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1 [L-NNA] for NO synthesis, and VIP(10-28) for VIP receptor).
2 ember of the Gi family in signaling from the VIP receptor.
3 is probably due to the higher expression of VIP receptors.
4 can was positive for tumors known to express VIP receptors.
5 consistent with expression of high affinity VIP receptors.
8 l and GrB expression is mediated through the VIP receptors 1 and 2 and cAMP signaling through exchang
10 e, differential expression of VIP receptor-1/VIP receptor-2 (VIPR1/VIPR2) was observed between B6 and
11 onstructs containing various portions of the VIP receptor 5'-flanking sequences linked to the lucifer
13 results, the authors conclude that Gs links VIP receptor activation to adenylyl cyclase and cyclic a
16 ike and wave discharges, whereas blockade of VIP receptors almost completely abolished this form of e
17 ned to investigate whether human PDC express VIP receptors and whether VIP has immunological effects
18 fect is mediated through the specific type 2 VIP receptor, and the cAMP/protein kinase A pathway.
19 eptor antagonist (exendin (9-39) amide) or a VIP receptor antagonist (hybrid of neurotensin 6-11 and
22 n in the presence of different levels of the VIP receptor antagonist, VIP(10-28), also delivered by m
24 9 mol/L, 3 x 10(-)9 mol/L, respectively) and VIP-receptor antagonist (IC50, 3 x 10(-)7 mol/L, 5 x 10(
26 retin and vasoactive intestinal polypeptide (VIP) receptors are responsible for the activation of ade
27 ibited reversibly by bath application of the VIP receptor-binding inhibitor L-8-K (5 microM) or the n
28 of VIP, acting through G(s)-protein-coupled VIP receptors, can induce LNCaP cell growth in the absen
33 R-mediated transcriptional repression of the VIP receptor gene, but basal transcription is also reduc
34 he type 1 vasoactive intestinal polypeptide (VIP) receptor gene is highly expressed in the lung where
35 d was identified as Gi3 through capture of a VIP/receptor/ Gi3 ternary complex by covalent cross-link
36 pha i3 antiserum suppressed formation of the VIP/receptor/Gi3 ternary complex, demonstrating its func
37 transfection of GR expression vector and the VIP receptor GR binding site linked to the thymidine kin
41 ed glucocorticoid receptor (GR) binds to the VIP receptor promoter between -21 and -36 bp relative to
42 ng receptor autoradiography, PACAP and PACAP/VIP receptor (PVR) mRNAs by reverse transcription polyme
44 active intestinal peptide (VIP) and a VPAC2 (VIP receptor subtype 2) receptor-specific agonist, Ro-25
45 erogeneity of these tumors, we also assessed VIP receptor subtype expression in seven well-characteri
49 lly distinct G-protein-associated receptors, VIP receptor type 1 (VPAC1) and VIP receptor type 2 (VPA
50 d receptors, VIP receptor type 1 (VPAC1) and VIP receptor type 2 (VPAC2), mediate the biological effe
51 r gene shows that 126 base pairs (bp) of the VIP receptor upstream sequences are sufficient to mediat
53 found in cutaneous nerves and mRNA, for the VIP receptor vasoactive intestinal peptide receptor type
55 oactive intestinal polypeptide (VIP) and the VIP receptor VPAC(2) (encoded by the gene Vipr2) may med
57 interact with vasoactive intestinal peptide (VIP) receptors (VPAC(1)-R and VPAC(2)-R) because of the
59 ng activity is mediated through the specific VIP receptor VPAC1, and involves both cAMP-dependent and
64 VIP regulation of AR activation, mediated by VIP receptors, was protein kinase A (PKA)-dependent, and
69 es, and to a lesser degree VPAC2, the type 2 VIP receptor, which is induced upon macrophage activatio
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