1 e frontal and temporal cortex from 15 AD,
15 VaD and 15 control brains.
2 When we examined AD
and VaD separately, subjects with late-life depressive sympt
3 ntribute to vascular abnormalities in AD
and VaD.
4 Chronic cerebral hypoperfusion
and VaD was induced by bilateral common carotid artery occlu
5 uences of chronic cerebral hypoperfusion
and VaD.
6 ortem brain tissue in AD, vascular
dementia (
VaD) and controls.
7 20 AD, 20 control and 15 Vascular
dementia (
VaD) brains by real-time PCR (RT-PCR).
8 , Alzheimer disease (AD), vascular
dementia (
VaD), and CIND by age.
9 lzheimer disease (AD) and vascular
dementia (
VaD), is likely to increase as the population ages.
10 disease (AD), and 35% of vascular
dementia (
VaD).
11 erebral hypoperfusion and vascular
dementia (
VaD).
12 on developed AD, and about 637,000
developed VaD.
13 In VaD, KLK6 protein level was significantly increased in t
14 symptoms had more than a 3-fold increase
in VaD risk (3.51 [2.44-5.05]).
15 k factor for dementia with stroke,
including VaD and AD with CVD.
16 patients with dementia and stroke,
including VaD as currently defined, may include patients with AD.
17 f moderate to severe AD and mild to
moderate VaD, but it has not been studied specifically in mixed d
18 ologically associated with increased risk
of VaD.
19 ogen mRNA levels were unaltered in the AD
or VaD groups compared to the controls.
20 entia or neurology clinic diagnosis of AD
or VaD.
21 ymptom category and risk of dementia, AD,
or VaD.
22 ed with dementia (5.5% with AD and 2.3%
with VaD).
23 stroke was 41% overall, 33% among those
with VaD, and 44% among those with AD with CVD.