ライフサイエンスコーパス: VEP

1 VEP acuity in the LCP-supplemented group was significant

2 VEP amplitude was a monotonically increasing function of

3 VEP amplitude was measured as a function of the size of

4 VEP amplitude, RNFL thicknesses provided by Cirrus and S

5 VEP is a method that can be used to assess brain functio

6 VEP latencies at week 16 were shorter in erythropoietin-

7 VEP latencies were found to decrease significantly durin

8 VEP latency and visual function tests that capture optic

9 VEP latency, PERG measurements, and macular thicknesses

10 VEP may be useful for early diagnosis of glaucoma.

11 VEP P100 latency was found superior to color vision and

12 VEP results can be predictive of visual recovery in trau

13 VEP Vernier acuity and grating acuity develop at differe

14 VEP Vernier acuity remains strikingly immature throughou

15 VEPs can be valuable in diagnosing optic neuropathies, n

16 VEPs recorded in humans showed significant phase locking

17 VEPs to checks and ZD stimulation were similar.

18 VEPs were analyzed at the pattern reversal rate using sp

19 VEPs were less pronounced in the infarction model, provi

20 VEPs were recorded from the visual cortices of five maca

21 VEPs were recorded in 16 healthy adults in response to a

22 VEPs were recorded in 35 infants between 5 and 15 months

23 VEPs were recorded to the onset of luminance increments

24 VEPs were repeated in nine subjects with 20/40 or better

25 ovea, (3) nystagmus intensity, (4) BCVA, (5) VEP asymmetry, (6) skin pigmentation, and (7) hair pigme

26 ut procedure of Sutter was used to obtain 60 VEP responses to a scaled checkerboard pattern.

27 re and 1 month after surgery, visual acuity, VEP, PERG, and 3 repetitions of scans using the RNFL and

28 The presence of cataracts affects VEP amplitude, RNFL, and macular measurements performed

29 At 1 month of age, VEP implicit times were somewhat shorter in Akt-DD trans

30 Beyond 6months of age, VEP latencies were consistently delayed in Akt-DD transg

31 other eye resulted in an increased amplitude VEP response.

32 Age, severity of amblyopia, and VEP amplitudes of positive peak (P)100, P1, and negative

33 tory and visual evoked potentials (BSAEP and VEP) and in sleep patterns.

34 crucial role in the generation of BSAEP and VEP, as well as in sleep disturbances.

35 Magnetic resonance imaging and VEP confirmed absence of decussation of retinofugal fibe

36 t the development of optomotor threshold and VEP acuity can occur in an experience-independent manner

37 tical auditory evoked potentials (CAEPs) and VEPs and a decrease in both REM sleep and SWS.

38 ic visual functions (motion perception), and VEPs were assessed repeatedly.

39 were acquired with a commercially available VEP unit using standard electrode recording techniques.

40 Time-averaged VEP central field latency was shorter by 6.1 ms (95% CI

41 any of the baseline, 1 year or time-averaged VEP variables.

42 A strong correlation was found between VEP latencies and motion perception.

43 The relation between VEP latency and blood glucose was determined.

44 The within-subject relationship between VEP Vernier acuity and grating acuity follows the same d

45 , and OCT) and demyelination (as measured by VEP).

46 It was possible to record a clear VEP from Akt-DD mice at all ages examined.

47 Either a delayed VEP is not a good indicator of damaged, as opposed to de

48 The delayed VEP responses of the patients with MAR to luminance incr

49 High-density VEPs evoked by a contrast reversing checkerboard were co

50 animals, we measured NMDA receptor-dependent VEP potentiation ipsilateral to the NDE during MD, which

51 rocessing can be detected with the dichoptic VEP method we describe.

52 monstrated by the PR-VEP latencies, our DRCD-VEP data show that the visual cortex is remarkably ready

53 nse to dynamic random dot correlograms (DRDC-VEP), appears to be at around the same time after birth

54 To ensure attentiveness during VEP assessments, subjects responded with a button press

55 rally presented IC stimuli resulted in early VEP modulation (88-100 msec) over lateral-occipital (LOC

56 primary analysis evaluated vaccine efficacy (VEP) as the percent reduction (vaccine vs placebo) in cu

57 Estimated VEP (mITT) was15.8% (-21.9, 41.8) at 60 months postinfec

58 In this study, we examined VEP plasticity in healthy control subjects and patients

59 In a second experiment, VEPs were obtained using slowed m-sequences (8 and 16 vi

60 Deprived-eye VEPs were no larger in the injected hemisphere than in t

61 ion speed were highly correlated with faster VEP latencies.

62 using full-field visual evoked potential (FF-VEP) versus the unaffected fellow eye at baseline.

63 Like the monkey neurons, human VEPs more typically phase locked to stimuli containing s

64 ected fellow eyes, no significant changes in VEP parameters or functional indices were observed.

65 th vernier offsets resulted in a decrease in VEP amplitude for both horizontal and vertical dispariti

66 RTT patients exhibited a similar decrease in VEP amplitude that was most striking in the later stages

67 mately 8.8 Hz, 2 minutes) designed to induce VEP potentiation.

68 s shorter latency than visible light-induced VEP, its amplitude increases with peak irradiance and pu

69 Shock-induced VEP is essential in extinguishing fibrillation but can r

70 Shock-induced VEPs may reinduce arrhythmias via a phase-singularity me

71 Sixty local VEP responses were recorded simultaneously.

72 41% of children, whereas an abnormal motion VEP, Worth 4-dot, or positive 4-PD base-out prism respon

73 lly strabismic monkeys had asymmetric motion VEP responses: AI = 0.57 +/- 0.22 in the Delayed Repair

74 The concordance between motion VEP symmetry and normal fusional vergence was significan

75 naturally strabismic monkeys also had motion VEP asymmetries of equivalent magnitude when tested usin

76 Delayed repair causes permanent motion VEP maldevelopment.

77 ric (F2) frequency component from the motion VEP.

78 Motion VEPs, random dot stereopsis, and bifoveal fusion were me

79 ths after the removal of the goggles, motion VEPs to horizontally oscillating grating stimuli were re

80 the development or maldevelopment of motion VEPs.

81 tric motion visual evoked potentials (motion VEPs).

82 ly Repair monkeys exhibited symmetric motion VEPs (AI < 0.25).

83 erebral cortex, measured as symmetric motion VEPs.

84 luding neuroaxonal loss (as measured by MRI, VEP, and OCT) and demyelination (as measured by VEP).

85 Multifocal VEPs were recorded from both eyes of six normal subjects

86 % to 70% of first-phase voltage) produced no VEP, because of an asymmetric reversal of the first-phas

87 riment 1: although adults showed evidence of VEP amplitude alternations between the eyes for cross-or

88 The amplitudes and latencies of VEP waveform components were quantified, and were relate

89 r this period, a significant prolongation of VEP latencies occurred in the asymptomatic fellow eye, a

90 study which showed significant shortening of VEP latencies between 6 months and 3 years without signi

91 Positive values of VEP resulted in a prolongation of the action potential d

92 that LTP similarly enhances the amplitude of VEPs, but in a way that generalizes across multiple stim

93 the effects of medication and mood state on VEP plasticity.

94 Multifocal electroretinogram (ERG) or VEP can provide an objective assessment of visual field

95 e was observed for LM- or achromatic-pathway VEP latency in subjects with diabetes.

96 The S-pathway VEP latency was correlated significantly with blood gluc

97 le of resolution (logMAR) units, and pattern VEP.

98 referred to as a virtual electrode pattern (VEP).

99 resulted in virtual electrode polarization (VEP).

100 changes in virtual electrode polarizations (VEPs) and propagation delay through the peri-infarct zon

101 ants by using sweep visual evoked potential (VEP) acuity as the functional outcome.

102 suggested that the visual evoked potential (VEP) amplitude can vary with stimulus duration.

103 ION, with a loss of visual evoked potential (VEP) amplitude that persisted in the long term.

104 investigate if the visual evoked potential (VEP) could be used as an unbiased, quantitative biomarke

105 ade tracers into the vagus evoked potential (VEP) focus in Pf of macaque monkeys.

106 t plasticity of the visual evoked potential (VEP) induced by repeated visual stimulation might reflec

107 ssive shortening of visual evoked potential (VEP) latencies and to determine whether this is associat

108 ss with a prolonged visual evoked potential (VEP) latency suggests that acute and persistent demyelin

109 cal examination and visual evoked potential (VEP) measurement, each patient had their optic nerves im

110 than for standard visually evoked potential (VEP) recordings, the eVEP has proven to be a reliable to

111 function, the sweep visual evoked potential (VEP) was used to evaluate cortical responses to grating

112 sual acuity (BCVA), visual evoked potential (VEP), and grading of skin and hair pigmentation were use

113 assessed using the visual evoked potential (VEP).

114 modulations in the visual evoked potentials (VEP) recorded.

115 and measurement of visual evoked potentials (VEP) was performed on each patient.

116 e basis of lesion, visual evoked potentials (VEP), and neuroimaging evidence, others contend that IC

117 ural feedback in visually evoked potentials (VEP).

118 ing enhancement of visual evoked potentials (VEP).

119 ured were visual evoked cortical potentials (VEPs) and multifocal (mf)ERGs, with both a standard fast

120 tials (spikes) and visual-evoked potentials (VEPs) align with the video impulses, particularly when h

121 cuity with sweep visually evoked potentials (VEPs) and for optotype acuity (Landolt C) with behaviora

122 stinjection, and visually evoked potentials (VEPs) and single-cell activity were recorded.

123 Visual Evoked Potentials (VEPs) following optic neuritis (ON) remain chronically p

124 tern-reversal (PR) visual evoked potentials (VEPs) have been found to be a sensitive indicator of vis

125 ual behavior and visually evoked potentials (VEPs) in binocular visual cortex of the same mice before

126 tagmus (OKN) and visually evoked potentials (VEPs) in one direction than to those in the opposite dir

127 Cortical visually evoked potentials (VEPs) in response to stimuli designed to selectively act

128 of neurons, and visually-evoked potentials (VEPs) in response to task light cues, while increasing c

129 RECENT FINDINGS: Visual evoked potentials (VEPs) may be useful as an objective measurement of refra

130 In awake mice, visual evoked potentials (VEPs) recorded in layer 4 of binocular visual cortex und

131 Visual evoked potentials (VEPs) to check reversal (163-18 arc min) and onset of si

132 Steady-state visual-evoked potentials (VEPs) to contrast reversing gratings were recorded over

133 Visual evoked potentials (VEPs) were measured in 10 patients with RP and in 10 age

134 Visual evoked potentials (VEPs) were recorded over three occipital sites to the on

135 Structural MRI, visual evoked potentials (VEPs), and optical coherence tomography (OCT) were used

136 etinography (ERG), visual evoked potentials (VEPs), spectral-domain optical coherence tomography (OCT

137 by spatial sweep visually evoked potentials (VEPs).

138 visual field, and visual evoked potentials (VEPs).

139 MD on its own potentiated VEPs contralateral to the NDE during MD and shifted ocul

140 PR-VEP latency is not affected by premature birth, demonstr

141 PR-VEP responses were recorded from 81 adults and 137 infan

142 ked response to pattern reversal stimuli (PR-VEP).

143 sual pathway, clearly demonstrated by the PR-VEP latencies, our DRCD-VEP data show that the visual co

144 nted as an Ensembl variant effect predictor (VEP) plugin, COCOS captures amino acid sequence alterati

145 At presentation, VEPs were a more sensitive indicator of optic pathway da

146 mal defibrillation waveforms did not produce VEPs because of an asymmetric effect of phase reversal o

147 s with a strong second phase (>70%) produced VEPs of reversed polarity.

148 ng-edge voltage of the first phase) produced VEPs similar to monophasic shocks.

149 Contrary to previous reports, prolonged VEP delays were present in a minority of patients with g

150 The authors recorded VEPs from 57 healthy full-term infants and 4 adults.

151 We recorded VEPs in response to strobe flash ganzfeld stimuli presen

152 itude and static visual functions recovered, VEP latency remained significantly prolonged, and motion

153 pearance of vernier onset-offset, and reduce VEP amplitude for both horizontal and vertical dispariti

154 ptic nerve pallor, whereas all had a reduced VEP in 1 or both eyes.

155 or testing, in vivo visual evoked responses (VEP) and single-unit cortical recordings.

156 We recorded pattern-reversal VEPs in Mecp2 heterozygous female mice and 34 girls with

157 The modulation block resulted in significant VEP plasticity in healthy control subjects.

158 ngs support the introduction of standardized VEP analysis in clinical and research settings to probe

159 nst temporal frequency (TF) for steady state VEP measurements as well as from the transient P1 peak.

160 The steady state VEPs were analyzed with discrete Fourier transforms to o

161 Step VEP acuity was 0.46 (95% CI: -0.13 to 1.06) logMAR units

162 Step VEP and GAC acuities correlated highly (r(2) = 0.60, P =

163 e equation: acuity(GAC) = (0.9 x acuity(step VEP)) - 0.37.

164 cuity assessed five times with both the step VEP and with Glasgow Acuity Cards (GAC).

165 The step VEP provides a rapid, objective means of estimating visu

166 Shocks producing strong VEPs resulted in postshock reentrant arrhythmias via a m

167 Sweep VEP acuity was the primary outcome.

168 Sweep VEP data were obtained from 16 healthy observers under b

169 Assessments of optotype acuity and sweep VEP acuity revealed amblyopic deficits in both pseudopha

170 t there is not a significant change in sweep VEP acuity estimates over an 8-second stimulus presentat

171 requency, contrast, and vernier offset sweep VEP tuning functions were measured at 5 to 7 months' cor

172 The sweep VEP acuities for the 16 subjects did not change signific

173 mates obtained by extrapolation of the sweep VEP are altered by this adaptation effect.

174 icantly affect the clinical use of the sweep VEP.

175 avioral testing with optotypes or with sweep VEPs.

176 The VEP consists of large adjacent areas of strong positive

177 The VEP plasticity was significantly impaired in patients wi

178 The VEP was strongest lateral and anterior to the habenuloin

179 Immediately preceding the IC effect, the VEP modulated with inducer eccentricity--the configurati

180 results suggested that N1-P2 complex in the VEP could be a neural marker for stereopsis and fNIRS de

181 previous SRP occludes TBS-induced LTP of the VEP evoked by the experienced stimulus, but not by unfam

182 ower recovery from the principal peak of the VEP response that was impacted by MECP2 mutation type.

183 plitudes for the P1 and N1 components of the VEP that were specific to Arabic numerals and to dot con

184 ency delays on the mfVEP test but not on the VEP test, presumably due to the mfVEP's ability to detec

185 Our tools, in particular the VEP, have been improved significantly through integratio

186 Unlike the VEP results, the mfVEP revealed a significant increase i

187 We sought to determine whether the VEP may be responsible for defibrillation failure by cre

188 The authors tested whether the VEP responses were asymmetrical because of abnormal eye

189 concentration (HbO) was correlated with the VEP amplitude during the checks and HD presentations.

190 The VEPs did not have sufficient power to reliably distingui

191 The VEPs obtained in our two volunteers with implants had a

192 The VEPs recorded during surface eye stimulation are similar

193 The VEPs were compared at sub- and supra-threshold stimulati

194 The VEPs were evoked by checkerboard reversal stimulation be

195 The VEPs were recorded with a 15' and 60' reversing checkerb

196 source of the asymmetrical amplitudes of the VEPs, and the visual cortex is at least one source respo

197 Their VEP responses showed a marked delay to increments but on

198 These VEP results are in general agreement with recent psychop

199 This VEP technique provides a rapid estimate of Vernier acuit

200 ty is a sensitive measure of amblyopia, this VEP test may be useful in the future to identify amblyop

201 Short-duration transient VEP objectively identified decreased visual function and

202 and 38 control eyes (19 subjects) underwent VEP, mfVEP, and visual field testing.

203 15 control children underwent swept vernier VEP acuity testing accompanied by a swept motion control

204 o investigate the specificity of the vernier VEP as a measure of positional acuity, evaluating the po

205 The vernier VEP paradigm, when applied in the manner described, can

206 Whereas VEP amplitude and static visual functions recovered, VEP