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1 are initiated by somatic inactivation of the VHL tumor suppressor gene.
2 d by germ line mutations that inactivate the VHL tumor suppressor gene.
3 sease is caused by germline mutations of the VHL tumor suppressor gene.
4 ting of tumors caused by inactivation of the VHL tumor suppressor gene.
5 is caused by mutational inactivation of the VHL tumor suppressor gene.
6 or may not have a germline mutation in their VHL tumor-suppressor gene.
7 lelic inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene.
8 ation/inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene.
9 in ligase activity of the von Hippel-Lindau (VHL) tumor suppressor gene.
10 ated with mutation of the von Hippel-Lindau (VHL) tumor suppressor gene.
11 lelic inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene.
12 s due to mutations in the Von Hippel-Lindau (VHL) tumor suppressor gene.
13 ted by the product of the von Hippel-Lindau (VHL) tumor suppressor gene.
14 s from germline and somatic mutations in the VHL tumor suppressor gene and is characterized by highly
15 ease is caused by germ-line mutations in the VHL tumor suppressor gene and is the most common cause o
16 Patients with germ line mutations in the VHL tumor suppressor gene are predisposed to the develop
17 tivating mutations of the von Hippel-Lindau (VHL) tumor suppressor gene are associated with inherited
18 epigenetic changes in the von Hippel-Lindau (VHL) tumor suppressor gene are common in sporadic conven
19 erm line mutations in the Von Hippel-Lindau (VHL) tumor suppressor gene are predisposed to the develo
22 mline inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene causes the von Hippel-Lindau
24 he E-cadherin (E-cad) and von Hippel-Lindau (VHL) tumor suppressor gene CpG island regions in normal
25 y explain why patients with mutations in the VHL tumor suppressor gene display cancerous lesions in s
30 e mutations in one of the three exons of the VHL tumor suppressor gene have been found in the majorit
32 ity of selectively targeting the loss of the VHL tumor suppressor gene in clear cell renal carcinoma
33 ghts into the role of the von-Hippel Lindau (VHL) tumor suppressor gene in hereditary and sporadic cl
37 In VHL disease, a germline mutation of the VHL tumor suppressor gene is inherited, and loss of func
45 r loss of function of the von Hippel-Lindau (VHL) tumor suppressor gene is regularly found in sporadi
46 he hereditary loss of the von Hippel-Lindau (VHL) tumor suppressor gene is the leading cause of death
47 The absence of functional von Hippel-Lindau (VHL) tumor suppressor gene leads to the development of n
48 to loss of chromosome 3p, which harbors the VHL tumor suppressor gene, loss of chromosome 14q, which
49 Loss of function in the von Hippel-Lindau (VHL) tumor suppressor gene occurs in familial and most s
50 ther allelic loss of the recently identified VHL tumor suppressor gene on chromosome 3p25-26 occurs i
51 is the result of a germline mutation of the VHL tumor suppressor gene on the short arm of chromosome
52 that mutations and allelic deletions of the VHL tumor suppressor gene play a role in the tumorigenes
54 ting mutations within the von Hippel-Lindau (VHL) tumor suppressor gene predispose patients to develo
55 Germline mutations in the von Hippel-Lindau (VHL) tumor suppressor gene predispose people to renal ca
56 Inheritance of an inactivated form of the VHL tumor suppressor gene predisposes patients to develo
57 The inactivation of the von Hippel-Lindau (VHL) tumor suppressor gene predisposes affected individu
62 in which mutations in the von Hippel-Lindau (VHL) tumor suppressor gene result in accumulation of hyp
63 hemangioblastomas in the von Hippel-Lindau (VHL) tumor suppressor gene syndrome, VHL disease, are un
65 ), cells deficient in the von Hippel-Lindau (VHL) tumor suppressor gene use glutamine to generate cit
67 by an inactivation of the von Hippel-Lindau (VHL) tumor-suppressor gene with subsequent stabilization
68 sease is caused by germline mutations in the VHL tumor suppressor gene, with Type 2B missense VHL mut
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