al

182 FN responsiveness, transfection of PCAF into U937 cells led to a large increase in IFN-alpha-inducibl

183 ic cells compared to the uninfected parental U937 cell line and decreased TLR message in alveolar mac

184 bited FAS-induced apoptosis in the monocytic U937 cell line and in fresh human monocytes.

185 rom rat and human alveolar macrophages and a U937 cell line by reducing the LPS-elicited phosphorylat

186 LL-AF9 on differentiation of the monoblastic U937 cell line by using a tetracycline-inducible express

187 hole system was then validated by monitoring U937 cell line over 88 h.

188 We used the U937 cell line to examine the modulation of adaptor prot

189 F-kappaB (RelA and RelB) in the promonocytic U937 cell line using dominant-negative IkappaBalpha sign

190 l motility, stable transfection of LSP1-null U937 cell line with an episomal expression vector carryi

191 macrophage-like cells, as exemplified by the U937 cell line, c-Jun may be functional in other cell ty

192 he FPR, expressed in the human promyelocytic U937 cell line, were characterized.

193 BPbeta mRNA complexes in the human monocytic U937 cell line.

194 vision and growth were similar for different U937 cell lines at all LSP1 levels.

195 on of cell growth regulation, we established U937 cell lines stably transfected with a truncated form

196 To assess the role of TIF1beta, U937 cell lines were made that expressed antisense-hamme

197 defective for intracellular growth in human U937 cell macrophages and Hartmannella and Acanthamoeba

198 d Hartmannella vermiformis amoebae and human U937 cell macrophages.

199 The overexpression of 7SL RNA in J774G8 or U937 cells made these cells resistant to Leishmania infe

200 scular smooth muscle, endothelial cells, and U937 cell membranes contain a high-affinity EET binding

201 14,15-epoxyeicosa-5(Z)-enoic acid binding to U937 cell membranes with K(i) values of 3.60 and 2.73 nM

202 5-EE8ZE-APSA labeled a single 47 kDa band in U937 cell membranes, smooth muscle and endothelial cells

203 In U937 cell membranes, the 47 kDa radiolabeling was inhibi

204 protein on two-dimensional Western blots of U937 cell membranes.

205 SDF-1 induced U937 cell migration in vitro and in vivo in a dose-depen

206 , the inhibitory role of endogenous ANXA1 on U937 cell migration in vitro was confirmed by the potent

207 U937 cell migration through an RA ST fibroblast monolaye

208 s of IL-1, TNF-alpha, and TGF-beta genes for U937 cells mixed with PBMC before and after the separati

209 U937 cell mRNA contains a 1.8-kb transcript detected wit

210 In producing cAMP in U937 cells, N-coumaroyldopamine and N-caffeoyldopamine w

211 cation of proliferating and nonproliferating U937 cells occurs by distinct mechanisms and suggest tha

212 own to decrease the number of alpha(2)MRs in U937 cells or by antibodies to alpha(2)MR.

213 gh viability of encapsulated human monocytic U937 cells over a period of 4 days.

214 In U937 cells, overexpression of Cdk9-dn sensitized cells t

215 min, we observed dynamic protrusions of the U937 cell plasma membrane into nearby hyaluronan matrix,

216 phosphodiester chimaeric oligonucleotides to U937 cells reduced mRNA levels to up to 19.8% that of th

217 n, while ectopic overexpression of SAMHD1 in U937 cells repressed HSV-1 replication.

218 L-60 cells, respectively (35% and 40% of the U937 cells, respectively).

219 Forced expression of GATA-1 in nonexpressing U937 cells resulted in a four- to fivefold enhancement o

220 alysis of beta2 integrin mRNA and protein in U937 cells revealed a 5- to 6-fold increase with hypoxia

221 125I-Glycodelin binding to whole U937 cells revealed a single, saturable site with a Kd =

222 Live-cell imaging with Lifeact-transfected U937 cells revealed that force triggers actin polymeriza

223 ication of serovar Typhi during infection of U937 cells, serovar Typhi caused significantly less apop

224 istogram (n = 150 cells) of MI-U937(+) or MI-U937( +) cells shows all cells move respectively faster

225 Knockdown of CHOP in U937 cells significantly reduced the synergistic effects

226 id not arise because of PMA treatment of the U937 cells, similar experiments were conducted with the

227 The U937 cells spontaneously released themselves from the ab

228 Lastly, U937 cells stably expressing a p21(CIP1/WAF1) antisense

229 DeltaB-U937, where U937 cells stably transfected with deleted basic domain

230 d coprecipitation of integrins and CX3CR1 in U937 cells, suggesting that FKN-CD induces ternary compl

231 hondria, ER, and cytosol of both MCF-7c3 and U937 cells, suggesting that Mcl-1 protein, unlike Bcl-2

232 or upregulation of myelomonocytic markers in U937 cells, suggesting that the biological activity we o

233 cells demonstrated that K145 accumulates in U937 cells, suppresses the S1P level, and inhibits SphK2

234 ecognized plasminogen-binding protein on the U937 cell surface.

235 In U937 cells, synergistic interactions between MG-132 and

236 to these cultured neutrophils and stimulated U937 cells that exhibited elevated levels of binding but

237 ssion were investigated using differentiated U937 cells that lack soluble guanylate cyclase.

238 For U937 cells, the 72-h IC(50) for both 2a and 2b was 53 mi

239 Tested in human leukemia U937 cells, the benzamide and anilide derivatives 1b, 1c

240 Furthermore, in U937 cells, the hydrogen peroxide scavenger catalase and

241 o CD32/FcgammaRII aggregation in transfected U937 cells, thus demonstrating that FDF03 can function a

242 imultaneous, but not sequential, exposure of U937 cells to 100 nM FP and 10 nM PMA significantly incr

243 Exposure of U937 cells to a low concentration of LAQ824 (30 nM) resu

244 ins also inhibited CR3-dependent adhesion of U937 cells to C3 opsonized erythrocytes, indicating a se

245 , Merle 48 showed sufficient activity on the U937 cells to confirm that it was PMA-like for growth an

246 activators significantly reduced adhesion of U937 cells to cultured human ECs.

247 Here we used human monocytic-like U937 cells to evaluate apolipoprotein E receptor 2 (ApoE

248 or ATP increased the adherence of monocytic U937 cells to HCAEC, an effect that was inhibited by ant

249 (OH)D(3) increased the adhesion of monocytic U937 cells to HUVEC (159% and 153%, respectively).

250 RAP blocked binding of U937 cells to immobilized APC.

251 xamine the response of human macrophage-like U937 cells to low-dose infections with L. pneumophila.

252 -acetate-induced terminal differentiation of U937 cells to macrophages and resulted in maintenance of

253 at subsequent exposure of paclitaxel-treated U937 cells to MEK/MAPK inhibitors induces perturbations

254 n of full-length Naip rescues the ability of U937 cells to sense flagellin.

255 Combined exposure of U937 cells to subtoxic concentrations of UCN-01 and L744

256 -selectin and enhanced adhesion of monocytic U937 cells to the HAECs.

257 However, coexposure of U937 cells to UCN-01 and PD184352 induced a marked incre

258 Differential centrifugation of COS-1 and U937 cells together with Western blot analysis demonstra

259 mediates this process using neutrophils and U937 cells transfected with the C5a receptor (U937-C5aR

260 actic cofactor for C5a using neutrophils and U937 cells transfected with the C5aR (U937-C5aR cells).

261 U937 cells treated with H2O2 were shown to produce high

262 of cell lysates and conditioned medium from U937 cells treated with oxLDL alone revealed an increase

263 protein kinase C (PKC) isoforms in LNCaP and U937 cells, two cell lines used extensively for probing

264 XIAP-overexpressing U937 cells (U937XIAP) were more resistant to TRAIL than

265 cytes with CR1-bound ICs were incubated with U937 cells under a variety of conditions, and subsequent

266 The increase in gene expression levels for U937 cells upon lipopolysaccharide induction could be ac

267 Enhanced apoptosis in U937 cells was associated with an early caspase-independ

268 Since the potent cytotoxicity for U937 cells was completely lost when L-3-deoxy-diC8PI was

269 mmunized with FI-RSV, an increased number of U937 cells was infected.

270 Furthermore, apoptosis of nonproliferating U937 cells was unaffected by the Cdt mutant possessing r

271 sis of monocytoid cells (human monocytes and U937 cells) was induced with either TNFalpha or cyclohex

272 e C5aR on these cells, which, in the case of U937 cells, was largely caused by NSP-mediated cleavage

273 Using formyl peptide receptor-transfected U937 cells, we further show that the time course of avid

274 Levels of enzyme activity in plasma-treated U937 cells were closely dependent on the severity of dia

275 U937 cells were cultured with ketone bodies (acetoacetat

276 11b promoter-driven luciferase activity when U937 cells were induced to differentiate into monocyteli

277 Monocyte-like U937 cells were labeled with fluo-3-acetoxymethyl ester

278 A, CR1, AHP, or C3b on both erythrocytes and U937 cells were measured by flow cytometry with appropri

279 O cells, and the human promonocyte cell line U937 cells were not susceptible to transduction by the p

280 U937 cells were pre-incubated with BSG extracts, exposed

281 12-O-tetradecanoylphorbol-13-acetate-matured U937 cells were treated chronically with insulin (1 nm,

282 Human U937 cells were used as a model of monocyte chemotaxis i

283 h antiproliferative IC(50)'s (>100 microM in U937 cells) were observed for analogues in which R' = CH

284 nt to initiate macrophage differentiation of U937 cells whereas blocking endogenous BLIMP-1 inhibits

285 ression of MHCII genes in human promonocytic U937 cells, which represent immature antigen-presenting

286 n MCP-1 and RANTES produced by monocytes and U937 cells, while a 2-fold increase in TNF-alpha product

287 Results showed that coculture of U937 cells with adipocytes led to a marked increase in o

288 Treatment of U937 cells with an IkappaBalpha phosphorylation inhibito

289 blasts were incubated with supernatants from U937 cells with B. burgdorferi or recombinant CD14, the

290 We treated U937 cells with CpB, then subjected membrane fractions t

291 U937 cells with forced ST6Gal-I displayed TNFR1 with ele

292 using U1 monocytic cells (latently infected U937 cells with HIV-1).

293 Similarly, pretreatment of U937 cells with lovastatin inhibited PMA-stimulated, but

294 ted PLD activity after stimulation of intact U937 cells with opsonized zymosan.

295 Cotreatment of U937 cells with PD184352 and UCN-01 resulted in the acti

296 Incubation of human macrophage-like U937 cells with preparations of FHA resulted in dose-dep

297 Pretreatment of matured U937 cells with rapamycin blocked chronic insulin + high

298 Treatment of U937 cells with tunicamycin/thapsigargin resulted in red

299 Loading U937 cells with vitamin C decreased intracellular levels

300 se lead compounds in a calcium flux assay in U937 cells yielded similar results although with reduced