ライフサイエンスコーパス: VLDL cholesterol

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1 es (+58 mg/dL), LDL cholesterol (+11 mg/dL), VLDL cholesterol (+8 mg/dL), glucose (+9 mg/dL), systoli

2 luding obesity, total cholesterol level, LDL/VLDL cholesterol and glucose/insulin ratio.

3 reased LDL and HDL cholesterol but increased VLDL cholesterol and no significant difference in extent

4 y available for lowering LDL cholesterol and VLDL cholesterol and raising HDL cholesterol.

5 accounted for major variations in plasma LDL/VLDL cholesterol and triglyceride levels, coincided with

6 BMI, systolic and diastolic blood pressure, VLDL cholesterol, and glucose parameters were higher in

7 hanges in concentrations of HDL cholesterol, VLDL cholesterol, and triglyceride, but not LDL choleste

8 sma TC, LDL-C, very-low-density lipoprotein (VLDL) cholesterol, and MDA than had the PC group after 8

9 low-density lipoprotein (VLDL)-lipoproteins, VLDL-cholesterol (C), VLDL-triglycerides, VLDL-diameter,

10 on dramatically decreased plasma VLDL TG and VLDL cholesterol concentrations but only moderately incr

11 ts reduce fasting plasma triacylglycerol and VLDL-cholesterol concentrations by 19% and 22%, respecti

12 more marked reduction in triacylglycerol and VLDL-cholesterol concentrations in subjects who consumed

13 sity-lipoprotein (VLDL) triacylglycerol, and VLDL-cholesterol concentrations were higher (P < 0.05-0.

14 entrations were adjusted for HDL-, LDL-, and VLDL-cholesterol concentrations, we calculated partial S

15 KO mice also exhibited higher plasma LDL and VLDL cholesterol content, increased circulating apolipop

16 Moreover, M-CSF did not augment beta-VLDL cholesterol deposition in macrophages from LDL rece

17 ficantly increased the hepatic production of VLDL-cholesterol fourfold, VLDL-triglyceride two and one

18 irst 6 months with respect to triglycerides, VLDL cholesterol, insulin, C peptide, and uric acid; the

19 Highly significant reduction in VLDL cholesterol levels and systolic BP was observed amo

20 tivation of Bmal1 led to elevated plasma LDL/VLDL cholesterol levels as a consequence of the disrupti

21 mice, which displayed strongly reduced serum VLDL cholesterol levels.

22 ein (HDL), and very-low-density lipoprotein (VLDL) cholesterol levels.

23 otal plasma cholesterol, LDL-cholesterol and VLDL-cholesterol levels, as well as obesity measures (bo

24 Plasma LDL/VLDL cholesterol or triglyceride levels of F2 mice were

25 at content and the production of TG-depleted VLDL cholesterol particles.

26 ity lipoprotein cholesterol (LDL cholesterol/VLDL cholesterol-TBARS) as indicators of lipid peroxidat

27 omen had an increased likelihood of elevated VLDL cholesterol, triacylglycerol, diastolic blood press

28 endothelial function, or total cholesterol, VLDL-cholesterol, triacylglycerol, apolipoprotein B, or

29 No significant effects were observed on VLDL-cholesterol, triacylglycerol, lipoprotein(a), and C

30 VLDL cholesterol, triglycerides, and 2-hour OGTT were hi

31 olesterol, and very-low-density-lipoprotein (VLDL)-cholesterol values were significantly lower than i

32 VLDL-cholesterol, VLDL-triglyceride, VLDL-apoB, apoCIII

33 ) cholesterol, very-low-density-lipoprotein (VLDL) cholesterol, VLDL3 cholesterol, lipoprotein(a), an

34 By contrast, uptake of VLDL-cholesterol was much higher than CM-cholesterol (P

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