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1 UES and esophageal body pressure responses, along with l
2 UES contraction and relaxation were the overriding respo
3 UES is not caused by reduced scleral hydraulic conductiv
4 UES possessed Na,K-ATPase activity consistent with a sin
5 UES pressure progressively declined with deeper stages o
6 UES shows a misleading cystlike appearance at CT and MR
10 n and high-resolution manometry to determine UES, lower esophageal sphincter, and intraesophageal pre
12 ow balloon distension activated the esophago-UES contractile reflex and esophago-LES relaxation respo
13 smaller proportion of patients with SERD had UES contractile reflexes in response to slow esophageal
14 ophageal reflux disease (SERD) have impaired UES and esophageal body responses to simulated reflux ev
15 nd complaints of regurgitation have impaired UES and esophageal responses to simulated liquid reflux
16 detected by immunoblots (except for beta2 in UES), their mRNAs were detected in UES and EB (beta2 and
17 ly with age while the magnitude of change in UES pressure remains unchanged, indicating a deleterious
18 beta2 in UES), their mRNAs were detected in UES and EB (beta2 and beta3), and in immature and fully
20 ater-induced UES contraction and air-induced UES relaxation were the predominant responses among indi
23 Hg/50-ms duration stimuli, the frequency of UES response to air stimulation as evidenced by mucosal
25 aryngoglottal Closure reflex; PGCR, Pharyngo-UES Contractile reflex; PUCR, and Reflexive Pharyngeal S
27 magnitude of the increase in poststimulation UES pressure in the elderly volunteers was similar to th
29 ents include an upstream essential sequence (UES) located upstream of the UAS(INO) element and a nega
30 responses of the upper esophageal sphincter (UES) and esophageal body to liquid reflux events prevent
31 l vestibule, and upper esophageal sphincter (UES) and intraluminal pharyngeal dimensions were measure
33 l stimulation on upper esophageal sphincter (UES) pressure and to determine the reproducibility of th
34 response of the upper esophageal sphincter (UES) to simulated or spontaneous gastroesophageal reflux
36 formulations: uncoated on an empty stomach (UES), uncoated with food (UFED), and film-coated (FC) wi
40 ct the airway by reflexively contracting the UES and clearing the esophagus of refluxate, respectivel
45 escalated from 100 mg to 340 mg daily in the UES group, from 60 mg to 100 mg twice daily in the UFED
48 thy nonsmoker volunteers and 7 patients with UES dysphagia using a concurrent manometric and video en
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