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1 VGLUT expression argues that dysplastic neurons may be g
2 VGLUT(2) -immunoreactive (IR) neurons in the MPG were ra
3 characterized antibodies against VGLUT(1) , VGLUT(2) , and calcitonin gene-related peptide (CGRP) we
6 he vesicular glutamate transporters-1 and 2 (VGLUT-1, VGLUT-2), or the vesicular GABA transporter (VG
7 essed for vesicular glutamate transporter-2 (VGLUT-2), tryptophan-hydroxylase (TrOH), glial fibrillar
14 ng the vesicular glutamate transporter eat-4/VGLUT, induction of neuropeptide expression, changes in
15 s, the vesicular glutamate transporter EAT-4/VGLUT, is expressed in 38 of the 118 anatomically define
18 Previously characterized antibodies against VGLUT(1) , VGLUT(2) , and calcitonin gene-related peptid
20 have ipsilateral descending axons, were also VGLUT-positive, as were the ventrally located VeMe inter
21 we studied VGLUTs type 1 and 2 (VGLUT(1) and VGLUT(2) , respectively) in neurons innervating the mous
23 ires GABA(A)Rs and NMDARs in PNs, as well as VGLUT and cAMP signaling in the multiglomerular inhibito
25 However, the demonstration of astrocytic VGLUT expression is largely based on immunostaining, and
29 Cs represent the first synthetically derived VGLUT inhibitors and are promising templates for the dev
31 on of ArcN neurons immunoreactive for either VGLUT-2 (74 +/- 21 versus 342 +/- 84 cells/section, P <
33 1) that most colorectal DRG neurons express VGLUT(2) , and to a lesser extent, VGLUT(1) ; 2) abundan
38 s express VGLUT(2) , and to a lesser extent, VGLUT(1) ; 2) abundance of VGLUT2-IR fibers innervating
40 study reports a previously unknown role for VGLUT as an acid-extruding protein when deposited in the
45 ransmission via a mechanism that may involve VGLUT inhibition rather than activation of mGlu2/3 recep
47 and current knowledge on the distribution of VGLUT isoforms in highly visual mammals, we examined the
49 wn that glucose stimulates the expression of VGLUT isoform 2 (VGLUT2) in beta cells via transcription
53 nocytochemistry showed a punctate pattern of VGLUT immunoreactivity throughout the entire cell body a
54 esses, whereas pharmacological inhibition of VGLUTs abolished mechanically and agonist-evoked Ca2+-de
58 s were also found to receive dual-phenotype (VGLUT + VGAT) inputs; these varied with season in a mann
60 ns of neurons, and VGLUT1 is the predominant VGLUT in the neocortex, hippocampus, and cerebellar cort
61 ns of neurons, and VGLUT1 is the predominant VGLUT in the neocortex, hippocampus, and cerebellar cort
62 ns of neurons, and VGLUT1 is the predominant VGLUT in the neocortex, hippocampus, and cerebellar cort
63 te transporter 2 (VGLUT2) is the predominant VGLUT isoform expressed in the basal forebrain and brain
64 found that vesicles with drastically reduced VGLUT expression were released with a lower probability.
68 Taken together, these data indicate that VGLUTs play a functional role in exocytotic glutamate re
70 substituted QDCs tested as inhibitors of the VGLUT system, the 6-PhCH=CH-QDC (K(i) = 167 microM), 6-P
71 f the key elements needed for binding to the VGLUT protein based on the structure-activity relationsh
73 Preobraschenski et al. (2014) show that the VGLUTs, in addition to transporting glutamate, also prov
74 les with glutamate and mammals express three VGLUT isoforms (VGLUT1-3) with distinct spatiotemporal e
75 favor of the expression of any of the three VGLUTs by gray matter protoplasmic astrocytes of the pri
76 e, is a known vesicular glutamate transport (VGLUT) inhibitor and has also been proposed as an mGlu2/
78 sponsible for vesicular glutamate transport (VGLUTs) that show no sequence similarity to the other tw
80 cluding the vesicular glutamate transporter (VGLUT) genes for glutamatergic neurons, the neuronal gly
81 cluding the vesicular glutamate transporter (VGLUT) genes for glutamatergic neurons, the neuronal gly
86 the lamprey vesicular glutamate transporter (VGLUT) provides an anatomical basis for the general dist
87 ressing the vesicular glutamate transporter (VGLUT) type 2 ( approximately 6%) but not those expressi
88 les via the vesicular glutamate transporter (VGLUT), a mechanism conserved across phyla, and this stu
91 unc-2) or a vesicular glutamate transporter (VGLUT; eat-4), the abundance of GLR-1 in the ventral ner
92 the three vesicular glutamate transporters (VGLUT 1-3) in the locus coeruleus (LC) and the dorsal ra
93 ression of vesicular glutamate transporters (VGLUTs) 1 and 2 accounts for the release of glutamate by
97 The three vesicular glutamate transporters (VGLUTs) are expressed in distinct populations of neurons
98 The three vesicular glutamate transporters (VGLUTs) are expressed in distinct populations of neurons
99 The three vesicular glutamate transporters (VGLUTs) are found in different populations of neurons, a
100 synapses, vesicular glutamate transporters (VGLUTs) are responsible for filling synaptic vesicles wi
103 of several vesicular glutamate transporters (VGLUTs) similarly revealed an unexpected molecular diver
106 s requires vesicular glutamate transporters (VGLUTs) to concentrate cytosolic glutamate in synaptic v
107 n requires vesicular glutamate transporters (VGLUTs) to sequester glutamate into synaptic vesicles.
108 ed whether vesicular glutamate transporters (VGLUTs) were differentially distributed among the two di
109 d with the vesicular glutamate transporters (VGLUTs), we characterize a chloride conductance that is
111 cular sorting will be critical to understand VGLUT's involvement in normal and pathological condition
112 the C. elegans central nervous system, using VGLUT-pHluorin to monitor synaptic vesicle exocytosis an
114 at conjugation of these gold nanoprobes with VGLUT-2 antibodies and polyethyleneimine (PEI) facilitat
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